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PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 11, p. e79977
... to the transition into reproductive senescence. The deficit in brain metabolism was sustained thereafter... 
MECHANISM | ASTROCYTE | MULTIDISCIPLINARY SCIENCES | LACTATE | DISEASE | HIPPOCAMPAL | ESTRADIOL | COGNITIVE IMPAIRMENT | ENERGY-METABOLISM | EXPRESSION | OLIGODENDROCYTES | Adaptation, Physiological | Ketone Oxidoreductases - metabolism | Neurons - pathology | Humans | Ketone Bodies - metabolism | Astrocytes - pathology | Glucose Transporter Type 1 - metabolism | Alzheimer Disease - pathology | Biological Transport, Active | Monocarboxylic Acid Transporters - metabolism | Aging - genetics | Female | Neurons - metabolism | Hexokinase - genetics | Disease Models, Animal | Gene Expression | Lactic Acid - metabolism | Mice, Transgenic | Hippocampus - pathology | Mitochondria - metabolism | Ketone Oxidoreductases - genetics | Aging - pathology | Blood-Brain Barrier - metabolism | Hippocampus - metabolism | Animals | Energy Metabolism | Glucose Transporter Type 1 - genetics | Alzheimer Disease - metabolism | Glucose - metabolism | Mice | Monocarboxylic Acid Transporters - genetics | Alzheimer Disease - genetics | Hexokinase - metabolism | Aging - metabolism | Astrocytes - metabolism | Lactates | Glucose metabolism | Enzymes | Brain | Neurons | Glucose | Alzheimer's disease | Dextrose | Neurosciences | Senescence | Adaptive systems | Dehydrogenases | Pyruvic acid | Ketone bodies | Fuels | Mitochondria | Bioenergetics | Blood-brain barrier | Rodents | Aging | Pharmaceutical sciences | Age | Glucose transporter | Phenotypes | Medical imaging | Neurodegenerative diseases | Ketones | Astrocytes | Pharmacology | Metabolism | Glucose transport | Fatty acids | Hexokinase | Utilization | Pharmacy | Glycolysis | Lactic acid | Laboratory animals | Alzheimers disease | Axonal transport | Transporter | Hippocampus | Animal cognition
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2016, Volume 311, Issue 2, pp. H347 - H363
Dramatic maturational changes in cardiac energy metabolism occur in the newborn period, with a shift from glycolysis to fatty acid oxidation... 
Lysine acetylation | Lysine succinylation | Myocardial fatty acid oxidation | Newborn heart | MITOCHONDRIAL-PROTEIN-ACETYLATION | BIVENTRICULAR WORKING HEARTS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | GLUCOSE-OXIDATION | lysine acetylation | NEONATAL RABBIT HEARTS | lysine succinylation | FATTY-ACID OXIDATION | INSULIN-RESISTANCE | CARDIAC-HYPERTROPHY | PERIPHERAL VASCULAR DISEASE | OVERLOAD HYPERTROPHY | myocardial fatty acid oxidation | CONTRACTILE FUNCTION | newborn heart | Phosphoglycerate Mutase - metabolism | Immunoprecipitation | Mitochondria, Heart - metabolism | 3-Hydroxyacyl CoA Dehydrogenases - metabolism | Immunoblotting | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Mitochondrial Proteins - metabolism | Adenosine Triphosphate - metabolism | Myocardium - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Lysine - metabolism | Acetylation | Fatty Acids - metabolism | Fetal Heart - metabolism | Animals, Newborn | Cell Line | Rabbits | Oxidation-Reduction | Acyl-CoA Dehydrogenase, Long-Chain - metabolism | Rats | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Animals | Energy Metabolism | Succinic Acid - metabolism | Glycolysis | Protein Processing, Post-Translational | Hexokinase - metabolism | In Vitro Techniques | Heart | Infants (Newborn) | Physiological aspects | Energy metabolism | Bioenergetics
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 3, p. e59825
... substrate availability and metabolism in a mouse model of familial Alzheimer's (3xTgAD). Results of these analyses indicated that ovarian hormones deprivation by ovariectomy (OVX... 
POSITRON-EMISSION-TOMOGRAPHY | HORMONE-THERAPY | MULTIDISCIPLINARY SCIENCES | BRAIN GLUCOSE-METABOLISM | AEROBIC GLYCOLYSIS | IN-VIVO | A-BETA | HEXOKINASE-ACTIVITY | AMYLOID-BETA-PROTEIN | RAT-BRAIN | ENERGY-METABOLISM | L-Lactate Dehydrogenase - metabolism | Body Weight | Positron-Emission Tomography | X-Ray Microtomography | Isoenzymes - metabolism | Amyloid beta-Peptides - metabolism | Female | Neurons - metabolism | Estrogens - metabolism | Skin Temperature | Disease Models, Animal | Ovary - metabolism | Estradiol - metabolism | Ovariectomy | Gene Expression Regulation | Mice, Transgenic | Mitochondria - metabolism | Hippocampus - metabolism | Animals | Lactates - metabolism | Brain - pathology | Glucose - metabolism | Glycolysis | Mice | Alzheimer Disease - genetics | Brain | Glucose metabolism | Neurons | Physiological aspects | Genetic engineering | Genetic aspects | Glucose | Gene expression | Estradiol | Alzheimer's disease | Dextrose | Cytochrome | Energy metabolism | Estrogens | Ketone bodies | Hormones | Proteins | Mitochondria | Bioenergetics | Aging | Availability | Glucose transporter | Preservation | Neurodegenerative diseases | Metabolism | Glucose transport | Substrates | Neurology | Sex hormones | Pharmacy | Transporter | Neuroimaging | Neurosciences | Senescence | 17β-Estradiol | Kinases | Energy resources | Rodents | Pharmaceutical sciences | Deprivation | Medical imaging | Ketones | Transgenic mice | CoA transferase | Pharmacology | Fuel technology | Hexokinase | Lactic acid | Respiration | Laboratory animals | Alzheimers disease | Alternative fuels
Journal Article
Journal Article
Journal Article
Molecular psychiatry, ISSN 1476-5578, 2018, Volume 24, Issue 9, pp. 1319 - 1328
Journal Article
Nature communications, ISSN 2041-1723, 2018, Volume 9, Issue 1, pp. 1480 - 17
Glycolytic reprogramming is a typical feature of many cancers; however, key regulators of glucose metabolism reengineering are poorly understood, especially in cutaneous squamous cell carcinoma (cSCC). Here, Homeobox A9 (HOXA9... 
OXYGEN | LUNG-CANCER | APOPTOSIS | MIR-365 | HEXOKINASE 2 | PROTEIN | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | MULTIDISCIPLINARY SCIENCES | GROWTH | TRANSCRIPTION | GENE-EXPRESSION | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Homeodomain Proteins - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Glucose Transporter Type 1 - metabolism | Male | MicroRNAs - metabolism | LIM Domain Proteins - metabolism | Case-Control Studies | Cellular Reprogramming | Glycolysis - genetics | Heterografts | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Female | Hexokinase - genetics | Protein-Serine-Threonine Kinases - metabolism | Skin Neoplasms - pathology | Promoter Regions, Genetic | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Protein-Serine-Threonine Kinases - genetics | Homeodomain Proteins - genetics | Skin Neoplasms - metabolism | Keratinocytes - pathology | Animals | Glucose Transporter Type 1 - genetics | Keratinocytes - metabolism | Mice, Nude | Skin Neoplasms - genetics | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Protein Binding | LIM Domain Proteins - genetics | Aged | MicroRNAs - genetics | Adaptor Proteins, Signal Transducing - metabolism | Hexokinase - metabolism | Homeobox | Glucose metabolism | Repressing | Regulators | Squamous cell carcinoma | Target recognition | Reengineering | Glycolysis | Tumor suppressor genes | Gene expression | Metabolism | Tumors
Journal Article
Cell death & disease, ISSN 2041-4889, 2014, Volume 5, Issue 5, pp. e1212 - e1212
Oxidative stress serves as an important regulator of both apoptosis and metabolic reprogramming in tumor cells. Chaetocin, a histone methyltransferase... 
YAP | ATM | Metabolism | JNK | Glioblastoma | ROS | YES-ASSOCIATED PROTEIN | GLIOMA-CELLS | ACTIVATION | DNA-DAMAGE | CANCER | CELL BIOLOGY | GROWTH | KAPPA-B AXIS | metabolism | UP-REGULATION | GLIOBLASTOMA CELLS | Reactive Oxygen Species - metabolism | Ataxia Telangiectasia Mutated Proteins - metabolism | Apoptosis - drug effects | Humans | Brain Neoplasms - pathology | JNK Mitogen-Activated Protein Kinases - metabolism | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Dose-Response Relationship, Drug | Glioma - genetics | Transfection | RNA Interference | Time Factors | Adenosine Triphosphate - metabolism | Glioma - pathology | Antineoplastic Agents - pharmacology | Brain Neoplasms - enzymology | Tumor Suppressor Proteins - metabolism | Glioma - enzymology | Pyruvate Kinase - metabolism | Lactic Acid - metabolism | Brain Neoplasms - genetics | Nuclear Proteins - metabolism | Phosphoproteins - genetics | Brain Neoplasms - drug therapy | Piperazines - pharmacology | Xenograft Model Antitumor Assays | p300-CBP Transcription Factors - metabolism | Tumor Protein p73 | Animals | Signal Transduction - drug effects | Tumor Burden - drug effects | Mice, Nude | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Glucose - metabolism | Cell Proliferation - drug effects | Mice | Enzyme Activation | Oxidative Stress - drug effects | Adaptor Proteins, Signal Transducing - metabolism | Hexokinase - metabolism | Glioma - drug therapy | Original
Journal Article