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PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 6, pp. e100701 - e100701
.... At present, there are no FDA-approved treatments for CIPN. The anti-diabetic drug metformin is the most widely used prescription drug in the world and improves glycemic control in diabetes patients... 
BREAST-CANCER | CELLS | NERVE-FIBERS | ACTIVATION | PAIN | CORTICAL-NEURONS | REGENERATION | MULTIDISCIPLINARY SCIENCES | INJURY | OVARIAN-CANCER | APOLIPOPROTEIN-E | Hyperalgesia - chemically induced | Hypoglycemic Agents - therapeutic use | Nerve Fibers - pathology | Neuralgia - complications | Peripheral Nervous System Diseases - chemically induced | Metformin - therapeutic use | Hyperalgesia - complications | Metformin - pharmacology | Mice, Inbred C57BL | Paclitaxel - adverse effects | Nerve Fibers - drug effects | Protective Agents - therapeutic use | Neuralgia - drug therapy | Hypoglycemic Agents - pharmacology | Animals | Hyperalgesia - drug therapy | Protective Agents - pharmacology | Cisplatin - adverse effects | Neuralgia - chemically induced | Peripheral Nervous System Diseases - complications | Peripheral Nervous System Diseases - drug therapy | Disease Models, Animal | Drugs | Prescribing | Chemotherapy | Analysis | Metformin | Diabetes | Drug therapy | Drug approval | Cisplatin | Diabetes therapy | Cancer | Neuroprotection | Neurosciences | Toxicity | Neurobiology | Neuropathy | Kinases | Peripheral neuropathy | Cancer therapies | Fibers | Pain | Rodents | Paclitaxel | Drug dosages | Tactile | Pain sensitivity | Nerve endings | Pain perception | Diabetes mellitus | Breast cancer | Metabolism | Apolipoproteins | Latency | Studies | Sensitivity | Hypotheses | Adhesives | Brain research | Mechanical stimuli | Laboratory animals | Index Medicus
Journal Article
Acta biomaterialia, ISSN 1742-7061, 2016, Volume 42, pp. 77 - 89
[Display omitted] The hostile environment after spinal cord injury (SCI) can compromise effects of regenerative therapies... 
Functional recovery | Self-assembling peptides | Tissue-preservation | Neural precursor cells | Cervical spinal cord injury | SCAR FORMATION | MATERIALS SCIENCE, BIOMATERIALS | NEUROLOGICAL RECOVERY | ENGINEERING, BIOMEDICAL | REGENERATION | TRANSPLANTATION | REPAIR | ADULT-RAT | GROWTH | STEM/PROGENITOR CELLS | PRECURSOR CELLS | Spinal Cord Injuries - drug therapy | Rats, Wistar | Pyramidal Tracts - drug effects | Cervical Cord - drug effects | Synapses - pathology | Neural Stem Cells - cytology | Choline O-Acetyltransferase - metabolism | Cervical Cord - physiopathology | Spinal Cord Injuries - pathology | Gliosis - pathology | Stem Cell Transplantation | Neural Stem Cells - transplantation | Behavior, Animal - drug effects | Biotin - metabolism | Female | Spinal Cord Injuries - therapy | Cicatrix - complications | Pyramidal Tracts - pathology | Motor Neurons - drug effects | Dextrans - metabolism | Cell Survival - drug effects | Synapses - drug effects | Cervical Cord - pathology | Hyperalgesia - complications | Neural Stem Cells - drug effects | Wounds and Injuries - complications | Wounds and Injuries - physiopathology | Gliosis - complications | Hyperalgesia - physiopathology | Peptides - pharmacology | Animals | Hyperalgesia - therapy | Cell Differentiation - drug effects | Wounds and Injuries - drug therapy | Cicatrix - pathology | Spinal Cord Injuries - physiopathology | Microscopy, Fluorescence | Peptides - therapeutic use | Biotin - analogs & derivatives | Transplantation | Spinal cord injuries | Peptides | Stem cells | Regenerative | Therapy | Spinal cord | Cysts | Differentiation | Injuries
Journal Article
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2006, Volume 21, Issue 5, pp. 686 - 698
Abstract Paclitaxel is a commonly used cancer chemotherapy drug that frequently causes painful peripheral neuropathies... 
Psychiatry | Allergy and Immunology | Interleukin-1 | Meninges | Glia | Macrophages | Tumor necrosis factor | Neuropathic pain | NERVOUS-SYSTEM | interleukin-1 | TNF RECEPTORS | NECROSIS-FACTOR-ALPHA | PAINFUL PERIPHERAL NEUROPATHY | ANTIINFLAMMATORY CYTOKINE | IMMUNOLOGY | glia | NEUROSCIENCES | SENSORY NEURONS | neuropathic pain | macrophages | tumor necrosis factor | INDUCED HYPERALGESIA | TAXOL | SPINAL-CORD GLIA | ALTERNATIVE ACTIVATION | meninges | Hyperalgesia - chemically induced | Interleukin-1beta - drug effects | Tumor Necrosis Factor-alpha - metabolism | Pain Threshold - physiology | Peripheral Nervous System Diseases - chemically induced | Spinal Cord - drug effects | Spinal Cord - metabolism | CD11b Antigen - drug effects | RNA, Messenger - analysis | Male | Ganglia, Spinal - cytology | Interleukin-10 - physiology | Meninges - metabolism | Neuroglia - drug effects | Peripheral Nervous System Diseases - prevention & control | Interleukin-1beta - metabolism | Interleukin-10 - administration & dosage | Plasmids - genetics | Pain Threshold - drug effects | Spinal Cord - cytology | Peripheral Nervous System Diseases - complications | Antineoplastic Agents, Phytogenic - adverse effects | Cytokines - immunology | Disease Models, Animal | Injections, Spinal | Hyperalgesia - prevention & control | Paclitaxel - adverse effects | Rats | Meninges - drug effects | Rats, Sprague-Dawley | Animals | Tumor Necrosis Factor-alpha - drug effects | Cytokines - drug effects | Interleukin-10 - genetics | Hyperalgesia - etiology | Neuroglia - metabolism | Receptors, Interleukin-1 - physiology | CD11b Antigen - metabolism | Ganglia, Spinal - drug effects | Receptors, Interleukin-1 - antagonists & inhibitors | Plasmids - administration & dosage | Ganglia, Spinal - metabolism | Genetic Therapy - methods | Genetic research | Gene expression | Gene therapy | Health aspects
Journal Article
Experimental neurology, ISSN 0014-4886, 2009, Volume 219, Issue 1, pp. 146 - 155
Cancer pain significantly affects the quality of cancer patients, and current treatments for this pain are limited. C-Jun N-terminal kinase (JNK) has been... 
NEUROPATHIC PAIN | MURINE MODEL | MECHANICAL ALLODYNIA | ACTIVATED PROTEIN-KINASE | N-TERMINAL KINASE | SPINAL NERVE LIGATION | FUNCTIONAL INTERACTIONS | CELL-CYCLE ARREST | PRIMARY SENSORY NEURONS | C-JUN | NEUROSCIENCES | Melanoma - complications | Skin Neoplasms - drug therapy | Analgesics, Opioid - pharmacology | Melanoma - enzymology | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Peripheral Nervous System Diseases - physiopathology | Skin Neoplasms - enzymology | Pain - enzymology | Pain - drug therapy | Pain - etiology | Sensory Receptor Cells - metabolism | Disease Models, Animal | Peripheral Nervous System Diseases - etiology | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Morphine - pharmacology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Peripheral Nervous System Diseases - enzymology | Activating Transcription Factor 3 - metabolism | Enzyme Inhibitors - therapeutic use | Peptides - pharmacology | Skin Neoplasms - complications | Animals | Sensory Receptor Cells - cytology | Sensory Receptor Cells - drug effects | Hyperalgesia - drug therapy | Melanoma - drug therapy | Cell Line, Tumor | Hyperalgesia - etiology | Cell Proliferation - drug effects | Mice | Pain Measurement | Hyperalgesia - enzymology | Peptides - therapeutic use | Allergy | Care and treatment | Pain | Peptides | Growth | Analysis | Melanoma | Fluorescence | Cancer pain | Allergic reaction | Skin cancer | Cancer
Journal Article
The Journal of pharmacology and experimental therapeutics, ISSN 0022-3565, 2006, Volume 319, Issue 3, pp. 1376 - 1385
Journal Article
Pain (Amsterdam), ISSN 0304-3959, 2008, Volume 135, Issue 3, pp. 232 - 239
Although it has been shown that pro-inflammatory cytokines such as interleukin-1β (IL-1β) facilitate perception of noxious inputs at the spinal level, the... 
Phosphorylation | Spinal cord | Rat | IL-1β | NMDA receptor | Hyperalgesia | rat | GLIAL ACTIVATION | SPINAL-CORD | IL-1 beta | PROINFLAMMATORY CYTOKINES | INTRADERMAL INJECTION | PERIPHERAL INFLAMMATION | GLUTAMATE UPTAKE | NEUROSCIENCES | DORSAL-HORN | CLINICAL NEUROLOGY | NEUROPATHIC PAIN | NITRIC-OXIDE | ANESTHESIOLOGY | hyperalgesia | spinal cord | phosphorylation | BEHAVIORAL HYPERALGESIA | Analgesics - pharmacology | Pain Threshold - physiology | Posterior Horn Cells - drug effects | Receptors, N-Methyl-D-Aspartate - metabolism | Male | Pain - immunology | Gliosis - physiopathology | Astrocytes - immunology | Inflammation - drug therapy | Inflammation Mediators - pharmacology | Pain - drug therapy | Reaction Time - drug effects | Pain Threshold - drug effects | Posterior Horn Cells - physiopathology | Gliosis - immunology | Interleukin 1 Receptor Antagonist Protein - pharmacology | Phosphorylation - drug effects | Disease Models, Animal | Interleukin-1beta - antagonists & inhibitors | Astrocytes - drug effects | Receptors, N-Methyl-D-Aspartate - drug effects | Reaction Time - immunology | Anti-Inflammatory Agents - pharmacology | Receptors, Interleukin-1 - immunology | Proto-Oncogene Proteins c-fos - metabolism | Interleukin-1beta - immunology | Rats | Inflammation - immunology | Rats, Sprague-Dawley | Proto-Oncogene Proteins c-fos - drug effects | Hyperalgesia - physiopathology | Hyperalgesia - immunology | Posterior Horn Cells - immunology | Animals | Hyperalgesia - drug therapy | Pain - physiopathology | Gliosis - drug therapy | Receptors, Interleukin-1 - antagonists & inhibitors | Inflammation - physiopathology | Interleukins | Methyl aspartate
Journal Article
Journal Article
Journal of neurophysiology, ISSN 0022-3077, 04/2015, Volume 114, Issue 1, pp. 199 - 208
The purpose of this study was to determine the effect of supplementing the diet of a mouse model of type 2 diabetes with menhaden (fish) oil or daily treatment... 
Type 2 diabetes | Diabetic neuropathy | Epidermal nerve fibers | Resolvin | Fish oil | Corneal nerve fibers | NERVE REGENERATION | IMPROVES INSULIN SENSITIVITY | PHYSIOLOGY | RISK-FACTORS | diabetic neuropathy | fish oil | corneal nerve fibers | epidermal nerve fibers | FATTY-ACIDS | NEUROSCIENCES | OBESITY | type 2 diabetes | DOCOSAHEXAENOIC ACID | INFLAMMATION | FIBER DAMAGE | resolvin | PERIPHERAL NEUROPATHY | CORNEAL CONFOCAL MICROSCOPY | Neurons - pathology | Diabetes Mellitus, Experimental - drug therapy | Neurites - physiology | Diabetes Mellitus, Experimental - diet therapy | Retinal Ganglion Cells - pathology | Hyperalgesia - diet therapy | Neuroprotective Agents - pharmacology | Diet, High-Fat | Neurons - physiology | Fish Oils - administration & dosage | Cornea - pathology | Diabetes Mellitus, Type 2 | Neurons - drug effects | Neurites - drug effects | Skin - pathology | Diabetes Mellitus, Experimental - physiopathology | Diabetic Neuropathies - physiopathology | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Ganglia, Spinal - physiology | Neural Conduction - physiology | Diabetic Neuropathies - drug therapy | Hot Temperature | Hyperalgesia - physiopathology | Docosahexaenoic Acids - pharmacology | Animals | Hyperalgesia - drug therapy | Diabetic Neuropathies - pathology | Diabetes Mellitus, Experimental - pathology | Skin - innervation | Cornea - innervation | Ganglia, Spinal - drug effects | Dietary Supplements | Diabetic Neuropathies - diet therapy | Nervous System Pathophysiology
Journal Article
Nature neuroscience, ISSN 1546-1726, 2013, Volume 16, Issue 2, pp. 183 - 192
A major unresolved issue in treating pain is the paradoxical hyperalgesia produced by the gold-standard analgesic morphine and other opiates. We found that... 
LAMINA-I NEURONS | NEUROTROPHIC FACTOR | P2X RECEPTORS | DOWN-REGULATION | OPIOID-RECEPTOR GENE | SPINAL-CORD-INJURY | UNDERLYING NEUROPATHIC PAIN | ALTERED CHLORIDE HOMEOSTASIS | NERVE INJURY | NEUROSCIENCES | INDUCED ABNORMAL PAIN | Brain-Derived Neurotrophic Factor - genetics | Biophysical Phenomena - drug effects | Motor Activity - drug effects | Male | Ribosome Inactivating Proteins, Type 1 - pharmacology | Touch | CD11b Antigen - genetics | Microglia - physiology | Time Factors | Chlorides - metabolism | Pain Threshold - drug effects | Biophysical Phenomena - genetics | Vocalization, Animal - drug effects | Brain-Derived Neurotrophic Factor - metabolism | Homeostasis - drug effects | Protein Synthesis Inhibitors - pharmacology | Spinal Cord - cytology | Neurons - drug effects | Membrane Potentials - drug effects | Hot Temperature - adverse effects | Microglia - drug effects | Mice, Inbred C57BL | Rats | Mice, Transgenic | Signal Transduction - genetics | Down-Regulation - drug effects | Rats, Sprague-Dawley | Symporters - metabolism | Gene Expression Regulation - drug effects | Narcotics - administration & dosage | Patch-Clamp Techniques | Rotarod Performance Test | Animals | Hyperalgesia - drug therapy | Signal Transduction - drug effects | Naloxone - pharmacology | Narcotic Antagonists - pharmacology | Mice | CD11b Antigen - metabolism | Receptors, Purinergic P2X4 - metabolism | Morphine - administration & dosage | Receptors, Purinergic P2X4 - genetics | Ion Channel Gating - drug effects
Journal Article