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Diabetologia, ISSN 1432-0428, 2011, Volume 54, Issue 6, pp. 1480 - 1490
Journal Article
PloS one, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53557
Erythropoietin (EPO) has multiple biological functions, including the modulation of glucose metabolism. However, the mechanisms underlying the action of EPO... 
ISCHEMIA-REPERFUSION INJURY | SIGNALING PATHWAYS | METABOLISM | RECOMBINANT-HUMAN-ERYTHROPOIETIN | MULTIDISCIPLINARY SCIENCES | HEMODIALYSIS-PATIENTS | GENE-EXPRESSION | IKK-BETA | INDUCED OBESITY | INDUCED INSULIN-RESISTANCE | PROTECTS | Erythropoietin - pharmacology | Inflammation - pathology | Liver - pathology | Liver - enzymology | Erythropoietin - therapeutic use | Gluconeogenesis - drug effects | Humans | Body Weight - drug effects | Insulin Receptor Substrate Proteins - metabolism | Insulin - blood | RNA, Messenger - metabolism | Obesity - genetics | Glucose Intolerance - blood | Inflammation - blood | Receptors, Erythropoietin - genetics | Glucose Intolerance - drug therapy | Inflammation - complications | Liver - drug effects | Fasting - blood | Diet, High-Fat | Proto-Oncogene Proteins c-akt - metabolism | Glucose Intolerance - genetics | Glucose Tolerance Test | Mice, Inbred C57BL | RNA, Messenger - genetics | Signal Transduction - genetics | Glucose Intolerance - complications | Feeding Behavior - drug effects | Glucose-6-Phosphatase - metabolism | Obesity - pathology | Gene Expression Regulation - drug effects | Animals | Gluconeogenesis - genetics | Signal Transduction - drug effects | Phosphoenolpyruvate Carboxykinase (ATP) - metabolism | Inflammation - genetics | Receptor, Insulin - metabolism | Mice | Blood Glucose - metabolism | Receptors, Erythropoietin - metabolism | Type 2 diabetes | Phosphatases | Erythropoietin | Diet | Glucose intolerance | Blood sugar | Analysis | Body weight | Inflammation | Insulin | Phosphorylation | Transcription factors | Liver | AKT protein | Glucose | Kinases | Blood | High fat diet | Interleukin 6 | Glucose metabolism | Rodents | Toll-like receptors | Inhibition | Gluconeogenesis | NF-κB protein | Fasting | Diabetes mellitus | Extracellular signal-regulated kinase | c-Jun protein | MAP kinase | TLR4 protein | Tumor necrosis factor-α | Metabolism | Substrates | 1-Phosphatidylinositol 3-kinase | Glucose tolerance | Intolerance | Insulin resistance
Journal Article
PloS one, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, p. e46986
.... Therefore, we investigated the positive and negative regulatory components controlling expression of the CCL2 gene using isolated rat islets and INS-1-derived beta-cell lines... 
ISLET TRANSPLANTATION | DIABETES-MELLITUS | STIMULATED INSULIN-SECRETION | INCREASED EXPRESSION | MULTIDISCIPLINARY SCIENCES | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | KINASE INHIBITOR | CHEMOKINE RECEPTORS | NF-KAPPA-B | DUAL-SPECIFICITY PHOSPHATASES | TISSUE FACTOR | Dual Specificity Phosphatase 1 - genetics | Interleukin-1beta - pharmacology | Insulin-Secreting Cells - secretion | Humans | RNA, Messenger - genetics | Transcriptional Activation - drug effects | Rats | Chemokine CCL2 - genetics | Receptors, Glucocorticoid - metabolism | RNA, Messenger - metabolism | Gene Expression Regulation - drug effects | Insulin-Secreting Cells - metabolism | Animals | I-kappa B Kinase - metabolism | Insulin-Secreting Cells - drug effects | Transcription Factor RelA - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Line, Tumor | Glucocorticoids - pharmacology | Dual Specificity Phosphatase 1 - metabolism | Insulin-Secreting Cells - cytology | Protein Kinase Inhibitors - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Phosphorylation | Glucocorticoids | Transplants & implants | Transcription | Regulatory sequences | Leukocytes | Kinases | Phosphatase | Recruitment | MAP kinase phosphatase | Proteins | Pathways | Rodents | Inhibition | Localization | Pancreas | Obesity | NF-κB protein | Deactivation | Preservation | Cytokines | Nutrition | Diabetes mellitus | MAP kinase | Inflammation | Pharmacology | IL-1β | Metabolism | Gene expression | Insulin | White blood cells | Signaling | Microscopy | Cell lines | Diabetes | Endocrinology | Chemokines | Monocyte chemoattractant protein 1
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, p. e0165459
Journal Article
Cell (Cambridge), ISSN 0092-8674, 11/2013, Volume 155, Issue 4, pp. 765 - 777
Journal Article
Nature genetics, ISSN 1546-1718, 1999, Volume 21, Issue 1, pp. 76 - 83
Journal Article
Lipids in health and disease, ISSN 1476-511X, 2015, Volume 14, Issue 1, p. 121
... systems eventually leading to metabolic defects (obesity, metabolic syndrome, and diabetes). Insulin is the primary regulator of carbohydrate, fat, and protein... 
Adipose tissue | Liver | Insulin resistance | Inflammation | Muscle | Hypothalamus | Palmitic acid | Fatty acids | Endotoxemia | Omega-3 fatty acids | METABOLIC SYNDROME | GASTROINTESTINAL SATIETY SIGNALS | HEALTHY-SUBJECTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INDUCED INFLAMMATION | GUT MICROBIOTA | DIET-INDUCED OBESITY | SKELETAL-MUSCLE | NUTRITION & DIETETICS | PPAR-GAMMA | ADIPOSE-TISSUE | GLUCOSE-PRODUCTION | Tumor Necrosis Factor-alpha - metabolism | Dietary Fats - metabolism | Liver - pathology | Humans | Tumor Necrosis Factor-alpha - genetics | Muscle, Skeletal - metabolism | Obesity - genetics | Adipose Tissue - metabolism | MAP Kinase Kinase 4 - metabolism | I-kappa B Kinase - metabolism | Liver - drug effects | Muscle, Skeletal - drug effects | Dietary Fats - administration & dosage | Fatty Acids - administration & dosage | Hypothalamus - drug effects | Insulin - genetics | Fatty Acids - metabolism | Liver - metabolism | Adipose Tissue - pathology | Gene Expression Regulation | Insulin Resistance | Hypothalamus - pathology | I-kappa B Kinase - genetics | Obesity - metabolism | Obesity - pathology | Insulin - metabolism | Hypothalamus - metabolism | Diet | MAP Kinase Kinase 4 - genetics | Muscle, Skeletal - pathology | Adipose Tissue - drug effects | Saturated fatty acids | Obesity | Phosphorylation | Adipocytes | Kinases | Metabolism | Gene expression | Insulin | Proteins | Hyperglycemia | Tumor necrosis factor-TNF | Diabetes | Binding sites
Journal Article