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Oncogene, ISSN 0950-9232, 01/2016, Volume 35, Issue 1, pp. 12 - 21
Journal Article
Cell, ISSN 0092-8674, 10/2017, Volume 171, Issue 2, pp. 358 - 371.e9
Journal Article
Nature, ISSN 0028-0836, 08/2015, Volume 524, Issue 7565, pp. 361 - 365
Journal Article
Journal Article
Nature, ISSN 0028-0836, 09/2017, Volume 549, Issue 7673, pp. 533 - 537
High-grade gliomas (HGG) are a devastating group of cancers, and represent the leading cause of brain tumour-related death in both children and adults.... 
AUTISM | PEPTIDES | PHOSPHORYLATION | DISINTEGRIN | SUBTYPES | MULTIDISCIPLINARY SCIENCES | OLIGODENDROCYTE PRECURSOR CELLS | MUTATION | HIPPOCAMPAL | SELF-RENEWAL | CLEAVAGE | Neoplasm Transplantation | Neurons - pathology | Oligodendroglia - metabolism | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Nerve Tissue Proteins - deficiency | Brain Neoplasms - pathology | ADAM10 Protein - metabolism | Tumor Microenvironment | Male | Phosphatidylinositol 3-Kinases - metabolism | Brain Neoplasms - metabolism | Glioma - metabolism | Cell Adhesion Molecules, Neuronal - deficiency | Glioma - genetics | Membrane Proteins - deficiency | ADAM10 Protein - antagonists & inhibitors | Heterografts | Glioma - pathology | Adult | Cell Adhesion Molecules, Neuronal - metabolism | Membrane Proteins - metabolism | Neurons - metabolism | Oligodendroglia - cytology | Child | Signal Transduction | Focal Adhesion Protein-Tyrosine Kinases - metabolism | Membrane Proteins - genetics | Brain Neoplasms - genetics | Nerve Tissue Proteins - genetics | Cell Adhesion Molecules, Neuronal - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Amyloid Precursor Protein Secretases - metabolism | Animals | Membrane Proteins - antagonists & inhibitors | Mice | Amyloid Precursor Protein Secretases - antagonists & inhibitors | Gliomas | Health aspects | Research | TOR protein | Brain | Phosphorylation | Transcription | Brain tumors | Brain cancer | Genes | Glioblastoma | Genomes | Kinases | Glial stem cells | Proteins | Signal transduction | Cell growth | Proteolysis | Glioma cells | Xenografts | Children | Secretion | Adhesion | 1-Phosphatidylinositol 3-kinase | Oligodendroglioma | Signaling | Proteomics | Focal adhesion kinase | Cancer | Index Medicus
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2015, Volume 149, Issue 7, pp. 1884 - 1895.e4
Background & Aims Inflammation may contribute to the formation, maintenance, and expansion of cancer stem cells (CSCs), which have the capacity for... 
Gastroenterology and Hepatology | Cyclooxygenase | Tumorigenesis | NSAIDs | Colon Cancer | Neoplastic Stem Cells - drug effects | Colorectal Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Male | NF-kappa B - metabolism | Dinoprostone - administration & dosage | Genes, APC | Heterografts | Transfection | Neoplastic Stem Cells - metabolism | RNA Interference | Inflammation Mediators - metabolism | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Tumor Cells, Cultured | Liver Neoplasms - secondary | Phosphatidylinositol 3-Kinase - metabolism | Colorectal Neoplasms - metabolism | Carcinoma - secondary | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Liver Neoplasms - prevention & control | Receptors, Prostaglandin E, EP4 Subtype - genetics | Cyclooxygenase 2 Inhibitors - pharmacology | Signal Transduction | Colorectal Neoplasms - prevention & control | Carcinoma - prevention & control | Mice, Transgenic | Tumor Burden | Mice, SCID | Dinoprostone - metabolism | Cell Movement - drug effects | Animals | NF-kappa B - genetics | Phosphatidylinositol 3-Kinase - genetics | Liver Neoplasms - metabolism | Cell Line, Tumor | Carcinoma - genetics | Mice, Inbred NOD | Mitogen-Activated Protein Kinases - genetics | Biomarkers, Tumor - genetics | Cell Proliferation - drug effects | Carcinoma - metabolism | Colorectal Neoplasms - pathology | Mitogen-Activated Protein Kinases - metabolism | Index Medicus | Abridged Index Medicus | cyclooxygenase | tumorigenesis | colon cancer
Journal Article
Cancer Discovery, ISSN 2159-8274, 07/2016, Volume 6, Issue 7, pp. 770 - 783
Journal Article
Nature, ISSN 0028-0836, 09/2014, Volume 514, Issue 7522, pp. 385 - 388
Journal Article
Leukemia, ISSN 0887-6924, 03/2016, Volume 30, Issue 3, pp. 728 - 739
The t(8;21) rearrangement, which creates the AML1-ETO fusion protein, represents the most common chromosomal translocation in acute myeloid leukemia (AML).... 
MUTATED C-CBL | CANCER GENOMICS | ONCOLOGY | T(8/21) AML | HEMATOPOIETIC STEM-CELLS | MOUSE MODEL | JUVENILE MYELOMONOCYTIC LEUKEMIA | ACQUIRED UNIPARENTAL DISOMY | SELF-RENEWAL | MYELOPROLIFERATIVE NEOPLASMS | HEMATOLOGY | TYROSINE-PHOSPHATASE | Myeloid Cells - cytology | RNA, Small Interfering - genetics | Translocation, Genetic | Oncogene Proteins, Fusion - metabolism | Proto-Oncogene Proteins c-cbl - metabolism | Cell Proliferation | Humans | Leukemia, Myeloid, Acute - metabolism | Preleukemia - pathology | MicroRNAs - metabolism | Protein Tyrosine Phosphatases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Proto-Oncogene Proteins c-akt - genetics | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Protein Tyrosine Phosphatases - genetics | Heterografts | Preleukemia - genetics | src-Family Kinases - metabolism | Myeloid Cells - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Transgenes | Chromosomes, Human, Pair 21 | Proto-Oncogene Proteins c-cbl - genetics | Core Binding Factor Alpha 2 Subunit - metabolism | Leukemia, Myeloid, Acute - pathology | Chromosomes, Human, Pair 8 | Thrombopoietin - pharmacology | Fetal Blood - cytology | Gene Expression Regulation, Leukemic | Fetal Blood - drug effects | Fetal Blood - metabolism | Mice, SCID | RUNX1 Translocation Partner 1 Protein | Preleukemia - metabolism | Animals | Oncogene Proteins, Fusion - genetics | Myeloid Cells - metabolism | Proto-Oncogene Proteins c-cbl - antagonists & inhibitors | Mice | MicroRNAs - genetics | src-Family Kinases - genetics | Core Binding Factor Alpha 2 Subunit - genetics | Leukemia, Myeloid, Acute - genetics | RNA, Small Interfering - metabolism | Molecular targeted therapy | Gene mutations | Translocation (Genetics) | Health aspects | Innovations | Index Medicus | AML1-ETO | miR-9 | UBASH3B | acute myeloid leukemia | CBL | Sts-1
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1480 - 17