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PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, p. e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment. Current understanding... 
MACROPHAGE INFILTRATION | PAIN | INDUCED MECHANICAL ALLODYNIA | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | DORSAL-ROOT GANGLIA | SPINAL-CORD | NERVE INJURY | SENSORY NEURONS | SATELLITE GLIAL-CELLS | PACLITAXEL | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1
Journal Article
European Spine Journal, ISSN 0940-6719, 12/2009, Volume 18, Issue 12, pp. 1978 - 1985
Altered dorsal root ganglion (DRG) function is associated with neuropathic pain following spinal nerve injury. However, compression of the cauda equina and... 
Tumor necrosis factor alpha | Surgical Orthopedics | Medicine & Public Health | Neurosurgery | Dorsal root ganglion | Neuropathic pain | Apoptosis | NUCLEUS PULPOSUS | SELECTIVE-INHIBITION | MECHANICAL ALLODYNIA | NECROSIS-FACTOR-ALPHA | RAT SCIATIC-NERVE | MOTOR DYSFUNCTION | CLINICAL NEUROLOGY | DISC HERNIATION | GANGLION APOPTOSIS | TNF-ALPHA | ORTHOPEDICS | CHRONIC-CONSTRICTION-INJURY | Tumor Necrosis Factor-alpha - metabolism | Pain Threshold - physiology | Spondylosis - pathology | Spinal Nerves - pathology | Caspase 3 - metabolism | Nerve Degeneration - physiopathology | Ganglia, Spinal - physiopathology | Peripheral Nervous System Diseases - physiopathology | Neuralgia - pathology | Spondylosis - physiopathology | Hyperalgesia - pathology | Radiculopathy - pathology | Low Back Pain - physiopathology | Spinal Nerve Roots - physiopathology | Low Back Pain - etiology | Sensory Receptor Cells - physiology | Neuralgia - etiology | Radiculopathy - physiopathology | Female | Peripheral Nervous System Diseases - pathology | Neuralgia - physiopathology | Low Back Pain - pathology | Lumbar Vertebrae - physiopathology | Disease Models, Animal | Biomarkers - metabolism | Peripheral Nervous System Diseases - etiology | Spinal Nerves - physiopathology | Sensory Receptor Cells - pathology | Biomarkers - analysis | Rats | Behavior, Animal - physiology | Rats, Sprague-Dawley | Nerve Degeneration - pathology | Hyperalgesia - physiopathology | Tumor Necrosis Factor-alpha - analysis | Animals | Ganglia, Spinal - pathology | Caspase 3 - analysis | Hyperalgesia - etiology | Lumbar Vertebrae - pathology | Apoptosis - physiology | Spinal Nerve Roots - pathology | Nerve Degeneration - etiology | Pain Measurement | Medical colleges | Cell research | Comparative analysis | Neurons | Tumors | Original
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 12/2013, Volume 28, Issue 12, pp. 2512 - 2522
Journal Article
Human Reproduction, ISSN 0268-1161, 04/2016, Volume 31, Issue 4, pp. 734 - 749
Journal Article
Experimental Neurology, ISSN 0014-4886, 2007, Volume 203, Issue 1, pp. 42 - 54
Paclitaxel-induced peripheral neuropathy (PN) can be a significant problem for patients receiving chemotherapeutic regimens for the treatment of breast,... 
Axonopathy | Neurotoxicity | Breast cancer | Ganglionopathy | Neuropathic pain | Microglia | axonopathy | TACTILE ALLODYNIA | breast cancer | SPINAL-CORD | SATELLITE CELLS | OVARIAN-CANCER | BONE CANCER PAIN | ACTIVATING TRANSCRIPTION FACTOR-3 | ganglionopathy | DORSAL-ROOT GANGLION | NEUROSCIENCES | NERVE GROWTH-FACTOR | neuropathic pain | neurotoxicity | MESSENGER-RNA | SCIATIC-NERVE | microglia | Hyperalgesia - chemically induced | Microglia - metabolism | Posterior Horn Cells - drug effects | Peripheral Nervous System Diseases - chemically induced | Posterior Horn Cells - metabolism | Injections, Intravenous | Antigens, Differentiation, Myelomonocytic - drug effects | Male | CD11b Antigen | Peripheral Nervous System Diseases - physiopathology | Glial Fibrillary Acidic Protein - metabolism | Peripheral Nerves - drug effects | Satellite Cells, Perineuronal - metabolism | Antigens, CD - metabolism | Chemotaxis, Leukocyte - physiology | Hyperalgesia - pathology | Activating Transcription Factor 3 - drug effects | Paclitaxel - toxicity | Chemotaxis, Leukocyte - drug effects | Microglia - pathology | Neurons, Afferent - pathology | Peripheral Nervous System Diseases - pathology | Disease Models, Animal | Schwann Cells - drug effects | Neurons, Afferent - drug effects | Microglia - drug effects | Rats | Schwann Cells - pathology | Schwann Cells - metabolism | Antigens, CD - drug effects | Activating Transcription Factor 3 - metabolism | Peripheral Nerves - pathology | Rats, Sprague-Dawley | Hyperalgesia - physiopathology | Peripheral Nerves - physiopathology | Antineoplastic Agents, Phytogenic - toxicity | Satellite Cells, Perineuronal - drug effects | Macrophages - metabolism | Animals | Antigens, Differentiation, Myelomonocytic - metabolism | Glial Fibrillary Acidic Protein - drug effects | Ganglia, Spinal - pathology | Posterior Horn Cells - pathology | Macrophages - drug effects | Neurons, Afferent - metabolism | Ganglia, Spinal - drug effects | Satellite Cells, Perineuronal - pathology | Ganglia, Spinal - metabolism | Antimitotic agents | Chemotherapy | Infiltration (Military science) | Neurosciences | Neurons | Oncology, Experimental | Lung cancer | Research | Macrophages | Antineoplastic agents | Intermediate filament proteins | Cancer
Journal Article
Molecular Pain, ISSN 1744-8069, 5/2016, Volume 12, p. 174480691664678
Background Chronic pain is often accompanied by short-term memory deficit and depression. Currently, it is believed that short-term memory deficit and... 
short-term memory deficit | peripheral nerve injury | depression | microglia | interleukin-1β | Neuropathic pain | MIRROR-IMAGE PAIN | NECROSIS-FACTOR-ALPHA | BLOOD-BRAIN-BARRIER | SRC-FAMILY KINASES | SYNAPTIC PLASTICITY | NEUROSCIENCES | SPINAL DORSAL-HORN | interleukin-1 beta | LONG-TERM POTENTIATION | GROWTH-FACTOR | HIPPOCAMPAL NEUROGENESIS | WORKING-MEMORY | Microglia - metabolism | Sciatic Nerve - injuries | Injections, Intravenous | Depression - pathology | Neuralgia - metabolism | Memory Disorders - metabolism | Male | Neuralgia - pathology | Hyperalgesia - pathology | Interleukin-1beta - blood | Behavior, Animal | Depression - etiology | Depression - metabolism | Time Factors | Interleukin-1beta - metabolism | Peripheral Nerve Injuries - metabolism | Receptors, Interleukin-1 - deficiency | Gene Deletion | Neuralgia - etiology | Microglia - pathology | Sciatic Nerve - drug effects | Memory Disorders - pathology | Hyperalgesia - metabolism | Microglia - drug effects | Sciatic Nerve - pathology | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | Rats | Antibodies, Neutralizing - pharmacology | Rats, Sprague-Dawley | Receptors, Interleukin-1 - metabolism | Peripheral Nerve Injuries - etiology | Spinal Cord Dorsal Horn - drug effects | Animals | Memory Disorders - etiology | Spinal Cord Dorsal Horn - pathology | Signal Transduction - drug effects
Journal Article
Pharmacological Reports, ISSN 1734-1140, 04/2018, Volume 70, Issue 2, pp. 294 - 303
The purpose of this study was to evaluate the participation of satellite glial cells (SGC), microglia and astrocytes in a model of streptozotocin-induced... 
Diabetes | Astrocytes | nSTZ model | Satellite glial cells | Microglia | METFORMIN | NERVE LIGATION MODEL | TACTILE ALLODYNIA | MECHANISMS | INDUCTION | ACTIVATING TRANSCRIPTION FACTOR-3 | GABAPENTIN | INSULIN-RESISTANCE | PHARMACOLOGY & PHARMACY | HYPERALGESIA | Microglia - metabolism | Pain Threshold - physiology | Diabetes Mellitus, Experimental - drug therapy | Spinal Cord - drug effects | Spinal Cord - metabolism | Gabapentin | Rats, Wistar | Neuroglia - pathology | Astrocytes - pathology | Neuralgia - metabolism | Activating Transcription Factor 3 | Male | Neuralgia - pathology | Hyperalgesia - pathology | Neuroglia - drug effects | Spinal Cord - pathology | Microglia - pathology | Pain Threshold - drug effects | Diabetes Mellitus, Experimental - metabolism | Disease Models, Animal | Animals, Newborn | Astrocytes - drug effects | Hyperalgesia - metabolism | Microglia - drug effects | Metformin - pharmacology | Rats | gamma-Aminobutyric Acid - pharmacology | Diabetic Neuropathies - drug therapy | Streptozocin - pharmacology | Neuralgia - drug therapy | Pain Measurement - methods | Animals | Hyperalgesia - drug therapy | Cyclohexanecarboxylic Acids - pharmacology | Diabetic Neuropathies - pathology | Ganglia, Spinal - pathology | Diabetes Mellitus, Experimental - pathology | Neuroglia - metabolism | Diabetic Neuropathies - metabolism | Ganglia, Spinal - drug effects | Amines - pharmacology | Astrocytes - metabolism | Ganglia, Spinal - metabolism
Journal Article
Molecular Brain, ISSN 1756-6606, 2012, Volume 5, Issue 1, pp. 16 - 16
It has been reported that remarkable and sustained activation of astrocytes and/or microglia occurs in cancer induced pain (CIP), which is different from... 
Neuroinflammation | Cancer induced pain | Glial activation | Rat | Toll-like receptor 4 | CORD | INNATE IMMUNE-RESPONSE | INJURY | MODEL | CYTOKINE EXPRESSION | NEUROSCIENCES | NEUROPATHIC PAIN | TOLL-LIKE RECEPTOR-4 | CELL ACTIVATION | CENTRAL-NERVOUS-SYSTEM | Inflammation - pathology | Microglia - metabolism | Neoplasm Transplantation | Posterior Horn Cells - metabolism | Spinal Cord - metabolism | Rats, Wistar | Astrocytes - pathology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Bone Neoplasms - pathology | Tibia - pathology | Hyperalgesia - pathology | Behavior, Animal | Inflammation - complications | Pain - complications | Pain - genetics | Time Factors | Carcinoma 256, Walker - pathology | Spinal Cord - pathology | Bone Neoplasms - physiopathology | Carcinoma 256, Walker - physiopathology | Microglia - pathology | Female | Bone Neoplasms - genetics | Cytokines - genetics | Tibia - physiopathology | Cytokines - metabolism | Carcinoma 256, Walker - complications | Hyperalgesia - complications | RNA, Messenger - genetics | Gene Expression Regulation | Rats | Bone Neoplasms - complications | Pain - pathology | Toll-Like Receptor 4 - metabolism | Hyperalgesia - physiopathology | Animals | Hyperalgesia - genetics | Pain - physiopathology | Posterior Horn Cells - pathology | Inflammation - genetics | Pain Threshold | Spinal Cord - physiopathology | CD11b Antigen - metabolism | Astrocytes - metabolism | Hypertrophy | Inflammation - physiopathology | Cancer pain | Bone cancer | Inflammation | Instrument industry | RNA | Medical research | Neurosciences | Pain | Cytokines | Rodents | Software | Behavior | Manuscripts
Journal Article
Journal Article
Neuropharmacology, ISSN 0028-3908, 04/2017, Volume 116, pp. 315 - 327
Joint pain is a major clinical problem mainly associated to osteoarthritis, and characterized by articular cartilage degradation resulting in a complex chronic... 
Dynorphin | Opioids | Anhedonia | Anxiety | Cognition | Osteoarthritis | KOR | AGONIST SALVINORIN | INDUCED EXPERIMENTAL OSTEOARTHRITIS | C57BL/6 MICE | ENDOCANNABINOID SYSTEM | NEUROSCIENCES | KAPPA-OPIOID-RECEPTOR | RAT MODEL | NEUROPATHIC PAIN | CORTICOTROPIN-RELEASING-FACTOR | MOOD DISORDERS | PHARMACOLOGY & PHARMACY | MONOSODIUM IODOACETATE MODEL | Arthralgia - pathology | Microglia - metabolism | Spinal Cord - metabolism | Enkephalins - metabolism | Epigenesis, Genetic | Hyperalgesia - psychology | Hyperalgesia - pathology | Enkephalins - genetics | Spinal Cord - pathology | Cognition - physiology | Microglia - pathology | Acetylation | Osteoarthritis - pathology | Receptors, Opioid, kappa - genetics | Arthralgia - psychology | Disease Models, Animal | Emotions - physiology | Protein Precursors - genetics | Hyperalgesia - metabolism | Mice, Inbred C57BL | Amygdala - metabolism | Receptors, Opioid, kappa - metabolism | Osteoarthritis - metabolism | Hippocampus - pathology | Chronic Pain - psychology | Chronic Pain - pathology | Amygdala - pathology | Corticotropin-Releasing Hormone - metabolism | Mice, Knockout | Protein Precursors - metabolism | Chronic Pain - metabolism | Hippocampus - metabolism | Osteoarthritis - psychology | Animals | Arthralgia - metabolism | Histones - metabolism | Corticotropin releasing hormone | Epigenetic inheritance | Analysis | Arthralgia | ACTH | Missing in action | Chronic pain | Artrosi | Cognició | Angoixa | Dolor | Neuropèptids
Journal Article