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Journal Article
Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7516, pp. 110 - 114
Mutations in isocitrate dehydrogenase 1 (IDH1) and IDH2 are among the most common genetic alterations in intrahepatic cholangiocarcinoma (IHCC), a deadly liver... 
PROGENITORS | INTRAHEPATIC CHOLANGIOCARCINOMA | HOMEOSTASIS | LIVER-REGENERATION | MULTIDISCIPLINARY SCIENCES | MUTATION | GROWTH | MICE | PROLIFERATION | EXPRESSION | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Hepatocyte Nuclear Factor 4 - antagonists & inhibitors | Humans | ras Proteins - metabolism | Hepatocytes - pathology | Male | Hepatocytes - metabolism | Bile Duct Neoplasms - enzymology | Cell Differentiation - genetics | Neoplasm Metastasis | Hepatocyte Nuclear Factor 4 - biosynthesis | Female | Bile Duct Neoplasms - genetics | Cell Lineage - genetics | Cholangiocarcinoma - enzymology | Disease Models, Animal | Cell Division - genetics | Proto-Oncogene Proteins - metabolism | Hepatocyte Nuclear Factor 4 - metabolism | Bile Ducts, Intrahepatic - pathology | Mutant Proteins - genetics | Isocitrate Dehydrogenase - genetics | Mice, Transgenic | Mutant Proteins - metabolism | Proto-Oncogene Proteins - genetics | Hepatocyte Nuclear Factor 4 - genetics | Mutation - genetics | Bile Ducts, Intrahepatic - enzymology | Cholangiocarcinoma - pathology | Animals | Cholangiocarcinoma - genetics | Stem Cells - pathology | Isocitrate Dehydrogenase - metabolism | Glutarates - metabolism | Mice | Bile Duct Neoplasms - pathology | Hepatocytes - enzymology | Index Medicus | Càncer | Diferenciació cel·lular | Gallbladder diseases | Metabolisme | Cell diferentiation | Metabolism | Malalties de la vesícula biliar | Cancer
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 01/2015, Volume 290, Issue 2, pp. 1170 - 1185
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1533 - 1544
The cause of organ failure is enigmatic for many degenerative diseases, including end-stage liver disease. Here, using a CCl4-induced rat model of irreversible... 
LONG-TERM | PATHOGENESIS | MEDICINE, RESEARCH & EXPERIMENTAL | NUCLEAR | MECHANISM | CHRONIC LIVER-DISEASE | CIRRHOSIS | RATS | HEPATOCYTE | EXPRESSION | TRANSPLANTATION | Liver Cirrhosis, Experimental - therapy | Liver Failure - etiology | Dependovirus - genetics | Genetic Therapy | Hepatocyte Nuclear Factor 1-alpha - genetics | Hepatocyte Nuclear Factor 3-beta - genetics | Rats, Inbred Lew | Transcriptome | Hepatocyte Nuclear Factor 1-alpha - biosynthesis | Liver Failure - pathology | Hepatocytes - pathology | Male | Gene Expression Profiling | Genetic Vectors - therapeutic use | Hepatocytes - metabolism | Gene Regulatory Networks | Hepatocyte Nuclear Factor 4 - physiology | Recombinant Fusion Proteins - metabolism | CCAAT-Enhancer-Binding Protein-alpha - biosynthesis | Hepatocyte Nuclear Factor 4 - biosynthesis | Liver Cirrhosis, Experimental - pathology | Liver Cirrhosis, Experimental - complications | Hepatocyte Nuclear Factor 3-beta - biosynthesis | Transcription Factors - physiology | Transduction, Genetic | PPAR alpha - biosynthesis | Down-Regulation | Cells, Cultured | Gene Expression Regulation | Rats | PPAR alpha - genetics | Hepatocyte Nuclear Factor 4 - genetics | Liver Cirrhosis, Experimental - genetics | Liver Failure - genetics | Disease Progression | Animals | Carbon Tetrachloride Poisoning - therapy | Liver Failure - therapy | CCAAT-Enhancer-Binding Protein-alpha - genetics | Carbon Tetrachloride Poisoning - genetics | Cell Dedifferentiation - genetics | Transcription factors | Liver diseases | Genetic research | Development and progression | Genetic aspects | Genetic transcription | Research | Properties | Hypertension | Cytochrome | Transplants & implants | Rodents | Stem cells | Metabolism | Laboratory animals | Liver cirrhosis | Index Medicus | Abridged Index Medicus | Hepatology | Gastroenterology
Journal Article
Journal Article
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 08/2016, Volume 304, pp. 18 - 29
Perfluorooctanoic acid (PFOA) and perfluorooctanesulfonic acid (PFOS), chemicals present in a multitude of consumer products, are persistent organic... 
Hepatocyte nuclear factor 4-alpha | Perfluorooctanoic acid | Human hepatocyte | Perfluorooctanesulfonic acid | Liver | RECEPTORS PPAR-ALPHA | PERFLUOROALKYL ACIDS | CELL-PROLIFERATION | DEVELOPMENTAL TOXICITY | POTASSIUM PERFLUOROOCTANESULFONATE | CYP7A1 DEFICIENCY | PHARMACOLOGY & PHARMACY | TOXICOLOGY | SERUM-LIPID LEVELS | CHOLESTEROL 7-ALPHA-HYDROXYLASE | HEPATIC STEATOSIS | TRANSCRIPTION FACTOR | Fluorocarbons - toxicity | Caprylates - toxicity | Gene Expression - drug effects | Down-Regulation | Humans | Male | RNA, Messenger - metabolism | Dose-Response Relationship, Drug | Hepatocyte Nuclear Factor 4 - drug effects | Up-Regulation - drug effects | Animals | Sequence Analysis, RNA | Hepatocyte Nuclear Factor 4 - biosynthesis | Alkanesulfonic Acids - toxicity | Cell Proliferation - drug effects | Mice | Real-Time Polymerase Chain Reaction | Hepatocytes - drug effects | RNA sequencing | Liver cancer | Occupational health and safety | RNA | Cell death | Analysis | Ammonium perfluorooctanoate | Persistent organic pollutants | Gene expression | Fatty acids | Index Medicus | ALCOHOL DEHYDROGENASE | APOPTOSIS | DMSO | 60 APPLIED LIFE SCIENCES | CARCINOGENESIS | CYTOCHROMES | MESSENGER-RNA | METABOLISM | TYROSINE | GENES | LIVER CELLS | PHOSPHATES | AMINOTRANSFERASES | LIPIDS | TRANSCRIPTION FACTORS | RODENTS | NEOPLASMS | CELL PROLIFERATION | CONCENTRATION RATIO | ALCOHOLS | ALANINES | LIVER | OCCUPATIONAL EXPOSURE | STEM CELLS | RECEPTORS | hepatocyte nuclear factor 4-alpha | perfluorooctanesulfonic acid | liver | human hepatocyte
Journal Article
Development (Cambridge), ISSN 0950-1991, 05/2018, Volume 145, Issue 10, pp. dev153619 - dev153619
Nephron progenitor cells (NPCs) are Six2-positive metanephric mesenchyme cells, which undergo self-renewal and differentiation to give rise to nephrons until... 
Epigenetics | Nephron progenitors | Histone deacetylase | Kidney development | EMBRYONIC-DEVELOPMENT | METANEPHRIC MESENCHYME | HDAC2 CONTROL | SELF-RENEWAL | DEVELOPMENTAL BIOLOGY | EPITHELIAL TRANSFORMATION | DNA-REPLICATION | NASCENT NEPHRONS | GENE-EXPRESSION | MOUSE KIDNEY | Transcriptional Activation - genetics | Homeodomain Proteins - metabolism | Humans | Hepatocyte Nuclear Factor 1-alpha - biosynthesis | Intercellular Signaling Peptides and Proteins - biosynthesis | Stem Cells - cytology | Kidney Diseases - genetics | Tumor Suppressor Protein p53 - genetics | Nephrons - cytology | Hepatocyte Nuclear Factor 4 - biosynthesis | HEK293 Cells | Histone Deacetylase 1 - genetics | Protein-Serine-Threonine Kinases - metabolism | Cell Line | Histone Deacetylase 2 - genetics | Cell Proliferation - genetics | Nephrons - embryology | Transcription Factors - genetics | Mice, Knockout | Transcription Factors - metabolism | LIM-Homeodomain Proteins - genetics | Animals | Organogenesis - genetics | Mice | Histone Deacetylase 2 - metabolism | Transcription, Genetic - genetics | Histone Deacetylase 1 - metabolism | Phenotypes | Kidneys | Mesenchyme | Transcription | p53 Protein | Lethality | Progenitor cells | Nephrons | Vesicles | Cell fate | Clonal deletion | Rodents | Stem cells | HDAC2 protein | Index Medicus | 203 | Stem Cells and Regeneration | 207
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2012, Volume 287, Issue 36, pp. 30181 - 30190
Journal Article