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1. Full Text Bioaccumulation and biotransformation of brominated and chlorinated contaminants and their metabolites in ringed seals ( Pusa hispida) and polar bears ( Ursus maritimus) from East Greenland
by Letcher, Robert J and Gebbink, Wouter A and Sonne, Christian and Born, Erik W and McKinney, Melissa A and Dietz, Rune
Environment International, ISSN 0160-4120, 2009, Volume 35, Issue 8, pp. 1118 - 1124
We report on the comparative bioaccumulation, biotransformation and/or biomagnification from East Greenland ringed seal ( ) blubber to polar bear ( ) tissues... 
East Greenland | Metabolites | Ringed seals | Bioaccumulation | Brominated and chlorinated contaminants | Polar bears | Biomagnification | POLYBROMINATED DIPHENYL ETHERS | HYDROCARBON CONTAMINANTS | ENVIRONMENTAL SCIENCES | BALTIC SEA | MARINE-ENVIRONMENT | METHYL SULFONE PCB | FLAME RETARDANTS | HYDROXYLATED METABOLITES | MONOOXYGENASE ACTIVITIES | POLYCHLORINATED-BIPHENYLS | ADIPOSE-TISSUE | Hydrocarbons, Brominated - metabolism | Hydrocarbons, Chlorinated - metabolism | Environmental Monitoring | Liver - metabolism | Phoca - metabolism | Pentachlorophenol - metabolism | Male | Environmental Exposure | Environmental Pollutants - metabolism | Polychlorinated Biphenyls - metabolism | Brain - metabolism | Adipose Tissue - metabolism | Halogenated Diphenyl Ethers - metabolism | Animals | Insecticides - metabolism | Dichlorodiphenyl Dichloroethylene - metabolism | Ursidae - metabolism | Biotransformation | Flame Retardants - metabolism | Polybrominated Biphenyls - metabolism | Female | Greenland | Natural resources | Polar bear | Hydroxides | Fireproofing agents | Polybrominated biphenyls | Polychlorinated biphenyls | Monomers | Brain | Bromination | Contaminants | Seals | Bears | Index Medicus
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2. Full Text Widespread and enzyme-independent Nε-acetylation and Nε-succinylation of proteins in the chemical conditions of the mitochondrial matrix
by Wagner, Gregory R and Payne, R. Mark
Journal of Biological Chemistry, ISSN 0021-9258, 10/2013, Volume 288, Issue 40, pp. 29036 - 29045
Alterations in mitochondrial protein acetylation are implicated in the pathophysiology of diabetes, the metabolic syndrome, mitochondrial disorders, and... 
Amino Acid Sequence | Sirtuin 3 - metabolism | Mice, Inbred C57BL | Computational Biology | Molecular Sequence Data | Mitochondria - metabolism | Coenzyme A - metabolism | Hydroxides - metabolism | Animals | Mitochondrial Proteins - metabolism | Mitochondrial Proteins - chemistry | Succinates - metabolism | Mice | Acetylation | Catalysis | Kinetics | Hydrogen-Ion Concentration | Index Medicus | Sirtuins | pH Regulation | Metabolic Regulation | Metabolic Diseases | Succinyl Coenzyme A | Succinylation | Metabolism | Acetyl Coenzyme A | Mitochondrial Metabolism | Nonenzymatic
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3. Full Text The immunobiology of aluminium adjuvants: how do they really work?
by Exley, Christopher and Siesjö, Peter and Eriksson, Håkan and Neurosurgery and Neurokirurgi and Lund University and Lunds universitet
Trends in Immunology, ISSN 1471-4906, 2010, Volume 31, Issue 3, pp. 103 - 109
Aluminium adjuvants potentiate the immune response, thereby ensuring the potency and efficacy of typically sparingly available antigen. Their concomitant... 
Allergy and Immunology | IMMUNE-RESPONSE | CYTOKINE PRODUCTION | HYDROXIDE ADJUVANT | MONOCLONAL-ANTIBODY | IMMUNOLOGY | VACCINE ADJUVANTS | NF-KAPPA-B | DRINKING-WATER | HUMAN BRAIN-CELLS | BIOLOGICAL-SYSTEMS | NALP3 INFLAMMASOME | Adjuvants, Immunologic - pharmacology | Aluminum Compounds - adverse effects | Reactive Oxygen Species - metabolism | Humans | Muscle, Skeletal - metabolism | Magnesium Compounds - immunology | Vaccines - metabolism | Adjuvants, Immunologic - adverse effects | T-Lymphocytes - drug effects | Adenosine Triphosphate - metabolism | Inflammation Mediators - metabolism | Aluminum Hydroxide - pharmacology | Adaptive Immunity - immunology | Phosphates - pharmacology | Magnesium Compounds - pharmacology | Phosphates - immunology | Aluminum Hydroxide - metabolism | Antigen-Presenting Cells - metabolism | Immunity, Innate - drug effects | Oxidation-Reduction | Phagocytes - drug effects | Antigen-Presenting Cells - drug effects | Vaccines - immunology | Inflammation - immunology | Aluminum Compounds - immunology | Phagocytes - metabolism | Phagocytes - immunology | Antigen-Presenting Cells - immunology | Inflammation - etiology | Aluminum Compounds - metabolism | Magnesium Compounds - metabolism | B-Lymphocytes - drug effects | Immunity, Innate - immunology | Extracellular Fluid - metabolism | Models, Immunological | Phosphates - metabolism | Adaptive Immunity - drug effects | Aluminum Hydroxide - adverse effects | Animals | B-Lymphocytes - immunology | Phosphates - adverse effects | Aluminum Hydroxide - immunology | T-Lymphocytes - immunology | Aluminum Compounds - pharmacology | Ligands | Immunization | Vaccines | Fluorides | Molecular weight | Index Medicus | Clinical Medicine | Neurologi | Neurology | Medical and Health Sciences | Medicin och hälsovetenskap | Surgery | Klinisk medicin | Kirurgi
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4. Full Text BENAZEPRIL, AN ANGIOTENSIN-CONVERTING ENZYME INHIBITOR, ALLEVIATES RENAL INJURY IN SPONTANEOUSLY HYPERTENSIVE RATS BY INHIBITING ADVANCED GLYCATION END-PRODUCT-MEDIATED PATHWAYS
by Liu, Xue-Ping and Pang, Yue-Jiu and Zhu, Wei-Wei and Zhao, Ting-Ting and Zheng, Min and Wang, Yi-Bing and Sun, Zhi-Jian and Sun, Siao-Jing
Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 03/2009, Volume 36, Issue 3, pp. 287 - 296
SUMMARY • Advanced glycation end-products (AGE) and their receptors (RAGE) have been implicated in renal damage in diabetes. The aim of the present study was... 
advanced glycation end-products | benazepril | rats | spontaneous hypertension | renal injury | Renal injury | Rats | Benazepril | Advanced glycation end-products | Spontaneous hypertension | SYSTEM | PHYSIOLOGY | RENIN | ACE-INHIBITOR | FACTOR-KAPPA-B | II/III IMMUNOREACTIVITY | DAMAGE | PATHOGENESIS | IN-VITRO | RECEPTOR RAGE | PHARMACOLOGY & PHARMACY | EXPRESSION | Kidney Diseases - physiopathology | Kidney Cortex - drug effects | Antihypertensive Agents - pharmacology | Phosphorylation | Reactive Oxygen Species - metabolism | Kidney Cortex - metabolism | Transforming Growth Factor beta1 - metabolism | NADPH Oxidases - metabolism | Rats, Inbred WKY | Hypertension - drug therapy | Male | Proteinuria - prevention & control | Female | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Blood Pressure - drug effects | Receptor for Advanced Glycation End Products | Disease Models, Animal | Kidney Diseases - metabolism | Rats, Inbred SHR | Angiotensin II - metabolism | Kidney Diseases - prevention & control | Proteinuria - metabolism | Kidney Cortex - pathology | Benzazepines - pharmacology | Hypertension - physiopathology | Hypertension - metabolism | Platelet-Derived Growth Factor - metabolism | Animals | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Glycation End Products, Advanced - metabolism | Hypertension - complications | Kidney Diseases - etiology | Oxidative Stress - drug effects | Vascular Cell Adhesion Molecule-1 - metabolism | Kidney Cortex - enzymology | Receptors, Immunologic - metabolism | Oxidative Stress, drug effects | Transforming Growth Factor beta1, metabolism | Hypertension, drug therapy | Signal Transduction, drug effects | Kidney Cortex, enzymology | Receptors, Immunologic, metabolism | Hypertension, physiopathology | Vascular Cell Adhesion Molecule-1, metabolism | Kidney Cortex, drug effects | Kidney Cortex, metabolism | Blood Pressure, drug effects | Glycosylation End Products, Advanced, metabolism | Proteinuria, metabolism | Angiotensin-Converting Enzyme Inhibitors, pharmacology | Angiotensin II, metabolism | Kidney Diseases, physiopathology | Hypertension, metabolism | Proteinuria, prevention and control | Platelet-Derived Growth Factor, metabolism | Antihypertensive Agents, pharmacology | Kidney Diseases, etiology | Benzazepines, pharmacology | Transcription Factor RelA, metabolism | NADPH Oxidase, metabolism | Hypertension, complications | Kidney Diseases, prevention and control | Kidney Diseases, metabolism | Kidney Cortex, pathology | Reactive Oxygen Species, metabolism | Immunohistochemistry | Oxidases | Hypertension | Hydroxides | Enzyme inhibitors | Angiotensin | Kidney diseases | Diabetes | Peptide hormones | Transforming growth factors | Index Medicus
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5. Full Text Oxygen activation at the plasma membrane: relation between superoxide and hydroxyl radical production by isolated membranes
by Eiri Heyno and Véronique Mary and Peter Schopfer and Anja Krieger-Liszkay
Planta, ISSN 0032-0935, 7/2011, Volume 234, Issue 1, pp. 35 - 45
Production of reactive oxygen species (hydroxyl radicals, superoxide radicals and hydrogen peroxide) was studied using EPR spin-trapping techniques and... 
Oxidases | Enzymes | Soybeans | Hypocotyls | Superoxides | Corn | Hydroxyl radicals | Cell membranes | Plants | Seedlings | Life Sciences | Reactive oxygen species | Electron paramagnetic resonance spectroscopy | Forestry | Zea | Ecology | Agriculture | Quinone reductase | Glycine | Peroxidase, plasma membrane | Plant Sciences | NAD(P)H oxidase | SYSTEM | NADPH OXIDASE | PEROXIDASE-ACTIVITIES | NEUTROPHILS | ELONGATION GROWTH | PLANT SCIENCES | EXTENSION GROWTH | MAYS L. ROOTS | GENERATION | INHIBITOR | BINDING | Plant Roots - metabolism | Reactive Oxygen Species - metabolism | NADPH Oxidases - metabolism | Hydroxyl Radical - metabolism | Cotyledon - metabolism | Soybeans - metabolism | Oxygen - metabolism | Hydrogen Peroxide - metabolism | Cell Membrane - enzymology | Superoxides - metabolism | Cell Membrane - metabolism | Hypocotyl - metabolism | Zea mays - metabolism | Peroxidase - metabolism | Hydrogen peroxide | Hydroxides | Quinone | Peroxidase | Superoxide | Index Medicus
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6. Full Text Synthesis and antioxidant capacities of hydroxyl derivatives of cinnamoylphenethylamine in protecting DNA and scavenging radicals
by Yang, Yang and Song, Zhi-Guang and Liu, Zai-Qun
Free Radical Research, ISSN 1071-5762, 04/2011, Volume 45, Issue 4, pp. 445 - 453
Abstract Cinnamoylphenethylamine (CNPA) derivatives including feruloylphenethylamine (FRPA), caffeoylphenethylamine (CFPA), cinnamoyltyramine (CNTA),... 
oxidation | Ceratostigma willmottianum | antioxidant | Cinnamoylphenethylamine | free radical | DNA | SECONDARY METABOLITES | CELLS | ACID-AMIDES | ANALOGS | BIOCHEMISTRY & MOLECULAR BIOLOGY | COUMAROYLTYRAMINE | QUANTITATION | STEMS | ROOTS | CONSTITUENTS | Glutathione - metabolism | Linoleic Acid - metabolism | Antioxidants - metabolism | Structure-Activity Relationship | Biphenyl Compounds - metabolism | Amidines - metabolism | Solutions - chemistry | Hydroxides - metabolism | Copper - chemistry | Copper - metabolism | Oxidation-Reduction - drug effects | Cinnamates - chemical synthesis | Cinnamates - metabolism | Free Radical Scavengers - chemical synthesis | Benzothiazoles - metabolism | Free Radical Scavengers - metabolism | Solutions - metabolism | Picrates - metabolism | Phenethylamines - metabolism | Antioxidants - chemical synthesis | DNA - metabolism | Sulfonic Acids - metabolism | beta Carotene - metabolism | Phenethylamines - chemical synthesis | Benzhydryl Compounds - metabolism | Index Medicus
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7. Full Text Calcium‐ and potassium‐permeable plasma membrane transporters are activated by copper in Arabidopsis root tips: linking copper transport with cytosolic hydroxyl radical production
by RODRIGO‐MORENO, ANA and ANDRÉS‐COLÁS, NURIA and POSCHENRIEDER, CHARLOTTE and GUNSÉ, BENET and PEÑARRUBIA, LOLA and SHABALA, SERGEY
Plant, Cell & Environment, ISSN 0140-7791, 04/2013, Volume 36, Issue 4, pp. 844 - 855
Transition metals such as copper can interact with ascorbate or hydrogen peroxide to form highly reactive hydroxyl radicals (OH • ), with numerous implications... 
Arabidopsis thaliana | toxicity | COPT1 | oxidative stress | Oxidative stress | Toxicity | HOMEOSTASIS | OXYGEN RADICALS | CELL-DEATH | PLANT SCIENCES | SIGNAL-TRANSDUCTION | CATION CHANNELS | EFFLUX | Ca2 | THALIANA | PROPIDIUM IODIDE | Potassium - metabolism | Meristem - cytology | Arabidopsis - physiology | Oxidative Stress | Calcium - metabolism | Meristem - physiology | Homeostasis | Plant Epidermis - metabolism | Arabidopsis Proteins - metabolism | Dose-Response Relationship, Drug | Biological Transport | Membrane Transport Proteins - genetics | Copper - metabolism | Meristem - metabolism | Plant Epidermis - cytology | Membrane Transport Proteins - metabolism | Cell Membrane - metabolism | Meristem - drug effects | Copper - toxicity | Plant Epidermis - physiology | Arabidopsis Proteins - genetics | Arabidopsis - drug effects | Cell Survival | Seedlings - cytology | Arabidopsis - cytology | Peroxides - analysis | Hydroxyl Radical - metabolism | Plant Epidermis - drug effects | Seedlings - drug effects | Gene Knockout Techniques | Verapamil - pharmacology | Arabidopsis - metabolism | Phenotype | Seedlings - physiology | Models, Biological | Cytosol - metabolism | Mutagenesis, Insertional | Seedlings - metabolism | Hydroxides | Hydrogen peroxide | Copper industry | Cell membranes | Erythromycin | Plants | Index Medicus
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8. Full Text Moving Fe2+from ferritin ion channels to catalytic OH centers depends on conserved protein cage carboxylates
by Rabindra K. Behera and Elizabeth C. Theil
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2014, Volume 111, Issue 22, pp. 7925 - 7930
Ferritin biominerals are protein-caged metabolic iron concentrates used for iron–protein cofactors and oxidant protection (Fe2+ and O2 sequestration). Fe2+... 
Enzymes | Biomineralogy | Active sites | Ions | Ion channels | Minerals | Ferritins | Carboxylates | Enzyme substrates | Metal ions | Ferrihydrite | Iron traffic | Oxidoreductase enzyme activity | Antioxidant | BioIron | antioxidant | H-CHAIN FERRITIN | MULTIDISCIPLINARY SCIENCES | FERROXIDASE SITE | IRON | IDENTIFICATION | PORES | TRANSPORT | ferrihydrite | MINERALIZATION | CHEMISTRY | INTERMEDIATE | APOFERRITIN | oxidoreductase enzyme activity | iron traffic | Heme - metabolism | Antioxidants - chemistry | Models, Chemical | Antioxidants - metabolism | Substrate Specificity | Ferric Compounds - chemistry | Oxidoreductases - chemistry | Hydroxides - metabolism | Ferritins - metabolism | Heme - chemistry | Metals - chemistry | Ferritins - genetics | Sulfates - chemistry | Conserved Sequence | Minerals - chemistry | Catalysis | Minerals - metabolism | Ion Channels - chemistry | Protein Structure, Tertiary | Hydroxides - chemistry | Mutagenesis, Site-Directed | Sulfates - metabolism | Ferric Compounds - metabolism | Oxidoreductases - metabolism | Protein Structure, Secondary | Iron - chemistry | Iron - metabolism | Anura | Metals - metabolism | Animals | Ion Channels - metabolism | Ferritins - chemistry | Index Medicus | Biological Sciences | Physical Sciences
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9. Full Text Astrocytes produce the antiinflammatory and neuroprotective agent hydrogen sulfide
by Lee, Moonhee and Schwab, Claudia and Yu, Sheng and McGeer, Edith and McGeer, Patrick L
Neurobiology of Aging, ISSN 0197-4580, 2009, Volume 30, Issue 10, pp. 1523 - 1534
Abstract Hydrogen sulfide (H2 S) is an essential physiological product in brain. We investigated the expression of cystathionine-β-synthase (CBS) and... 
Neurology | Internal Medicine | Alzheimer disease | Parkinson disease | Aging | Neuroinflammation | NaSH | Cystathionine-β-synthase | Microglia | CELLS | ALZHEIMERS-DISEASE BRAIN | OXIDATIVE STRESS | MOUSE | H2S | CYSTATHIONINE BETA-SYNTHASE | NEUROMODULATOR | TOXICITY | NEUROSCIENCES | Cystathionine-beta-synthase | GERIATRICS & GERONTOLOGY | VASORELAXANT | Hydrogen Sulfide - metabolism | Tumor Necrosis Factor-alpha - metabolism | Glycine - analogs & derivatives | Microglia - metabolism | Cystathionine beta-Synthase - antagonists & inhibitors | Humans | Brain - enzymology | Nitrites - metabolism | Cystathionine gamma-Lyase - metabolism | Male | NF-kappa B - metabolism | Astrocytes - enzymology | Neuroprotective Agents - metabolism | Brain - metabolism | Microglia - immunology | Astrocytes - immunology | Aged, 80 and over | Alkynes - pharmacology | Female | Interleukin-6 - metabolism | Hydroxylamine - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Microglia - enzymology | Cystathionine beta-Synthase - metabolism | Cystathionine gamma-Lyase - antagonists & inhibitors | Glycine - pharmacology | Cell Line, Tumor | Aged | Brain - immunology | Astrocytes - metabolism | Hydrogen sulfide | Enzymes | Nervous system diseases | Hydroxides | Parkinson's disease | Anti-inflammatory drugs | Nitrites | Physiological aspects | Alzheimer's disease | Index Medicus
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10. Full Text Sugars as hydroxyl radical scavengers: proof‐of‐concept by studying the fate of sucralose in Arabidopsis
by Matros, Andrea and Peshev, Darin and Peukert, Manuela and Mock, Hans‐Peter and Van den Ende, Wim
The Plant Journal, ISSN 0960-7412, 06/2015, Volume 82, Issue 5, pp. 822 - 839
Substantial formation of reactive oxygen species ( ROS ) is inevitable in aerobic life forms. Due to their extremely high reactivity and short lifetime,... 
Arabidopsis thaliana | stress | hydroxyl radical | sucralose | ROS | membrane protection | sugar | COLD TOLERANCE | RAFFINOSE | MECHANISMS | PLANT SCIENCES | SUCROSE METABOLISM | IN-VITRO | GENE | FRUCTANS | PLANTS | ACCUMULATION | OXIDATIVE STRESS TOLERANCE | Sucrose - chemistry | Reactive Oxygen Species - metabolism | Free Radical Scavengers - metabolism | Oxidation-Reduction | Oxidative Stress | Hordeum - metabolism | Sucrose - analogs & derivatives | Hydroxyl Radical - metabolism | Sucrose - pharmacokinetics | Arabidopsis - metabolism | Arabidopsis Proteins - metabolism | Tissue Distribution | Tandem Mass Spectrometry | Plant Leaves - metabolism | Hydroxyl Radical - chemistry | Sucrose - metabolism | Physiological aspects | Sucralose | Enzymes | Hydrogen peroxide | Hydroxides | Metabolism | Sugar | Botany | Index Medicus
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11. Full Text Sodic alkaline stress mitigation by interaction of nitric oxide and polyamines involves antioxidants and physiological strategies in Solanum lycopersicum
by Gong, Biao and Li, Yan and Li, Xiu and Bloszies, Sean and Wen, Dan and Sun, Shasha and Wei, Min and Yang, Fengjuan and Shi, Qinghua and Wang, Xiufeng
Free Radical Biology and Medicine, ISSN 0891-5849, 06/2014, Volume 71, pp. 36 - 48
Nitric oxide (NO) and polyamines (PAs) are two kinds of important signal in mediating plant tolerance to abiotic stress. In this study, we observed that both... 
Oxidative stress | Free radicals | Alleviating effects | Nitric oxide | Tomato | Polyamines | Alkaline stress | ABIOTIC STRESS | CHLOROPHYLL FLUORESCENCE | SEEDLINGS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SALT TOLERANCE | METABOLISM | ENDOCRINOLOGY & METABOLISM | PLANTS | ARABIDOPSIS | CUCUMIS-SATIVUS | Adaptation, Physiological | Reactive Oxygen Species - metabolism | Calcium - metabolism | Antioxidants - metabolism | Polyamines - antagonists & inhibitors | Stress, Physiological | Plant Roots - genetics | Seedlings - genetics | Sodium Hydroxide - pharmacology | Plant Roots - drug effects | Sodium-Hydrogen Exchangers - metabolism | Biological Transport | Gene Expression Regulation, Plant | Lycopersicon esculentum - genetics | Sodium-Hydrogen Exchangers - genetics | Lycopersicon esculentum - drug effects | Lycopersicon esculentum - metabolism | Plant Roots - metabolism | Signal Transduction | Nitric Oxide - antagonists & inhibitors | Imidazoles - pharmacology | Seedlings - drug effects | Polyamines - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Mitoguazone - pharmacology | Hydroponics | Benzoates - pharmacology | Mitogen-Activated Protein Kinases - genetics | Nitric Oxide - metabolism | Mitogen-Activated Protein Kinases - metabolism | Seedlings - metabolism | Antioxidants | Analysis | Physiological aspects | Stress (Physiology) | Tomatoes | Photosynthesis | Index Medicus
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