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Human molecular genetics, ISSN 1460-2083, 10/2016, Volume 25, Issue 20, pp. 4556 - 4565
Lipid traits (total, low-density and high-density lipoprotein cholesterol, and triglycerides) are risk factors for cardiovascular disease. DNA methylation is... 
Biochemistry & Molecular Biology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Carnitine O-Palmitoyltransferase - genetics | Cholesterol - blood | Epigenesis, Genetic | Humans | Amino Acid Transport System y+ - genetics | Male | Genetic Loci | ATP Binding Cassette Transporter, Subfamily G, Member 1 - metabolism | Cardiovascular Diseases - enzymology | Cardiovascular Diseases - genetics | Cholesterol - chemistry | ras GTPase-Activating Proteins - genetics | DNA Methylation | Amino Acid Transport System y+ - metabolism | ATP Binding Cassette Transporter, Subfamily G, Member 1 - genetics | Carnitine O-Palmitoyltransferase - metabolism | Female | Lipid Metabolism - genetics | Sterol Regulatory Element Binding Protein 2 - genetics | Sterol Regulatory Element Binding Protein 2 - metabolism | Sterol Regulatory Element Binding Protein 1 - metabolism | Genetic Predisposition to Disease | Phosphoric Monoester Hydrolases - genetics | ras GTPase-Activating Proteins - metabolism | Cardiovascular Diseases - metabolism | Genetic Association Studies | Ubiquitin-Protein Ligases - metabolism | Cholesterol - metabolism | Sequence Analysis, DNA | Triglycerides - metabolism | Carrier Proteins - genetics | Carrier Proteins - metabolism | Sterol Regulatory Element Binding Protein 1 - genetics | Triglycerides - blood | CpG Islands | Triglycerides - genetics | Phosphoric Monoester Hydrolases - metabolism | Ubiquitin-Protein Ligases - genetics | Index Medicus | Epigènesi | ADN
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 02/2017, Volume 114, Issue 9, pp. 2349 - 2354
.... The understanding of the molecular mechanisms underlying RV replication and pathogenesis has been hampered by the lack of an entirely plasmid-based reverse genetics system... 
SEE COMMENTARY | Vaccine | Reporter virus | Rotavirus | Reverse genetics | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Luciferases - metabolism | Cricetulus | Humans | Methyltransferases - metabolism | Reverse Genetics - methods | DNA, Complementary - genetics | Methyltransferases - genetics | Multienzyme Complexes - metabolism | Green Fluorescent Proteins - genetics | Orthoreovirus - metabolism | Vaccinia virus - genetics | Viral Proteins - metabolism | Luciferases - genetics | Base Sequence | Nucleotidyltransferases - metabolism | Genes, Reporter | Cell Line | Green Fluorescent Proteins - metabolism | Phosphoric Monoester Hydrolases - genetics | Transduction, Genetic | Vaccinia virus - metabolism | Orthoreovirus - genetics | Orthoreovirus, Mammalian - genetics | Gene Expression Regulation | Viral Nonstructural Proteins - genetics | Viral Proteins - genetics | DNA-Directed RNA Polymerases - genetics | Multienzyme Complexes - genetics | Plasmids - metabolism | Animals | Epithelial Cells - virology | Plasmids - chemistry | Cell Line, Tumor | Nucleotidyltransferases - genetics | Viral Nonstructural Proteins - metabolism | DNA-Directed RNA Polymerases - metabolism | Phosphoric Monoester Hydrolases - metabolism | DNA, Complementary - metabolism | Orthoreovirus, Mammalian - metabolism | Genetic aspects | Gene expression | Health aspects | Rotaviruses | Viruses | Genetics | Microbiology | Pathogenesis | Plasmids | Medical immunity | Index Medicus | reporter virus | Biological Sciences | reverse genetics | vaccine | rotavirus
Journal Article
PloS one, ISSN 1932-6203, 06/2017, Volume 12, Issue 6, pp. e0178925 - e0178925
We previously showed that p21Cip1 transits through the nucleolus on its way from the nucleus to the cytoplasm and that DNA damage inhibits this transit and... 
Proto-Oncogene Proteins c-mdm2 - genetics | Humans | Protein Multimerization | Cyclin E - genetics | Cyclin-Dependent Kinase Inhibitor p21 - deficiency | Tumor Suppressor Protein p53 - genetics | Organelle Biogenesis | Organelles - genetics | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cysteine Endopeptidases - metabolism | Proliferating Cell Nuclear Antigen - genetics | SUMO-1 Protein - genetics | Cell Nucleolus - metabolism | Promyelocytic Leukemia Protein - metabolism | Proto-Oncogene Proteins c-mdm2 - metabolism | Promyelocytic Leukemia Protein - genetics | Cyclin-Dependent Kinase 2 - metabolism | Cell Nucleolus - genetics | Signal Transduction | HCT116 Cells | Gene Expression Regulation | Tumor Suppressor Protein p53 - metabolism | Cyclin-Dependent Kinase 2 - genetics | Ubiquitin-Conjugating Enzymes - genetics | Receptors, Cytoplasmic and Nuclear - genetics | Protein Transport | Karyopherins - metabolism | Cysteine Endopeptidases - genetics | Karyopherins - genetics | Protein Binding | SUMO-1 Protein - metabolism | Cyclin E - metabolism | DNA Damage | Proliferating Cell Nuclear Antigen - metabolism | Organelles - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Ubiquitin-Conjugating Enzymes - deficiency | GTP-binding protein | Regulators | Complex formation | p53 Protein | DNA damage | Biosynthesis | Cancer therapies | Nuclei | Accumulation | Proteins | Nucleoli | Cell cycle | Proliferating cell nuclear antigen | Cyclin E | Protein transport | Localization | Damage accumulation | Deoxyribonucleic acid--DNA | Translocation | Enzymes | MDM2 protein | Hydrolase | Cyclin-dependent kinase 2 | Cyclin-dependent kinase inhibitor p21 | Depletion | Microscopy | Disruption | Intracellular | Cytoplasm | Impact damage | Apoptosis | Index Medicus | Biosíntesi | Biologia molecular | RNA | Citoplasma | Ribosomes | Mutació (Biologia) | Molecular biology | Mutation (Biology) | Deoxyribonucleic acid | DNA
Journal Article
Development, ISSN 0950-1991, 06/2008, Volume 135, Issue 11, pp. 2013 - 2022
The COP9 signalosome (CSN) is required for the full activity of cullin-RING E3 ubiquitin ligases (CRLs) in eukaryotes. CSN exerts its function on CRLs by... 
COP9 signalosome | Cell cycle | DNA Damage | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Protein Kinases - metabolism | Cell Cycle - genetics | Genomic Instability | G2 Phase - genetics | Protein Kinases - genetics | Oligonucleotide Array Sequence Analysis | Peptide Hydrolases - genetics | Ubiquitins - genetics | Plant Roots - genetics | Seedlings - genetics | Immunoblotting | Multiprotein Complexes - genetics | Seedlings - growth & development | Arabidopsis Proteins - metabolism | Multiprotein Complexes - metabolism | Cyclin B - genetics | Flow Cytometry | ATP-Binding Cassette Transporters - genetics | Gene Expression Regulation, Developmental | Gene Expression Regulation, Plant | ATP-Binding Cassette Transporters - metabolism | Cullin Proteins - metabolism | Nuclear Proteins - genetics | Plant Roots - growth & development | Ubiquitins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Peptide Hydrolases - metabolism | Cell Division - genetics | In Situ Nick-End Labeling | Arabidopsis Proteins - genetics | G2 Phase - physiology | Plant Roots - metabolism | Protein-Serine-Threonine Kinases - genetics | Ubiquitin-Protein Ligases - metabolism | COP9 Signalosome Complex | Nuclear Proteins - metabolism | Cell Division - physiology | Cullin Proteins - genetics | Cell Cycle - physiology | Cyclin B - metabolism | Ubiquitin-Protein Ligases - genetics | Seedlings - metabolism | Index Medicus
Journal Article
Journal Article
Nature, ISSN 0028-0836, 07/2010, Volume 466, Issue 7303, pp. 243 - 247