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Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 11/2009, Volume 13, Issue 11‐12, pp. 4505 - 4521
The extracellular sulfatases Sulf1 and Sulf2 remove specific 6‐O‐sulfate groups from heparan sulfate, thereby modulating numerous signalling pathways... 
behaviour | synaptic plasticity | neurite outgrowth | heparan sulfate proteoglycans | Sulf1 | Sulf2 | Heparan sulfate proteoglycans | Behaviour | Neurite outgrowth | Synaptic plasticity | MEDICINE, RESEARCH & EXPERIMENTAL | NERVOUS-SYSTEM | CRYSTAL-STRUCTURE | AXON GUIDANCE | CELL-SURFACE | CELL BIOLOGY | HB-GAM | HEPARAN-SULFATE | MICE | LONG-TERM POTENTIATION | GROWTH-ASSOCIATED MOLECULE | Neurons - pathology | Brain - embryology | Synaptic Transmission - physiology | Neurites - enzymology | Brain - enzymology | Hydrocephalus - pathology | Embryo Loss - enzymology | Sulfotransferases - deficiency | Behavior, Animal | Nervous System Malformations - enzymology | Embryo Loss - pathology | Hydrocephalus - physiopathology | Nervous System Malformations - physiopathology | Sulfatases - metabolism | Long-Term Potentiation - physiology | Hippocampus - enzymology | Hippocampus - ultrastructure | Sulfotransferases - metabolism | Hydrocephalus - enzymology | Animals, Newborn | Mice, Inbred C57BL | Sulfatases - deficiency | Hippocampus - pathology | Hydrocephalus - complications | Embryo Loss - physiopathology | Neurites - pathology | Phenotype | Animals | Neuronal Plasticity | Neurons - enzymology | Nervous System Malformations - complications | Mice | Hippocampus - physiopathology | Extracellular Space - enzymology | Cerebellum | Spine | Spatial discrimination learning | Homeostasis | Nervous system | Lethality | Sulfates | Proteins | Signal transduction | Embryogenesis | Functionals | Rodents | Plasticity | Growth factors | Heparan sulfate | Enzymes | Phenotypes | Brain architecture | Redundancy | Axonogenesis | Plasticity (cerebellar) | Anatomy | Embryos | Embryonic growth stage | Plasticity (synaptic) | Microscopy | Stem cells | Viability | Hippocampus | Spatial memory
Journal Article
Nature Genetics, ISSN 1061-4036, 08/2012, Volume 44, Issue 8, pp. 934 - 940
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2006, Volume 26, Issue 37, pp. 9593 - 9602
Hydrocephalus is a common and variegated pathology often emerging in newborn children after genotoxic insults during pregnancy (Hicks and D'Amato, 1980). Cre... 
Cre genotoxicity | Hydrocephalus | Cre recombinase | Cortex | Nestin Cre | Neuronal progenitor cell | cortex | SURVIVAL | nestin Cre | NERVOUS-SYSTEM | EPENDYMAL DENUDATION | hydrocephalus | MOUSE | NEURAL STEM | CONGENITAL HYDROCEPHALUS | KNOCKOUT MICE | neuronal progenitor cell | MAMMALIAN-CELLS | NEUROSCIENCES | RECOMBINASE ACTIVITY | GENE | Ependyma - pathology | Nestin | Receptors, Estrogen - metabolism | Cell Proliferation | Microcephaly - genetics | Nuclear Localization Signals - genetics | Brain - enzymology | Aneuploidy | Brain - abnormalities | Microcephaly - enzymology | Nervous System Malformations - enzymology | Stem Cells - enzymology | Hydrocephalus - physiopathology | Intermediate Filament Proteins - genetics | Nervous System Malformations - physiopathology | Integrases - metabolism | Microcephaly - physiopathology | Hydrocephalus - enzymology | Hydrocephalus - genetics | Nervous System Malformations - genetics | Cell Differentiation - physiology | Gene Expression Regulation, Developmental - physiology | Biomarkers - metabolism | Receptors, Estrogen - genetics | Brain - physiopathology | Nuclear Localization Signals - metabolism | Ependyma - metabolism | Mice, Transgenic | Nerve Tissue Proteins - genetics | Ependyma - abnormalities | Nerve Tissue Proteins - metabolism | Animals | Cell Death - physiology | Neurons - enzymology | Tamoxifen - pharmacology | Mice | Integrases - genetics | Intermediate Filament Proteins - metabolism | Selective Estrogen Receptor Modulators - pharmacology
Journal Article
Neurosurgery, ISSN 0148-396X, 10/2009, Volume 65, Issue 4, pp. 702 - 708
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e64455
Mammalian target of rapamycin (mTOR) is a protein kinase that senses nutrient availability, trophic factors support, cellular energy level, cellular stress,... 
MAMMALIAN TARGET | NERVOUS-SYSTEM | TEMPORAL-LOBE EPILEPSY | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MOUSE MODEL | PROTEIN S6 PHOSPHORYLATION | GENE-EXPRESSION | HIPPOCAMPAL-NEURONS | TUBEROUS-SCLEROSIS | INDUCED SEIZURES | Rats, Wistar | Status Epilepticus - enzymology | TOR Serine-Threonine Kinases - metabolism | Astrocytes - pathology | Seizures - drug therapy | Status Epilepticus - chemically induced | Brain - enzymology | Status Epilepticus - drug therapy | Male | Hippocampus - drug effects | Cell Nucleus - enzymology | Kainic Acid | Seizures - pathology | Spatio-Temporal Analysis | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Astrocytes - drug effects | Sirolimus - therapeutic use | Subcellular Fractions - drug effects | Status Epilepticus - pathology | Rats | Hippocampus - pathology | Phosphoserine - metabolism | Sirolimus - pharmacology | Subcellular Fractions - metabolism | Brain - drug effects | Sirolimus - administration & dosage | Animals | Signal Transduction - drug effects | Ribosomal Protein S6 - metabolism | Neurons - enzymology | Brain - pathology | Cell Nucleus - drug effects | Astrocytes - metabolism | Brain | Rapamycin | Seizures (Medicine) | Neurons | Status epilepticus | Protein kinases | TOR protein | Neurosciences | Animal models | Energy levels | Laboratories | Epilepsy | Activation | Kinases | Rodents | Pretreatment | Trophic factors | Seizures | Astrocytes | Mammals | Metabolism | Signaling | Neurotransmitters | Brain-derived neurotrophic factor | Acids | Protein kinase | Nutrient availability | Kainic acid | Cellular stress response
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 08/2012, Volume 32, Issue 34, pp. 11511 - 11523
Hydrocephalus formation is a frequent complication of neuropathological insults associated with neuroinflammation. However, the mechanistic role of... 
IMMUNE-RESPONSE | CELLS | STREPTOCOCCUS-PNEUMONIAE | INFLAMMATION | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | CONGENITAL HYDROCEPHALUS | NEUROSCIENCES | BRAIN | TRANSGENIC MICE | LIPOCALIN-2 | Glial Fibrillary Acidic Protein - genetics | Cell Adhesion Molecules - genetics | Lateral Ventricles - pathology | Age Factors | Humans | Brain - enzymology | Hydrocephalus - pathology | Astrocytes - enzymology | Brain - growth & development | I-kappa B Proteins - metabolism | Cerebral Cortex - cytology | Complement System Proteins - metabolism | Doxycycline - administration & dosage | I-kappa B Kinase - metabolism | Microarray Analysis | Statistics, Nonparametric | Glioma, Subependymal - etiology | Complement System Proteins - genetics | Hydrocephalus - enzymology | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Gene Expression Regulation, Developmental - physiology | Animals, Newborn | Astrocytes - drug effects | Glioma, Subependymal - pathology | Microscopy, Electron, Scanning | NF-KappaB Inhibitor alpha | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation, Developmental - drug effects | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | Hydrocephalus - complications | Signal Transduction - genetics | I-kappa B Kinase - genetics | Lateral Ventricles - growth & development | Enzyme Activation - drug effects | Cell Adhesion Molecules - metabolism | Chemokines - genetics | Lateral Ventricles - ultrastructure | Animals | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Brain - pathology | Chemokines - metabolism | Mice | Enzyme Activation - genetics | Encephalitis - etiology
Journal Article
Molecular Genetics and Metabolism, ISSN 1096-7192, 12/2017, Volume 122, pp. 41 - 48
The mucopolysaccharidosis (MPS) disorders are ultra-rare lysosomal storage disorders associated with progressive accumulation of glycosaminoglycans (GAGs) in... 
Mucopolysaccharidosis | Diagnostic imaging | Neurological disease | Hydrocephalus | Surgery | Spinal cord compression | MEDICINE, RESEARCH & EXPERIMENTAL | MORQUIO-BRAILSFORD | IMAGING FINDINGS | IVA MORQUIO | CHILDREN | MAROTEAUX-LAMY-SYNDROME | ENDOCRINOLOGY & METABOLISM | GENETICS & HEREDITY | NORMAL-PRESSURE HYDROCEPHALUS | SCORING SYSTEM | SPINAL-CORD COMPRESSION | OF-THE-LITERATURE | Brain - diagnostic imaging | Postoperative Complications - etiology | Humans | Postoperative Complications - prevention & control | Brain - enzymology | Lysosomes - enzymology | Mucopolysaccharidoses - etiology | Mucopolysaccharidoses - pathology | Brain - metabolism | Nerve Compression Syndromes - etiology | Lysosomes - metabolism | Glycosaminoglycans - toxicity | Hydrocephalus - diagnostic imaging | Mucopolysaccharidoses - complications | Spinal Cord Compression - diagnostic imaging | Intraoperative Neurophysiological Monitoring - methods | Neuroimaging - methods | Nerve Compression Syndromes - surgery | Neurosurgical Procedures - adverse effects | Brain - cytology | Congresses as Topic | Glycosaminoglycans - metabolism | Hydrocephalus - etiology | Spinal Cord Compression - surgery | Treatment Outcome | Spinal Cord Compression - etiology | Nerve Compression Syndromes - diagnostic imaging | Hydrocephalus - surgery | Neurosurgical Procedures - methods | Enzymes | Medical colleges | Brain damage | Nervous system diseases | Glycosaminoglycans
Journal Article