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Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 11/2017, Volume 24, Issue 11, pp. 1948 - 1962
High Mobility Group A1 (HMGA1) is an architectural chromatin protein whose overexpression is a feature of malignant neoplasias with a causal role in cancer... 
TRANSFORMATION | HMGA PROTEINS | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RESISTANCE | REQUIRES | DEATH | TRANSCRIPTIONAL REGULATION | EXPRESSION | MTOR | TRANSGENIC MICE | CELL BIOLOGY | Neoplasms - metabolism | Skin Neoplasms - pathology | Green Fluorescent Proteins - metabolism | Cell Proliferation | Cell Survival | Microtubule-Associated Proteins - metabolism | Humans | Gene Silencing | Protein-Serine-Threonine Kinases - genetics | Autophagy-Related Protein-1 Homolog - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | HMGA1a Protein - metabolism | Autophagy | Gene Knockdown Techniques | Skin Neoplasms - metabolism | Animals | Autophagy-Related Protein 5 - metabolism | Transcription, Genetic | Mice | HeLa Cells | Neoplasms - pathology | Autophagy-Related Protein-1 Homolog - genetics | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | TOR protein | Cell proliferation | Chromatin | Deregulation | Transcription | Mobility | Phagosomes | A1 protein | Kinases | DNA repair | Skin cancer | Proteins | Cell growth | Protein folding | Deoxyribonucleic acid--DNA | Cell survival | Stability | Organelles | Survival | High mobility group proteins | Depletion | Cell death | Tumor suppressor genes | Viability | Phagocytosis | Cancer | Apoptosis | Original Paper
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2008, Volume 28, Issue 10, pp. 1825 - 1830
Journal Article
Hepatology, ISSN 0270-9139, 05/2018, Volume 67, Issue 5, pp. 1823 - 1841
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 05/2017, Volume 394, pp. 22 - 32
Hepatitis B virus X protein (HBx) plays an important role in the progression of hepatocellular carcinoma. Here we reported that overexpression of HBx in... 
High mobility group box 1 | Calmodulin dependent protein kinase | Hepatocellular carcinoma | Metastasis | Hepatitis B virus X protein | CELLS | DNA-BINDING | PHOSPHATIDYLINOSITOL 3-KINASE | HMGB1 | RELEASE | HBX PROTEIN | LIVER-CANCER | IN-VITRO | ONCOLOGY | NUCLEAR FACTOR | NF-KAPPA-B | Hepatitis B - metabolism | Hepatitis B - genetics | Humans | Lung Neoplasms - metabolism | Middle Aged | Hepatitis B - virology | Male | Antineoplastic Agents - therapeutic use | Viral Load | Hepatitis B - complications | Time Factors | HMGB1 Protein - metabolism | Liver Neoplasms - pathology | Calcium Chelating Agents - pharmacology | HMGB1 Protein - antagonists & inhibitors | Liver Neoplasms - virology | Carcinoma, Hepatocellular - virology | Host-Pathogen Interactions | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - antagonists & inhibitors | Cell Movement - drug effects | Lung Neoplasms - prevention & control | Gene Expression Regulation, Viral | Hepatitis B virus - genetics | Liver Neoplasms - metabolism | Cell Line, Tumor | Mice, Inbred NOD | Hepatitis B virus - metabolism | DNA, Viral - genetics | Carcinoma, Hepatocellular - metabolism | Prognosis | Calcium-Calmodulin-Dependent Protein Kinase Type 4 - metabolism | Carcinoma, Hepatocellular - secondary | Liver Neoplasms - therapy | Transfection | Lung Neoplasms - secondary | Trans-Activators - genetics | Female | Calcium Signaling | Proportional Hazards Models | Lung Neoplasms - virology | Mice, SCID | Antibodies - pharmacology | Xenograft Model Antitumor Assays | Animals | Calcium-Calmodulin-Dependent Protein Kinase Kinase - antagonists & inhibitors | Calcium-Calmodulin-Dependent Protein Kinase Kinase - metabolism | Trans-Activators - metabolism | Carcinoma, Hepatocellular - therapy | Protein Kinase Inhibitors - pharmacology | Medical colleges | Chromosomal proteins | Liver | Antibodies | Development and progression | Hepatoma | Viral antibodies | International cooperation | Analysis | Cellular signal transduction | Hepatitis B virus | Health aspects | Hepatitis B | Protein kinases | Calmodulin
Journal Article
Seminars in Arthritis and Rheumatism, ISSN 0049-0172, 2016, Volume 46, Issue 4, pp. 444 - 450
Journal Article
Journal of Thrombosis and Haemostasis, ISSN 1538-7933, 01/2007, Volume 5, Issue 1, pp. 109 - 116
Background: Sepsis is a life‐threatening disorder resulting from systemic inflammatory and coagulatory responses to infection. High‐mobility group box 1... 
high‐mobility group box 1 protein | thrombin | protein C | disseminated intravascular coagulation | sepsis | thrombomodulin | Disseminated intravascular coagulation | Protein C | Sepsis | Thrombin | Thrombomodulin | High-mobility group box 1 protein | TISSUE-DAMAGE | HMGB1 | NECROTIC CELLS | high-mobility group box 1 protein | ENDOTHELIAL-CELLS | HUMAN THROMBOMODULIN | MOBILITY GROUP BOX-1 | PERIPHERAL VASCULAR DISEASE | C ACTIVATION | HEMATOLOGY | EXPRESSION | SEVERE SEPSIS | Hemolysis - drug effects | Thrombosis - chemically induced | Kidney - pathology | Humans | Male | Monocytes - metabolism | Repressor Proteins - toxicity | High Mobility Group Proteins - toxicity | Repressor Proteins - pharmacology | Inflammation - blood | Kidney - metabolism | Coagulants - pharmacology | Coagulants - toxicity | Disseminated Intravascular Coagulation - pathology | Disseminated Intravascular Coagulation - metabolism | Thrombosis - blood | Repressor Proteins - metabolism | Cytokines - blood | Disease Models, Animal | Lung - pathology | Kidney - drug effects | High Mobility Group Proteins - metabolism | HMGB1 Protein | Cells, Cultured | Rats | Disseminated Intravascular Coagulation - chemically induced | Enzyme Activation - drug effects | Disseminated Intravascular Coagulation - blood | Rats, Sprague-Dawley | Monocytes - drug effects | Thrombosis - pathology | Thrombosis - metabolism | Animals | Blood Coagulation - drug effects | High Mobility Group Proteins - pharmacology | Lung - drug effects | Blood Coagulation Tests | Protein C - metabolism | Thromboplastin - metabolism | Fibrin - metabolism | Proteins | Thrombosis | Blood clot
Journal Article
PLOS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, p. e0164258
The HMGA1 architectural transcription factor is an oncogene overexpressed in the vast majority of human cancers. HMGA1 is a highly connected node in the... 
BREAST-CANCER | PATHWAY CHOICE | TRANSCRIPTION FACTOR HMGA2 | GENE | MULTIDISCIPLINARY SCIENCES | HISTONE H1 | LIGATION | DEPENDENT PROTEIN-KINASE | EXPRESSION | NUCLEOTIDE EXCISION-REPAIR | CANCER STEM-CELLS | Chromatin - metabolism | HMGA1a Protein - genetics | Phosphorylation | Humans | Recombinant Proteins - chemistry | Substrate Specificity | Ku Autoantigen - metabolism | Chromatography, High Pressure Liquid | Recombinant Proteins - biosynthesis | HMGA1a Protein - metabolism | Comet Assay | Recombinant Proteins - isolation & purification | HMGA2 Protein - metabolism | MCF-7 Cells | DNA Repair | HMGA2 Protein - genetics | Cell Line, Tumor | DNA Ligase ATP - metabolism | Histones - metabolism | Microscopy, Fluorescence | Chromatin - chemistry | Cellular proteins | Chromatin | Chromosomal proteins | Ligases | DNA damage | Genes | DNA | Gene expression | DNA repair | Transcription factors | Homology | Biochemistry | Kinases | Cancer therapies | Proteins | Architectural engineering | Cell cycle | Physiology | Life sciences | Damage | Repair | Deoxyribonucleic acid--DNA | Cell survival | DNA ligase (ATP) | Breast cancer | Pharmacology | Substrates | High mobility group proteins | Chemotherapy | DNA-dependent protein kinase | Stem cells | Non-homologous end joining | Genetic engineering | Histone H1 | Cancer | Deoxyribonucleic acid
Journal Article
The Journal of Pathology, ISSN 0022-3417, 11/2017, Volume 243, Issue 3, pp. 376 - 389
Cancer stem cells/cancer‐initiating cells (CICs) and their microenvironmental niche play a vital role in malignant tumour recurrence and metastasis.... 
high‐mobility group box 1 (HMGB1) | Toll‐like receptor 4 (TLR4) | breast cancer‐initiating cell (BCIC) | autophagy | cancer‐associated fibroblast (CAF) | high-mobility group box 1 (HMGB1) | breast cancer-initiating cell (BCIC) | Toll-like receptor 4 (TLR4) | cancer-associated fibroblast (CAF) | INTERFERES | ACTIVATION | METASTASIS | UNCONVENTIONAL PROTEIN SECRETION | TUMOR-CELLS | HMGB1 | PATHOLOGY | CHEMOTHERAPY | INHIBITION | ONCOLOGY | GROWTH | EXPRESSION | Neoplasm Recurrence, Local - metabolism | Cancer-Associated Fibroblasts - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Middle Aged | Tumor Microenvironment - physiology | Autophagy | Cell Transformation, Neoplastic - metabolism | Toll-Like Receptor 4 - metabolism | Breast Neoplasms - metabolism | Neoplasm Recurrence, Local - pathology | Cancer-Associated Fibroblasts - pathology | Neoplastic Stem Cells - metabolism | Breast Neoplasms - pathology | HMGB1 Protein - metabolism | Aged, 80 and over | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Female | Aged | Cell Transformation, Neoplastic - pathology | Analysis | Breast cancer | Cancer cells | Immunohistochemistry | Mobility | Phagosomes | TLR4 protein | Tissues | HMGB1 protein | Metastases | Proteins | Stem cells | Microtubule-associated protein 1 | Toll-like receptors | Fibroblasts | Tumorigenesis | Light levels | Phagocytosis | Cancer | Tumors | Immune system
Journal Article