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Hypertension, ISSN 0194-911X, 11/2010, Volume 56, Issue 5, pp. 885 - 892
Journal Article
Journal Article
Science, ISSN 0036-8075, 2/2011, Volume 331, Issue 6018, pp. 768 - 772
Endocrine tumors such as aldosterone-producing adrenal adenomas (APAs), a cause of severe hypertension, feature constitutive hormone production and... 
Depolarization | Hypertension | Somatic mutation | Cell growth | Hyperaldosteronism | REPORTS | Loss of heterozygosity | Adenoma | Genetic mutation | Sequencing | Tumors | REMEDIABLE ALDOSTERONISM | HUMAN BREAST | COLORECTAL CANCERS | ADRENOCORTICAL-CELLS | MULTIDISCIPLINARY SCIENCES | CONDUCTION | GLOMERULOSA CELLS | IDENTIFICATION | MOLECULAR-BASIS | SELECTIVITY | POTASSIUM CHANNEL | Adrenal Glands - pathology | Potassium - metabolism | Cell Proliferation | Humans | Hyperaldosteronism - pathology | Hyperplasia | Protein Multimerization | Zona Glomerulosa - pathology | Male | Hyperaldosteronism - metabolism | Sodium - metabolism | Aldosterone - metabolism | Adrenocortical Adenoma - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Female | Hypertension - genetics | Adrenal Cortex Neoplasms - metabolism | Cell Line | G Protein-Coupled Inwardly-Rectifying Potassium Channels - chemistry | Hyperaldosteronism - genetics | Mutant Proteins - genetics | Mutant Proteins - metabolism | Zona Glomerulosa - metabolism | Adrenal Cortex Neoplasms - pathology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | Adrenocortical Adenoma - pathology | Adrenocortical Adenoma - metabolism | Hypertension - metabolism | Adrenal Cortex Neoplasms - genetics | Mutant Proteins - chemistry | Mutation | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
Journal Article
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 06/2017, Volume 102, Issue 6, pp. 1792 - 1796
Journal Article
Journal Article
Journal of Molecular Endocrinology, ISSN 0952-5041, 07/2017, Volume 59, Issue 1, pp. R47 - R63
Primary aldosteronism (PA), the most common form of secondary hypertension, is caused in the majority of cases by unilateral aldosterone-producing adenoma... 
Calcium channels | Familial hyperaldosteronism | Primary aldosteronism | Somatic mutations | Aldosterone-producing adenoma | ATPase | Germline mutations | Potassium channels | Wnt/β-catenin pathway | aldosterone-producing adenoma | HYPERALDOSTERONISM TYPE-II | MEMBRANE CA2+ ATPASE | familial hyperaldosteronism | calcium channels | Wnt/beta-catenin pathway | germline mutations | PCR-SSCP ANALYSIS | somatic mutations | MOUSE ADRENAL-CORTEX | GLUCOCORTICOID-REMEDIABLE ALDOSTERONISM | potassium channels | K+ CHANNEL MUTATIONS | primary aldosteronism | ENDOCRINOLOGY & METABOLISM | CLINICAL-PRACTICE GUIDELINE | KCNJ5 MUTATIONS | POTASSIUM CHANNEL | Adrenal Glands - pathology | Sodium-Potassium-Exchanging ATPase - genetics | Plasma Membrane Calcium-Transporting ATPases - genetics | Adenoma - genetics | Humans | Hyperaldosteronism - pathology | Hyperaldosteronism - metabolism | Adrenal Hyperplasia, Congenital - genetics | Adenoma - metabolism | Plasma Membrane Calcium-Transporting ATPases - metabolism | Hyperaldosteronism - diagnosis | Adrenal Hyperplasia, Congenital - diagnosis | Hypokalemia - metabolism | Adrenal Hyperplasia, Congenital - pathology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Hypertension - genetics | Hypertension - diagnosis | Hypokalemia - pathology | Adenoma - diagnosis | Gene Expression | Genetic Predisposition to Disease | Adrenal Hyperplasia, Congenital - metabolism | Hyperaldosteronism - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | beta Catenin - metabolism | Hypertension - pathology | beta Catenin - genetics | Hypertension - metabolism | Hypokalemia - diagnosis | Sodium-Potassium-Exchanging ATPase - metabolism | Calcium Channels, L-Type - genetics | Adrenal Glands - metabolism | Adenoma - pathology | Calcium Channels, L-Type - metabolism | Mutation | Hypokalemia - genetics
Journal Article
Hypertension, ISSN 0194-911X, 11/2015, Volume 66, Issue 5, pp. 1014 - 1022
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2012, Volume 303, Issue 4, pp. H486 - H495
Cheema Y, Zhao W, Zhao T, Khan MU, Green KD, Ahokas RA, Gerling IC, Bhattacharya SK, Weber KT. Reverse remodeling and recovery from cachexia in rats with... 
Cardiomyocyte necrosis | Oxidative stress | Intracellular calcium overloading | Mitochondria | VENTRICULAR ASSIST DEVICES | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | mitochondria | HEART-FAILURE | CARDIAC MYOCYTES | intracellular calcium overloading | NECROSIS-FACTOR-ALPHA | cardiomyocyte necrosis | ZINC DYSHOMEOSTASIS | COUPLED CALCIUM | IN-VIVO | PERIPHERAL VASCULAR DISEASE | MAGNESIUM-DEFICIENCY | EXPRESSION | oxidative stress | Mitochondria, Heart - metabolism | Calcium - metabolism | Mitochondria, Heart - pathology | Heart Failure - physiopathology | Hyperaldosteronism - complications | Cardiomegaly - etiology | Cardiomegaly - pathology | Male | Muscle, Skeletal - metabolism | Hyperaldosteronism - metabolism | Dinoprost - metabolism | Recovery of Function | Necrosis | Ventricular Remodeling | Muscular Atrophy - physiopathology | Time Factors | Myocardium - metabolism | Cachexia - etiology | Dinoprost - analogs & derivatives | Cachexia - metabolism | Heart Failure - etiology | Muscular Atrophy - etiology | Disease Models, Animal | Mitochondrial Membrane Transport Proteins - metabolism | Cachexia - genetics | Muscular Atrophy - pathology | Gene Expression Regulation | Hyperaldosteronism - genetics | Cardiomegaly - physiopathology | Rats | Heart Failure - genetics | Myocardium - pathology | Heart Failure - metabolism | Heart Failure - pathology | Rats, Sprague-Dawley | Hydrogen Peroxide - metabolism | Myocytes, Cardiac - pathology | Animals | Cachexia - pathology | Muscle, Skeletal - physiopathology | Myocytes, Cardiac - metabolism | Muscle, Skeletal - pathology | Cachexia - physiopathology | Hyperaldosteronism | Care and treatment | Cachexia | Integrative Cardiovascular Physiology and Pathophysiology
Journal Article