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1. Full Text Kaposi's Sarcoma-Associated Herpesvirus Utilizes an Actin Polymerization-Dependent Macropinocytic Pathway To Enter Human Dermal Microvascular Endothelial and Human Umbilical Vein Endothelial Cells
by Hari Raghu and Neelam Sharma-Walia and Mohanan Valiya Veettil and Sathish Sadagopan and Bala Chandran
Journal of Virology, ISSN 0022-538X, 05/2009, Volume 83, Issue 10, pp. 4895 - 4911
Life Sciences & Biomedicine | Science & Technology | Virology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Miscellaneous | Microbiology | Humans | Actins - metabolism | Clathrin - drug effects | Amiloride - analogs & derivatives | Filipin - pharmacology | Cytochalasin D - pharmacology | Virus Internalization - drug effects | Fibroblasts - ultrastructure | Endothelial Cells - ultrastructure | Immediate-Early Proteins - metabolism | Pinocytosis - drug effects | Herpesvirus 8, Human - metabolism | Acetophenones - pharmacology | Benzopyrans - pharmacology | Endothelial Cells - virology | Umbilical Veins - cytology | Herpesvirus 8, Human - physiology | Fibroblasts - virology | rab GTP-Binding Proteins - metabolism | Cells, Cultured | DNA, Viral - metabolism | Chlorpromazine - pharmacology | Herpesvirus 8, Human - genetics | Clathrin - metabolism | Gene Expression Regulation, Viral | Amiloride - pharmacology | Trans-Activators - metabolism | Umbilical Veins - virology | Hydrogen-Ion Concentration | Index Medicus | Virus-Cell Interactions
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2. Full Text AXL-Mediated Productive Infection of Human Endothelial Cells by Zika Virus
by Liu, Shufeng and DeLalio, Leon J and Isakson, Brant E and Wang, Tony T
Circulation research, ISSN 0009-7330, 11/2016, Volume 119, Issue 11, pp. 1183 - 1189
endothelial cells | infection | virus internalization | Zika virus | axl receptor tyrosine kinase | blood | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Cricetinae | Human Umbilical Vein Endothelial Cells - drug effects | Humans | Cercopithecus aethiops | Zika Virus - drug effects | Human Umbilical Vein Endothelial Cells - virology | Receptor Protein-Tyrosine Kinases - metabolism | Dose-Response Relationship, Drug | Animals | Human Umbilical Vein Endothelial Cells - enzymology | Zika Virus - enzymology | Anilides - pharmacology | HEK293 Cells | Zika Virus Infection - enzymology | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Pyridines - pharmacology | Endothelial Cells - virology | Endothelial Cells - enzymology | Vero Cells | Cell Line, Transformed | Endothelial Cells - drug effects | Index Medicus | AXL receptor tyrosine kinase | Vascular Disease | virus entry | productive infection | Vascular Type | Basic Science Research | Endothelium
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3. Full Text HEXIM1 and NEAT1 Long Non-coding RNA Form a Multi-subunit Complex that Regulates DNA-Mediated Innate Immune Response
by Morchikh, Mehdi and Cribier, Alexandra and Raffel, Raoul and Amraoui, Sonia and Cau, Julien and Severac, Dany and Dubois, Emeric and Schwartz, Olivier and Bennasser, Yamina and Benkirane, Monsef
Molecular cell, ISSN 1097-2765, 08/2017, Volume 67, Issue 3, pp. 387 - 399.e5
HEXIM1 | DNA-PK | innate immune response | herpesvirus | paraspeckles | cGAS | NEAT1 | interferon stimulatory DNA | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Octamer Transcription Factors - immunology | Intracellular Signaling Peptides and Proteins - metabolism | Herpesvirus 8, Human - immunology | RNA, Long Noncoding - immunology | PTB-Associated Splicing Factor - genetics | RNA Interference | Calcium-Binding Proteins - immunology | Octamer Transcription Factors - genetics | Nucleotidyltransferases - metabolism | Intracellular Signaling Peptides and Proteins - genetics | DNA-Binding Proteins | Signal Transduction | Membrane Proteins - genetics | RNA-Binding Proteins - immunology | PTB-Associated Splicing Factor - metabolism | Nuclear Matrix-Associated Proteins - metabolism | PTB-Associated Splicing Factor - immunology | DNA - metabolism | Host-Pathogen Interactions | Nuclear Matrix-Associated Proteins - genetics | Transcription Factors | HeLa Cells | RNA-Binding Proteins - metabolism | Calcium-Binding Proteins - genetics | RNA-Binding Proteins - genetics | Multiprotein Complexes | Human Umbilical Vein Endothelial Cells - immunology | Intracellular Signaling Peptides and Proteins - immunology | Human Umbilical Vein Endothelial Cells - virology | Interferon Regulatory Factor-3 - genetics | Ku Autoantigen - genetics | Transfection | HEK293 Cells | Membrane Proteins - metabolism | Nuclear Proteins - genetics | Octamer Transcription Factors - metabolism | Interferon Regulatory Factor-3 - immunology | Calcium-Binding Proteins - metabolism | DNA - immunology | Nuclear Matrix-Associated Proteins - immunology | Ku Autoantigen - immunology | Membrane Proteins - immunology | Nuclear Proteins - metabolism | Ku Autoantigen - metabolism | RNA, Long Noncoding - genetics | Immunity, Innate | Nuclear Proteins - immunology | DNA - genetics | Interferon Regulatory Factor-3 - metabolism | Nucleotidyltransferases - genetics | Protein Binding | Nucleotidyltransferases - immunology | RNA, Long Noncoding - metabolism | RNA sequencing | Immune response | RNA | DNA | Genetic research | Interferon | Biological response modifiers | Mass spectrometry | Index Medicus | Human Umbilical Vein Endothelial Cells | Nuclear Matrix-Associated Proteins | Herpesvirus 8, Human | RNA-Binding Proteins | Life Sciences | Interferon Regulatory Factor-3 | Genetics | Calcium-Binding Proteins | Intracellular Signaling Peptides and Proteins | PTB-Associated Splicing Factor | Nuclear Proteins | Membrane Proteins | Nucleotidyltransferases | Ku Autoantigen | Octamer Transcription Factors | RNA, Long Noncoding
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4. Full Text Flavivirus NS1 Triggers Tissue-Specific Vascular Endothelial Dysfunction Reflecting Disease Tropism
by Puerta-Guardo, Henry and Glasner, Dustin R and Espinosa, Diego A and Biering, Scott B and Patana, Mark and Ratnasiri, Kalani and Wang, Chunling and Beatty, P. Robert and Harris, Eva
Cell reports (Cambridge), ISSN 2211-1247, 02/2019, Volume 26, Issue 6, pp. 1598 - 1613.e8
yellow fever virus | West Nile virus | disease tropism | dengue virus | vascular leak | Japanese encephalitis virus | Zika virus | Flavivirus | NS1 | endothelial permeability | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Liver - virology | Liver - pathology | Zika Virus - genetics | Yellow fever virus - pathogenicity | Humans | Brain - virology | Cell Membrane Permeability | Male | Dengue - pathology | West Nile virus - metabolism | Lung - virology | Viral Nonstructural Proteins - chemistry | West Nile virus - genetics | Encephalitis Virus, Japanese - metabolism | Dengue Virus - pathogenicity | Yellow fever virus - metabolism | Umbilical Veins - pathology | Endothelial Cells - virology | Glycocalyx - chemistry | Zika Virus - metabolism | Dengue - genetics | Zika Virus - pathogenicity | Cell Line | Gene Expression | Lung - pathology | Viral Nonstructural Proteins - genetics | Dermis - virology | Organ Specificity | West Nile virus - pathogenicity | Encephalitis Virus, Japanese - genetics | Dengue Virus - genetics | Glycocalyx - virology | Animals | Virus Replication | Brain - pathology | Dengue - metabolism | Dermis - pathology | Umbilical Veins - virology | Viral Nonstructural Proteins - metabolism | Mice | Dengue Virus - metabolism | Encephalitis Virus, Japanese - pathogenicity | Primary Cell Culture | Endothelial Cells - pathology | Yellow fever virus - genetics | Index Medicus
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5. Full Text Atorvastatin inhibits the apoptosis of human umbilical vein endothelial cells induced by angiotensin II via the lysosomal-mitochondrial axis
by Chang, Ye and Li, Yuan and Ye, Ning and Guo, Xiaofan and Li, Zhao and Sun, Guozhe and Sun, Yingxian
Apoptosis (London), ISSN 1360-8185, 9/2016, Volume 21, Issue 9, pp. 977 - 996
Biochemistry, general | Mitochondria | Biomedicine | Atorvastatin | Lysosomes | Cancer Research | Oncology | HUVEC | Angiotensin II | Cell Biology | Virology | Apoptosis | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Lysosomes - drug effects | Angiotensin II - genetics | Human Umbilical Vein Endothelial Cells - drug effects | Reactive Oxygen Species - metabolism | Angiotensin II - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Signal Transduction | Apoptosis - drug effects | Atorvastatin Calcium - pharmacology | Cytochromes c - metabolism | Humans | Mitochondria - metabolism | Mitochondria - drug effects | Lysosomes - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Cytochrome c | Proteins | Angiotensin | Cathepsins | Mitochondrial DNA | Antilipemic agents | Endothelium | Index Medicus
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6. Full Text Kaposi's Sarcoma-Associated Herpesvirus Infection Promotes Invasion of Primary Human Umbilical Vein Endothelial Cells by Inducing Matrix Metalloproteinases
by Li-Wu Qian and Jianping Xie and Fengchun Ye and Shou-Jiang Gao
Journal of Virology, ISSN 0022-538X, 07/2007, Volume 81, Issue 13, pp. 7001 - 7010
Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Miscellaneous | Microbiology | Virology | Gene Expression Regulation, Enzymologic - drug effects | Phenylalanine - analogs & derivatives | Phenylalanine - pharmacology | Humans | Autocrine Communication - drug effects | Neovascularization, Pathologic - virology | Transcription Factor AP-1 - metabolism | Neovascularization, Pathologic - pathology | Neovascularization, Pathologic - enzymology | Protease Inhibitors - pharmacology | RNA, Messenger - biosynthesis | Umbilical Veins - enzymology | Herpesvirus 8, Human - metabolism | Neoplasm Invasiveness - pathology | Umbilical Veins - pathology | Endothelial Cells - virology | Collagenases - biosynthesis | Response Elements | Cells, Cultured | Thiophenes - pharmacology | Sarcoma, Kaposi - pathology | Up-Regulation - drug effects | RNA, Neoplasm - biosynthesis | Sarcoma, Kaposi - enzymology | Paracrine Communication - drug effects | Umbilical Veins - virology | Endothelial Cells - pathology | Endothelial Cells - enzymology | Cell Transformation, Viral | Index Medicus | Pathogenesis and Immunity
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7. Full Text EGCG protects against homocysteine-induced human umbilical vein endothelial cells apoptosis by modulating mitochondrial-dependent apoptotic signaling and PI3K/Akt/eNOS signaling pathways
by Liu, Shumin and Sun, Zhengwu and Chu, Peng and Li, Hailong and Ahsan, Anil and Zhou, Ziru and Zhang, Zonghui and Sun, Bin and Wu, Jingjun and Xi, Yalin and Han, Guozhu and Lin, Yuan and Peng, Jinyong and Tang, Zeyao
Apoptosis (London), ISSN 1360-8185, 5/2017, Volume 22, Issue 5, pp. 672 - 680
Biochemistry, general | Epigallocatechin gallate | Reactive oxygen species | Biomedicine | Protein kinase B | Cancer Research | Oncology | Homocysteine | Cell Biology | Virology | Apoptosis | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Atherosclerosis - pathology | Hyperhomocysteinemia - pathology | Human Umbilical Vein Endothelial Cells - drug effects | Homocysteine - toxicity | Apoptosis - drug effects | Atherosclerosis - genetics | Humans | Atherosclerosis - complications | Catechin - administration & dosage | Mitochondria - drug effects | Nitric Oxide Synthase Type III - genetics | Atherosclerosis - diet therapy | Phosphatidylinositol 3-Kinases - genetics | Signal Transduction - drug effects | Antioxidants - administration & dosage | Oncogene Protein v-akt - genetics | Hyperhomocysteinemia - genetics | Catechin - analogs & derivatives | Hyperhomocysteinemia - diet therapy | Hyperhomocysteinemia - complications | Antioxidants | Yuan (China) | Catechin | Transmission electron microscopes | Nitric oxide | Pharmacy | Atherosclerosis | Development and progression | Protein kinases | Endothelium | Index Medicus
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8. Full Text Transcriptional response of human umbilical vein endothelial cell to H9N2 influenza virus infection
by Wang, Wei and Mu, Xiang and Zhao, Lihong and Wang, Jianfang and Chu, Yaocheng and Feng, Xuejian and Feng, Bo and Wang, Xiaohong and Zhang, Jianjun and Qiao, Jian
Virology (New York, N.Y.), ISSN 0042-6822, 2015, Volume 482, pp. 117 - 127
Infectious Disease | H9N2 virus | Human umbilical vein endothelial cell | Microarray | Gene expression profile | Life Sciences & Biomedicine | Science & Technology | Virology | Host-Pathogen Interactions | Microarray Analysis | Humans | Cells, Cultured | Gene Expression Profiling | Endothelial Cells - virology | Influenza A Virus, H9N2 Subtype - physiology | Virus diseases | Avian influenza | Genetic research | Avian influenza viruses | Genetic aspects | Genetic transcription | Biological response modifiers | Health aspects | Endothelium | Anopheles | Genes | Interferon | Index Medicus
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9. Full Text Comparative analysis of putative novel microRNA expression profiles induced by enterovirus 71 and coxsackievirus A16 infections in human umbilical vein endothelial cells using high-throughput sequencing
by Song, Jie and Hu, Yajie and Zheng, Huiwen and Guo, Lei and Huang, Xing and Jiang, Xi and Li, Weiyu and Li, Jiaqi and Yang, Zening and Dong, Shaozhong and Liu, Longding
Infection, genetics and evolution, ISSN 1567-1348, 09/2019, Volume 73, pp. 401 - 410
Coxsackievirus A16 (CA16) | High-throughput sequencing | Hand, foot and mouth disease (HFMD) | Enterovirus 71 (EV71) | microRNAs (miRNAs) | Life Sciences & Biomedicine | Infectious Diseases | Science & Technology | Human Umbilical Vein Endothelial Cells | Gene Regulatory Networks - genetics | Humans | Cells, Cultured | Computational Biology | Gene Expression Profiling - methods | Hand, Foot and Mouth Disease - genetics | Coxsackievirus Infections - virology | Enterovirus - pathogenicity | Coxsackievirus Infections - genetics | Enterovirus Infections - virology | Host-Pathogen Interactions - genetics | Enterovirus Infections - genetics | MicroRNAs - genetics | Enterovirus A, Human - pathogenicity | High-Throughput Nucleotide Sequencing - methods | Gene Ontology | Hand, Foot and Mouth Disease - virology | Medical research | Medical colleges | MicroRNA | Genetic research | Medicine, Experimental | Development and progression | Foot-and-mouth disease | Comparative analysis | Coxsackievirus infections | Health aspects | Endothelium | Index Medicus
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10. Full Text Phosphocreatine protects against LPS-induced human umbilical vein endothelial cell apoptosis by regulating mitochondrial oxidative phosphorylation
by Sun, Zhengwu and Sun, Zhengwu and Lan, Xiaoyan and Lan, Xiaoyan and Ahsan, Anil and Ahsan, Anil and Xi, Yalin and Xi, Yalin and Liu, Shumin and Liu, Shumin and Zhang, Zonghui and Zhang, Zonghui and Chu, Peng and Chu, Peng and Song, Yushu and Song, Yushu and Piao, Fengyuan and Piao, Fengyuan and Peng, Jinyong and Peng, Jinyong and Lin, Yuan and Lin, Yuan and Han, Guozhu and Han, Guozhu and Tang, Zeyao and Tang, Zeyao
Apoptosis (London), ISSN 1360-8185, 3/2016, Volume 21, Issue 3, pp. 283 - 297
Biochemistry, general | Human umbilical vein endothelial cells | Biomedicine | Oxidative phosphorylation | Lipopolysaccharide | Cancer Research | Oncology | Mitochondrial apoptosis | Phosphocreatine | Cell Biology | Virology | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Creatine Kinase, Mitochondrial Form - metabolism | Phosphocreatine - therapeutic use | Mitochondria - enzymology | Atherosclerosis - drug therapy | Human Umbilical Vein Endothelial Cells - drug effects | Reactive Oxygen Species - metabolism | Mitochondrial Swelling - drug effects | Apoptosis - drug effects | Cytochromes c - metabolism | Humans | bcl-2-Associated X Protein - metabolism | Endothelium, Vascular - drug effects | Membrane Potential, Mitochondrial - drug effects | Endothelium, Vascular - physiology | Oxidative Phosphorylation - drug effects | Proto-Oncogene Proteins c-bcl-2 - metabolism | Human Umbilical Vein Endothelial Cells - physiology | Caspases - metabolism | Adenosine Triphosphate - metabolism | Lipopolysaccharides - pharmacology | Phosphocreatine - pharmacology | Mitochondria - physiology | Cytoprotection | Niacinamide | Yuan (China) | Enzymes | Drugstores | Permeability | Muscle proteins | Cells | Endothelium | Purines | Pharmacy | Atherosclerosis | Physiological aspects | Adenosine triphosphate | Apoptosis | Index Medicus
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