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Paediatric Respiratory Reviews, ISSN 1526-0542, 2016, Volume 22, pp. 52 - 59
Summary Prader Willi syndrome, resulting from the partial deletion or lack of expression of a region of genes on the paternal chromosome 15, has a number of... 
Pediatrics | Pulmonary/Respiratory | growth hormone | Prader Willi syndrome | sleep disordered breathing | aspiration | OBSTRUCTIVE SLEEP-APNEA | ABNORMALITIES | PULMONARY-FUNCTION | DEATH | ADENOTONSILLECTOMY | GROWTH-HORMONE THERAPY | RESPIRATORY SYSTEM | INFANTS | BREATHING DISORDERS | PEDIATRICS | DIAGNOSTIC-CRITERIA | EXCESSIVE DAYTIME SLEEPINESS | Prader-Willi Syndrome - epidemiology | Muscle Hypotonia - epidemiology | Sleep Apnea, Central - epidemiology | Sleep Apnea, Obstructive - physiopathology | Sleep Apnea Syndromes - physiopathology | Sleep Apnea Syndromes - epidemiology | Disorders of Excessive Somnolence - physiopathology | Humans | Human Growth Hormone - therapeutic use | Scoliosis - physiopathology | Prader-Willi Syndrome - physiopathology | Sleep Apnea, Obstructive - epidemiology | Prader-Willi Syndrome - drug therapy | Scoliosis - epidemiology | Hypercapnia - physiopathology | Obesity - physiopathology | Sleep Apnea, Central - physiopathology | Death, Sudden | Hypoventilation - epidemiology | Hypoventilation - physiopathology | Disorders of Excessive Somnolence - epidemiology | Respiratory Aspiration - epidemiology | Muscle Hypotonia - physiopathology | Respiratory Aspiration - physiopathology | Obesity - epidemiology | Hypoxia - physiopathology | Sleep disorders | Complications and side effects | Somatotropin | Sleep | Prader-Willi syndrome | Children's hospitals | Children | Health aspects
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, p. e0167359
Therapeutic hypercapnia has the potential for neuroprotection after global cerebral ischemia. Here we further investigated the effects of different degrees of... 
FOCAL CEREBRAL ISCHEMIA/REPERFUSION | THERAPEUTIC HYPERCAPNIA | HEAD-INJURY | ADULT RATS | BARRIER PERMEABILITY | EDEMA | MULTIDISCIPLINARY SCIENCES | PROTEIN EXPRESSION | BLOOD-FLOW | PERINATAL-PERIOD | REOXYGENATION | Hypercapnia - physiopathology | Carbon Dioxide - metabolism | Carotid Artery, Common - physiopathology | Cerebrovascular Circulation - drug effects | Brain Ischemia - therapy | Humans | Blood Gas Analysis | Blood-Brain Barrier - physiopathology | Carbon Dioxide - therapeutic use | Rats | Brain Ischemia - physiopathology | Brain Injuries - therapy | Brain Injuries - physiopathology | Blood-Brain Barrier - drug effects | Animals | Brain Edema - chemically induced | Ligation | Brain Edema - physiopathology | Carotid Artery, Common - drug effects | Aquaporin 4 - genetics | Hypoxia - physiopathology | Brain Edema - blood | Disease Models, Animal | Hypercapnia | Hypoxia | Brain damage | Analysis | Risk factors | Aquaporin 4 | Brain | Neuroprotection | Flow cytometry | Traumatic brain injury | Carbon dioxide | Kinases | Blood | Western blotting | Proteins | Hypoxemia | Ischemia | Education | Cerebral blood flow | Rodents | Blood pressure | Carotid artery | Edema | Damage assessment | Neurons | Aquaporins | Cortex | Biotin | Permeability | Metabolism | Blood flow | DNA nucleotidylexotransferase | Cytometry | Ostomy | Brain research | Ventilation | Anesthesiology | Laboratory animals | Immunofluorescence | Brain injury | Hippocampus | Apoptosis
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