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Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2014, Volume 72, pp. 138 - 145
Abstract While inhibition of class I/IIb histone deacetylases (HDACs) protects the mammalian heart from ischemia reperfusion (IR) injury, class selective... 
Cardiovascular | Histone deacetylase | FOXO | Superoxide dismutase | Isolated heart | Catalase | Ischemia reperfusion | HDAC INHIBITORS | CARDIAC & CARDIOVASCULAR SYSTEMS | ACETYLATION | INJURY | FOXO TRANSCRIPTION FACTORS | HYPOXIA | CELL BIOLOGY | SUPEROXIDE-DISMUTASE | Superoxide Dismutase - genetics | Myocardial Contraction - drug effects | Male | Myocardial Reperfusion Injury - enzymology | Forkhead Transcription Factors - metabolism | Histone Deacetylase 1 - antagonists & inhibitors | Indoles - pharmacology | Benzamides - pharmacology | Histone Deacetylase 1 - genetics | Hydroxamic Acids - pharmacology | Myocardial Reperfusion Injury - genetics | Organ Culture Techniques | Superoxide Dismutase - metabolism | Heart - physiopathology | Histone Deacetylase 2 - genetics | Histone Deacetylases - genetics | Catalase - genetics | Histone Deacetylase 2 - antagonists & inhibitors | Histone Deacetylase 6 | Gene Expression Regulation | Rats | Histone Deacetylases - metabolism | Forkhead Transcription Factors - genetics | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Catalase - metabolism | Animals | Heart - drug effects | Histone Deacetylase Inhibitors - pharmacology | Pyridines - pharmacology | Histone Deacetylase 2 - metabolism | Forkhead Box Protein O3 | Myocardial Reperfusion Injury - prevention & control | Histone Deacetylase 1 - metabolism | Antioxidants | Ischemia | Superoxide | Index Medicus | ischemia reperfusion | isolated heart | histone deacetylase | superoxide dismutase | catalase
Journal Article
Oncogene, ISSN 0950-9232, 04/2014, Volume 33, Issue 17, pp. 2157 - 2168
Many tumor suppressor genes (TSGs) are silenced through synergistic layers of epigenetic regulation including abnormal DNA hypermethylation of promoter CpG... 
DNMTs | tumor suppressor genes | NuRD | WNT signaling | colon cancer | silencing | METHYLTRANSFERASE | HYPERMETHYLATION | DNA METHYLATION | HISTONE DEACETYLASE INHIBITION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CANCER | DAMAGE | CELL BIOLOGY | FACTOR CHD4 | CHROMATIN-REMODELING COMPLEX | ONCOLOGY | DEMETHYLATION | GENETICS & HEREDITY | EXPRESSION | Histone Deacetylase 2 - genetics | Promoter Regions, Genetic | Gene Expression | Autoantigens - metabolism | Epigenesis, Genetic | HCT116 Cells | Humans | Gene Expression Regulation, Neoplastic | Azacitidine - analogs & derivatives | Mi-2 Nucleosome Remodeling and Deacetylase Complex - genetics | Mi-2 Nucleosome Remodeling and Deacetylase Complex - metabolism | DNA (Cytosine-5-)-Methyltransferases - antagonists & inhibitors | Wnt Proteins - metabolism | Autoantigens - genetics | Gene Knockdown Techniques | DNA (Cytosine-5-)-Methyltransferases - metabolism | Azacitidine - pharmacology | Protein Binding | Colorectal Neoplasms | Histone Deacetylase 2 - metabolism | Genes, Tumor Suppressor | Histone Deacetylase 1 - genetics | Apoptosis | Histone Deacetylase 1 - metabolism | Colon cancer | Gene mutations | Methyltransferases | Oncology, Experimental | Genetic research | Tumor suppressor genes | Genetic aspects | Research | Properties | Identification and classification | Cancer | Drug therapy | Genes | Colorectal cancer | Tumors | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, pp. e102684 - e102684
Selective histone deacetylase (HDAC) inhibitors have emerged as a potential anti-latency therapy for persistent human immunodeficiency virus type 1 (HIV-1)... 
RECRUITMENT | THERAPY | LONG TERMINAL REPEAT | MULTIDISCIPLINARY SCIENCES | IMMUNODEFICIENCY-VIRUS TYPE-1 | CD4(+) T-CELLS | HISTONE DEACETYLASE INHIBITORS | ESTABLISHMENT | IDENTIFICATION | NF-KAPPA-B | EXPRESSION | Anti-HIV Agents - pharmacology | RNA, Small Interfering - genetics | HIV-1 - pathogenicity | Virus Latency | Humans | HIV Infections - enzymology | CD4-Positive T-Lymphocytes - enzymology | HIV-1 - physiology | Gene Deletion | Histone Deacetylase 1 - antagonists & inhibitors | CD4-Positive T-Lymphocytes - virology | Histone Deacetylase 1 - genetics | Histone Deacetylase 2 - genetics | Promoter Regions, Genetic | Histone Deacetylases - genetics | Signal Transduction | HIV-1 - drug effects | Histone Deacetylase 2 - antagonists & inhibitors | HIV Infections - genetics | HIV Infections - virology | Gene Expression Regulation | Histone Deacetylases - deficiency | Host-Pathogen Interactions | Histone Deacetylase 2 - deficiency | Histone Deacetylase 1 - deficiency | HIV Infections - drug therapy | Histone Deacetylase Inhibitors - pharmacology | CD4-Positive T-Lymphocytes - drug effects | Cell Line, Transformed | RNA, Small Interfering - metabolism | RNA | T cells | HIV (Viruses) | Health aspects | HIV infection | Analysis | Histone deacetylase | Transcription factors | Transcription | Viruses | Infections | Lymphocytes T | Kinases | Gene expression | Ribonucleic acid--RNA | CD4 antigen | Latency | Virology | Immunology | Depletion | Inhibitors | Lymphocytes | Antiretroviral agents | Human immunodeficiency virus--HIV | Cell cycle | Inhibition | HDAC2 protein | Index Medicus | Ribonucleic acid | HIV | Human immunodeficiency virus
Journal Article
Blood, ISSN 0006-4971, 04/2013, Volume 121, Issue 17, pp. 3459 - 3468
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, pp. e16727 - e16727
Journal Article