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1. Advances in malignant hematology
by Saba, Hussain I and Mufti, Ghulam
2011, 1. Aufl., ISBN 1405196262, xxi, 416 p., [4] p. of col. plates
Leukemia | Lymphomas | Myelodysplastic syndromes
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2. Full Text IL-15 triggers an antiapoptotic pathway in human intraepithelial lymphocytes that is a potential new target in celiac disease-associated inflammation and lymphomagenesis
by Malamut, Georgia and El Machhour, Raja and Montcuquet, Nicolas and Martin-Lannerée, Séverine and Dusanter-Fourt, Isabelle and Verkarre, Virginie and Mention, Jean-Jacques and Rahmi, Gabriel and Kiyono, Hiroshi and Butz, Eric A and Brousse, Nicole and Cellier, Christophe and Cerf-Bensussan, Nadine and Meresse, Bertrand
The Journal of clinical investigation, ISSN 0021-9738, 06/2010, Volume 120, Issue 6, pp. 2131 - 2143
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Phosphorylation | Apoptosis - drug effects | Enterocytes - metabolism | Humans | Intestines - metabolism | Leukemia - metabolism | STAT3 Transcription Factor - pharmacology | Interleukin-15 - immunology | Intestines - immunology | Inflammation - complications | Signal Transduction - immunology | Inflammation - metabolism | Lymphocytes - immunology | Celiac Disease - immunology | Leukemia - complications | Celiac Disease - complications | Adult | Janus Kinase 3 - metabolism | Intestine, Small - immunology | STAT3 Transcription Factor - metabolism | Cytokines - immunology | Janus Kinase 3 - immunology | Lymphocytes - metabolism | Cytokines - metabolism | Inflammation - immunology | Protein Binding - immunology | Enterocytes - immunology | Interleukin-15 - pharmacology | Apoptosis - immunology | Signal Transduction - drug effects | Leukemia - immunology | Interleukin-15 - metabolism | Intestine, Small - metabolism | STAT3 Transcription Factor - immunology | Celiac Disease - metabolism | Cytokines - pharmacology | Care and treatment | Lymphocytes | Development and progression | Interleukin-15 | Inflammation | Non-Hodgkin's lymphomas | T cells | Properties | Health aspects | Index Medicus | Abridged Index Medicus
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3. Full Text Ocular adnexal IgG4‐related disease: comparative analysis with mucosa‐associated lymphoid tissue lymphoma and other chronic inflammatory conditions
by Go, Heounjeong and Kim, Ji Eun and Kim, Young A and Chung, Ho Kyung and Khwarg, Sang In and Kim, Chul‐Woo and Jeon, Yoon Kyung
Histopathology, ISSN 0309-0167, 01/2012, Volume 60, Issue 2, pp. 296 - 312
regulatory T cells | MALT lymphoma | IgG4‐related sclerosing dacryoadenitis | IgG4‐related sclerosing disease | lacrimal gland | FoxP3 | ocular adnexa | Mikulicz’s disease | IgG4-related sclerosing dacryoadenitis | Lacrimal gland | Mikulicz's disease | Regulatory T cells | IgG4-related sclerosing disease | Ocular adnexa | Pathology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Investigative techniques, diagnostic techniques (general aspects) | Medical sciences | Pathology. Cytology. Biochemistry. Spectrometry. Miscellaneous investigative techniques | T-Lymphocytes, Regulatory - metabolism | Adnexal Diseases - diagnosis | Humans | Middle Aged | Male | T-Lymphocytes, Regulatory - pathology | Orbital Diseases - diagnosis | Forkhead Transcription Factors - metabolism | Adult | Female | Lymphoma, B-Cell, Marginal Zone - pathology | Retrospective Studies | Eyelid Diseases - pathology | Orbital Diseases - metabolism | Diagnosis, Differential | Immunophenotyping | Eyelid Diseases - metabolism | Lacrimal Apparatus Diseases - metabolism | Lacrimal Apparatus Diseases - diagnosis | Lymphoma, B-Cell, Marginal Zone - diagnosis | Adnexal Diseases - pathology | Eyelid Diseases - diagnosis | Lymphoma, B-Cell, Marginal Zone - metabolism | Orbital Diseases - pathology | Adnexal Diseases - metabolism | Aged | Lacrimal Apparatus Diseases - pathology | Immunoglobulin G - metabolism | Medical colleges | Oncology, Experimental | Immunoglobulin G | Ophthalmology | Non-Hodgkin's lymphomas | Research | Comparative analysis | T cells | Histochemistry | Cancer | Cell proliferation | Translocation | Trisomy | Lymphocytes B | Mucosal-associated lymphoid tissue | Differential diagnosis | Inflammation | Lymphocytes T | Lymphoma | Index Medicus
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4. Full Text High CD206 levels in Hodgkin lymphoma‐educated macrophages are linked to matrix‐remodeling and lymphoma dissemination
by Arlt, Annekatrin and Bonin, Frederike and Rehberg, Thorsten and Perez‐Rubio, Paula and Engelmann, Julia C and Limm, Katharina and Reinke, Sarah and Dullin, Christian and Sun, Xueni and Specht, Rieke and Maulhardt, Markus and Linke, Franziska and Bunt, Gertrude and Klapper, Wolfram and Vockerodt, Martina and Wilting, Jörg and Pukrop, Tobias and Dettmer, Katja and Gronwald, Wolfram and Oefner, Peter J and Spang, Rainer and Kube, Dieter
Molecular oncology, ISSN 1574-7891, 03/2020, Volume 14, Issue 3, pp. 571 - 589
CD206 | macrophages | tumor microenvironment | lymphoma | Life Sciences & Biomedicine | Oncology | Science & Technology | Up-Regulation | Proteome - genetics | Membrane Glycoproteins - metabolism | Hodgkin Disease - pathology | Humans | Tumor Microenvironment | Culture Media, Conditioned - pharmacology | Monocytes - metabolism | Chorioallantoic Membrane - pathology | Antigens, CD - metabolism | CD40 Antigens - metabolism | Neoplasm Metastasis - immunology | Lymphoma, B-Cell - immunology | Membrane Glycoproteins - immunology | Receptors, Immunologic - immunology | Lymphoma, B-Cell - metabolism | Collagen Type I - metabolism | Hodgkin Disease - metabolism | Receptors, Cell Surface - metabolism | Chorioallantoic Membrane - metabolism | Interleukin-13 - metabolism | Chick Embryo | Xenograft Model Antitumor Assays | B7-H1 Antigen - metabolism | Macrophages - metabolism | Animals | Cell Differentiation - drug effects | RNA-Seq | Antigens, Differentiation, Myelomonocytic - metabolism | Fluorescent Antibody Technique | Lymphoma, B-Cell - pathology | Cell Line, Tumor | Hodgkin Disease - immunology | Macrophages - drug effects | Proteome - metabolism | Receptors, Immunologic - metabolism | Culture Media, Conditioned - metabolism | Flow cytometry | Cell culture | Antigens | Phenotypes | Lymphatic system | Immunoglobulins | Collagen (type I) | CD40 antigen | Surface antigens | Immunomodulation | Glycerol | Zebrafish | Mannose | Interleukin 13 | Cell differentiation | Macrophages | Gene expression | Cell surface | Lymphoma | CD163 antigen | Monocytes | Endocytosis | Solid tumors | Hodgkin's disease | Index Medicus
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5. Full Text Epstein–Barr virus infection induces an increase of T regulatory type 1 cells in Hodgkin lymphoma patients
by Morales, Olivier and Mrizak, Dhafer and François, Violaine and Mustapha, Rami and Miroux, Céline and Depil, Stéphane and Decouvelaere, Anne‐Valérie and Lionne‐Huyghe, Pauline and Auriault, Claude and Launoit, Yvan and Pancré, Véronique and Delhem, Nadira
British journal of haematology, ISSN 0007-1048, 09/2014, Volume 166, Issue 6, pp. 875 - 890
IL10 | Tr1 | chemokines | Epstein–Barr Virus | Hodgkin lymphoma | Epstein-Barr Virus | Chemokines | Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | Tumors | Biomarkers - metabolism | Hodgkin Disease - virology | Up-Regulation | Cytokines - metabolism | Th2 Cells - virology | Epstein-Barr Virus Infections - immunology | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Th2 Cells - immunology | T-Lymphocytes, Regulatory - immunology | Young Adult | Phenotype | Adolescent | Adult | Female | Hodgkin Disease - immunology | Aged | Chemokines - metabolism | Child | T-Lymphocytes, Regulatory - virology | Virus diseases | Proteins | Analysis | Lymphomas | Epstein-Barr virus | T cells | Gene expression | Health aspects | Index Medicus | T-Lymphocytes, Regulatory/virology | T-Lymphocytes, Regulatory/immunology | Th2 Cells/immunology | Chemokines/metabolism | Biomarkers/metabolism | Life Sciences | Th2 Cells/virology | Hodgkin Disease/virology | Epstein-Barr Virus Infections/immunology | Hodgkin Disease/immunology | Cytokines/metabolism
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6. Full Text CD19 CAR T cell product and disease attributes predict leukemia remission durability
by Finney, Olivia C and Finney, Olivia C and Brakke, Hannah and Brakke, Hannah M and Rawlings-Rhea, Stephanie and Rawlings-Rhea, Stephanie and Hicks, Roxana and Hicks, Roxana and Doolittle, Danielle and Doolittle, Danielle and Lopez, Marisa and Lopez, Marisa and Futrell, Ben and Futrell, Robert B and Orentas, Rimas J and Orentas, Rimas J and Li, Daniel and Li, Daniel and Gardner, Rebecca and Gardner, Rebecca A and Jensen, Michael C and Jensen, Michael C
The Journal of clinical investigation, ISSN 0021-9738, 05/2019, Volume 129, Issue 5, pp. 2123 - 2132
Oncology | Immunology | Cancer immunotherapy | Leukemias | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Tumor Necrosis Factor-alpha - metabolism | CD8-Positive T-Lymphocytes - cytology | Recurrence | Cell Proliferation | Humans | Hepatitis A Virus Cellular Receptor 2 - metabolism | Child, Preschool | Infant | Male | Antigens, CD - metabolism | Young Adult | Adult | Female | Child | Receptors, Chimeric Antigen - metabolism | Immunotherapy, Adoptive | Lymphocyte Activation | Kaplan-Meier Estimate | Hematopoietic Stem Cell Transplantation | Leukemia - therapy | Programmed Cell Death 1 Receptor - metabolism | Immunophenotyping | Gene Expression Regulation, Leukemic | Remission Induction | Disease-Free Survival | Phenotype | Leukemia - immunology | Adolescent | K562 Cells | Antigens, CD19 - metabolism | Medical research | Care and treatment | Usage | Leukemia | Clinical trials | Medicine, Experimental | Research | T cells | Antigens | Oncology, Experimental | Transplantation | B cells | Non-Hodgkin's lymphomas | Health aspects | Hematopoietic stem cells | Cancer | Cell proliferation | Pediatrics | CD8 antigen | Effector cells | Stem cell transplantation | Lymphocytes T | CD223 antigen | Cancer therapies | Adoptive transfer | Cell activation | Lymphocytes | Immunotherapy | Peripheral blood | Bone marrow | Remission | Failure | Phenotypes | Cell survival | Young adults | CD19 antigen | Minimal residual disease | Tumor necrosis factor-α | Patients | Lymphoma | Hemopoiesis | Chimeric antigen receptors | Survival analysis | Chemotherapy | Aplasia | Lymphocytes B | Influenza | Stem cells | Index Medicus | Abridged Index Medicus
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7. Full Text PD-1 Blockade with Nivolumab in Relapsed or Refractory Hodgkin's Lymphoma
by Ansell, Stephen M and Lesokhin, Alexander M and Borrello, Ivan and Halwani, Ahmad and Scott, Emma C and Gutierrez, Martin and Schuster, Stephen J and Millenson, Michael M and Cattry, Deepika and Freeman, Gordon J and Rodig, Scott J and Chapuy, Bjoern and Ligon, Azra H and Zhu, Lili and Grosso, Joseph F and Kim, Su Young and Timmerman, John M and Shipp, Margaret A and Armand, Philippe
The New England journal of medicine, ISSN 0028-4793, 01/2015, Volume 372, Issue 4, pp. 311 - 319
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Recurrence | Hodgkin Disease - metabolism | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | STAT Transcription Factors - metabolism | Programmed Cell Death 1 Ligand 2 Protein - metabolism | Programmed Cell Death 1 Receptor - metabolism | Male | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Janus Kinases - metabolism | Antineoplastic Agents - administration & dosage | Stem Cell Transplantation | Antibodies, Monoclonal - administration & dosage | Antineoplastic Agents - adverse effects | Hodgkin Disease - therapy | Immunotherapy | Hodgkin Disease - drug therapy | Immunoconjugates - therapeutic use | Adult | Female | Reed-Sternberg Cells - drug effects | Medical research | Autografts | Transplants & implants | Transcription | PD-1 protein | Toxicity | Body weight | Blocking antibodies | Stat3 protein | Stem cell transplantation | Lymphoma | Chromosome 9 | PD-L1 protein | Tomography | PD-L2 protein | Janus kinase | Ligands | Drug therapy | Hodgkin's disease | Cancer | Apoptosis | Tumors | Index Medicus | Abridged Index Medicus
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8. Full Text The pan-deacetylase inhibitor panobinostat induces cell death and synergizes with everolimus in Hodgkin lymphoma cell lines
by Lemoine, Manuela and Derenzini, Enrico and Buglio, Daniela and Medeiros, L. Jeffrey and Davis, R. Eric and Zhang, Jiexin and Ji, Yuan and Younes, Anas
Blood, ISSN 0006-4971, 04/2012, Volume 119, Issue 17, pp. 4017 - 4025
Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | AMP-Activated Protein Kinases - metabolism | TOR Serine-Threonine Kinases - metabolism | Apoptosis - drug effects | Hodgkin Disease - pathology | Humans | Glucose Transporter Type 1 - metabolism | Vascular Endothelial Growth Factor A - metabolism | Hypoxia-Inducible Factor 1 - metabolism | STAT5 Transcription Factor - metabolism | Flow Cytometry | Hodgkin Disease - drug therapy | Indoles | Phosphorylation - drug effects | Everolimus | Hydroxamic Acids - pharmacology | Immunosuppressive Agents - pharmacology | Protein-Serine-Threonine Kinases - metabolism | Panobinostat | Sirolimus - analogs & derivatives | Cytokines - metabolism | Hodgkin Disease - metabolism | STAT6 Transcription Factor - metabolism | Sirolimus - pharmacology | Blotting, Western | Drug Synergism | Cell Line, Tumor | Histone Deacetylase Inhibitors - pharmacology | Cell Proliferation - drug effects | Chemokines - metabolism | Cell Cycle - drug effects | Index Medicus | Abridged Index Medicus | Lymphoid Neoplasia
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9. Full Text Frequent mutation of histone-modifying genes in non-Hodgkin lymphoma
by Morin, Ryan D and Mendez-Lago, Maria and Mungall, Andrew J and Goya, Rodrigo and Mungall, Karen L and Corbett, Richard D and Johnson, Nathalie A and Severson, Tesa M and Chiu, Readman and Field, Matthew and Jackman, Shaun and Krzywinski, Martin and Scott, David W and Trinh, Diane L and Tamura-Wells, Jessica and Li, Sa and Firme, Marlo R and Rogic, Sanja and Griffith, Malachi and Chan, Susanna and Yakovenko, Oleksandr and Meyer, Irmtraud M and Zhao, Eric Y and Smailus, Duane and Moksa, Michelle and Chittaranjan, Suganthi and Rimsza, Lisa and Brooks-Wilson, Angela and Spinelli, John J and Ben-Neriah, Susana and Meissner, Barbara and Woolcock, Bruce and Boyle, Merrill and McDonald, Helen and Tam, Angela and Zhao, Yongjun and Delaney, Allen and Zeng, Thomas and Tse, Kane and Butterfield, Yaron and Birol, Inana and Holt, Rob and Schein, Jacqueline and Horsman, Douglas E and Moore, Richard and Jones, Steven J. M and Connors, Joseph M and Hirst, Martin and Gascoyne, Randy D and Marra, Marco A
Nature (London), ISSN 0028-0836, 08/2011, Volume 476, Issue 7360, pp. 298 - 303
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | Chromatin - metabolism | Humans | Histone Acetyltransferases - genetics | Lymphoma, Non-Hodgkin - enzymology | Neoplasm Proteins - metabolism | Loss of Heterozygosity - genetics | DNA-Binding Proteins - metabolism | Lymphoma, Follicular - genetics | Histone Acetyltransferases - metabolism | Neoplasm Proteins - genetics | MEF2 Transcription Factors | Histone-Lysine N-Methyltransferase - genetics | Myogenic Regulatory Factors - genetics | MADS Domain Proteins - genetics | Lymphoma, Large B-Cell, Diffuse - enzymology | Myogenic Regulatory Factors - metabolism | Lymphoma, Non-Hodgkin - genetics | DNA-Binding Proteins - genetics | Mutation - genetics | Genome, Human - genetics | MADS Domain Proteins - metabolism | Histone Methyltransferases | Histone-Lysine N-Methyltransferase - metabolism | Lymphoma, Follicular - enzymology | Histones - metabolism | Chromatin - genetics | Lymphoma, Large B-Cell, Diffuse - genetics | Gene mutations | Physiological aspects | Genetic aspects | DNA binding proteins | Research | Non-Hodgkin's lymphomas | B cells | Genetics | Genomes | Mutation | Molecular weight | Methods | Binding sites | Index Medicus
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