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Journal of Bone and Mineral Research, ISSN 0884-0431, 10/2017, Volume 32, Issue 10, pp. 2049 - 2061
Journal Article
Comparative Biochemistry and Physiology, Part C, ISSN 1532-0456, 11/2016, Volume 189, pp. 54 - 60
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 03/2010, Volume 298, Issue 3, pp. F485 - F499
Riccardi D, Brown EM. Physiology and pathophysiology of the calcium-sensing receptor in the kidney. Am J Physiol Renal Physiol 298: F485-F499, 2010. First... 
Activating mutation | Thick ascending limb | Autosomal dominant hypoparathyroidism | Familial hypocalciuric hypercalcemia | Distal convoluted tubule | Hypercalcemia | Calcimimetic | Proximal tubule | Neonatal severe primary hyperparathyroidism | Hypercalciuria | 1,25-dihydroxyvitamin D | Parathyroid hormone | Inactivating mutation | Collecting duct | Hyperparathyroidism | Hypocalcemia | Polymorphism | neonatal severe primary hyperparathyroidism | PHYSIOLOGY | BONE-MINERAL DENSITY | NEONATAL SEVERE HYPERPARATHYROIDISM | polymorphism | hyperparathyroidism | PARATHYROID-HORMONE SECRETION | proximal tubule | parathyroid hormone | EXTRACELLULAR CA2+-SENSING RECEPTOR | VASOPRESSIN-ELICITED WATER | VITAMIN-D METABOLISM | 1,25-dihydroxyvitamin D-3 | calcimimetic | AUTOSOMAL-DOMINANT HYPOCALCEMIA | thick ascending limb | THICK ASCENDING LIMBS | hypercalcemia | collecting duct | hypocalcemia | inactivating mutation | UROLOGY & NEPHROLOGY | autosomal dominant hypoparathyroidism | APICAL K+ CHANNELS | hypercalciuria | distal convoluted tubule | activating mutation | familial hypocalciuric hypercalcemia | Calcium - metabolism | Autoantibodies - blood | Humans | Homeostasis | Structure-Activity Relationship | Receptors, Calcium-Sensing - metabolism | Hyperparathyroidism, Primary - metabolism | Hyperparathyroidism, Primary - physiopathology | Kidney - metabolism | Parathyroid Hormone - metabolism | Kidney - physiopathology | Bartter Syndrome - metabolism | Kidney - drug effects | Hypercalciuria - physiopathology | Hypercalcemia - metabolism | Receptors, Calcium-Sensing - immunology | Animals | Hypercalcemia - physiopathology | Signal Transduction - drug effects | Hypercalciuria - metabolism | Receptors, Calcium-Sensing - drug effects | Protein Conformation | Receptors, Calcium-Sensing - genetics | Bartter Syndrome - physiopathology | Mutation | Parathyroid Glands - metabolism | Autoimmunity | Physiological aspects | Cell receptors | Kidneys | Research | 1,25-dihydroxyvitamin D3 | Reviews
Journal Article
American Journal of Physiology - Endocrinology And Metabolism, ISSN 0193-1849, 04/2012, Volume 302, Issue 8, pp. 1009 - 1015
Journal Article
Endocrinology, ISSN 0013-7227, 09/2013, Volume 154, Issue 9, pp. 3031 - 3042
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 11/2017, Volume 232, Issue 11, pp. 2985 - 2995
Patients with end‐stage renal disease (ESRD) have elevated circulating calcium (Ca) and phosphate (Pi), and exhibit accelerated progression of calcific aortic... 
Annexin VI | calcification | calcific aortic valve disease | matrix vesicles | PHYSIOLOGY | MECHANISM | PROTEOME | VASCULAR CALCIFICATION | ATHEROSCLEROSIS | CELL BIOLOGY | CALCIUM | MINERALIZATION | OSTEOBLASTS | SMOOTH-MUSCLE-CELLS | CHRONIC KIDNEY-DISEASE | PROGRESSION | Calcinosis - genetics | Up-Regulation | Aortic Valve Stenosis - genetics | Extracellular Vesicles - ultrastructure | Calcium - metabolism | Homeodomain Proteins - metabolism | Humans | Alkaline Phosphatase - metabolism | Extracellular Matrix - metabolism | Male | RNA, Messenger - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Extracellular Vesicles - metabolism | Aortic Valve - pathology | Protein Interaction Maps | Aortic Valve - ultrastructure | Calcinosis - etiology | Aortic Valve Stenosis - etiology | Female | Proteomics - methods | Calcinosis - metabolism | Microscopy, Electron, Transmission | Alkaline Phosphatase - genetics | Kidney Failure, Chronic - diagnosis | RNA, Messenger - genetics | Cells, Cultured | Aortic Valve - metabolism | Rats, Sprague-Dawley | Homeodomain Proteins - genetics | Annexin A6 - metabolism | Extracellular Matrix - ultrastructure | Animals | Aortic Valve Stenosis - pathology | Hypercalcemia - diagnosis | Hypercalcemia - etiology | Kidney Failure, Chronic - complications | Aortic Valve Stenosis - metabolism | Aged | Calcinosis - pathology | Core Binding Factor Alpha 1 Subunit - genetics | Phosphates | Heart valve diseases | Chronic kidney failure | RNA | Analysis | Calcification | Development and progression | Binding proteins | Electron microscopy | Protein binding | Enrichment | Calcification (ectopic) | End-stage renal disease | CD9 antigen | Media (enrichment) | Calcium | Interstitial cells | mRNA | Aldosterone | Proteins | Vesicles | Biomedical materials | Msx2 protein | Aorta | Extracellular matrix | Biocompatibility | Rheumatic heart disease | Localization | Binding | Cardiovascular system | LAMP-1 protein | Cbfa-1 protein | LAMP-2 protein | CD63 antigen | Matrix vesicles | Patients | Calcium phosphates | Hypercalcemia | Transmission electron microscopy | Proteomics | Kidney diseases | Position (location) | Aortic valve | Kidney transplantation | Original | Original s
Journal Article
Diabetologia, ISSN 0012-186X, 5/2018, Volume 61, Issue 5, pp. 1180 - 1192
Journal Article