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Molecular pain, ISSN 1744-8069, 1/2010, Volume 6, Issue 1, pp. 4 - 4
Background: The cooling agents menthol and icilin act as agonists at TRPM8 and TRPA1. In vitro, activation of TRPM8 by icilin and cold, but not menthol, is... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Hyperalgesia - chemically induced | Pain Threshold - physiology | Rats, Wistar | Nociceptors - physiology | Male | Antipruritics - pharmacology | Group VI Phospholipases A2 - metabolism | Pyrones - pharmacology | Dose-Response Relationship, Drug | Group VI Phospholipases A2 - antagonists & inhibitors | Sensory Receptor Cells - physiology | Group VI Phospholipases A2 - genetics | Foot - physiology | Pain Threshold - drug effects | Pyrimidinones - pharmacology | Transient Receptor Potential Channels - metabolism | Hyperalgesia - metabolism | Thermosensing - drug effects | Thermosensing - physiology | Foot - innervation | Rats | TRPM Cation Channels - agonists | Transient Receptor Potential Channels - agonists | Cold Temperature - adverse effects | Hyperalgesia - physiopathology | Mice, Knockout | Naphthalenes - pharmacology | Pain Measurement - methods | Animals | Menthol - pharmacology | Sensory Receptor Cells - drug effects | Transient Receptor Potential Channels - genetics | TRPA1 Cation Channel | TRPM Cation Channels - genetics | Mice | TRPM Cation Channels - metabolism | Nociceptors - drug effects | Allergy | Physiological aspects | Ion channels | Menthol | Research | Allergic reaction | Health aspects | Risk factors | Statistical analysis | Biomedical research | Cold | Rodents | Experiments | Manuscripts | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0170814 - e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, pp. e100701 - e100701
Chemotherapy-induced peripheral neuropathy (CIPN) characterized by loss of sensory sensitivity and pain in hands and feet is the major dose-limiting toxicity of many chemotherapeutics... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Hyperalgesia - chemically induced | Hypoglycemic Agents - therapeutic use | Nerve Fibers - pathology | Neuralgia - complications | Peripheral Nervous System Diseases - chemically induced | Metformin - therapeutic use | Hyperalgesia - complications | Metformin - pharmacology | Mice, Inbred C57BL | Paclitaxel - adverse effects | Nerve Fibers - drug effects | Protective Agents - therapeutic use | Neuralgia - drug therapy | Hypoglycemic Agents - pharmacology | Animals | Hyperalgesia - drug therapy | Protective Agents - pharmacology | Cisplatin - adverse effects | Neuralgia - chemically induced | Peripheral Nervous System Diseases - complications | Peripheral Nervous System Diseases - drug therapy | Disease Models, Animal | Drugs | Prescribing | Chemotherapy | Analysis | Metformin | Diabetes | Drug therapy | Drug approval | Cisplatin | Diabetes therapy | Cancer | Neuroprotection | Neurosciences | Toxicity | Neurobiology | Neuropathy | Kinases | Peripheral neuropathy | Cancer therapies | Fibers | Pain | Rodents | Paclitaxel | Drug dosages | Tactile | Pain sensitivity | Nerve endings | Pain perception | Diabetes mellitus | Breast cancer | Metabolism | Apolipoproteins | Latency | Studies | Sensitivity | Hypotheses | Adhesives | Brain research | Mechanical stimuli | Laboratory animals | Index Medicus
Journal Article
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2007, Volume 282, Issue 36, pp. 26089 - 26100
Although principally produced by the pancreas to degrade dietary proteins in the intestine, trypsins are also expressed in the nervous system and in epithelial... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Hyperalgesia - chemically induced | Inflammation - chemically induced | Inflammation - pathology | Receptors, Proteinase-Activated - metabolism | Capsaicin - pharmacology | Edema - genetics | Receptor, PAR-1 - metabolism | Humans | Male | Receptor, PAR-1 - deficiency | Receptor, PAR-2 - deficiency | Hyperalgesia - pathology | Inflammation - metabolism | Trypsin Inhibitors - chemistry | Trypsin - pharmacology | Trypsin - genetics | Granulocytes - pathology | Granulocytes - metabolism | Nociceptors - pathology | Recombinant Proteins - metabolism | Hyperalgesia - metabolism | Rats | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Recombinant Proteins - pharmacology | Trypsin - metabolism | Rats, Sprague-Dawley | Mice, Knockout | Edema - metabolism | Animals | Hyperalgesia - genetics | Calcium Signaling - drug effects | Enteropeptidase - chemistry | Ganglia, Spinal - pathology | Inflammation - genetics | Receptor, PAR-2 - physiology | Receptors, Thrombin - metabolism | Mice | Nociceptors - metabolism | Aprotinin - chemistry | Edema - chemically induced | Pain Measurement | Edema - pathology | Trypsin - chemistry | Ganglia, Spinal - metabolism | Index Medicus | Nociceptors/metabolism | Aprotinin/chemistry | Edema/chemically induced | Recombinant Proteins/chemistry | Receptor, PAR-1/deficiency | Enteropeptidase/chemistry | Capsaicin/pharmacology | Inflammation/chemically induced | Receptors, Proteinase-Activated/metabolism | Granulocytes/metabolism | Trypsin Inhibitors/chemistry | Ganglia, Spinal/metabolism | Receptor, PAR-2/deficiency | Calcium Signaling/drug effects | Trypsin/chemistry | Hyperalgesia/chemically induced | Receptors, Thrombin/metabolism
Journal Article
Molecular pain, ISSN 1744-8069, 9/2012, Volume 8, Issue 1, pp. 71 - 71
...) signaling to both chemotherapy-induced neuropathy and CB2 agonist efficacy. Results: AM1710 (0.1, 1 or 5 mg/kg i.p.) suppressed the maintenance of mechanical and cold allodynia in the cisplatin and paclitaxel models... 
Chemokine | Chemotherapy | Cold allodynia | Endocannabinoid | CXCR4 | Cannabilactone | AM1710 | Hyperalgesia | Mechanical allodynia | Neuropathic pain | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Hyperalgesia - chemically induced | Peripheral Nervous System Diseases - chemically induced | Pyrazoles - therapeutic use | Receptor, Cannabinoid, CB2 - agonists | Heterocyclic Compounds - pharmacology | Male | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Cryopyrin-Associated Periodic Syndromes - chemically induced | Pyrazoles - chemistry | Heterocyclic Compounds - chemistry | Piperidines - pharmacology | Time Factors | Indoles - pharmacology | Chromones - pharmacology | Peripheral Nervous System Diseases - complications | Chromones - therapeutic use | Cryopyrin-Associated Periodic Syndromes - complications | Disease Models, Animal | Pyrazoles - pharmacology | Piperidines - chemistry | Hyperalgesia - metabolism | Receptors, CXCR4 - antagonists & inhibitors | Hyperalgesia - complications | Paclitaxel - adverse effects | Rats | Treatment Outcome | Cryopyrin-Associated Periodic Syndromes - drug therapy | Rats, Sprague-Dawley | Receptors, CXCR4 - metabolism | Chromones - chemistry | Peripheral Nervous System Diseases - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Hyperalgesia - drug therapy | Signal Transduction - drug effects | Piperidines - therapeutic use | Cisplatin - adverse effects | Indoles - therapeutic use | Heterocyclic Compounds - therapeutic use | Indoles - chemistry | Peripheral Nervous System Diseases - drug therapy | Cryopyrin-Associated Periodic Syndromes - metabolism | Antimitotic agents | Models | Antineoplastic agents | Cancer | Health sciences | Pain | Cold | Human immunodeficiency virus--HIV | Behavior | Drug dosages | Manuscripts | Chemokines | Index Medicus
Journal Article