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Obstetrics and Gynecology, ISSN 0029-7844, 2013, Volume 121, Issue 1, pp. 97 - 105
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 01/2010, Volume 31, Issue 1, pp. 1 - 7
Journal Article
Brain Research, ISSN 0006-8993, 2007, Volume 1188, Issue 1, pp. 122 - 131
Abstract Elevated homocysteine levels resulting from vitamin B deficiencies have been hypothesized to contribute to functional decline. To investigate the... 
Neurology | Motor activity | Motor coordination | Anxiety | Vitamin B12 | Homocysteine | Folate | Spatial learning | homocysteine | NERVOUS-SYSTEM | NEURODEGENERATIVE DISORDERS | FOLATE-DEFICIENCY | ALZHEIMERS-DISEASE | DNA-DAMAGE | RATS | METHYLENETETRAHYDROFOLATE-REDUCTASE | spatial learning | FOLIC-ACID | NEUROSCIENCES | FRUIT BATS | motor activity | anxiety | HOMOCYSTEINE LEVELS | folate | vitamin B12 | motor coordination | Folic Acid Deficiency - metabolism | Memory Disorders - physiopathology | Age Factors | Hyperhomocysteinemia - metabolism | Memory Disorders - metabolism | Learning Disorders - physiopathology | Male | Hyperkinesis - physiopathology | Brain - growth & development | Hyperkinesis - metabolism | Learning Disorders - etiology | Eating - physiology | Brain - metabolism | Space Perception - physiology | Exploratory Behavior - physiology | Hyperhomocysteinemia - physiopathology | Movement - physiology | Vitamin B 12 Deficiency - complications | Female | Vitamin B 12 Deficiency - metabolism | Motor Skills Disorders - etiology | Disease Models, Animal | Brain - physiopathology | Drinking - physiology | Homocysteine - blood | Vitamin B 12 Deficiency - physiopathology | Hyperhomocysteinemia - etiology | Folic Acid Deficiency - physiopathology | Motor Skills Disorders - metabolism | Movement Disorders - metabolism | Animals | Folic Acid Deficiency - complications | Memory Disorders - etiology | Movement Disorders - physiopathology | Aging - physiology | Movement Disorders - etiology | Learning Disorders - metabolism | Mice | Motor Skills Disorders - physiopathology | Vitamin B Deficiency - physiopathology | Body Weight - physiology | Vitamin B Deficiency - psychology | Youth | Teenagers | Diet | Folic acid | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 08/2003, Volume 108, Issue 8, pp. 933 - 938
Journal Article
Journal of Neural Transmission, ISSN 0300-9564, 5/2007, Volume 114, Issue 5, pp. 571 - 575
Pseudoexfoliation syndrome (PEX) is a systemic disorder characterized by the deposition of an abnormal fibrillar material in ocular and various extraocular... 
Neurology | Medicine & Public Health | Glaucoma, exfoliation syndrome, vitamins, homocysteine, malabsorption | Pharmacology/Toxicology | Psychiatry | Glaucoma | Exfoliation syndrome | Malabsorption | Homocysteine | Vitamins | homocysteine | exfoliation syndrome | glaucoma | ALZHEIMERS-DISEASE | AQUEOUS-HUMOR | COMPONENTS | NEUROSCIENCES | PEPTIDE | CLINICAL NEUROLOGY | OPEN-ANGLE GLAUCOMA | vitamins | RISK-FACTOR | malabsorption | FIBRILLOPATHY | FOLATE | Exfoliation Syndrome - blood | Glaucoma - etiology | Eye - pathology | Prospective Studies | Vitamin B 12 Deficiency - blood | Humans | Middle Aged | Exfoliation Syndrome - etiology | Male | Case-Control Studies | Hyperhomocysteinemia - physiopathology | Vitamin B Deficiency - blood | Vitamin B 12 Deficiency - complications | Female | Vitamin B 6 Deficiency - complications | Glaucoma - physiopathology | Folic Acid Deficiency - blood | Eye - metabolism | Hyperhomocysteinemia - blood | Exfoliation Syndrome - physiopathology | Vitamin B 6 Deficiency - blood | Vitamin B 6 Deficiency - physiopathology | Vitamin B 12 Deficiency - physiopathology | Folic Acid Deficiency - physiopathology | Glaucoma - blood | Folic Acid Deficiency - complications | Vitamin B Deficiency - complications | Aged | Vitamin B Deficiency - physiopathology | Hyperhomocysteinemia - complications | Eye - physiopathology | Evaluation | Ophthalmology | Index Medicus
Journal Article
Neurotoxicology, ISSN 0161-813X, 05/2015, Volume 48, pp. 44 - 49
N O exposure of 1.5 L/min for 90 min for 1 month results in myelin loss in spinal cord and subcortical white mater. Nitrous oxide (N O) toxicity can result in... 
Cobalamin | Nitrous oxide | Hyperhomocysteinemia | Myelin loss | Homocysteine | SUBACUTE COMBINED DEGENERATION | TOTALLY GASTRECTOMIZED RATS | PROLONGED EXPOSURE | NEUROSCIENCES | DEFICIENCY | DAMAGE | MYELONEUROPATHY | VITAMIN-B12 | SYNTHASE | MULTIPLE-SCLEROSIS | GLUTATHIONE | PHARMACOLOGY & PHARMACY | TOXICOLOGY | Spinal Cord - metabolism | Rats, Wistar | Vitamin B 12 Deficiency - psychology | Vitamin B 12 Deficiency - blood | Antioxidants - metabolism | Male | Vitamin B 12 Deficiency - pathology | Motor Activity | Brain - metabolism | Hyperhomocysteinemia - physiopathology | Time Factors | Vitamin B 12 Deficiency - chemically induced | Spinal Cord - pathology | Behavior, Animal - drug effects | Demyelinating Diseases - chemically induced | Demyelinating Diseases - psychology | Demyelinating Diseases - pathology | Muscle Strength | Hyperhomocysteinemia - psychology | Disease Models, Animal | Demyelinating Diseases - physiopathology | Demyelinating Diseases - blood | Hyperhomocysteinemia - blood | Hyperhomocysteinemia - pathology | Brain - physiopathology | Homocysteine - blood | Vitamin B 12 Deficiency - physiopathology | Vitamin B 12 - blood | Animals | Brain - pathology | Nitrous Oxide | Spinal Cord - physiopathology | Oxidative Stress - drug effects | Hyperhomocysteinemia - chemically induced | Glutathione - blood | Anesthetics | Antioxidants | Oxidative stress | Folic acid | Index Medicus
Journal Article
FASEB Journal, ISSN 0892-6638, 02/2015, Volume 29, Issue 2, pp. 418 - 432
Journal Article
European Journal of Nutrition, ISSN 1436-6207, 3/2018, Volume 57, Issue 2, pp. 557 - 568
The aim of this trial was to investigate the influence of a dietetic product consisting of a unique combination of l-arginine with the vitamins B6, folic acid... 
Chemistry | Nutrition | Atherosclerosis | Blood pressure | Endothelial function | l -Arginine | B vitamins | l-Arginine | L-Arginine | OXIDATIVE STRESS | FOLIC-ACID | SUPPLEMENTATION | ASYMMETRICAL DIMETHYLARGININE | NUTRITION & DIETETICS | GLUCOSE-METABOLISM | VASCULAR FUNCTION | NITRIC-OXIDE | CARDIOVASCULAR-DISEASE | DOUBLE-BLIND | DYSFUNCTION | Blood Pressure | Overweight - complications | Follow-Up Studies | Humans | Middle Aged | Essential Hypertension - complications | Male | Hyperhomocysteinemia - physiopathology | Prehypertension - complications | Female | Dietary Supplements - adverse effects | Hypertension - diet therapy | Body Mass Index | Severity of Illness Index | Vascular Resistance | Vitamin B Complex - adverse effects | Double-Blind Method | Vitamin B Complex - therapeutic use | Endothelium, Vascular - physiopathology | Disease Progression | Essential Hypertension - physiopathology | Hypertension - physiopathology | Essential Hypertension - diet therapy | Arginine - adverse effects | Hypertension - complications | Hyperhomocysteinemia - diet therapy | Prehypertension - physiopathology | Arginine - therapeutic use | Blood Pressure Monitoring, Ambulatory | Hyperhomocysteinemia - complications | Prehypertension - diet therapy | Cohort Studies | Hypertension | Diet therapy | Arginine | Analysis | Homocysteine | Folic acid | Endothelium | Statistical analysis | Vitamin B12 | Vitamin B6 | Vitamins | Index Medicus | Original Contribution
Journal Article
Diabetes, ISSN 0012-1797, 12/2015, Volume 64, Issue 12, pp. 4260 - 4271
We determined the mechanism of severity during type 1 diabetic (T1D) stroke (ischemia-reperfusion [IR] injury) that affects potential markers associated with... 
HYPERHOMOCYSTEINEMIA | ACTIVATION | DNA METHYLATION | METABOLIC MEMORY | CEREBRAL-ARTERY OCCLUSION | AKITA MICE | ENDOCRINOLOGY & METABOLISM | MELLITUS | FOCAL ISCHEMIA | BLOOD-BRAIN-BARRIER | EXPRESSION | Neurons - pathology | Vascular Remodeling | Capillary Permeability | Neuroglia - pathology | Epigenesis, Genetic | Blood-Brain Barrier - physiopathology | Male | Stroke - physiopathology | DNA (Cytosine-5-)-Methyltransferases - metabolism | Stroke - pathology | Diabetic Angiopathies - physiopathology | Matrix Metalloproteinase 9 - metabolism | Diabetic Angiopathies - pathology | Matrix Metalloproteinase 9 - genetics | Mice, Mutant Strains | Cell Death | Neuroglia - immunology | Neurons - metabolism | Reperfusion Injury - metabolism | Stroke - immunology | Disease Models, Animal | Endothelium, Vascular - immunology | Biomarkers - metabolism | Diabetic Angiopathies - metabolism | Blood-Brain Barrier - immunology | Brain Edema - etiology | Reperfusion Injury - pathology | Mice, Inbred C57BL | Endothelium, Vascular - physiopathology | Neurons - immunology | Nerve Tissue Proteins - genetics | Blood-Brain Barrier - metabolism | Nerve Tissue Proteins - metabolism | Blood-Brain Barrier - pathology | DNA (Cytosine-5-)-Methyltransferases - genetics | Stroke - metabolism | Animals | Endothelium, Vascular - metabolism | Diabetic Angiopathies - immunology | Reperfusion Injury - immunology | Reperfusion Injury - physiopathology | Endothelium, Vascular - pathology | Neuroglia - metabolism | Nitric Oxide Synthase - metabolism | Epigenetic inheritance | Care and treatment | Ischemia | Type 1 diabetes | Analysis | Influence | Research | Gene expression | Epigenetics | Diabetes | Permeability | Rodents | Nitric oxide | Index Medicus | Abridged Index Medicus | Complications
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, pp. e63710 - e63710
Background and aims: Homocysteine levels predict heart failure incidence in prospective epidemiological studies and correlate with severity of heart failure in... 
IMPAIRS ANGIOGENESIS | ENDOTHELIAL PROGENITOR CELLS | MURINE MODEL | HYPERHOMOCYSTEINEMIA LEADS | INNATE IMMUNE-RESPONSES | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | NITRIC-OXIDE | APO A-I | STRESS | PLASMA HOMOCYSTEINE | Myocardial Infarction - genetics | Homocystinuria - therapy | Hyperhomocysteinemia - therapy | Heart Failure - physiopathology | Heart Failure - therapy | Myocardial Infarction - therapy | Homocystinuria - physiopathology | Hyperhomocysteinemia - physiopathology | Hypercholesterolemia - pathology | Diet, High-Fat | Myocardial Infarction - pathology | Receptors, LDL - deficiency | Adenoviridae - genetics | Female | Myocardial Infarction - physiopathology | Hypercholesterolemia - therapy | Cystathionine beta-Synthase - genetics | Transgenes | Heart - physiopathology | Receptors, LDL - genetics | Hyperhomocysteinemia - pathology | Mice, Inbred C57BL | Homocysteine - blood | Heart Failure - genetics | Cystathionine beta-Synthase - metabolism | Heart Failure - pathology | Ventricular Remodeling - physiology | Diastole - physiology | Homocystinuria - pathology | Mice, Knockout | Animals | Hyperhomocysteinemia - genetics | Hypercholesterolemia - physiopathology | Mice | Hypercholesterolemia - genetics | Genetic Vectors | Homocystinuria - genetics | Surveys | Heart failure | Heart | Hypercholesterolemia | Collagen | Genes | Homocysteine | Epidemiology | Heart attack | Cholesterol | Myocardial infarction | Oxidative stress | Animal models | Heart attacks | High cholesterol diet | Cardiovascular disease | Biology | Kinases | Remodeling | Angiogenesis | Reduction | Ischemia | Rodents | Attenuation | Lipoprotein (low density) receptors | Capillaries | Heart diseases | Time constant | Failure | Gene transfer | Coronary artery | Cardiomyocytes | Studies | Lipoproteins | Diet | Correlation analysis | Nitric oxide | Myocardium | Low density lipoprotein receptors | Receptor density | Healing | Infarction | Ventricle | Index Medicus
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 08/2013, Volume 1832, Issue 8, pp. 1149 - 1158
Elevated levels of homocysteine produce detrimental effects in humans but its role in preterm birth is not known. Here we used a mouse model of... 
Prostaglandin E2 | Niacin receptor | Preterm birth | Cyclooxygenase-2 | Homocysteine | LARGE-CONDUCTANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PREVENTION | CYSTATHIONINE BETA-SYNTHASE | MECHANISMS | FOLIC-ACID | VASCULAR-DISEASE | NICOTINIC-ACID | HOMOCYSTEINE TOXICITY | BIOPHYSICS | DEFICIENT | K+ CHANNELS | Uterus - pathology | Up-Regulation | Premature Birth - genetics | Pregnancy Complications - blood | Receptors, Oxytocin - genetics | Humans | Uterus - metabolism | Hyperhomocysteinemia - metabolism | Homocysteine - metabolism | Premature Birth - blood | Homocysteine - genetics | Hyperhomocysteinemia - physiopathology | Premature Birth - physiopathology | Cyclooxygenase 2 - genetics | Myometrium - metabolism | Uterus - physiopathology | Female | Cystathionine beta-Synthase - genetics | Cell Line | Receptors, Nicotinic - metabolism | Trophoblasts - metabolism | Cyclooxygenase 2 Inhibitors - pharmacology | Dinoprostone - genetics | Mice, Inbred C57BL | Homocysteine - blood | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - metabolism | Cystathionine beta-Synthase - metabolism | Pregnancy Complications - physiopathology | Dinoprostone - metabolism | Placenta - metabolism | Pregnancy | Placenta - physiopathology | Receptors, Oxytocin - metabolism | Animals | Muscle Contraction | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - genetics | Myometrium - physiopathology | Cyclooxygenase 2 - metabolism | Pregnancy Complications - genetics | Hyperhomocysteinemia - genetics | Mice | Receptors, Nicotinic - genetics | Pregnant women | Analysis
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/1998, Volume 18, Issue 4, pp. 535 - 541
The incremental elastic modulus of the common carotid and radial arteries is increased in patients with end-stage renal disease (ESRD), independently of blood... 
Pulse wave velocity | End-stage renal disease | Endothelin | Homocysteine | homocysteine