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Brain, Behavior, and Immunity, ISSN 0889-1591, 10/2018, Volume 73, pp. 520 - 532
Functional pain syndromes, such as fibromyalgia and temporomandibular disorder, are associated with enhanced catecholamine tone and decreased levels of... 
Allodynia | Epinephrine | Mitogen-activated protein kinase (MAPK) | Catecholamines | Neuroinflammation | Astrocyte | Extracellular signal-regulated kinases (ERK) | Chronic pain | Hyperalgesia | Microglia | Beta-adrenergic receptor (βAR) | AR | p38 | Catechol-O-methyltransferase (COMT) | Beta 2-adrenergic receptor (β | Idiopathic pain | Norepinephrine | Tumor necrosis factor alpha (TNFα) | Beta 3-adrenergic receptor (β | Functional pain | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Phosphorylation | Spinal Cord - metabolism | Receptors, Adrenergic, beta-2 - physiology | Receptors, Adrenergic, beta-3 - drug effects | Male | Receptors, Adrenergic, beta-2 - drug effects | Pain - metabolism | Catechol O-Methyltransferase Inhibitors - metabolism | Catechol O-Methyltransferase - metabolism | Temporomandibular Joint Disorders - metabolism | Temporomandibular Joint Disorders - physiopathology | Interleukin-1beta - metabolism | Catechols - pharmacology | Female | p38 Mitogen-Activated Protein Kinases - metabolism | Receptors, Adrenergic, beta - metabolism | Interleukin-6 - metabolism | Receptors, Adrenergic, beta-3 - physiology | Mitogen-Activated Protein Kinases | Fibromyalgia - metabolism | Cytokines - metabolism | Hyperalgesia - metabolism | Rats | Imidazoles - pharmacology | Receptors, Adrenergic, beta-3 - metabolism | Rats, Sprague-Dawley | Receptors, Adrenergic, beta-2 - metabolism | Animals | Pain - physiopathology | Etanercept - pharmacology | Neuroglia - metabolism | Pyridines - pharmacology | Fibromyalgia - physiopathology | Propanolamines - pharmacology
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2011, Volume 31, Issue 29, pp. 10516 - 10528
Journal Article
Brain Behavior and Immunity, ISSN 0889-1591, 02/2018, Volume 68, pp. 132 - 145
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2013, Volume 33, Issue 13, pp. 5603 - 5611
Visceral afferents expressing transient receptor potential (TRP) channels TRPV1 and TRPA1 are thought to be required for neurogenic inflammation and... 
ACTIVATION | SUBPOPULATIONS | PHOSPHORYLATION | SENSITIZATION | PROTEIN-KINASE | HYPERALGESIA | EXPRESSION | NEUROSCIENCES | SENSORY NEURONS | ERK | Calcium - metabolism | Male | Nodose Ganglion - metabolism | RNA, Messenger - metabolism | Amidines - metabolism | Time Factors | TRPV Cation Channels - antagonists & inhibitors | Pancreatitis, Chronic - drug therapy | Exploratory Behavior - drug effects | Monocytes - pathology | Pain - etiology | Sensory Receptor Cells - metabolism | Transient Receptor Potential Channels - metabolism | Calcitonin Gene-Related Peptide - metabolism | Neutrophil Infiltration - drug effects | Disease Models, Animal | Injections, Intraperitoneal | Pancreas - pathology | Analgesics, Opioid - therapeutic use | Pancreas - metabolism | Pain - pathology | Disease Progression | Pancreatitis, Chronic - pathology | Analysis of Variance | Transient Receptor Potential Channels - genetics | Ganglia, Spinal - pathology | Mice | Transient Receptor Potential Channels - antagonists & inhibitors | Oximes - therapeutic use | Pancreatitis, Chronic - chemically induced | Monocytes - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | TRPV Cation Channels - metabolism | Antigens, Differentiation - metabolism | Pyridines - therapeutic use | Sensory Receptor Cells - pathology | Pain - prevention & control | Mice, Inbred C57BL | Ceruletide - toxicity | Pancreas - drug effects | Pain Measurement - drug effects | TRPV Cation Channels - genetics | Gene Expression Regulation - drug effects | Nodose Ganglion - pathology | Animals | Sensory Receptor Cells - drug effects | Pancreatitis, Chronic - complications | TRPA1 Cation Channel | Morphine - therapeutic use | Ganglia, Spinal - metabolism | Peroxidase - metabolism | Index Medicus
Journal Article
Pain, ISSN 0304-3959, 2015, Volume 156, Issue 12, pp. 2572 - 2584
Treating neuropathic pain continues to be a major clinical challenge and underlying mechanisms of neuropathic pain remain elusive. We have recently... 
Neural excitability | Nerve injury | Wnt | Ryk | Synaptic plasticity | DORSAL-HORN NEURONS | NERVOUS-SYSTEM | THERMAL HYPERALGESIA | ROOT GANGLION NEURONS | EPHB1 RECEPTOR | BETA-CATENIN | NEUROSCIENCES | CLINICAL NEUROLOGY | HYPEREXCITABILITY | ANESTHESIOLOGY | LONG-TERM POTENTIATION | Wnt3A Protein - metabolism | Posterior Horn Cells - metabolism | Sciatic Nerve - injuries | Neuralgia - genetics | Spinal Cord - metabolism | Neuralgia - metabolism | Receptors, N-Methyl-D-Aspartate - metabolism | Wnt3A Protein - antagonists & inhibitors | Neuronal Plasticity - drug effects | Wnt Signaling Pathway - physiology | Male | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - drug effects | Cyclic AMP Response Element-Binding Protein - drug effects | Wnt Proteins - metabolism | src-Family Kinases - drug effects | Neuronal Plasticity - physiology | src-Family Kinases - metabolism | Nerve Fibers, Unmyelinated - drug effects | Pain Threshold - drug effects | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Behavior, Animal - drug effects | Sensory Receptor Cells - metabolism | Real-Time Polymerase Chain Reaction | Calcium Signaling | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Locomotion - drug effects | Receptors, N-Methyl-D-Aspartate - drug effects | Hyperalgesia - metabolism | Signal Transduction | Rats | Behavior, Animal - physiology | Receptor Protein-Tyrosine Kinases - metabolism | Rats, Sprague-Dawley | Patch-Clamp Techniques | Animals | MAP Kinase Signaling System - drug effects | Wnt Signaling Pathway - drug effects | Hyperalgesia - genetics | Receptor Protein-Tyrosine Kinases - genetics | Cyclic AMP Response Element-Binding Protein - metabolism | Wnt Proteins - antagonists & inhibitors | Astrocytes - metabolism | Ganglia, Spinal - metabolism | Protein Kinase C | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 6, pp. 2084 - 2095.e3
Background & Aims The transient receptor potential (TRP) channel family includes transducers of mechanical and chemical stimuli for visceral sensory neurons.... 
Gastroenterology and Hepatology | SENSITIZATION | PELVIC COLONIC AFFERENTS | INFLAMMATORY PAIN | MICE | HYPERALGESIA | COLD | GASTROENTEROLOGY & HEPATOLOGY | IRRITABLE-BOWEL-SYNDROME | SENSORY NEURONS | BRADYKININ | HYPERSENSITIVITY | Hyperalgesia - chemically induced | Immunohistochemistry | Intestinal Mucosa - metabolism | Intestinal Mucosa - physiopathology | Mechanotransduction, Cellular - drug effects | Capsaicin - pharmacology | Neuroanatomical Tract-Tracing Techniques | Receptor, PAR-2 - metabolism | Colon - drug effects | Afferent Pathways - physiopathology | Colon - innervation | Male | Ganglia, Spinal - physiopathology | Nodose Ganglion - metabolism | Trinitrobenzenesulfonic Acid | Pelvis - innervation | RNA, Messenger - metabolism | Transient Receptor Potential Channels - deficiency | Action Potentials | Nodose Ganglion - physiopathology | Splanchnic Nerves - physiopathology | In Situ Hybridization | Colitis - chemically induced | Nodose Ganglion - drug effects | Colon - physiopathology | Female | Transient Receptor Potential Channels - metabolism | Bradykinin - pharmacology | Disease Models, Animal | Afferent Pathways - metabolism | Hyperalgesia - metabolism | Mice, Inbred C57BL | Reverse Transcriptase Polymerase Chain Reaction | Pressure | Stimulation, Chemical | Colon - metabolism | Hyperalgesia - physiopathology | Mice, Knockout | Animals | Colitis - physiopathology | Transient Receptor Potential Channels - genetics | TRPA1 Cation Channel | Colitis - metabolism | Splanchnic Nerves - metabolism | Mice | Ganglia, Spinal - drug effects | Pain Measurement | Ganglia, Spinal - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 02/2016, Volume 7, Issue 1, pp. 10604 - 10604
Journal Article
Pain, ISSN 0304-3959, 09/2016, Volume 157, Issue 9, pp. 2012 - 2023
Journal Article
Pain, ISSN 0304-3959, 07/2014, Volume 155, Issue 7, pp. 1293 - 1302
Lack of interleukin-17 leads to a modulated micro-environment and amelioration of mechanical hypersensitivity after peripheral nerve injury in mice.... 
Nerve injury | Animal model | Inflammation | Interleukin-17 | Neuropathic pain | CELLS | BRAIN-INJURY | SPINAL-CORD | NEUROSCIENCES | BETA-ENDORPHIN | CLINICAL NEUROLOGY | INFILTRATION | IMMUNE | BEHAVIORAL HYPERSENSITIVITY | HOST-DEFENSE | ANESTHESIOLOGY | INFLAMMATORY RESPONSE | Tumor Necrosis Factor-alpha - metabolism | Sciatic Nerve - injuries | Neuralgia - genetics | Peripheral Nerve Injuries - immunology | Enkephalins - metabolism | Interleukin-17 - immunology | Neuralgia - metabolism | Interleukin-13 - immunology | Behavior, Animal | beta-Endorphin - metabolism | Neuralgia - immunology | Interleukin-2 - immunology | Interleukin-1beta - metabolism | Peripheral Nerve Injuries - metabolism | Tumor Necrosis Factor-alpha - immunology | Interleukin-10 - metabolism | Dynorphins - metabolism | Interleukin-6 - metabolism | Macrophages - immunology | Cytokines - immunology | Disease Models, Animal | Interleukin-2 - metabolism | Nociception | Central Nervous System Sensitization - immunology | Cytokines - metabolism | Hyperalgesia - metabolism | Mice, Inbred C57BL | Neutrophils - immunology | Interleukin-17 - genetics | Interleukin-1beta - immunology | Inflammation - immunology | Interleukin-13 - metabolism | Mice, Knockout | Hyperalgesia - immunology | Animals | Hyperalgesia - genetics | Interleukin-6 - immunology | Inflammation - genetics | T-Lymphocytes - immunology | Mice | Central Nervous System Sensitization - genetics | Peripheral Nerve Injuries - genetics | Interleukin-10 - immunology | Peroxidase - metabolism | Interleukins | Allergy | Allergic reaction | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 05/2004, Volume 24, Issue 18, pp. 4300 - 4312
Inflammatory proteases (mast cell tryptase and trypsins) cleave protease-activated receptor 2 (PAR(2)) on spinal afferent neurons and cause persistent... 
Protein kinase C | Inflammation | TRPV1 | Substance P | Hyperalgesia | Protease-activated receptors | protease-activated receptors | THERMAL HYPERALGESIA | ROOT GANGLION NEURONS | PAIN PATHWAY | RAT SENSORY NEURONS | NEUROSCIENCES | inflammation | SIGNALING PATHWAY | KINASE-C ACTIVATION | MYENTERIC NEURONS | MOLECULAR-CLONING | hyperalgesia | substance P | THROMBIN RECEPTOR | protein kinase C | MAST-CELL TRYPTASE | Enzyme Activators - pharmacology | Hyperalgesia - chemically induced | Spinal Cord - drug effects | Spinal Cord - metabolism | Calcium - metabolism | Capsaicin - pharmacology | Receptors, Drug - genetics | Humans | Receptor, PAR-2 - metabolism | Type C Phospholipases - metabolism | Male | Neurons - cytology | RNA, Messenger - metabolism | Receptors, Drug - agonists | Type C Phospholipases - antagonists & inhibitors | Kidney - metabolism | Protein Kinase C - metabolism | Neurons - metabolism | Calcitonin Gene-Related Peptide - metabolism | Neurons - drug effects | Receptor, PAR-2 - genetics | Hyperalgesia - metabolism | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Kidney - cytology | Protein Kinase C - antagonists & inhibitors | Rats, Sprague-Dawley | Receptors, Drug - metabolism | Mice, Knockout | Animals | Receptor, PAR-2 - agonists | Substance P - metabolism | Mice | Index Medicus | Cellular | Molecular
Journal Article
Journal Article
Journal of Affective Disorders, ISSN 0165-0327, 2013, Volume 151, Issue 3, pp. 1041 - 1052
Journal Article