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Nature (London), ISSN 1476-4687, 2018, Volume 561, Issue 7724, pp. 547 - 550
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e15457
Spinal muscular atrophy (SMA) is a major genetic cause of death in childhood characterized by marked muscle weakness. To investigate mechanisms underlying... 
ANTERIOR HORN | PROTEIN | MOTOR-NEURON DEGENERATION | BIOLOGY | AXONAL NEUROPATHY | MICE | AMYOTROPHIC-LATERAL-SCLEROSIS | WERDNIG-HOFFMANN DISEASE | SENSORY NEURONS | END-PLATE | MOTONEURONS | Immunohistochemistry | Spinal Cord - metabolism | Neuromuscular Junction - metabolism | Electrophysiology | Spinal Nerve Roots - metabolism | Male | Muscle, Skeletal - metabolism | Axons - physiology | Ganglia, Spinal - physiopathology | Synapses - genetics | Hindlimb - physiopathology | Muscular Atrophy, Spinal - genetics | Spinal Nerve Roots - physiopathology | Female | Muscular Atrophy, Spinal - physiopathology | Disease Models, Animal | Proprioception - physiology | Motor Neurons - physiology | Muscular Atrophy, Spinal - metabolism | Receptors, Cholinergic - metabolism | Synapses - physiology | Hindlimb - innervation | Mice, Transgenic | Neuromuscular Junction - physiopathology | Disease Progression | Animals | Muscle, Skeletal - physiopathology | Mice | Spinal Cord - physiopathology | Ganglia, Spinal - metabolism | Analysis | Models | Spinal muscular atrophy | Neurosciences | Spinal cord | Disease | Circuits | Pathogenesis | SMN protein | Neurobiology | Defects | Proteins | Atrophy | Reduction | Rodents | Sensory neurons | Children | Quantitative analysis | Motor neurons | Neurons | Proprioception | Muscles | Amyotrophic lateral sclerosis | Muscle contraction | Microglia | Microscopy | Neuromuscular junctions | Glutamic acid transporter | Transporter | Synapses | Animal models
Journal Article
Journal of Applied Physiology, ISSN 8750-7587, 10/2012, Volume 113, Issue 8, pp. 1311 - 1322
Arterial blood pressure and heart rate responses to static contraction of the hindlimb muscles are greater in rats whose femoral arteries were previously... 
Peripheral arterial disease | Nerve growth factor | Hindlimb ischemia | ASIC3 | Muscle pressor reflex | ACID-SENSING ION | SPORT SCIENCES | PHYSIOLOGY | MYOCARDIAL-INFARCTION | ROOT GANGLION NEURONS | EXERCISE PRESSOR REFLEX | peripheral arterial disease | GROUP-III | SENSORY NEURONS | SKELETAL-MUSCLE | nerve growth factor | hindlimb ischemia | GROUP-IV AFFERENTS | muscle pressor reflex | GROWTH-FACTOR | RESPIRATORY RESPONSES | Reflex - physiology | Pressoreceptors - physiopathology | Male | Muscle, Skeletal - innervation | Muscle, Skeletal - metabolism | Ganglia, Spinal - physiopathology | Sympathetic Nervous System - physiopathology | Sympathetic Nervous System - metabolism | Arteries - metabolism | Hypoxia - metabolism | Hindlimb - physiopathology | Sensory Receptor Cells - physiology | Blood Pressure - physiology | Pressoreceptors - metabolism | Sensory Receptor Cells - metabolism | Nerve Fibers, Myelinated - metabolism | Nerve Growth Factor - metabolism | Acid Sensing Ion Channels | Femoral Artery - physiopathology | Lactic Acid - metabolism | Tendons - metabolism | Rats | Hindlimb - innervation | Neurons, Afferent - physiology | Rats, Sprague-Dawley | Tendons - innervation | Tendons - physiopathology | Animals | Nerve Fibers, Unmyelinated - metabolism | Arteries - innervation | Arteries - physiopathology | Muscle Contraction - physiology | Muscle, Skeletal - physiopathology | Femoral Artery - metabolism | Hypoxia - physiopathology | Neurons, Afferent - metabolism | Hindlimb - metabolism | Ganglia, Spinal - metabolism | Highlighted Topic
Journal Article
The American Journal of Sports Medicine, ISSN 1552-3365, 2016, Volume 16, Issue 1, pp. 7 - 12
This study investigates the biomechanical failure prop erties of five architecturally different skeletal muscles and examines the role muscle structure plays... 
Rabbits | Hindlimb | Muscles - physiopathology | Rupture | Tendon Injuries - physiopathology | Stress, Mechanical | Sprains and Strains - physiopathology | Biomechanical Phenomena | Muscles - anatomy & histology | Tendons - physiopathology | Animals | Models, Biological | Muscles - injuries | Tendons - anatomy & histology
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e23083
Inflammation is known to be responsible for the sensitization of peripheral sensory neurons, leading to spontaneous pain and invalidating pain... 
TETRODOTOXIN-RESISTANT | RESISTANT SODIUM-CHANNEL | PERSISTENT NA+ CURRENT | RAT | NA(V)1.9 KNOCKOUT MICE | DRG NEURONS | MOUSE | BIOLOGY | PRIMARY SENSORY NEURONS | ROOT GANGLIA NEURONS | EXPRESSION | Immunohistochemistry | Sodium Channels - physiology | Gene Knockdown Techniques | Forelimb - physiopathology | Hindlimb - physiopathology | Time Factors | Sodium Channels - metabolism | Sensory Receptor Cells - metabolism | Forelimb - drug effects | Gene Expression | Arthritis, Experimental - physiopathology | Mice, Inbred C57BL | Rats | Forelimb - metabolism | NAV1.9 Voltage-Gated Sodium Channel | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Mice, Knockout | Animals | Pain - physiopathology | Hindlimb - drug effects | Edema - physiopathology | Mice | Sodium Channels - genetics | Carrageenan | Edema - chemically induced | Chronic Disease | Hindlimb - metabolism | Ganglia, Spinal - metabolism | Inflammation - physiopathology | Allergy | Care and treatment | Carrageenin | Pain | Analysis | Genetic engineering | Inflammation | Allergic reaction | Animal models | Antisense | Arthritis | mRNA | Kinases | Dorsal root ganglia | Analgesics | Rodents | Animal tissues | Sensory neurons | Sodium channels (voltage-gated) | Localization | Pain perception | Neurons | Hypersensitivity | Excitability | Ganglia | Inflammatory bowel disease | Ankle | Gene silencing | Heat | Sodium | Freund's adjuvant | Weight reduction | Immunoreactivity | Axonal transport | Life Sciences | Biomolecules | Biochemistry, Molecular Biology
Journal Article
Scientific reports, ISSN 2045-2322, 2017, Volume 7, Issue 1, pp. 2811 - 16
Journal Article
Journal Article
Spine (Philadelphia, Pa. 1976), ISSN 0362-2436, 2013, Volume 38, Issue 17, pp. 1459 - 1465
Journal Article
Circulation research, ISSN 1524-4571, 2017, Volume 120, Issue 9, pp. 1453 - 1465
RATIONALE:Angiogenesis occurs after ischemic injury to skeletal muscle, and enhancing this response has been a therapeutic goal. However, to appropriately... 
Erythrocyte | Microvascular dysfunction | smooth muscle | Angiogenesis | Microcirculation | CAPILLARY NETWORKS | CARDIAC & CARDIOVASCULAR SYSTEMS | angiogenesis | LIMB ISCHEMIA | OXYGEN-TRANSPORT | THERAPEUTIC ANGIOGENESIS | microcirculation | FLOW | erythrocyte | SKELETAL-MUSCLE | microvascular dysfunction | BLOOD-CELL DYNAMICS | IMAGE-ANALYSIS | IN-VIVO | MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Microvessels - physiopathology | Venules - physiopathology | Hindlimb | Image Interpretation, Computer-Assisted | Male | Venules - diagnostic imaging | Cell Hypoxia | Time Factors | Arterioles - physiopathology | Microvessels - diagnostic imaging | Hypoxia - diagnostic imaging | Disease Models, Animal | Ischemia - diagnostic imaging | Muscle, Skeletal - blood supply | Microscopy, Video - methods | Capillaries - physiopathology | Mice, Inbred C57BL | Cellular Microenvironment | Ischemia - blood | Ischemia - physiopathology | Microscopy, Confocal - methods | Regional Blood Flow | Oxygen - blood | Hypoxia - blood | Animals | Erythrocytes - metabolism | Hypoxia - physiopathology | Arterioles - diagnostic imaging | Capillaries - diagnostic imaging | Neovascularization, Physiologic | Blood cells | Erythrocytes | Smooth muscle | Skeletal muscle | Blood | Vascularization | Musculoskeletal system | Ischemia | Actin | Spatial heterogeneity | Hypoxia | Microvasculature | Circulatory system | Capillaries | Structure-function relationships
Journal Article
Nature neuroscience, ISSN 1546-1726, 2008, Volume 11, Issue 11, pp. 1294 - 1301
Cellular abnormalities in amyotrophic lateral sclerosis (ALS) are not limited to motor neurons. Astrocyte dysfunction also occurs in human ALS and transgenic... 
GLIAL-RESTRICTED PRECURSORS | TRANSGENIC RAT MODEL | GLUTAMATE TRANSPORTERS | ADULT RATS | MOUSE MODEL | EXTEND SURVIVAL | ALS MODEL | AMYOTROPHIC-LATERAL-SCLEROSIS | HNT NEURONS | NEUROSCIENCES | STEM-CELL GRAFTS | Superoxide Dismutase - genetics | Phrenic Nerve - physiopathology | Humans | Glial Fibrillary Acidic Protein - metabolism | Cell Movement - physiology | Hindlimb - physiopathology | Motor Neuron Disease - therapy | Spinal Cord - pathology | Motor Neuron Disease - physiopathology | Cell Differentiation - physiology | Cell Survival - physiology | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Neural Conduction - genetics | Animals, Genetically Modified | Excitatory Amino Acid Transporter 2 - deficiency | Rats | Cell Transplantation | Spinal Cord - surgery | Motor Neuron Disease - pathology | Action Potentials - physiology | Astrocytes - physiology | Animals | Green Fluorescent Proteins - biosynthesis | Microfilament Proteins | Muscle, Skeletal - physiopathology | Stem Cells - physiology | Spinal Cord - physiopathology | Physiological aspects | Motor neurons | Nervous system | Care and treatment | Degeneration | Astrocytes | astroglia | grafting | neurodegeneration | ALS | neural precursor cell | progenitor | amyotrophic lateral sclerosis | SOD1 | non-cell autonomous | stem cell | motor neuron | neuroprotection | transplantation | lineage-restricted precursor | glial precursor | replacement | astrocyte
Journal Article
Nature medicine, ISSN 1546-170X, 2016, Volume 22, Issue 2, pp. 138 - 145
Journal Article