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Pain Physician, ISSN 1533-3159, 05/2009, Volume 12, Issue 3, pp. 679 - 684
Opioids have been and continue to be used for the treatment of chronic pain. Evidence supports the notion that opioids can be safely administered in patients... 
Complications of opioid therapy | NMDA receptor induced hyperalgesia | Buprenorphine and hyperalgesia | Spinal dynorphin induced hyperalgesia | NMDA receptor antagonists | Opioid hyperalgesia | Opioid detoxification | Office-based detoxification | Tolerance | Descending facilitation and hyperalgesia | Hyperalgesia | PROTEIN-KINASE-C | descending facilitation and hyperalgesia | buprenorphine and hyperalgesia | office-based detoxification | CLINICAL NEUROLOGY | DEPENDENCE | KETAMINE | CLONIDINE | NEUROPATHIC PAIN | spinal dynorphin induced hyperalgesia | opioid detoxification | MORPHINE-TOLERANCE | BUPRENORPHINE | ENHANCEMENT | ANESTHESIOLOGY | CANCER PAIN | hyperalgesia | complications of opioid therapy | tolerance | ANALGESIA | Hyperalgesia - chemically induced | Receptors, N-Methyl-D-Aspartate - drug effects | Central Nervous System - metabolism | Inactivation, Metabolic - physiology | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Drug Tolerance - physiology | Pain - chemically induced | Hyperalgesia - physiopathology | Analgesics, Opioid - adverse effects | Buprenorphine - therapeutic use | Hyperalgesia - drug therapy | Narcotic Antagonists - therapeutic use | Analgesics, Opioid - administration & dosage | Pain - drug therapy | Pain - physiopathology | Central Nervous System - drug effects | Central Nervous System - physiopathology | Dynorphins - metabolism
Journal Article
Journal of applied physiology (1985), ISSN 1522-1601, 2013, Volume 114, Issue 6, pp. 725 - 733
Journal Article
Journal Article
2002, Progress in inflammation research., ISBN 3764362375, xii, 258
Book
Gastroenterology, ISSN 0016-5085, 2015, Volume 148, Issue 1, pp. 148 - 157.e7
Background & Aims Chronic stress alters the hypothalamic–pituitary–adrenal axis, increases gut motility, and increases the perception of visceral pain. We... 
Gastroenterology and Hepatology | HPA | Epigenetic Modification | Pain Sensitivity | Visceral Hyperalgesia | METHYLATION | SENSORY FIBERS | WATER AVOIDANCE STRESS | GUT | RECEPTOR | TRPV1 | GASTROENTEROLOGY & HEPATOLOGY | EXPOSURE | Epigenesis, Genetic | E1A-Associated p300 Protein - genetics | Male | E1A-Associated p300 Protein - metabolism | Ganglia, Spinal - physiopathology | Receptors, Glucocorticoid - metabolism | Visceral Pain - diagnosis | RNA Interference | Time Factors | Histones | Transcription, Genetic | Acetylation | Disease Models, Animal | Abdominal Pain - diagnosis | DNA (Cytosine-5-)-Methyltransferase 1 | Visceral Pain - physiopathology | Abdominal Pain - metabolism | Rats, Sprague-Dawley | Visceral Pain - genetics | Receptor, Cannabinoid, CB1 - genetics | Hyperalgesia - genetics | Abdominal Pain - genetics | Pain Threshold | Chronic Disease | Stress, Psychological - complications | Stress, Psychological - physiopathology | Hyperalgesia - psychology | Stress, Psychological - psychology | DNA (Cytosine-5-)-Methyltransferases - metabolism | TRPV Cation Channels - metabolism | DNA Methylation | Stress, Psychological - metabolism | Visceral Pain - psychology | Hyperalgesia - diagnosis | Promoter Regions, Genetic | Hyperalgesia - metabolism | Abdominal Pain - physiopathology | Gene Expression Regulation | Visceral Pain - metabolism | TRPV Cation Channels - genetics | Hyperalgesia - physiopathology | Abdominal Pain - psychology | DNA (Cytosine-5-)-Methyltransferases - genetics | Receptor, Cannabinoid, CB1 - metabolism | Stress, Psychological - genetics | Animals | Receptors, Glucocorticoid - genetics | Pain Measurement | Ganglia, Spinal - metabolism | Stress (Psychology) | Epigenetic inheritance | Genes | epigenetic modification | pain sensitivity | Visceral hyperalgesia
Journal Article