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PloS one, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0170814 - e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment. Current understanding... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
Scientific reports, ISSN 2045-2322, 02/2017, Volume 7, Issue 1, pp. 42304 - 42304
Multiple sclerosis (MS)-induced neuropathic pain deteriorates quality of life in patients but is often refractory to treatment. In experimental autoimmune... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Analgesics - pharmacology | Neuralgia - complications | Pregabalin - therapeutic use | Demyelinating Diseases - complications | Encephalomyelitis, Autoimmune, Experimental - immunology | Motor Activity - drug effects | Neuralgia - pathology | Gray Matter - drug effects | Hyperalgesia - pathology | Myelin Sheath - metabolism | Peripheral Nervous System - pathology | Peptides - metabolism | Microglia - pathology | Neuralgia - physiopathology | Demyelinating Diseases - pathology | Lumbar Vertebrae - physiopathology | Analgesics - therapeutic use | Demyelinating Diseases - physiopathology | Encephalomyelitis, Autoimmune, Experimental - physiopathology | Encephalomyelitis, Autoimmune, Experimental - pathology | Sensory Receptor Cells - pathology | Microglia - drug effects | Hyperalgesia - complications | Pregabalin - pharmacology | Peripheral Nervous System - physiopathology | Lumbar Vertebrae - drug effects | Neuralgia - drug therapy | Hyperalgesia - physiopathology | Mice, Knockout | Animals | Encephalomyelitis, Autoimmune, Experimental - complications | Ganglia, Spinal - pathology | Gray Matter - pathology | Peripheral Nervous System - injuries | Lumbar Vertebrae - pathology | Ganglia, Spinal - drug effects | Acid Sensing Ion Channels - metabolism | Ganglia, Spinal - metabolism | Sodium channels | Spinal cord | Multiple sclerosis | Central nervous system | Acidity | Neuropathy | Peripheral neuropathy | Pertussis toxin | Experimental allergic encephalomyelitis | Quality of life | Pertussis | Side effects | Pain | Analgesics | Rodents | Ion channels | Acidosis | Index Medicus
Journal Article
Journal of bone and mineral research, ISSN 0884-0431, 12/2013, Volume 28, Issue 12, pp. 2512 - 2522
Journal Article
Molecular pain, ISSN 1744-8069, 05/2016, Volume 12, p. 174480691664678
Journal Article
eJournal
Neuropharmacology, ISSN 0028-3908, 04/2017, Volume 116, pp. 315 - 327
Joint pain is a major clinical problem mainly associated to osteoarthritis, and characterized by articular cartilage degradation resulting in a complex chronic... 
Dynorphin | Opioids | Anhedonia | Anxiety | Cognition | Osteoarthritis | KOR | Pharmacology & Pharmacy | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Arthralgia - pathology | Microglia - metabolism | Spinal Cord - metabolism | Enkephalins - metabolism | Epigenesis, Genetic | Hyperalgesia - psychology | Hyperalgesia - pathology | Enkephalins - genetics | Spinal Cord - pathology | Cognition - physiology | Microglia - pathology | Acetylation | Osteoarthritis - pathology | Receptors, Opioid, kappa - genetics | Arthralgia - psychology | Disease Models, Animal | Emotions - physiology | Protein Precursors - genetics | Hyperalgesia - metabolism | Mice, Inbred C57BL | Amygdala - metabolism | Receptors, Opioid, kappa - metabolism | Osteoarthritis - metabolism | Hippocampus - pathology | Chronic Pain - psychology | Chronic Pain - pathology | Amygdala - pathology | Corticotropin-Releasing Hormone - metabolism | Mice, Knockout | Protein Precursors - metabolism | Chronic Pain - metabolism | Hippocampus - metabolism | Osteoarthritis - psychology | Animals | Arthralgia - metabolism | Histones - metabolism | Corticotropin releasing hormone | Epigenetic inheritance | Analysis | Arthralgia | ACTH | Missing in action | Chronic pain | Index Medicus | Artrosi | Cognició | Angoixa | Dolor | Neuropèptids
Journal Article
Journal Article
Molecular brain, ISSN 1756-6606, 2012, Volume 5, Issue 1, pp. 44 - 44
Background: Trigeminal neuropathic pain attacks can be excruciating for patients, even after being lightly touched. Although there are rodent trigeminal nerve... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Inflammation - pathology | Neurons - pathology | Neuralgia - complications | Tetrazoles - pharmacology | Minocycline - pharmacology | Male |