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Nature, ISSN 0028-0836, 04/2015, Volume 520, Issue 7546, pp. 192 - 197
The metabolism of endothelial cells during vessel sprouting remains poorly studied. Here we report that endothelial loss of CFT1A, a rate-limiting enzyme of... 
OXIDATION | DISTRIBUTIONS | GLYCOLYSIS | REDUCTIVE GLUTAMINE-METABOLISM | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | TUMOR | PROLIFERATION | GROWTH-FACTOR | CANCER | CULTURE | Carnitine O-Palmitoyltransferase - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Neovascularization, Pathologic - pathology | Nucleotides - chemistry | Carnitine O-Palmitoyltransferase - antagonists & inhibitors | Adenosine Triphosphate - metabolism | Carnitine O-Palmitoyltransferase - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Nucleotides - biosynthesis | Nucleotides - pharmacology | Oxidation-Reduction - drug effects | DNA - biosynthesis | Carnitine O-Palmitoyltransferase - deficiency | Fatty Acids - metabolism | Disease Models, Animal | Carbon - metabolism | Fatty Acids - chemistry | Human Umbilical Vein Endothelial Cells - drug effects | Endothelial Cells - metabolism | Gene Silencing | Acetic Acid - pharmacology | Blood Vessels - cytology | Citric Acid Cycle | Animals | Retinopathy of Prematurity - drug therapy | Endothelial Cells - cytology | Neovascularization, Pathologic - drug therapy | Blood Vessels - drug effects | Cell Line, Tumor | Glucose - metabolism | Human Umbilical Vein Endothelial Cells - pathology | Cell Proliferation - drug effects | Mice | Neovascularization, Pathologic - metabolism | Retinopathy of Prematurity - metabolism | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Retinopathy of Prematurity - pathology | Physiological aspects | Deoxyribonucleotides | Fatty acids | Carbon | Endothelium | Proteins | Angiogenesis | Protein synthesis | Glucose | Kinases | Deoxyribonucleic acid--DNA | Cells | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 2012, Volume 14, Issue 3, pp. 249 - 256
The shear-responsive transcription factor Kruppel-like factor 2 (KLF2) is a critical regulator of endothelial gene expression patterns induced by... 
MECHANISM | ANGIOGENESIS | IN-VIVO | DISEASE | MICRORNAS | MICROPARTICLES | TRANSCRIPTION FACTOR | EXPRESSION | KLF2 | FLOW | CELL BIOLOGY | Exosomes - metabolism | Apolipoproteins E - deficiency | Human Umbilical Vein Endothelial Cells - metabolism | Lovastatin - pharmacology | Atherosclerosis - genetics | Coculture Techniques | Humans | Endothelial Cells - transplantation | Stress, Mechanical | MicroRNAs - metabolism | Lovastatin - analogs & derivatives | Aorta - metabolism | Transfection | Kruppel-Like Transcription Factors - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Exosomes - ultrastructure | Atherosclerosis - pathology | Exosomes - transplantation | Endothelial Cells - metabolism | Cells, Cultured | Signal Transduction - genetics | Microscopy, Electron | Reverse Transcriptase Polymerase Chain Reaction | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Gene Expression Regulation - drug effects | Microscopy, Confocal | Animals | Endothelial Cells - cytology | Apolipoproteins E - genetics | Mice | MicroRNAs - genetics | Kruppel-Like Transcription Factors - genetics | Transcription factors | MicroRNA | Atherosclerosis | Physiological aspects | Genetic aspects | Research | Gene expression | Risk factors | Index Medicus
Journal Article
Journal Article
Journal Article
Aging Cell, ISSN 1474-9718, 06/2016, Volume 15, Issue 3, pp. 428 - 435
Journal Article
Biomaterials, ISSN 0142-9612, 2011, Volume 32, Issue 35, pp. 9218 - 9230
Abstract Recent study showed that mesenchymal stem cells (MSC) could inhibit apoptosis of endothelial cells in hypoxic condition, increase their survival, and... 
Advanced Basic Science | Dentistry | Human umbilical vein endothelial cells | Cardiac patch | Human mesenchymal stem cells | Laser-induced forward transfer cell printing | Cardiac regeneration | MATERIALS SCIENCE, BIOMATERIALS | GENE DELIVERY | MYOCARDIAL-INFARCTION | ANGIOGENESIS | ENGINEERING, BIOMEDICAL | MUSCLE | MATURATION | NANOPARTICLES | THERAPY | ENGINEERED HEART-TISSUE | SCAFFOLDS | EXPRESSION | Neovascularization, Physiologic - drug effects | Capillaries - pathology | Human Umbilical Vein Endothelial Cells - metabolism | Heart - physiology | Humans | Capillaries - drug effects | Regenerative Medicine - methods | Tissue Scaffolds - chemistry | Mesenchymal Stromal Cells - cytology | Lasers | Human Umbilical Vein Endothelial Cells - cytology | Myocardial Infarction - pathology | Myocardial Infarction - physiopathology | Mesenchymal Stromal Cells - drug effects | Human Umbilical Vein Endothelial Cells - drug effects | Implants, Experimental | Cell Separation | Cells, Cultured | Mesenchymal Stromal Cells - metabolism | Rats | Immunophenotyping | Regeneration - physiology | Cell Movement - drug effects | Regeneration - drug effects | Animals | Fibrosis | Heart - drug effects | Heart Function Tests - drug effects | Urethane - pharmacology | Urea | Biological products | Tissue engineering | Stem cells | Lectins | Transplantation | Endothelium | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, pp. e111083 - e111083
Background: Heat stress can be acutely cytotoxic, and heat stress-induced apoptosis is a prominent pathological feature of heat-related illnesses, although the... 
HYPERTHERMIA | OXIDATIVE STRESS | SMOOTH-MUSCLE | HELA-CELLS | RYANODINE RECEPTOR | CA2+ RELEASE | MULTIDISCIPLINARY SCIENCES | CALCIUM | ENDOPLASMIC-RETICULUM STRESS | SARCOPLASMIC-RETICULUM | HEPATOCELLULAR INJURY | Reactive Oxygen Species - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Calcium - metabolism | Calcium Signaling - physiology | Human Umbilical Vein Endothelial Cells - cytology | Humans | Mitochondria - metabolism | Apoptosis - physiology | Heat-Shock Response - physiology | Apoptosis Regulatory Proteins - metabolism | Endoplasmic Reticulum Stress - physiology | Initiators | Pathogenesis | Ryanodine receptors | Cytotoxicity | Smooth muscle | Confocal | Caspase-3 | Calcium influx | Fragmentation | Proteins | Signal transduction | Elevation | Mitochondria | Illnesses | Heat stress | Trauma care | Deoxyribonucleic acid--DNA | Inositol 1,4,5-trisphosphate receptors | Gene expression | Calcium (mitochondrial) | Trauma | Heat | Caspase-9 | Heat tolerance | Endoplasmic reticulum | Fluo-3 | Viability | Hyperthermia | Oxidative stress | Flow cytometry | Reactive oxygen species | Intensive care | Hydrogen peroxide | Calcium | Laboratories | Fluorescence | Confocal microscopy | Activation | Ca2+-transporting ATPase | DNA fragmentation | Initiation factor eIF-2 | Poly(ADP-ribose) polymerase | Caspase | Heatstroke | Fever | Stress | Endothelial cells | Cytometry | Signaling | Microscopy | Cell death | Apaf-1 protein | Cytoplasm | Scanning microscopy | Apoptosis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article