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Scientific reports, ISSN 2045-2322, 02/2016, Volume 6, Issue 1, pp. 20831 - 20831
Hydrogen sulfide is a highly toxic gas-second only to carbon monoxide as a cause of inhalational deaths. Its mechanism of toxicity is only partially known, and... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Mitochondria, Heart - metabolism | Sulfides - antagonists & inhibitors | Oxidative Stress | Humans | Male | Neurons - cytology | F2-Isoprostanes - metabolism | Mitochondria, Heart - drug effects | Electron Transport Complex IV - metabolism | Brain - metabolism | Cobamides - pharmacology | Sulfides - toxicity | F2-Isoprostanes - antagonists & inhibitors | Myocardium - metabolism | Potassium Cyanide - toxicity | Potassium Cyanide - antagonists & inhibitors | Cell Differentiation | Neurons - metabolism | Hydroxyl Radical - antagonists & inhibitors | Induced Pluripotent Stem Cells - cytology | Neurons - drug effects | Fibroblasts - metabolism | Induced Pluripotent Stem Cells - metabolism | Antidotes - pharmacology | Induced Pluripotent Stem Cells - drug effects | Hydrogen Sulfide - toxicity | Mice, Inbred C57BL | Rats | Hydroxyl Radical - metabolism | Brain - drug effects | Hydrogen Sulfide - antagonists & inhibitors | Animals | Fibroblasts - drug effects | Fibroblasts - cytology | Mice | Drosophila melanogaster | Apoptosis | Sulfide | Oxidative stress | Reactive oxygen species | Hydrogen | Toxicity | Vitamin B12 | Isoprostanes | Hydrogen sulfide | Mitochondria | Rodents | Stem cells | Hydroxyl radicals | Poisoning | Carbon monoxide | Respiration | Electron transport | Pluripotency | Index Medicus
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 11/2011, Volume 334, Issue 6058, pp. 982 - 986
Bacteria become highly tolerant to antibiotics when nutrients are limited. The inactivity of antibiotic targets caused by starvation-induced growth arrest is... 
Starvation | Cell growth | Biofilms | Antibiotics | Microbiology | Cell death | DNA | REPORTS | Bacteria | Infections | Overproduction | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Oxidative Stress | Escherichia coli - drug effects | Pseudomonas aeruginosa - growth & development | Biofilms - growth & development | Drug Resistance, Bacterial | Ofloxacin - therapeutic use | Pseudomonas aeruginosa - physiology | Anti-Bacterial Agents - therapeutic use | Ofloxacin - pharmacology | Female | Escherichia coli - growth & development | Escherichia coli - physiology | Superoxide Dismutase - metabolism | Biofilms - drug effects | Pseudomonas Infections - drug therapy | Serine - analogs & derivatives | Mice, Inbred C57BL | Drug Tolerance | Hydroxyquinolines - metabolism | Hydroxyl Radical - metabolism | Pseudomonas aeruginosa - drug effects | Pseudomonas Infections - microbiology | Catalase - metabolism | Animals | Pseudomonas aeruginosa - genetics | Escherichia coli - genetics | Serine - pharmacology | Anti-Bacterial Agents - pharmacology | Mice | Mutation | Physiological aspects | Dosage and administration | Bacterial infections | Drug therapy | Microbial mats | Nutrients | Index Medicus | Antibiotic tolerance | Biotechnology | Serine | Escherichia coli | Ofloxacin | Hydroxyquinolines | Bacteriology | Life Sciences | Hydroxyl Radical | Microbiology and Parasitology | Anti-Bacterial Agents | Catalase | Computer Science | Pseudomonas aeruginosa | Pseudomonas Infections | Superoxide Dismutase
Journal Article
Angewandte Chemie (International ed.), ISSN 1433-7851, 01/2019, Volume 58, Issue 4, pp. 946 - 956
Tailored to the specific tumour microenvironment, which involves acidity and the overproduction of hydrogen peroxide, advanced nanotechnology has been... 
Fenton reaction | reaction environment | external energy fields | nanomaterials selection | chemodynamic therapy | Physical Sciences | Chemistry | Chemistry, Multidisciplinary | Science & Technology | Therapy | Hydrogen peroxide | Hydroxyl radicals | Nanomaterials | Acidity | Nanotechnology | Tumors | Index Medicus
Journal Article
European journal of pharmacology, ISSN 0014-2999, 01/2012, Volume 675, Issue 1-3, pp. 6 - 14
Reactive oxygen species production is necessary to induce cell death following hypoxia/reoxygenation but the effect of reactive oxygen species produced during... 
Mitochondria | Reactive oxygen species | Permeability transition pore | Adult cardiomyocytes | Hypoxia/reoxygenation | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Reactive Oxygen Species - metabolism | Mitochondrial Membrane Transport Proteins - antagonists & inhibitors | Rats, Wistar | Cyclosporine - pharmacology | Male | Reactive Oxygen Species - agonists | Cell Hypoxia | Membrane Transport Modulators - pharmacology | Hydroxyl Radical - agonists | Superoxides - metabolism | Superoxides - antagonists & inhibitors | Cell Death - drug effects | Hydroxyl Radical - antagonists & inhibitors | Cell Survival - drug effects | Mitochondrial Membrane Transport Proteins - metabolism | Superoxides - agonists | Cells, Cultured | Rats | Hydroxyl Radical - metabolism | Antioxidants - pharmacology | Hydrogen Peroxide - metabolism | Myocardial Reperfusion Injury - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Animals | Myocytes, Cardiac - drug effects | Hydrogen Peroxide - agonists | Mitochondrial Membrane Transport Proteins - agonists | Myocytes, Cardiac - metabolism | Kinetics | Oxidative Stress - drug effects | Hydrogen Peroxide - antagonists & inhibitors | Myocardial Reperfusion Injury - prevention & control | Hydrogen peroxide | Cell death | Analysis | Heart cells | Biochemistry | Mitochondrial DNA | Superoxide | Permeability | Index Medicus | Myocytes, Cardiac | Reactive Oxygen Species | Oxidative Stress | Cell Survival | Hydrogen Peroxide | Biochemistry, Molecular Biology | Cyclosporine | Membrane Transport Modulators | Antioxidants | Life Sciences | Mitochondrial Membrane Transport Proteins | Hydroxyl Radical | Superoxides | Cell Death | Myocardial Reperfusion Injury
Journal Article
Chemical communications (Cambridge, England), ISSN 1359-7345, 05/2014, Volume 50, Issue 37, pp. 4843 - 4845
Journal Article
Journal Article