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Journal Article
Journal of lipid research, ISSN 1539-7262, 2017, Volume 58, Issue 8, pp. 1536 - 1547
HDL plays crucial roles at multiple stages of the pathogenesis of atherosclerosis. AMP-activated protein kinase (AMPK) is a therapeutic candidate for the... 
Apolipoprotein E deficient | Adenosine monophosphate-activated protein kinase | Atherosclerosis | Reverse cholesterol transport | atherosclerosis | reverse cholesterol transport | SR-BI | PROTEIN-KINASE | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | apolipoprotein E deficient | IDENTIFICATION | REDUCES ATHEROSCLEROSIS | HDL | EFFLUX | PARAOXONASE-1 | adenosine monophosphate-activated protein kinase | EXPRESSION | Cell Line | AMP-Activated Protein Kinases - metabolism | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Mice, Inbred C57BL | Lipoproteins, HDL - metabolism | Body Weight - drug effects | Male | Hepatocytes - metabolism | Enzyme Activation - drug effects | Atherosclerosis - metabolism | Gene Expression Regulation - drug effects | Atherosclerosis - enzymology | Animals | ATP Binding Cassette Transporter, Subfamily G, Member 1 - genetics | CD36 Antigens - metabolism | Lipid Peroxidation - drug effects | Mice | Macrophages, Peritoneal - metabolism | Hepatocytes - drug effects | ATP Binding Cassette Transporter 1 - genetics | Phosphates | AMP | Liver | Paraoxonase 1 | Lipid peroxidation | Inflammation | Glucose | Paraoxonase | AMP-activated protein kinase | Kinases | Macrophages | Cholesterol | High fat diet | ABCA1 protein | Antioxidants | Lipoproteins | Cell activation | Apolipoprotein E | Arteriosclerosis | ATP-binding protein | Peroxidase | Metformin | Lipoproteins (high density)
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 03/2010, Volume 55, Issue 13, pp. 1283 - 1299
Coronary heart disease remains a major cause of worldwide morbidity and mortality despite therapeutic advances that control many risk factors such as low-density lipoprotein cholesterol to levels... 
coronary artery disease | prognosis | high-density lipoprotein | prevention | CHOLESTERYL-ESTER TRANSFER | CARDIAC & CARDIOVASCULAR SYSTEMS | TRANSFER PROTEIN INHIBITOR | PROLIFERATOR-ACTIVATED RECEPTOR | MODERATE ALCOHOL-CONSUMPTION | ELEVATED OXIDATIVE STRESS | TYPE-2 DIABETES-MELLITUS | EXTENDED-RELEASE NIACIN | CYTOKINE-INDUCED EXPRESSION | APOLIPOPROTEIN-A-I | LOW HDL-CHOLESTEROL | Cholesterol Ester Transfer Proteins - antagonists & inhibitors | Humans | Coronary Disease - physiopathology | Coronary Artery Disease - blood | Niacin - pharmacology | Cholesterol, HDL - physiology | Diabetic Angiopathies - physiopathology | Thiazolidinediones - therapeutic use | Coronary Artery Disease - physiopathology | Lipoproteins, HDL - drug effects | Smoking - physiopathology | Thiazolidinediones - pharmacology | Coronary Disease - blood | Glycemic Index | Endothelium, Vascular - physiopathology | Diabetic Angiopathies - blood | Diabetic Angiopathies - drug therapy | Vitamin B Complex - pharmacology | Lipoproteins, HDL - physiology | Animals | Coronary Artery Disease - therapy | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Acetyl-CoA C-Acetyltransferase | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Coronary Disease - drug therapy | Apolipoprotein A-I - physiology | Biological Transport - physiology | Exercise - physiology | Cardiovascular disease | Heart attacks | Apolipoproteins | Cholesterol
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2017, Volume 69, Issue 7, pp. 789 - 800
... (hazard ratio per 1 SD [HR/SD]: 1.40; 95% confidence interval [CI]: 1.17 to 1.67), apoC-III (HR/SD: 1.38; 95% CI: 1.17 to 1.63), and apoE (HR/SD: 1.31; 95% CI: 1.13 to 1.52). Associations were independent of high-density lipoprotein... 
Cardiovascular | Internal Medicine | mass spectrometry | triglycerides | proteomics | lipidomics | CARDIAC & CARDIOVASCULAR SYSTEMS | RISK | OF-FUNCTION MUTATIONS | REMNANT CHOLESTEROL | METABOLISM | LDL | DISEASE | C-III | SECRETION | PLASMA TRIGLYCERIDES | Apolipoprotein C-III - antagonists & inhibitors | Prospective Studies | Cardiovascular Diseases - prevention & control | Humans | Middle Aged | Male | Apolipoproteins - blood | Incidence | Lipoproteins, VLDL - blood | Mass Spectrometry | Cardiovascular Diseases - blood | Cardiovascular Diseases - epidemiology | Female | Aged | Medical colleges | Research institutes | Blood lipids | Apolipoproteins | Low density lipoproteins | Analysis | Mass spectrometry | Heart attack | Blood proteins | Studies | Lipids | Low density lipoprotein | Statins | HDL-C, high-density lipoprotein cholesterol | MS, mass spectrometry | PC, phosphatidylcholine | LDL-C, low-density lipoprotein cholesterol | CE, cholesterol ester | CVD, cardiovascular disease | DAG, diacylglycerol | VLDL, very-low-density lipoprotein | TAG, triacylglycerol | TG, triglyceride | TRL, triglyceride-rich lipoprotein | apo, apolipoprotein | FCS, familial chylomicronemia syndrome | MRM, multiple reaction monitoring | HDL, high-density lipoprotein | LDL, low-density lipoprotein | Original Investigation
Journal Article
JAMA : the journal of the American Medical Association, ISSN 0098-7484, 11/2012, Volume 308, Issue 23, pp. 1 - 10
Journal Article
by Chen, W and Stambolian, D and Edwards, A. O and Branham, K. E and Othman, M and Jakobsdottir, J and Tosakulwong, N and Pericak-Vance, M. A and Campochiaro, P. A and Klein, M. L and Tan, P. L and Conley, Y. P and Kanda, A and Kopplin, L and Li, Y and Augustaitis, K. J and Karoukis, A. J and Scott, W. K and Agarwal, A and Kovach, J. L and Schwartz, S. G and Postel, E. A and Brooks, M and Baratz, K. H and Brown, W. L and Brucker, A. J and Orlin, A and Brown, G and Ho, A and Regillo, C and Donoso, L and Tian, L and Kaderli, B and Hadley, D and Hagstrom, S. A and Peachey, N. S and Klein, R and Klein, B. E. K and Gotoh, N and Yamashiro, K and Ferris, F and Fagerness, J. A and Reynolds, R and Farrer, L. A and Kim, I. K and Miller, J. W and Corton, M and Carracedo, A and Sanchez-Salorio, M and Pugh, E. W and Doheny, K. F and Brion, M and DeAngelis, M. M and Weeks, D. E and Zack, D. J and Chew, E. Y and Heckenlively, J. R and Yoshimura, N and Iyengar, S. K and Francis, P. J and Katsanis, N and Seddon, J. M and Haines, J. L and Gorin, M. B and Abecasis, G. R and Swaroop, A and Johnson, R. N and Ai, E and McDonald, H. R and Stolarczuk, M and Pavan, P. R and Billiris, K. K and Iyer, M and Menosky, M. M and Pautler, S. E and Millard, S. M and Hubbard, B and Aaberg, T and DuBois, L and Lyon, A and Anderson-Nelson, S and Jampol, L. M and Weinberg, D. V and Munana, A and Rozenbajgier, Z and Orth, D and Cohen, J and MacCumber, M and Figliulo, C and Porcz, L and Folk, J and Boldt, H. C and Russell, S. R and Ivins, R and Hinz, C. J and Barr, C. C and Bloom, S and Jaegers, K and Kritchman, B and Whittington, G and ... and Complications of Age-Related Macular Degeneration Prevention Trial (CAPT) Research Group and Complications Age-Related Macular and Complications of Age-Related Macular Degeneration Prevention Trial Research Group
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2010, Volume 107, Issue 16, pp. 7401 - 7406
Journal Article
Journal Article
Circulation (New York, N.Y.), ISSN 0009-7322, 2013, Volume 127, Issue 8, pp. 891 - 904
...). High-density lipoprotein from healthy subjects (HDLHealthy) has been proposed to exert endothelial antiapoptotic effects that may represent an important antiatherogenic property of the lipoprotein... 
clusterin | apoptosis | apolipoprotein C-III | proteomics | high-density lipoprotein | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | SCAVENGER RECEPTOR-BI | STATISTICAL-MODEL | PLAQUE EROSION | HDL | CHOLESTEROL EFFLUX | PERIPHERAL VASCULAR DISEASE | A-I | BINDING | APOLIPOPROTEIN-C-III
Journal Article
2007, ISBN 3527317171, xxii, 530
Christopher Fielding is Neider Professor of Cardiovascular Physiology at the University of California at San Francisco (UCSF). He graduated from University... 
High density lipoproteins | Lipoproteins | Lipoproteins, HDL
Book
Journal of the American College of Cardiology, ISSN 0735-1097, 2013, Volume 61, Issue 4, pp. 427 - 436
Objectives The aim of this study was to test the hypothesis that elevated nonfasting remnant cholesterol is a causal risk factor for ischemic heart disease independent of reduced high-density lipoprotein (HDL) cholesterol... 
Cardiovascular | Internal Medicine | atherosclerosis | cardiovascular disease | lipoproteins | myocardial infarction | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | PREDICTIVE-VALUE | MENDELIAN RANDOMIZATION | PARTICLE CHOLESTEROL | DIABETES-MELLITUS | DENSITY-LIPOPROTEIN CHOLESTEROL | NONFASTING TRIGLYCERIDES | CARDIOVASCULAR-DISEASE | HDL CHOLESTEROL | CORONARY-ARTERY-DISEASE | Myocardial Ischemia - genetics | Myocardial Ischemia - metabolism | Lipoproteins, HDL - genetics | Humans | Middle Aged | Risk Factors | Lipoproteins, HDL - metabolism | Genotype | Male | Data Collection | Cholesterol - metabolism | Cholesterol - genetics | Genetic Variation - physiology | Triglycerides - metabolism | Time Factors | Denmark - epidemiology | Female | Aged | Triglycerides - genetics | Causality | Odds Ratio | Myocardial Ischemia - epidemiology | Analysis and chemistry | Blood cholesterol | Low density lipoproteins | Atherosclerosis | Myocardial ischemia | Universities and colleges | Heart attack | Risk factors | Blood | Cholesterol | Heart | Lipoproteins (low density) | Cardiovascular disease | Density | Confidence intervals | Variables | Randomization | Ischemia | Population | Blood pressure | Heart diseases | Lipoproteins (high density) | Age | Deoxyribonucleic acid--DNA | Hypertension | Enzymes | Health risks | Triglycerides | Risk analysis | Apolipoproteins | Low density lipoprotein | Coronary artery disease | Studies | Genetic variance | Lipoproteins | Diabetes | Cardiovascular diseases
Journal Article