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PROTEOMICS, ISSN 1615-9853, 04/2013, Volume 13, Issue 7, pp. 1096 - 1107
Journal Article
Radiation Research, ISSN 0033-7587, 2015, Volume 183, Issue 1, pp. 82 - 93
Recently several laboratories have reported that radiation induces senescence in endothelial cells. Senescent cells can secrete multiple growth-regulatory... 
OXIDATIVE STRESS | ANGIOGENESIS | DNA-DAMAGE | ATHEROSCLEROSIS | DOUBLE-STRAND BREAKS | REPAIR | BIOPHYSICS | X-RAYS | BIOLOGY | SMOOTH-MUSCLE-CELLS | IONIZING-RADIATION | NORMAL-TISSUES | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Apoptosis - drug effects | Apoptosis - radiation effects | Heterocyclic Compounds, 3-Ring - pharmacology | Humans | Cellular Senescence - drug effects | NF-kappa B - metabolism | Death Domain Receptor Signaling Adaptor Proteins - genetics | Neovascularization, Physiologic - radiation effects | DNA Breaks, Double-Stranded - radiation effects | Radiation Tolerance - radiation effects | DNA Breaks, Double-Stranded - drug effects | Human Umbilical Vein Endothelial Cells - cytology | Cellular Senescence - radiation effects | Signal Transduction - radiation effects | Human Umbilical Vein Endothelial Cells - drug effects | Interleukin-6 - genetics | Gene Expression Regulation - drug effects | Radiation Tolerance - drug effects | Phenotype | Signal Transduction - drug effects | Gene Expression Regulation - radiation effects | Cell Proliferation - drug effects | Pyridines - pharmacology | Human Umbilical Vein Endothelial Cells - radiation effects | Dose-Response Relationship, Radiation | Cell Proliferation - radiation effects
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 81, pp. 62 - 70
Abstract Age-associated cardiovascular diseases are at least partially ascribable to vascular cell senescence. Replicative senescence (RS) and stress-induced... 
Cardiovascular | Vascular aging | miRNA | Senescence | Mitochondria | lncRNA | Endothelial cells | LncRNA | MiRNA | CARDIAC & CARDIOVASCULAR SYSTEMS | LONG NONCODING RNAS | TELOMERASE | P16(INK4A) | CELLULAR SENESCENCE | CANCER | FAMILY | CELL BIOLOGY | DNA | GENE-EXPRESSION | PROTEINS | BINDING | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Molecular Sequence Data | Male | MicroRNAs - metabolism | Aorta - metabolism | RNA - genetics | G2 Phase Cell Cycle Checkpoints - radiation effects | G2 Phase Cell Cycle Checkpoints - drug effects | Ultraviolet Rays | Aging - genetics | Mitochondria - genetics | Cellular Senescence | Base Sequence | Human Umbilical Vein Endothelial Cells - cytology | Myocytes, Smooth Muscle - radiation effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | RNA - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Hydrogen Peroxide - pharmacology | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Mitochondria - metabolism | RNA, Long Noncoding - genetics | Animals | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Mice | MicroRNAs - genetics | Aorta - cytology | Human Umbilical Vein Endothelial Cells - radiation effects | RNA, Long Noncoding - metabolism | Aging - metabolism | Analysis | Mitochondrial DNA | Endothelium
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2015, Volume 5, Issue 1, pp. 18398 - 18398
The conventional photosensitizers used in photodynamic therapy (PDT), such as haematoporphyrin (HP), have not yet reached satisfactory therapeutic effects on... 
CARCINOMA-CELLS | PDT | IN-VITRO | PORT-WINE STAINS | PULSED DYE-LASER | PHARMACOKINETICS | HEMATOPORPHYRIN | MULTIDISCIPLINARY SCIENCES | DEATH | GROWTH-FACTOR | AUTOPHAGY | Reactive Oxygen Species - metabolism | Gene Expression - drug effects | Apoptosis - drug effects | Apoptosis - radiation effects | Membrane Potential, Mitochondrial - radiation effects | Photochemotherapy - methods | Humans | Caspase 3 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Membrane Potential, Mitochondrial - drug effects | Light | Caspase 3 - genetics | Signal Transduction - radiation effects | Proto-Oncogene Proteins c-akt - metabolism | Cytokines - genetics | Perylene - analogs & derivatives | Cell Line | Human Umbilical Vein Endothelial Cells - drug effects | Cytokines - metabolism | Radiation-Sensitizing Agents - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Gene Expression - radiation effects | Blotting, Western | Perylene - pharmacology | Signal Transduction - drug effects | Human Umbilical Vein Endothelial Cells - radiation effects | Wine | Photodynamic therapy | Toxicity | Caspase | AKT protein | Inflammation | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Mitochondria | Cell activation | Hypericin | Stains | Vascular endothelial growth factor | Umbilical vein | Apoptosis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, p. e30674
Many studies suggest that far-infrared (FIR) therapy can reduce the frequency of some vascular-related diseases. The non-thermal effect of FIR was recently... 
OXIDATIVE STRESS | NITRIC-OXIDE SYNTHASE | ZINC-FINGER | ARTERIOVENOUS-FISTULA | BIOLOGY | BALLOON ANGIOPLASTY | VENOUS STENOSIS | HEMODIALYSIS | SMOOTH-MUSCLE-CELLS | GROWTH-FACTOR | ACUTE PROMYELOCYTIC LEUKEMIA | Infrared Rays - therapeutic use | Human Umbilical Vein Endothelial Cells - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Up-Regulation - radiation effects | Cells, Cultured | Phosphatidylinositol 3-Kinases - metabolism | Down-Regulation - drug effects | Promyelocytic Leukemia Zinc Finger Protein | Human Umbilical Vein Endothelial Cells - physiology | Phototherapy - methods | Cell Nucleus - metabolism | Signal Transduction - drug effects | Cell Nucleus - radiation effects | Kruppel-Like Transcription Factors - metabolism | Protein Transport - radiation effects | Cell Proliferation - drug effects | Signal Transduction - radiation effects | Human Umbilical Vein Endothelial Cells - radiation effects | Oncogene Protein v-akt - metabolism | Active Transport, Cell Nucleus - radiation effects | Vascular Endothelial Growth Factor A - pharmacology | Cell Proliferation - radiation effects | RNA | Analysis | Nitric oxide | Leukemia | Vascular endothelial growth factor | Phosphotransferases | Endothelium | Cell proliferation | Therapy | Oxidative stress | Cell culture | Reactive oxygen species | Transcription factors | Phosphorylation | Transcription | Pathogenesis | Hemodialysis | AKT protein | Cytotoxicity | Smooth muscle | Cardiovascular disease | Kinases | Proteins | Promyeloid leukemia | Signal transduction | Biological effects | Cell growth | Transfection | Infrared radiation | Physiology | Oxidation | Inhibition | Zinc finger proteins | Translocation | Internal medicine | Pulmonary arteries | Exposure | siRNA | Zinc | Nuclear transport | Endothelial cells | 1-Phosphatidylinositol 3-kinase | Medicine | Studies | Hospitals | Ribonucleic acids | Wortmannin | Nitrotyrosine | Umbilical vein
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 7, p. e22182
Despite wide margins and high dose irradiation, unresectable malignant glioma (MG) is less responsive to radiation and is uniformly fatal. We previously found... 
INVASION | CELL MOTILITY | ANGIOGENESIS | PATHWAY | ANALOGS | CANCER METASTASIS | BIOLOGY | AKT | MECHANISMS | IDENTIFICATION | LYSOPHOSPHATIDIC ACID RECEPTORS | Receptors, Lysophosphatidic Acid - metabolism | Cell Death - radiation effects | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Molecular Targeted Therapy | Glioma - metabolism | Neovascularization, Pathologic - pathology | Glioma - pathology | Human Umbilical Vein Endothelial Cells - cytology | Lysophospholipids - pharmacology | Neovascularization, Pathologic - radiotherapy | Cell Death - drug effects | Receptors, Lysophosphatidic Acid - antagonists & inhibitors | Cell Survival - drug effects | Human Umbilical Vein Endothelial Cells - drug effects | Phosphoric Diester Hydrolases - metabolism | Endothelial Cells - metabolism | Radiation-Sensitizing Agents - pharmacology | Endothelial Cells - radiation effects | Cell Survival - radiation effects | Cell Movement - radiation effects | Glioma - blood supply | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Endothelial Cells - cytology | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Mice | Neovascularization, Pathologic - metabolism | Human Umbilical Vein Endothelial Cells - radiation effects | Endothelial Cells - drug effects | Animal experimentation | Ionizing radiation | Blood-vessels | Gliomas | Phosphonates | Radiotherapy | Tumors | Cancer | Endothelium | Cell culture | Brain | Motility | Leukocyte migration | Radiation | AKT protein | Metastasis | Cancer therapies | Stomach cancer | Proteins | Angiogenesis | Signal transduction | Receptors | Radiosensitization | Vascular endothelial growth factor | Quantitative analysis | Phospholipase A2 | Melanoma | Gene expression | Survival | Radiation dosage | Irradiation | Scientific imaging | Viability | Mass spectrometry | Cell migration | Animal models | Phosphorylation | Brain tumors | Irradiated | Oncology | Biology | Kinases | Cell adhesion & migration | Glioma cells | Cell survival | siRNA | Radiation therapy | Radioprotection | Chromatography | Endothelial cells | Medicine | Phospholipase | Radiosensitizers | Acids | Lysophosphatidic acid | Cell lines | Microvasculature | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e70024
The etiology of radiation-induced cardiovascular disease (CVD) after chronic exposure to low doses of ionizing radiation is only marginally understood. We have... 
PROGENITOR CELLS | MORTALITY EXPERIENCE | ACCELERATES SENESCENCE | LABEL-FREE | MULTIDISCIPLINARY SCIENCES | PLECKSTRIN HOMOLOGY DOMAIN | TELOMERASE INACTIVATION | IONIZING-RADIATION | PROTEOMIC ANALYSIS | PROTEIN-KINASE B | REPLICATIVE SENESCENCE | TOR Serine-Threonine Kinases - metabolism | Humans | Phosphatidylinositol 3-Kinases - metabolism | Time Factors | Human Umbilical Vein Endothelial Cells - cytology | Cellular Senescence - radiation effects | Proteomics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Signal Transduction - radiation effects | Human Umbilical Vein Endothelial Cells - radiation effects | Proto-Oncogene Proteins c-akt - metabolism | Dose-Response Relationship, Radiation | Cell Proliferation - radiation effects | Ionizing radiation | Aging | Research | Cardiovascular diseases | Health aspects | Risk factors | Endothelium | TOR protein | Heart | GTP-binding protein | Senescence | Galactosidase | Cardiovascular disease | AKT protein | Biology | Inactivation | Chronic exposure | Proteins | Signal transduction | Etiology | Atherosclerosis | Radiation effects | Medical imaging | Deactivation | Mortality | Environmental health | Exposure | Endothelial cells | Radiation dosage | 1-Phosphatidylinositol 3-kinase | Gamma rays | Signaling | Cyclin-dependent kinase inhibitor p21 | Protein arrays | Coronary vessels | Cytoskeleton | Umbilical vein | Biological Sciences | Naturvetenskap | Biokemi och molekylärbiologi | Biologiska vetenskaper | Biochemistry and Molecular Biology | Natural Sciences | Cellbiologi | Cell Biology
Journal Article