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The Journal of biological chemistry, ISSN 1083-351X, 2009, Volume 284, Issue 36, pp. 24035 - 24048
..., macrophages need double stimulation with ligands to both Toll-like receptors (TLRs) for IL-1β... 
GENE | LEUCINE-RICH PROTEOGLYCANS | CASPASE-1 | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE IMMUNE-RESPONSE | HYALURONAN | IL-1-BETA | SECRETION | ATP | NALP3 INFLAMMASOME | Toll-Like Receptor 2 - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Immunity, Innate - genetics | Extracellular Matrix - metabolism | Caspase 1 - metabolism | Male | Ureteral Obstruction - metabolism | Interleukin-1beta - genetics | Inflammation - metabolism | Kidney - metabolism | Carrier Proteins - immunology | Signal Transduction | Proteoglycans - immunology | Extracellular Matrix Proteins - genetics | Proteoglycans - metabolism | Toll-Like Receptor 4 - genetics | Biglycan | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Ureteral Obstruction - immunology | Macrophages - metabolism | Receptors, Purinergic P2 - metabolism | Toll-Like Receptor 2 - immunology | Mice | Proteoglycans - genetics | Extracellular Matrix - immunology | Kidney - immunology | Caspase 1 - immunology | Extracellular Matrix - genetics | Receptors, Purinergic P2 - genetics | Ureteral Obstruction - genetics | Interleukin-1beta - biosynthesis | Lung - metabolism | Receptors, Purinergic P2 - immunology | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Carrier Proteins - biosynthesis | Interleukin-1beta - immunology | Inflammation - immunology | Protein Structure, Tertiary - genetics | Receptors, Purinergic P2X7 | Carrier Proteins - genetics | Immunity, Innate - immunology | Animals | Caspase 1 - genetics | Extracellular Matrix Proteins - immunology | Receptors, Purinergic P2X4 | Inflammation - genetics | Lung - immunology | Animal models | Reactive oxygen species | Oligomerization | Proteoglycans | Transcription | Lung | Inflammation | Macrophages | Kidney | Stress | Caspase-1 | Purine P2X receptors | Signal transduction | Cell activation | TLR2 protein | Interleukin 1 | Toll-like receptors | Extracellular matrix | Sepsis | Cooperativity | Injuries | Glycobiology and Extracellular Matrices
Journal Article
Cancer research (Chicago, Ill.), ISSN 0008-5472, 08/2013, Volume 73, Issue 16, pp. 5016 - 5028
TGF-beta has limited effects on ovarian cancer cells, but its contributions to ovarian tumor growth might be mediated through elements of the tumor... 
CROSS-TALK | MATRIX | STEM-CELLS | CONTRIBUTE | INHIBITION | ONCOLOGY | MEMBRANE | TRANSCRIPTION | STROMAL CELLS | EXPRESSION | FIBROBLASTS | Up-Regulation | Receptors, Transforming Growth Factor beta - genetics | Epithelial Cells - metabolism | Stromal Cells - pathology | Humans | Ovarian Neoplasms - pathology | Transcriptome | NF-kappa B - metabolism | Cell Movement - genetics | Ovarian Neoplasms - genetics | Matrix Metalloproteinase 9 - metabolism | Versicans - genetics | Tumor Microenvironment - genetics | Matrix Metalloproteinase 9 - genetics | Hyaluronan Receptors - metabolism | Female | Ovarian Neoplasms - metabolism | Extracellular Matrix Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Signal Transduction | Neoplasm Invasiveness | Extracellular Matrix Proteins - genetics | Stromal Cells - metabolism | Protein-Serine-Threonine Kinases - genetics | Epithelial Cells - pathology | Smad Proteins - genetics | Fibroblasts - pathology | Hyaluronan Receptors - genetics | Disease Progression | Transforming Growth Factor beta - genetics | NF-kappa B - genetics | Receptors, Transforming Growth Factor beta - metabolism | Cell Line, Tumor | Versicans - metabolism | Smad Proteins - metabolism | Transforming Growth Factor beta - metabolism | cancer-associated fibroblast | TGF-β | versican | tumor microenvironment | ovarian cancer
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2012, Volume 287, Issue 39, pp. 32800 - 32824
.... Importantly, hyaluronan (HA) stimulates the CD44v3 (an HA receptor) interaction with Oct4-Sox2-Nanog leading to both a complex formation and the nuclear translocation of three CSC transcription factors... 
SOX2 | IN-VITRO | ISOFORMS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | MARKER NANOG | NF-KAPPA-B | TRANSCRIPTIONAL REGULATION | CD44 | BREAST | MICRORNA | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Homeodomain Proteins - metabolism | Humans | Retinal Dehydrogenase - metabolism | Gene Expression Regulation, Neoplastic | Cell Survival - genetics | Drug Resistance, Neoplasm | Mouth Neoplasms - metabolism | Neoplasm Proteins - metabolism | SOXB1 Transcription Factors - metabolism | Octamer Transcription Factor-3 - genetics | Neoplastic Stem Cells - metabolism | SOXB1 Transcription Factors - genetics | Isoenzymes - metabolism | Hyaluronan Receptors - metabolism | Neoplastic Stem Cells - pathology | Antineoplastic Agents - pharmacology | Epigenesis, Genetic - drug effects | Neoplasm Proteins - genetics | Mouth Neoplasms - genetics | Cell Survival - drug effects | Nanog Homeobox Protein | Isoenzymes - genetics | Hyaluronic Acid - genetics | MicroRNAs - biosynthesis | Signal Transduction - genetics | Cisplatin - pharmacology | Retinal Dehydrogenase - genetics | Hyaluronan Receptors - genetics | Homeodomain Proteins - genetics | RNA, Neoplasm - biosynthesis | Signal Transduction - drug effects | Epigenesis, Genetic - genetics | Hyaluronic Acid - metabolism | Octamer Transcription Factor-3 - metabolism | Mouth Neoplasms - pathology | RNA, Neoplasm - genetics | MicroRNAs - genetics | Hyaluronan | Self-renewal | MicroRNA | Signal Transduction | Extracellular Matrix | Head and Neck Cancer | Differentiation | Cancer Stem Cells
Journal Article
PloS one, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, p. e0132977
Cancer stem cells are cancer cells characterized by stem cell properties and represent a small population of tumor cells that drives tumor development,... 
TESTIS ANTIGEN BORIS | HEPATOCELLULAR-CARCINOMA | SIDE POPULATION | IN-VITRO | INDUCED APOPTOSIS | TELOMERASE | MULTIDISCIPLINARY SCIENCES | IMPRINTED SITES | CTCF | IDENTIFICATION | TUMOR-INITIATING CELLS | ATP Binding Cassette Transporter, Sub-Family G, Member 2 | RNA, Small Interfering - genetics | Cell Proliferation | Epithelial Cells - metabolism | Polycomb Repressive Complex 1 - metabolism | Homeodomain Proteins - metabolism | Humans | Retinal Dehydrogenase - metabolism | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | Epithelial-Mesenchymal Transition - genetics | Neoplasm Proteins - metabolism | SOXB1 Transcription Factors - metabolism | DNA-Binding Proteins - metabolism | Octamer Transcription Factor-3 - genetics | Polycomb Repressive Complex 1 - genetics | Telomerase - genetics | ATP-Binding Cassette Transporters - genetics | Neoplastic Stem Cells - metabolism | SOXB1 Transcription Factors - genetics | Isoenzymes - metabolism | Hyaluronan Receptors - metabolism | ATP-Binding Cassette Transporters - metabolism | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Telomerase - metabolism | Neoplasm Proteins - genetics | DNA-Binding Proteins - antagonists & inhibitors | Nanog Homeobox Protein | Signal Transduction | Isoenzymes - genetics | Spheroids, Cellular - metabolism | Epithelial Cells - pathology | Retinal Dehydrogenase - genetics | DNA-Binding Proteins - genetics | Organ Specificity | Hyaluronan Receptors - genetics | Homeodomain Proteins - genetics | Phenotype | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Biomarkers, Tumor - genetics | Cell Movement | RNA, Small Interfering - metabolism | RNA | Genes | Colorectal cancer | Stem cells | Genetic aspects | Gene expression | Cancer | Protein binding | Neurosciences | Germ cells | Telomerase reverse transcriptase | Populations | Dyes | Mesenchyme | Oct-4 protein | Gene regulation | Oncology | Spheres | Drug resistance | Tissues | Metastases | DNA-binding protein | CD44 antigen | Cell cycle | Life sciences | Colon | Telomerase | Growth factors | Deoxyribonucleic acid--DNA | Medical research | Antigens | Cell survival | Tumor cells | Invasiveness | Cervix | Survival | Brain research | Molecular modelling | Medical prognosis | Epigenetics | Breast | Tumors | Deoxyribonucleic acid | DNA
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2013, Volume 288, Issue 21, pp. 14824 - 14838
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e14062
Background: The cancer stem cell theory hypothesizes that cancers are perpetuated by cancer stem cells (CSC) or tumor initiating cells (TIC) possessing... 
COLON-CANCER | INITIATING CELLS | POPULATION | MARKER | MULTIDISCIPLINARY SCIENCES | TUMOR | HYALURONAN | CHEMORESISTANCE | FLOW-CYTOMETRY | IDENTIFICATION | LINES | Immunohistochemistry | Humans | Lung Neoplasms - metabolism | Middle Aged | Glycoproteins - metabolism | Peptides - genetics | Immunoblotting | Lung Neoplasms - pathology | Male | Transplantation, Heterologous | Gene Expression Profiling | Antigens, CD - genetics | Antigens, CD - metabolism | Neoplasms, Experimental - pathology | Octamer Transcription Factor-3 - genetics | Flow Cytometry | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Hyaluronan Receptors - metabolism | Neoplasms, Experimental - genetics | Neoplastic Stem Cells - pathology | Female | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Glycoproteins - genetics | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | AC133 Antigen | Hyaluronan Receptors - genetics | Animals | Polycomb Repressive Complex 1 | Mice, Nude | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Aged | Mice | Neoplasms, Experimental - metabolism | Squamous cell carcinoma | Lung cancer, Non-small cell | Analysis | Stem cells | Flow cytometry | Biotechnology | Laboratories | Heart surgery | Oct-4 protein | Lung cancer | Colorectal cancer | Stem cell transplantation | Proteins | Allografts | Epidermal growth factor | CD44 antigen | Xenografts | Cell cycle | CD34 antigen | Subpopulations | Cell survival | Tumor cells | Non-small cell lung carcinoma | Tumor cell lines | Cisplatin | Studies | Polymerase chain reaction | Pathology | Cytometry | Properties (attributes) | Medical prognosis | Biomarkers | In vivo methods and tests | Pluripotency | Tumors | Apoptosis | Cancer
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 7, p. e69808
Several studies have demonstrated the potential for vector-mediated gene transfer to the brain. Helper-dependent (HD) human (HAd) and canine (CAV-2)... 
GREEN FLUORESCENT PROTEIN | STIMULATION | DENDRITIC CELLS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | GUIDELINES | NEURONS | RECEPTOR | TRANSDUCTION | CENTRAL-NERVOUS-SYSTEM | GENE-TRANSFER | Cell Cycle - genetics | Mesencephalon - cytology | Ataxia Telangiectasia Mutated Proteins - metabolism | Humans | Transcriptional Activation | Neurons - cytology | Gene Expression Profiling | Neural Stem Cells - cytology | Wnt Proteins - metabolism | Cell Differentiation - genetics | Wnt Proteins - genetics | DNA Damage - genetics | Endocytosis - genetics | Interferons - genetics | Toll-Like Receptors - metabolism | Neurons - virology | Transduction, Genetic | Neural Stem Cells - virology | Adenoviruses, Human - physiology | Genetic Vectors - metabolism | Signal Transduction - genetics | Transcriptome - genetics | Lentivirus | Down-Regulation - genetics | Genetic Vectors - genetics | Animals | Toll-Like Receptors - genetics | Dogs | Interferons - metabolism | Immunity - genetics | Neural Stem Cells - metabolism | Adenoviruses, Canine - physiology | Viral genetics | Neurons | Genes | Genomics | Genetic transcription | DNA microarrays | Genetic vectors | Adenoviruses | High-definition television | Gene therapy | Tumor proteins | Hyaluronic acid | Health aspects | Brain | Transcription | Toxicity | p53 Protein | DNA damage | Central nervous system | Homeostasis | Genomes | Kinases | Proteins | Morphogenesis | Hyaluronan | Receptors | Pathways | Human immunodeficiency virus--HIV | Toll-like receptors | Biocompatibility | Growth factors | Deoxyribonucleic acid--DNA | Expression vectors | Brain diseases | Immune system | Cell survival | Immune response | Gene transfer | Cloning | Inflammation | Gene expression | Vectors (Biology) | Neural stem cells | In vivo methods and tests | Interferon | Life Sciences | Biochemistry, Molecular Biology | Deoxyribonucleic acid | HIV | DNA | Human immunodeficiency virus
Journal Article
PloS one, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0169452 - e0169452
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 03/2012, Volume 16, Issue 3, pp. 483 - 495
The fibrocytes are thought to serve as a source of newly deposited collagens I and III during reparative processes and in certain fibrotic disorders, but their... 
Endo180 | matrix internalization | scavenger receptor | cell adhesion | collagen receptor | fibrocyte | fibrosis | extracellular matrix | Scavenger receptor | Fibrocyte | Cell adhesion | Fibrosis | Extracellular matrix | Collagen receptor | Matrix internalization | MEDICINE, RESEARCH & EXPERIMENTAL | RETICULAR BASEMENT-MEMBRANE | PERIPHERAL-BLOOD FIBROCYTES | MONONUCLEAR-CELLS | CIRCULATING FIBROCYTES | SCAVENGER RECEPTORS | RECEPTOR-ASSOCIATED PROTEIN/ENDO180 | CELL BIOLOGY | MESENCHYMAL PROGENITOR | GENE-EXPRESSION | LUNG FIBROBLASTS | NEPHROGENIC SYSTEMIC FIBROSIS | Mannose-Binding Lectins - metabolism | Biglycan - genetics | Humans | Extracellular Matrix - metabolism | RNA, Messenger - analysis | Gene Expression Profiling | Lectins, C-Type - genetics | Extracellular Matrix - genetics | Glucuronosyltransferase - genetics | Lectins, C-Type - metabolism | RNA, Messenger - biosynthesis | Versicans - genetics | Protein Isoforms - metabolism | Heparan Sulfate Proteoglycans - genetics | Cell Differentiation | Collagen - genetics | Hyaluronan Synthases | Decorin - genetics | Fibroblasts - metabolism | Receptors, Scavenger - metabolism | Gene Expression | Receptors, Cell Surface - metabolism | Biglycan - metabolism | Collagen - metabolism | Mannose-Binding Lectins - genetics | Glucuronosyltransferase - metabolism | Receptors, Scavenger - genetics | Heparan Sulfate Proteoglycans - metabolism | Fibroblasts - cytology | Versicans - metabolism | Primary Cell Culture | Connective Tissue Cells - metabolism | Decorin - metabolism | Connective Tissue Cells - cytology | Protein Isoforms - genetics | Receptors, Cell Surface - genetics | Flow cytometry | Decorin | Pathogenesis | Perlecan | Collagens | Labelling | Mannose | Remodeling | Hyaluronan synthase | Proteins | Hyaluronan | Receptors | Fibroblasts | Scavenger receptors | Versican | Antigens | Immunoglobulins | Level (quantity) | Wound healing | Proteoglycans | Cloning | Gene expression | Mannose receptors | Asthma | CD163 antigen | Properties (attributes) | Collagen | Hyaluronic acid | Original
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2011, Volume 108, Issue 4, pp. 1397 - 1402
Tumors are often heterogeneous, being composed of multiple cell types with different phenotypic and molecular properties. Cancer stem-like cells (CSCs) are a... 
Tumor cell line | MicroRNA | Stem cells | Cell lines | Transformed cell line | Breast cancer | Cells | Cancer | Tumors | Mesenchymal stem cells | Cellular transformation | Cancer stem cells equilibrium | Inflammation | cancer stem cells equilibrium | cellular transformation | ZEB1 | REPRESSION | inflammation | MULTIDISCIPLINARY SCIENCES | MESENCHYMAL TRANSITION | IDENTIFICATION | MIR-200 FAMILY | SIGNATURE | Prostatic Neoplasms - metabolism | Receptors, Estrogen - metabolism | Humans | Male | Transplantation, Heterologous | Gene Expression Profiling | Breast Neoplasms - metabolism | Neoplasms, Experimental - pathology | Cell Differentiation - genetics | Prostatic Neoplasms - genetics | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | Hyaluronan Receptors - metabolism | Neoplasms, Experimental - genetics | src-Family Kinases - metabolism | Antibodies - immunology | Neoplastic Stem Cells - pathology | Female | Interleukin-6 - metabolism | Cell Line | Prostatic Neoplasms - pathology | Receptors, Estrogen - genetics | Antineoplastic Agents, Hormonal - pharmacology | Interleukin-6 - genetics | Antibodies - pharmacology | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Interleukin-6 - immunology | Tamoxifen - pharmacology | Cell Line, Tumor | Mice | MicroRNAs - genetics | Cell Transformation, Neoplastic - drug effects | Neoplasms, Experimental - metabolism | src-Family Kinases - genetics | Care and treatment | Cancer cells | Physiological aspects | Development and progression | Genetic aspects | Research | Cell transformation | Health aspects | Interleukin-6 | Biological Sciences
Journal Article
PloS one, ISSN 1932-6203, 2018, Volume 13, Issue 12, p. e0209583
Tumor necrosis factor-like weak inducer of apoptosis (TWEAK), along with its receptor fibroblast growth factor-inducible (Fn... 
PATHOGENESIS | STIMULATION | OPHTHALMOPATHY | MULTIDISCIPLINARY SCIENCES | DISEASE | CYTOKINE | TWEAK/FN14 PATHWAY | FN14 | EXPRESSION | T-LYMPHOCYTES | AXIS | Inflammation - pathology | Interleukin-8 - genetics | TWEAK Receptor - genetics | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Apoptosis - genetics | Male | Interleukin-1beta - genetics | Inflammation - blood | Adult | Female | Cytokine TWEAK - blood | Fibroblasts - metabolism | Interleukin-6 - genetics | TWEAK Receptor - blood | Gene Expression Regulation - genetics | Chemokine CCL2 - genetics | Signal Transduction - genetics | Graves Ophthalmopathy - genetics | Fibroblasts - pathology | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Graves Ophthalmopathy - blood | Inflammation - genetics | Cytokine TWEAK - genetics | Graves Ophthalmopathy - pathology | Care and treatment | Cytokines | Graves' disease | Fibroblast growth factors | Genetic aspects | Research | Gene expression | T cells | Risk factors | Tumor necrosis factor receptors | TWEAK protein | Fibroblast growth factor | Transcription | Disease | Pathogenesis | Interleukin | Arthritis | Thyroid gland | Kinases | Tissues | Interleukin 6 | Proteins | Angiogenesis | Hyaluronan | Cell growth | Surgery | Fibroblasts | Tumor necrosis factor-TNF | Bioindicators | Growth factors | Interleukin 8 | Thyroid | Inflammation | Tumor necrosis factor-α | Patients | Myogenesis | Medicine | Signaling | Government agencies | Monocytes | Hospitals | Monocyte chemoattractant protein | Biomarkers | Diagnostic systems | Hyaluronic acid | Monocyte chemoattractant protein 1 | Apoptosis | Tumors
Journal Article