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PloS one, ISSN 1932-6203, 2008, Volume 3, Issue 8, p. e2986
...). Less is known about the implications of common genetic variation in the LDLR gene regarding the variability of cholesterol levels and risk of CAD. Methods... 
METAANALYSIS | THERAPY | EVENTS | MULTIDISCIPLINARY SCIENCES | PHENOTYPE | POLYMORPHISM | HYPERCHOLESTEROLEMIA | PREVALENCE | ASSOCIATION | Receptors, LDL - genetics | Oligonucleotide Array Sequence Analysis | Risk Assessment | Humans | Risk Factors | Cholesterol, LDL - drug effects | Chromosome Mapping | Random Allocation | Case-Control Studies | Genetic Variation | Cholesterol, LDL - genetics | Coronary Disease - prevention & control | Anticholesteremic Agents - therapeutic use | Coronary Disease - genetics | Cholesterol, LDL - blood | Polymorphism, Single Nucleotide | Chromosomes, Human, Pair 10 | Medical research | Hypercholesterolemia | Blood cholesterol | Low density lipoproteins | Genes | Medicine, Experimental | Genetic research | Genetics | Genetic aspects | Single nucleotide polymorphisms | Coronary heart disease | Risk factors | Lipoproteins (low density) | Risk | Lipids | Cardiovascular disease | Genomes | Single-nucleotide polymorphism | Gene polymorphism | Epidemiology | Consortia | Confidence intervals | Randomization | Population | Lipoprotein (low density) receptors | Children | Heart diseases | Statistical analysis | Mortality | Coronary artery | Health risks | Environmental health | Genetic diversity | Regression analysis | Metabolism | LDLR gene | Apolipoproteins | Coronary artery disease | Low density lipoprotein | Cholesterol | Studies | Genetic variance | Hospitals | Coronary vessels | Alleles | Biomarkers | Receptor density | Adults | Mutation | Polymorphism
Journal Article
Cell metabolism, ISSN 1550-4131, 2012, Volume 15, Issue 5, pp. 665 - 674
Nonalcoholic fatty liver disease (NAFLD) is associated with increased cardiovascular and liver-related mortality. NAFLD is characterized by both triglyceride... 
RAT-LIVER | RISK-FACTORS | MACROPHAGE APOPTOSIS | PROTEIN-KINASE | ENDOCRINOLOGY & METABOLISM | HMG-COA REDUCTASE | STEATOHEPATITIS | 3-HYDROXY-3-METHYLGLUTARYL COENZYME | PREVALENCE | MICRORNA EXPRESSION | MODULATION | CELL BIOLOGY | Sirtuin 1 - metabolism | Up-Regulation | Cholesterol - blood | Humans | Middle Aged | Sterol Esterase - metabolism | Male | MicroRNAs - metabolism | Desmosterol - metabolism | Cardiovascular Diseases - genetics | Cholesterol - genetics | Sirtuin 1 - genetics | Case-Control Studies | Phosphorylation - genetics | Hydroxymethylglutaryl CoA Reductases - metabolism | Adenylate Kinase - metabolism | Non-alcoholic Fatty Liver Disease | Sterol O-Acyltransferase - metabolism | Adult | Female | Lipid Metabolism - genetics | Sterol Regulatory Element Binding Protein 2 - genetics | Sterol Regulatory Element Binding Protein 2 - metabolism | Fatty Liver - genetics | Receptors, LDL - genetics | Gene Expression | Fatty Liver - metabolism | Cardiovascular Diseases - metabolism | Fatty Liver - blood | Liver - metabolism | Receptors, LDL - metabolism | Cholesterol - metabolism | Cholesterol, LDL - genetics | Phenotype | Sterol Esterase - genetics | Desmosterol - blood | Cholesterol, LDL - metabolism | MicroRNAs - genetics | Sterol O-Acyltransferase - genetics | Adenylate Kinase - genetics | Hydroxymethylglutaryl CoA Reductases - genetics | Enzymes | Liver diseases | Low density lipoproteins | Genes | Esters | Triglycerides | Cholesterol | MicroRNA | Fatty liver | Blood cholesterol | Physiological aspects | Hydrolases | Blood lipids | Health aspects | Statins | atherosclerosis | Nonalcoholic steatohepatitis | cholesterol | Nonalcoholic fatty liver disease | fatty liver | lipogenesis | hypercholesterolemia | HMG CoA reductase
Journal Article
Nature (London), ISSN 1476-4687, 2010, Volume 464, Issue 7287, pp. 409 - 412
Journal Article
by Debette, Stéphanie and Kamatani, Yoichiro and Metso, Tiina M and Kloss, Manja and Chauhan, Ganesh and Engelter, Stefan T and Pezzini, Alessandro and Thijs, Vincent and Markus, Hugh S and Dichgans, Martin and Wolf, Christiane and Dittrich, Ralf and Touzé, Emmanuel and Southerland, Andrew M and Samson, Yves and Abboud, Shérine and Béjot, Yannick and Caso, Valeria and Bersano, Anna and Gschwendtner, Andreas and Sessa, Maria and Cole, John and Lamy, Chantal and Medeiros, Elisabeth and Beretta, Simone and Bonati, Leo H and Grau, Armin J and Michel, Patrik and Majersik, Jennifer J and Sharma, Pankaj and Kalashnikova, Ludmila and Nazarova, Maria and Dobrynina, Larisa and Bartels, Eva and Guillon, Benoit and van den Herik, Evita G and Fernandez-Cadenas, Israel and Jood, Katarina and Nalls, Michael A and De Leeuw, Frank-Erik and Jern, Christina and Cheng, Yu-Ching and Werner, Inge and Metso, Antti J and Lichy, Christoph and Lyrer, Philippe A and Brandt, Tobias and Boncoraglio, Giorgio B and Wichmann, Heinz-Erich and Gieger, Christian and Johnson, Andrew D and Böttcher, Thomas and Castellano, Maurizio and Arveiler, Dominique and Ikram, M Arfan and Breteler, Monique M B and Padovani, Alessandro and Meschia, James F and Kuhlenbäumer, Gregor and Rolfs, Arndt and Worrall, Bradford B and Ringelstein, Erich-Bernd and Zelenika, Diana and Tatlisumak, Turgut and Lathrop, Mark and Leys, Didier and Amouyel, Philippe and Dallongeville, Jean and International Stroke Genetics Consortium and the CADISP group and CADISP Grp and Int Stroke Genetics Consortium and CADISP Group and Institutionen för neurovetenskap och fysiologi, sektionen för klinisk neurovetenskap och rehabilitering and Sahlgrenska akademin and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy and Institute of Neuroscience and Physiology, Department of Clinical Neuroscience and Rehabilitation
Nature genetics, ISSN 1546-1718, 2014, Volume 47, Issue 1, pp. 78 - 83
.... No confirmed genetic susceptibility factors have been identified using candidate gene approaches... 
VASCULAR RISK-FACTORS | IDENTIFIES 3 | METAANALYSIS | AORTIC-ANEURYSMS | GENE | GENETICS & HEREDITY | INTRACRANIAL ANEURYSM | LOCI | ISCHEMIC-STROKE | MIGRAINE | GENOME-WIDE ASSOCIATION | Myocardial Infarction - epidemiology | Follow-Up Studies | Humans | Middle Aged | Brain Ischemia - genetics | Microfilament Proteins - physiology | Male | Vertebral Artery Dissection - genetics | Adult | Female | Hypertension - epidemiology | Odds Ratio | Microfilament Proteins - genetics | Hypercholesterolemia - epidemiology | Migraine Disorders - epidemiology | Brain Ischemia - epidemiology | Genetic Predisposition to Disease | Genome-Wide Association Study | Risk Factors | Genetic Pleiotropy | Vertebral Artery Dissection - epidemiology | Carotid Artery, Internal, Dissection - epidemiology | Carotid Artery, Internal, Dissection - genetics | Obesity - epidemiology | Alleles | Polymorphism, Single Nucleotide | Finland - epidemiology | Genetic aspects | Research | Genetic susceptibility | Genomics | Risk factors | Hematoma | Studies | Stroke | Genealogy | Genomes | Genetic testing | Dissection | Chromosomes | Deoxyribonucleic acid--DNA | Meta-analysis | Bioengineering | Brain Ischemia | Neurons and Cognition | Neurobiology | Computer Vision and Pattern Recognition | Medical Imaging | Life Sciences | Carotid Artery, Internal, Dissection | Image Processing | Imaging | Computer Science | Hypertension | Obesity | Vertebral Artery Dissection | Signal and Image processing | Myocardial Infarction | Hypercholesterolemia | Microfilament Proteins | Migraine Disorders | Finland | Engineering Sciences | Medicinska grundvetenskaper | Basic Medicine
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2018, Volume 72, Issue 6, pp. 662 - 680
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 4, p. e94697
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e60729
...). FH is primarily caused by genetic variations in Low Density Lipoprotein Receptor (LDLR), Apolipoprotein B (APOB) or Proprotein Convertase Subtilisin/Kexin type 9... 
POPULATION | COMBINED HYPERLIPIDEMIA | GROWTH-HORMONE | RISK-FACTOR | CHOLESTEROL | UPSTREAM STIMULATORY FACTOR | MULTIDISCIPLINARY SCIENCES | LIPOPROTEIN RECEPTOR GENE | MUTATIONS | CORONARY-HEART-DISEASE | HARDY-WEINBERG EQUILIBRIUM | Receptors, LDL - genetics | Gene Frequency | Humans | Middle Aged | Genotype | Male | Apolipoproteins B - genetics | Malaysia | Proprotein Convertases - genetics | Hyperlipoproteinemia Type II - metabolism | Hyperlipoproteinemia Type II - pathology | Serine Endopeptidases - genetics | Adult | Female | Lipid Metabolism - genetics | Polymorphism, Single Nucleotide | Cohort Studies | Hyperlipoproteinemia Type II - genetics | Proprotein Convertase 9 | Hypercholesterolemia | Low density lipoproteins | Genes | Genetic aspects | Research | Single nucleotide polymorphisms | Cardiovascular diseases | Apolipoproteins | Cholesterol | Lipids | Risk | Cardiovascular disease | Single-nucleotide polymorphism | Family medical history | Mines | Apolipoprotein B | Atherosclerosis | Lipoprotein (low density) receptors | Heart diseases | Deoxyribonucleic acid--DNA | Health risks | Subtilisin | Genetic diversity | LDLR gene | Metabolism | Disease control | APOB gene | Hereditary diseases | Medicine | Studies | Genetic variance | Kexin | DNA microarrays | Genotyping | Growth hormones | Receptor density | Mutation | Genetic testing | Risk management | Deoxyribonucleic acid | DNA
Journal Article
Nature communications, ISSN 2041-1723, 2014, Volume 5, Issue 1, p. 3983
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology: Journal of the American Heart Association, ISSN 1079-5642, 06/2004, Volume 24, Issue 6, pp. 1062 - 1067
OBJECTIVE—To investigate the effect of complement deficiency on atherogenesis and lipidemia, we used mice deficient in the third complement component (C3−/−)... 
Complement | Factor B | Hyperlipidemia | Atherosclerosis | atherosclerosis | hyperlipidemia | ACTIVATION | C-3 | COMPONENTS | PUTATIVE THIOESTER | factor B | DEFICIENCY | ALTERNATIVE PATHWAY | LDL | PERIPHERAL VASCULAR DISEASE | ACYLATION-STIMULATING PROTEIN | complement | HEMATOLOGY | ASP | ADIPOSE-TISSUE | Apolipoproteins E - deficiency | Aortic Diseases - blood | Male | Arteriosclerosis - genetics | Complement C3 - deficiency | Complement Factor B - genetics | Receptors, LDL - deficiency | Cholesterol, LDL - blood | Female | Complement Pathway, Alternative - genetics | Arteriosclerosis - blood | Complement C3 - genetics | Complement Factor B - deficiency | Aortic Diseases - pathology | Receptors, LDL - genetics | Complement Factor B - physiology | Genetic Predisposition to Disease | Hyperlipoproteinemia Type II - blood | Hyperlipoproteinemia Type IV - genetics | Mice, Knockout | Hyperlipoproteinemia Type IV - blood | Aortic Diseases - genetics | Animals | Lipoproteins, VLDL - blood | Apolipoproteins E - genetics | Triglycerides - blood | Mice | Arteriosclerosis - pathology | Complement Pathway, Classical - genetics | Complement C3 - physiology | Crosses, Genetic | Hyperlipoproteinemia Type II - genetics | Research Support | Comparative Study | Genetic | Apolipoproteins E/deficiency/genetics | LDL Cholesterol/blood | Alternative/genetics | MEDICIN OCH HÄLSOVETENSKAP | Arteriosclerosis/blood/ genetics/pathology | Crosses | Receptors | Hyperlipoproteinemia Type IV/blood/genetics | VLDL/blood | MEDICAL AND HEALTH SCIENCES | Aortic Diseases/blood/ genetics/pathology | Complement Pathway | Complement C3/ deficiency/genetics/physiology | Knockout | Classical/genetics | Lipoproteins | Hypercholesterolemia | LDL/deficiency/genetics | Complement Factor B/ deficiency/genetics/physiology | Familial/blood/genetics | Triglycerides/blood | Non-U.S. Gov't
Journal Article