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Nature, ISSN 0028-0836, 04/2015, Volume 520, Issue 7546, pp. 192 - 197
The metabolism of endothelial cells during vessel sprouting remains poorly studied. Here we report that endothelial loss of CFT1A, a rate-limiting enzyme of... 
OXIDATION | DISTRIBUTIONS | GLYCOLYSIS | REDUCTIVE GLUTAMINE-METABOLISM | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | TUMOR | PROLIFERATION | GROWTH-FACTOR | CANCER | CULTURE | Carnitine O-Palmitoyltransferase - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Neovascularization, Pathologic - pathology | Nucleotides - chemistry | Carnitine O-Palmitoyltransferase - antagonists & inhibitors | Adenosine Triphosphate - metabolism | Carnitine O-Palmitoyltransferase - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Nucleotides - biosynthesis | Nucleotides - pharmacology | Oxidation-Reduction - drug effects | DNA - biosynthesis | Carnitine O-Palmitoyltransferase - deficiency | Fatty Acids - metabolism | Disease Models, Animal | Carbon - metabolism | Fatty Acids - chemistry | Human Umbilical Vein Endothelial Cells - drug effects | Endothelial Cells - metabolism | Gene Silencing | Acetic Acid - pharmacology | Blood Vessels - cytology | Citric Acid Cycle | Animals | Retinopathy of Prematurity - drug therapy | Endothelial Cells - cytology | Neovascularization, Pathologic - drug therapy | Blood Vessels - drug effects | Cell Line, Tumor | Glucose - metabolism | Human Umbilical Vein Endothelial Cells - pathology | Cell Proliferation - drug effects | Mice | Neovascularization, Pathologic - metabolism | Retinopathy of Prematurity - metabolism | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Retinopathy of Prematurity - pathology | Physiological aspects | Deoxyribonucleotides | Fatty acids | Carbon | Endothelium | Proteins | Angiogenesis | Protein synthesis | Glucose | Kinases | Deoxyribonucleic acid--DNA | Cells | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 2012, Volume 14, Issue 3, pp. 249 - 256
The shear-responsive transcription factor Kruppel-like factor 2 (KLF2) is a critical regulator of endothelial gene expression patterns induced by... 
MECHANISM | ANGIOGENESIS | IN-VIVO | DISEASE | MICRORNAS | MICROPARTICLES | TRANSCRIPTION FACTOR | EXPRESSION | KLF2 | FLOW | CELL BIOLOGY | Exosomes - metabolism | Apolipoproteins E - deficiency | Human Umbilical Vein Endothelial Cells - metabolism | Lovastatin - pharmacology | Atherosclerosis - genetics | Coculture Techniques | Humans | Endothelial Cells - transplantation | Stress, Mechanical | MicroRNAs - metabolism | Lovastatin - analogs & derivatives | Aorta - metabolism | Transfection | Kruppel-Like Transcription Factors - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Exosomes - ultrastructure | Atherosclerosis - pathology | Exosomes - transplantation | Endothelial Cells - metabolism | Cells, Cultured | Signal Transduction - genetics | Microscopy, Electron | Reverse Transcriptase Polymerase Chain Reaction | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Gene Expression Regulation - drug effects | Microscopy, Confocal | Animals | Endothelial Cells - cytology | Apolipoproteins E - genetics | Mice | MicroRNAs - genetics | Kruppel-Like Transcription Factors - genetics | Transcription factors | MicroRNA | Atherosclerosis | Physiological aspects | Genetic aspects | Research | Gene expression | Risk factors | Index Medicus
Journal Article
Journal Article
Nature Cell Biology, ISSN 1465-7392, 10/2011, Volume 13, Issue 10, pp. 1202 - 1213
Angiogenesis, the growth of new blood vessels, involves specification of endothelial cells to tip cells and stalk cells, which is controlled by Notch... 
LYMPHATIC ENDOTHELIUM | KINASE-ACTIVITY | VASCULAR NETWORK FORMATION | ENDOTHELIAL-GROWTH-FACTOR | FACTOR-C | MOUSE EMBRYOS | EXTRACELLULAR-MATRIX | MONOCLONAL-ANTIBODY | TUMOR ANGIOGENESIS | FACTOR RECEPTOR-3 | CELL BIOLOGY | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Receptors, Notch - antagonists & inhibitors | Transfection | RNA Interference | Time Factors | Forkhead Transcription Factors - metabolism | Vascular Endothelial Growth Factor D - genetics | Phosphatidylinositol 3-Kinase - metabolism | Vascular Endothelial Growth Factor Receptor-3 - deficiency | Transduction, Genetic | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Rhombencephalon - blood supply | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Forkhead Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Mice, Knockout | Antibodies - pharmacology | Macrophages - metabolism | Vascular Endothelial Growth Factor D - metabolism | Animals | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Vascular Endothelial Growth Factor C - metabolism | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Vascular Endothelial Growth Factor B - genetics | Mice | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-3 - genetics | Endothelial Cells - drug effects | Physiological aspects | Blood vessels | Cellular signal transduction | Growth factor receptors | Research | Vascular endothelial growth factor | Index Medicus | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2015, Volume 125, Issue 12, pp. 4514 - 4528
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2015, Volume 35, Issue 7, pp. 1623 - 1634
Journal Article
Circulation Research, ISSN 0009-7330, 05/2015, Volume 116, Issue 11, pp. 1753 - 1764
RATIONALE:The miR-143/145 cluster is highly expressed in smooth muscle cells (SMCs), where it regulates phenotypic switch and vascular homeostasis. Whether it... 
microRNAs | endothelial cells | smooth muscle myocytes | angiogenesis effect | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | PDGF-B | BREAST-CANCER | MORPHOGENESIS | GROWTH-FACTOR-BETA | TUNNELING NANOTUBES | IN-VIVO | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | PERICYTES | Human Umbilical Vein Endothelial Cells - metabolism | Blood Vessels - metabolism | Coculture Techniques | Humans | MicroRNAs - metabolism | Human Umbilical Vein Endothelial Cells - physiology | Dioxoles - pharmacology | Transforming Growth Factor beta - antagonists & inhibitors | Benzamides - pharmacology | Biological Transport - drug effects | Hexokinase - genetics | Myocytes, Smooth Muscle - metabolism | Human Umbilical Vein Endothelial Cells - ultrastructure | Microscopy, Electron, Scanning | Cell Surface Extensions - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Cell Communication - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Microscopy, Confocal | Transforming Growth Factor beta - pharmacology | Animals | Cell Surface Extensions - drug effects | Cell Communication - drug effects | Integrin beta Chains - metabolism | MicroRNAs - genetics | Myocytes, Smooth Muscle - ultrastructure | Integrin beta Chains - genetics | Blood Vessels - physiology | Hexokinase - metabolism | Transforming Growth Factor beta - metabolism | Index Medicus
Journal Article
Journal Article