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Journal of Neurochemistry, ISSN 0022-3042, 09/2012, Volume 122, Issue 5, pp. 941 - 951
J. Neurochem. (2012) 122, 941951. Abstract In vitro and in vivo models of Parkinsons disease (PD) suggest that increased oxidant production leads to... 
hydroethidine | mitochondria | glycolysis | reactive oxygen species | NADPH OXIDASE | OXYGEN SPECIES PRODUCTION | OXIDATIVE STRESS | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | DOPAMINERGIC-NEURONS | PARAQUAT | NEUROSCIENCES | BRAIN MITOCHONDRIA | REACTIVE OXYGEN | ANIMAL-MODELS | CELL-CULTURE | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Insecticides - pharmacology | Rats | Herbicides - pharmacology | Adrenergic Agents - pharmacology | Dose-Response Relationship, Drug | Neurotoxins - pharmacology | Animals | Oligomycins - pharmacology | Oxidopamine - pharmacology | Time Factors | Dopamine Agents - pharmacology | Oxygen Consumption - drug effects | Adenosine Triphosphate - metabolism | Dopaminergic Neurons - metabolism | Proton Ionophores - pharmacology | Rotenone - pharmacology | Superoxides - metabolism | Dopaminergic Neurons - drug effects | Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone - pharmacology | Paraquat - pharmacology | Cell Line, Transformed | Energy Metabolism - drug effects | Glucose metabolism | Herbicides | Parkinson's disease | Cell death | Analysis | Superoxide | Mitochondrial DNA | Universities and colleges | Cells | Neurochemistry | Mitochondria | Neurotransmitters | Bioenergetics | Parkinsons disease | Apoptosis | Index Medicus | Dopamine | Neurodegenerative diseases | 6-Hydroxydopamine | Acidification | Data processing | Oxygen consumption | Rotenone | Toxicants | MPP super(+) | Oxidants | Glycolysis | Paraquat | Oxidation | Movement disorders
Journal Article
Toxicology, ISSN 0300-483X, 2006, Volume 217, Issue 2, pp. 139 - 146
It was originally shown by Woerner and Schrenk [Woerner, W., Schrenk, D., 1998. 2,3,7,8-Tetrachlorodibenzo- -dioxin suppresses apoptosis and leads to... 
Involvement of ERK | c-Src | Anti-apoptotic action of TCDD | Stress response | SURVIVAL | ACTIVATION | HEPATOCYTES | DNA-DAMAGE | C-SRC KINASE | stress response | ARYL-HYDROCARBON RECEPTOR | PATHWAY | GROWTH-FACTOR-ALPHA | 2,3,7,8-TETRACHLORODIBENZO-P-DIOXIN TCDD | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | TOXICOLOGY | involvement of ERK | anti-apoptotic action of TCDD | Nitriles - pharmacology | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Adaptation, Physiological - drug effects | RNA, Messenger - metabolism | Glutathione - pharmacology | Phenanthrolines - pharmacology | Ultraviolet Rays | Time Factors | Polychlorinated Dibenzodioxins - pharmacology | Nitro Compounds - pharmacology | Female | Flavonoids - pharmacology | DNA Fragmentation - drug effects | bcl-2-Associated X Protein - genetics | Transforming Growth Factor alpha - pharmacology | Hydrogen Peroxide - toxicity | Cell Line | Butadienes - pharmacology | Epithelial Cells - radiation effects | Glutathione - analogs & derivatives | RNA, Messenger - genetics | bcl-2-Associated X Protein - metabolism | Ferric Compounds - pharmacology | Pyrimidines - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Pyrroles - pharmacology | Benzoflavones - pharmacology | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Lipopolysaccharides - pharmacology | alpha 1-Antitrypsin - pharmacology | Epidermal Growth Factor - pharmacology | Quaternary Ammonium Compounds - pharmacology | Nitrogen oxide | Herbicides | Epidermal growth factor | Dioxin | Chemical properties | Transforming growth factors | Tumor proteins | Index Medicus
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2012, Volume 33, Issue 4, pp. 767 - 785
Abstract This study aims to elucidate the processes underlying neuroprotection of kaempferol in models of rotenone-induced acute toxicity. We demonstrate that... 
Neurology | Internal Medicine | Oxidative stress | Neuroprotection | Mitochondria | Parkinson's disease | Kaempferol | Mitochondrial toxins | Autophagy | UBIQUITIN-PROTEASOME SYSTEM | PROTEIN | ALZHEIMERS-DISEASE | ALPHA-SYNUCLEIN | SUBSTANTIA-NIGRA | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | CELL-DEATH | GERIATRICS & GERONTOLOGY | COMPLEX-I INHIBITION | BRAIN | Electric Stimulation | Reactive Oxygen Species - metabolism | Rats, Wistar | Apoptosis - drug effects | Microtubule-Associated Proteins - metabolism | Humans | Caspase 3 - metabolism | Membrane Potential, Mitochondrial - drug effects | Protein Carbonylation - drug effects | Autophagy - drug effects | Dose-Response Relationship, Drug | Drug Interactions | Neuroprotective Agents - pharmacology | Transfection | Time Factors | Rotenone - pharmacology | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Cerebral Cortex - drug effects | Neuroblastoma - pathology | Insecticides - pharmacology | Cell Survival - drug effects | Microscopy, Electron, Transmission | Enzyme Inhibitors - pharmacology | Rats | Excitatory Amino Acid Antagonists - pharmacology | Gene Expression Regulation - drug effects | Animals | Neuroblastoma - ultrastructure | Oxygen Consumption - drug effects | Cell Line, Tumor | Kaempferols - pharmacology | Dizocilpine Maleate - pharmacology | In Vitro Techniques | Antioxidants | Herbicides | Analysis | Resveratrol | Fluorescence | Models | Mitochondrial DNA | Cells | Electron microscopy | Protein binding | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, pp. e0132832 - e0132832
Anthropogenic and natural stressors often interact to affect organisms. Amphibian populations are undergoing unprecedented declines and extinctions with... 
CHYTRIDIOMYCOSIS IMPACTS | IMMUNE DEFENSES | MULTIDISCIPLINARY SCIENCES | DISEASE | GROWTH | SUSCEPTIBILITY | BATRACHOCHYTRIUM-DENDROBATIDIS INFECTION | ANTIMICROBIAL PEPTIDE DEFENSES | EXTINCTION | GLOBAL DECLINE | EXPOSURE | Chytridiomycota - physiology | Glycine - analogs & derivatives | Species Specificity | Toluidines - pharmacology | Colony Count, Microbial | Endosulfan - pharmacology | Anura - microbiology | Larva - drug effects | Anura - growth & development | Larva - growth & development | Metamorphosis, Biological - drug effects | Insecticides - pharmacology | Metamorphosis, Biological - physiology | Permethrin - pharmacology | 2,4-Dichlorophenoxyacetic Acid - pharmacology | Herbicides - pharmacology | Atrazine - pharmacology | Chytridiomycota - pathogenicity | Host-Pathogen Interactions | Animals | Glycine - pharmacology | Survival Analysis | Chlorpyrifos - pharmacology | Carbaryl - pharmacology | Larva - microbiology | Drug Combinations | Fungi | Frogs | Herbicides | Insecticides | Amphibians | Endosulfan | Extinct species | Permethrin | Disease | Human influences | Amphibia | Biology | Infections | Reptiles & amphibians | Glyphosate | Endangered & extinct species | Agrochemicals | Risk assessment | Cascades | Population | Metamorphosis | Species | Chlorpyrifos | Pathogens | 2,4-D | Extinction | Mortality | Toads | Pesticides | Exposure | Anthropogenic factors | Population decline | Biological diversity | Studies | Infectious diseases | Environmental science | Carbaryl | Acetochlor | Internet | Atrazine | Environmental protection | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 02/2017, Volume 114, Issue 7, pp. E1091 - E1100
Journal Article
Neuroscience Research, ISSN 0168-0102, 2003, Volume 46, Issue 4, pp. 523 - 532
Paraquat (1,1′-dimethyl-4,4′-bipyridinium, PQ) is a herbicide to possibly induce Parkinson's disease (PD), since a strong correlation has been found between... 
Paraquat | Neurotoxicity | Parkinson's disease | Organotypic midbrain slice | Dopamine neurons | PESTICIDE EXPOSURE | paraquat | NITRIC-OXIDE SYNTHASE | HERBICIDE PARAQUAT | ALPHA-SYNUCLEIN | CEREBROSPINAL-FLUID | NEUROSCIENCES | CELL-DEATH | dopamine neurons | neurotoxicity | PARKINSONS-DISEASE MORTALITY | organotypic midbrain slice | BETA-CARBOLINIUM | MESENCEPHALIC CULTURES | ENVIRONMENTAL RISK-FACTORS | Tyrosine 3-Monooxygenase - metabolism | NADPH Dehydrogenase - metabolism | Rats, Wistar | Mesencephalon - metabolism | Selegiline - pharmacology | Dose-Response Relationship, Drug | Quinoxalines - pharmacology | Neuroprotective Agents - pharmacology | Caspases - pharmacology | Mesencephalon - drug effects | Protein Synthesis Inhibitors - pharmacology | Organ Culture Techniques | Animals, Newborn | NG-Nitroarginine Methyl Ester - pharmacology | Dopamine Agonists - pharmacology | Dopamine Uptake Inhibitors - pharmacology | 1-Methyl-4-phenylpyridinium - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Excitatory Amino Acid Antagonists - pharmacology | Herbicides - toxicity | Piperazines - pharmacology | Cycloheximide - pharmacology | Azepines - pharmacology | Immunohistochemistry - methods | Animals | Cell Count - methods | Analysis of Variance | Dizocilpine Maleate - pharmacology | Paraquat - toxicity | Ergolines - pharmacology | Index Medicus
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 02/2018, Volume 96, Issue 2, pp. 253 - 264
Oxidative stress and neural degeneration have been shown to be involved in the pathogenesis of Parkinson's disease (PD). The P2Y6 purinergic receptor (P2Y6R)... 
Parkinson's disease | P2Y6R | MPP | oxidative stress | SH‐SY5Y | SH-SY5Y | LIPID-PEROXIDATION | SIGNALING MOLECULE | SUBSTANTIA-NIGRA | NEUROSCIENCES | PROTECTS | INHIBITION | PATHWAY | P2Y RECEPTOR | SUPEROXIDE-DISMUTASE | PARKINSONS-DISEASE | Malondialdehyde - metabolism | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | 1-Methyl-4-phenylpyridinium - pharmacology | Humans | Herbicides - pharmacology | Thiourea - pharmacology | Transfection | Apyrase - pharmacology | Receptors, Purinergic P2 - genetics | Receptors, Purinergic P2 - metabolism | Superoxide Dismutase-1 - metabolism | Cell Line, Tumor | Purinergic P2 Receptor Antagonists - pharmacology | Purinergic P2 Receptor Agonists - pharmacology | Cell Death - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Oxidative Stress - drug effects | Uridine Diphosphate - pharmacology | Isothiocyanates - pharmacology | Neuroblastoma - pathology | Thiourea - analogs & derivatives | Nitric Oxide Synthase Type II - metabolism | RNA, Small Interfering - metabolism | Oxidative stress | Neurons | Cell death | Nitric oxide | Biochemistry | Superoxide | Mitogens | Reactive oxygen species | Pathogenesis | Superoxide dismutase | Kinases | Neurodegeneration | Down-regulation | Degeneration | Inhibition | Movement disorders | Neurodegenerative diseases | Extracellular signal-regulated kinase | Pharmacology | Inflammation | siRNA | Malondialdehyde | Nitric-oxide synthase | Microglia | Signaling | Protein kinase | Apyrase | Viability | Apoptosis | Index Medicus
Journal Article
Journal Article
Journal of the Science of Food and Agriculture, ISSN 0022-5142, 01/2017, Volume 97, Issue 2, pp. 488 - 496
BACKGROUNDPrevious studies indicated that tomato is a rich source of phytochemicals that act on different tumours. In this research, the phytochemical... 
HT‐29 cancer cells | carotenoids | fatty acids | Mediterranean diet | olive oil | sterols | tomato varieties | Lycopersicum esculentum | cytotoxicity | phenolics | HT-29 cancer cells | ANTIOXIDANT ACTIVITIES | FOOD SCIENCE & TECHNOLOGY | FATTY-ACIDS | CANCER | COLON-CARCINOMA CELLS | PHYTOSTEROLS | FRUIT | HEALTH-BENEFITS | AGRICULTURE, MULTIDISCIPLINARY | VEGETABLES | CHEMISTRY, APPLIED | Plant Extracts - chemistry | Carotenoids - analysis | Cell Proliferation | Phytochemicals - therapeutic use | Species Specificity | Plant Extracts - pharmacology | Humans | Carotenoids - pharmacology | Phytosterols - pharmacology | Dietary Fats, Unsaturated - pharmacology | Linoleic Acid - analysis | Olive Oil - pharmacology | Carotenoids - therapeutic use | Lycopersicon esculentum - classification | Colorectal Neoplasms - drug therapy | Linoleic Acid - therapeutic use | Phytosterols - analysis | Linoleic Acid - pharmacology | Phytotherapy | Antineoplastic Agents, Phytogenic - therapeutic use | Antioxidants - analysis | Phytosterols - therapeutic use | Phenols - pharmacology | Dietary Fats, Unsaturated - therapeutic use | Phytochemicals - analysis | Fruit - chemistry | Antioxidants - pharmacology | Drug Synergism | HT29 Cells | Antioxidants - therapeutic use | Phenols - analysis | Phenols - therapeutic use | Dietary Fats, Unsaturated - analysis | Olive Oil - therapeutic use | Colorectal Neoplasms - diet therapy | Lycopersicon esculentum - chemistry | Antineoplastic Agents, Phytogenic - pharmacology | Phytochemicals - pharmacology | Plant Extracts - therapeutic use | Chemical industry | Herbicides | Pesticides industry | Unsaturated fatty acids | Sterols | Colorectal cancer | Tomatoes | Lycopene | Tumors | Phytochemicals | Index Medicus | Biotechnology | Carotenoids | In vitro testing | Foods | Cherries | Olive oil | Fruits | Cancer
Journal Article
Journal of Biochemical and Molecular Toxicology, ISSN 1095-6670, 09/2018, Volume 32, Issue 9, pp. e22196 - n/a
Glutathione‐ S ‐transferases (GSTs) have a function in xenobiotic metabolism. They are a significant multifunctional family with a wide variety of catalytic... 
inhibition | transferase | Van Lake fish | pesticides | glutathione | purification | glutathione-S-transferase | CHALCALBURNUS-TARICHII PALLAS | CHLORPYRIFOS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TOXICOLOGY | HISTOPATHOLOGY | Glycine - analogs & derivatives | Glycine - metabolism | Liver - enzymology | Molecular Weight | Nitriles - pharmacology | Species Specificity | Glutathione Transferase - isolation & purification | Dimethylamines - pharmacology | Fish Proteins - isolation & purification | Pyrethrins - pharmacology | Fungicides, Industrial - pharmacology | Liver - growth & development | Dimethylamines - metabolism | Fungicides, Industrial - metabolism | Glutathione Transferase - antagonists & inhibitors | Enzyme Inhibitors - toxicity | Fish Proteins - antagonists & inhibitors | Pesticides - metabolism | Pyrethrins - metabolism | 2,4-Dichlorophenoxyacetic Acid - metabolism | Binding, Competitive | Nitriles - metabolism | Enzyme Inhibitors - metabolism | Fish Proteins - chemistry | Water Pollutants, Chemical - pharmacology | 2,4-Dichlorophenoxyacetic Acid - pharmacology | Fish Proteins - metabolism | Glutathione Transferase - metabolism | Glutathione Transferase - chemistry | Dichlorvos - pharmacology | Water Pollutants, Chemical - metabolism | Animals | Pyridines - metabolism | Turkey | Glycine - pharmacology | Saline Waters | Lakes | Pyridines - pharmacology | Kinetics | Cyprinidae - growth & development | Dichlorvos - metabolism | Pesticides - pharmacology | Herbicides | Glutathione transferase | Analysis | Liver | Pesticides | Physiological aspects | Xenobiotics | Chromatography | Enzyme activity | Metabolism | Glyphosate | Enzymatic activity | Dichlorvos | Dichlorophenoxyacetic acid | Fish | Catalysis | Inhibition | Glutathione | Cyhalothrin | Index Medicus
Journal Article