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Molecular Neurobiology, ISSN 0893-7648, 5/2016, Volume 53, Issue 4, pp. 2451 - 2467
...). During transsulfuration pathways, Hcy is metabolized into hydrogen sulfide (H2S), which is a synaptic modulator, as well as a neuro-protective agent... 
Cerebrovascular pathology | Neurology | Neurosciences | Biomedicine | Blood–brain barrier dysfunction | Alzheimer’s disease | Neurobiology | Homocysteine | Cell Biology | Dementia | COGNITIVE PERFORMANCE | NEUROSCIENCES | MATRIX METALLOPROTEINASES | INDUCED MEMORY IMPAIRMENT | RISK-FACTOR | Blood-brain barrier dysfunction | RECEPTOR TRAFFICKING | MATRIX-METALLOPROTEINASE-9 | RAT-BRAIN | Alzheimer's disease | PLASMA HOMOCYSTEINE | EXPRESSION | Memory - drug effects | Glial Fibrillary Acidic Protein - genetics | Cadherins - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Blood-Brain Barrier - physiopathology | Claudin-5 - metabolism | Microvessels - pathology | Male | Synapses - pathology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Antigens, CD - genetics | Alzheimer Disease - pathology | Antigens, CD - metabolism | Matrix Metalloproteinase 9 - metabolism | Cadherins - genetics | Synapses - drug effects | Alzheimer Disease - physiopathology | Hydrogen Sulfide - pharmacology | Cerebrovascular Circulation - drug effects | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Alzheimer Disease - drug therapy | Microvessels - drug effects | Claudin-5 - genetics | Cystathionine beta-Synthase - metabolism | Permeability | Blood-Brain Barrier - drug effects | Nerve Tissue Proteins - genetics | Blood-Brain Barrier - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Blood-Brain Barrier - pathology | Hydrogen Sulfide - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Intercellular Adhesion Molecule-1 - genetics | Avoidance Learning - drug effects | Alzheimer Disease - genetics | Dizocilpine Maleate - pharmacology | Hydrogen sulfide | Methyl aspartate | Brain | RNA | Neurons | Intermediate filament proteins | cerebrovascular pathology | dementia | blood brain barrier dysfunction
Journal Article
British journal of pharmacology, ISSN 0007-1188, 2013, Volume 169, Issue 8, pp. 1795 - 1809
Background and Purpose Atherosclerosis is associated with reduced vascular hydrogen sulfide (H2S) biosynthesis... 
arteriosclerosis | hydrogen sulfide | inflammation | endothelial dysfunction | oxidative stress | ACTIVATION | ATHEROSCLEROSIS | ISCHEMIA | VASORELAXANT | ENDOTHELIAL-CELLS | PHARMACOLOGY & PHARMACY | GENERATION | HYPERTENSION | NF-KAPPA-B | RECEPTOR-1 | Tumor Necrosis Factor-alpha - metabolism | Humans | Male | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Aorta - metabolism | Nitric Oxide Synthase Type II | Organothiophosphorus Compounds - pharmacology | Foam Cells - drug effects | Diet, High-Fat | Superoxides - metabolism | Female | Apolipoproteins E | Phosphorylation - drug effects | Interleukin-6 - metabolism | Scavenger Receptors, Class E - drug effects | NF-KappaB Inhibitor alpha | Atherosclerosis - drug therapy | Atherosclerosis - chemically induced | Down-Regulation | Cells, Cultured | I-kappa B Proteins | Morpholines - pharmacology | Cholesterol - metabolism | Atherosclerosis - metabolism | Scavenger Receptors, Class E - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Macrophages - metabolism | Animals | Endothelium, Vascular - metabolism | Macrophages - drug effects | Mice | Oxidative Stress - drug effects | Vascular Cell Adhesion Molecule-1 - metabolism | Hydrogen sulfide | RNA | Low density lipoproteins | Atherosclerosis | Lectins | Physiological aspects | Exhibitions | Acetylcholine | Superoxide | Cholesterol | Phosphorylation | Rodents | Endothelium | Research Papers
Journal Article
Journal Article
Scientific reports, ISSN 2045-2322, 2016, Volume 6, Issue 1, pp. 20831 - 20831
Hydrogen sulfide is a highly toxic gas-second only to carbon monoxide as a cause of inhalational deaths... 
OXYGEN | COBALAMIN PRECURSOR COBINAMIDE | OXIDATIVE STRESS | INHIBITION | MULTIDISCIPLINARY SCIENCES | NITRIC-OXIDE | H2S | MODEL | CELLULAR BIOENERGETICS | DAMAGE | IPS CELLS | Mitochondria, Heart - metabolism | Sulfides - antagonists & inhibitors | Oxidative Stress | Humans | Male | Neurons - cytology | F2-Isoprostanes - metabolism | Mitochondria, Heart - drug effects | Electron Transport Complex IV - metabolism | Brain - metabolism | Cobamides - pharmacology | Sulfides - toxicity | F2-Isoprostanes - antagonists & inhibitors | Myocardium - metabolism | Potassium Cyanide - toxicity | Potassium Cyanide - antagonists & inhibitors | Cell Differentiation | Neurons - metabolism | Hydroxyl Radical - antagonists & inhibitors | Induced Pluripotent Stem Cells - cytology | Neurons - drug effects | Fibroblasts - metabolism | Induced Pluripotent Stem Cells - metabolism | Antidotes - pharmacology | Induced Pluripotent Stem Cells - drug effects | Hydrogen Sulfide - toxicity | Mice, Inbred C57BL | Rats | Hydroxyl Radical - metabolism | Brain - drug effects | Hydrogen Sulfide - antagonists & inhibitors | Animals | Fibroblasts - drug effects | Fibroblasts - cytology | Mice | Drosophila melanogaster | Apoptosis | Sulfide | Oxidative stress | Reactive oxygen species | Hydrogen | Toxicity | Vitamin B12 | Isoprostanes | Hydrogen sulfide | Mitochondria | Rodents | Stem cells | Hydroxyl radicals | Poisoning | Carbon monoxide | Respiration | Electron transport | Pluripotency | Index Medicus
Journal Article
Journal of neurochemistry, ISSN 1471-4159, 2010, Volume 113, Issue 1, pp. 14 - 26
...) are well established as messenger molecules throughout the body, gasotransmitters, based on striking alterations in mice lacking the appropriate biosynthetic enzymes. Hydrogen sulfide (H2S... 
cystathionase | S‐sulfhydration | EDRF | hydrogen sulfide | cystathionine γ‐lyase | cystathionine β‐synthase | Hydrogen sulfide | Cystathionine γ-lyase | Cystathionase | Cystathionine β-synthase | S-sulfhydration | cystathionine gamma-lyase | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEUROSCIENCES | MICE LACKING | SMOOTH-MUSCLE | HEME OXYGENASE | HYPERPOLARIZING FACTOR | CARBON-MONOXIDE | CHAIN FATTY-ACIDS | GAMMA-CYSTATHIONASE | AMINO-ACID OXIDASE | CYSTATHIONINE-BETA-SYNTHASE | cystathionine beta-synthase | Hydrogen Sulfide - metabolism | Hydrogen Sulfide - pharmacology | Endothelium-Dependent Relaxing Factors - pharmacology | Humans | Hydrogen Sulfide - chemistry | Models, Molecular | Cystathionine gamma-Lyase - metabolism | Endothelium, Vascular - drug effects | Hypertension - drug therapy | Cystathionine beta-Synthase - metabolism | Carbon Monoxide - metabolism | Mice, Knockout | Animals | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Signal Transduction - physiology | Blood Pressure - drug effects | Mice | Nitric Oxide - metabolism | Cystathionine gamma-Lyase - deficiency | Enzymes | Neurosciences | Carbon monoxide | Endothelium-derived relaxing factors | Proteins | Nitric oxide | Chemical reactions | Biochemistry | cystathionine γ-lyase | KATP | cystathionine β-synthase | hydropersulfide | EDHF | GAPDH
Journal Article