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Gastroenterology, ISSN 0016-5085, 2009, Volume 137, Issue 6, pp. 2084 - 2095.e3
Background & Aims The transient receptor potential (TRP) channel family includes transducers of mechanical and chemical stimuli for visceral sensory neurons.... 
Gastroenterology and Hepatology | SENSITIZATION | PELVIC COLONIC AFFERENTS | INFLAMMATORY PAIN | MICE | HYPERALGESIA | COLD | GASTROENTEROLOGY & HEPATOLOGY | IRRITABLE-BOWEL-SYNDROME | SENSORY NEURONS | BRADYKININ | HYPERSENSITIVITY | Hyperalgesia - chemically induced | Immunohistochemistry | Intestinal Mucosa - metabolism | Intestinal Mucosa - physiopathology | Mechanotransduction, Cellular - drug effects | Capsaicin - pharmacology | Neuroanatomical Tract-Tracing Techniques | Receptor, PAR-2 - metabolism | Colon - drug effects | Afferent Pathways - physiopathology | Colon - innervation | Male | Ganglia, Spinal - physiopathology | Nodose Ganglion - metabolism | Trinitrobenzenesulfonic Acid | Pelvis - innervation | RNA, Messenger - metabolism | Transient Receptor Potential Channels - deficiency | Action Potentials | Nodose Ganglion - physiopathology | Splanchnic Nerves - physiopathology | In Situ Hybridization | Colitis - chemically induced | Nodose Ganglion - drug effects | Colon - physiopathology | Female | Transient Receptor Potential Channels - metabolism | Bradykinin - pharmacology | Disease Models, Animal | Afferent Pathways - metabolism | Hyperalgesia - metabolism | Mice, Inbred C57BL | Reverse Transcriptase Polymerase Chain Reaction | Pressure | Stimulation, Chemical | Colon - metabolism | Hyperalgesia - physiopathology | Mice, Knockout | Animals | Colitis - physiopathology | Transient Receptor Potential Channels - genetics | TRPA1 Cation Channel | Colitis - metabolism | Splanchnic Nerves - metabolism | Mice | Ganglia, Spinal - drug effects | Pain Measurement | Ganglia, Spinal - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Blood, ISSN 0006-4971, 2013, Volume 122, Issue 11, pp. 1853 - 1862
Journal Article
European Spine Journal, ISSN 0940-6719, 12/2009, Volume 18, Issue 12, pp. 1978 - 1985
Altered dorsal root ganglion (DRG) function is associated with neuropathic pain following spinal nerve injury. However, compression of the cauda equina and... 
Tumor necrosis factor alpha | Surgical Orthopedics | Medicine & Public Health | Neurosurgery | Dorsal root ganglion | Neuropathic pain | Apoptosis | NUCLEUS PULPOSUS | SELECTIVE-INHIBITION | MECHANICAL ALLODYNIA | NECROSIS-FACTOR-ALPHA | RAT SCIATIC-NERVE | MOTOR DYSFUNCTION | CLINICAL NEUROLOGY | DISC HERNIATION | GANGLION APOPTOSIS | TNF-ALPHA | ORTHOPEDICS | CHRONIC-CONSTRICTION-INJURY | Tumor Necrosis Factor-alpha - metabolism | Pain Threshold - physiology | Spondylosis - pathology | Spinal Nerves - pathology | Caspase 3 - metabolism | Nerve Degeneration - physiopathology | Ganglia, Spinal - physiopathology | Peripheral Nervous System Diseases - physiopathology | Neuralgia - pathology | Spondylosis - physiopathology | Hyperalgesia - pathology | Radiculopathy - pathology | Low Back Pain - physiopathology | Spinal Nerve Roots - physiopathology | Low Back Pain - etiology | Sensory Receptor Cells - physiology | Neuralgia - etiology | Radiculopathy - physiopathology | Female | Peripheral Nervous System Diseases - pathology | Neuralgia - physiopathology | Low Back Pain - pathology | Lumbar Vertebrae - physiopathology | Disease Models, Animal | Biomarkers - metabolism | Peripheral Nervous System Diseases - etiology | Spinal Nerves - physiopathology | Sensory Receptor Cells - pathology | Biomarkers - analysis | Rats | Behavior, Animal - physiology | Rats, Sprague-Dawley | Nerve Degeneration - pathology | Hyperalgesia - physiopathology | Tumor Necrosis Factor-alpha - analysis | Animals | Ganglia, Spinal - pathology | Caspase 3 - analysis | Hyperalgesia - etiology | Lumbar Vertebrae - pathology | Apoptosis - physiology | Spinal Nerve Roots - pathology | Nerve Degeneration - etiology | Pain Measurement | Medical colleges | Cell research | Comparative analysis | Neurons | Tumors | Original
Journal Article
Nature (London), ISSN 1476-4687, 2018, Volume 561, Issue 7724, pp. 547 - 550
Journal Article
Pain, ISSN 0304-3959, 06/2009, Volume 143, Issue 3, pp. 172 - 178
Females with Irritable Bowel Syndrome (IBS) and Temporomandibular Disorder (TMD) are characterized by enhanced sensitivity to experimental pain. One possible... 
Irritable Bowel Syndrome | Dysfunction | Temporomandibular Disorder | Pain modulation | Diffuse Noxious Inhibitory Control | Psychophysics | Focal heat pain | FIBROMYALGIA SYNDROME | TENSION-TYPE HEADACHE | STRESS-INDUCED ANALGESIA | HORN CONVERGENT NEURONS | EXPERIMENTALLY EVOKED PAIN | NEUROSCIENCES | CLINICAL NEUROLOGY | NOXIOUS INHIBITORY CONTROLS | VISCERAL HYPERSENSITIVITY | AGE-RELATED DIFFERENCES | TEMPORAL SUMMATION | ANESTHESIOLOGY | 2ND PAIN | Pain Threshold - physiology | Sensation Disorders - physiopathology | Humans | Sensation Disorders - psychology | Hyperalgesia - psychology | Young Adult | Neural Inhibition - physiology | Temporomandibular Joint Disorders - physiopathology | Irritable Bowel Syndrome - psychology | Adult | Female | Neural Pathways - physiopathology | Nerve Net - physiopathology | Sensation Disorders - complications | Hot Temperature - adverse effects | Conditioning (Psychology) - physiology | Hyperalgesia - complications | Irritable Bowel Syndrome - physiopathology | Temporomandibular Joint Disorders - psychology | Hyperalgesia - physiopathology | Temporomandibular Joint Disorders - complications | Pain Measurement - methods | Irritable Bowel Syndrome - complications | Adolescent | Central Nervous System - physiopathology | Observer Variation | Care and treatment | Pain | Irritable bowel syndrome | psychophysics | Diffuse noxious inhibitory control | focal heat pain | dysfunction
Journal Article