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Critical Care Medicine, ISSN 0090-3493, 06/2010, Volume 38 Suppl, Identification and Prevention of Common Adverse Drug Events in the Intensive Care Unit, Issue 6, pp. S253 - S264
Electrolyte imbalances are common in critically ill patients. Although multiple disease states typically encountered in the intensive care unit may be... 
Hypercalcemia | Critical illness | Hyperkalemia | Hyponatremia | Hypernatremia | Hypermagnesemia | Hyperphosphatemia | Hypokalemia | Hypophosphatemia | Water-electrolyte imbalance | Hypocalcemia | Hypomagnesemia | HOSPITALIZED-PATIENTS | hypernatremia | critical illness | hyperkalemia | AMPHOTERICIN-B NEPHROTOXICITY | DRUG-INDUCED HYPERKALEMIA | CONVERTING ENZYME-INHIBITORS | hypercalcemia | CATION-EXCHANGE RESIN | hypokalemia | hypocalcemia | hyperphosphatemia | hyponatremia | HEPARIN-INDUCED HYPERKALEMIA | TRIMETHOPRIM-SULFAMETHOXAZOLE THERAPY | hypomagnesemia | hypermagnesemia | RENAL TUBULAR-ACIDOSIS | NEPHROGENIC DIABETES-INSIPIDUS | hypophosphatemia | CRITICALLY-ILL PATIENTS | water-electrolyte imbalance | CRITICAL CARE MEDICINE | Critical Care - methods | Hypernatremia - physiopathology | Hyponatremia - chemically induced | Hypocalcemia - physiopathology | Magnesium Deficiency - physiopathology | United States | Humans | Hypocalcemia - chemically induced | Magnesium Deficiency - chemically induced | Hypokalemia - chemically induced | Hyponatremia - physiopathology | Hypophosphatemia - chemically induced | Hypokalemia - physiopathology | Intensive Care Units | Hyperphosphatemia - chemically induced | Water-Electrolyte Imbalance - chemically induced | Hyperkalemia - physiopathology | Drug-Related Side Effects and Adverse Reactions - etiology | Magnesium - metabolism | Hypercalcemia - chemically induced | Hypernatremia - chemically induced | Hypercalcemia - physiopathology | Hypophosphatemia - physiopathology | Water-Electrolyte Imbalance - physiopathology | Hyperkalemia - chemically induced | Hyperphosphatemia - physiopathology | Drug-Related Side Effects and Adverse Reactions - physiopathology | Index Medicus | Abridged Index Medicus
Journal Article
Expert Opinion on Drug Metabolism & Toxicology, ISSN 1742-5255, 12/2012, Volume 8, Issue 12, pp. 1531 - 1548
Introduction: Pancreas-kidney transplant is an effective treatment for patients with insulin-dependent dabetes and chronic renal failure. Reduction in... 
pancreas transplant | immunosuppressive drugs | toxicological effects | metabolic adverse effects | Immunosuppressive drugs | Toxicological effects | Metabolic adverse effects | Pancreas transplant | CALCINEURIN-INHIBITOR WITHDRAWAL | RISK-FACTORS | BONE-MINERAL DENSITY | POSTTRANSPLANTATION DIABETES-MELLITUS | BIOCHEMISTRY & MOLECULAR BIOLOGY | COATED MYCOPHENOLATE SODIUM | RENAL-TRANSPLANTATION | INSULIN SENSITIVITY | KIDNEY-TRANSPLANTATION | GLUCOSE-METABOLISM | PHARMACOLOGY & PHARMACY | TOXICOLOGY | RABBIT ANTITHYMOCYTE GLOBULIN | Pneumonia - physiopathology | Gastrointestinal Diseases - physiopathology | Renal Insufficiency, Chronic - etiology | Pancreas Transplantation | Humans | Immunosuppressive Agents - pharmacokinetics | Gastrointestinal Diseases - etiology | Bone Diseases - etiology | Diabetes Mellitus, Type 2 - etiology | Nervous System Diseases - physiopathology | Sirolimus - adverse effects | Hyperkalemia - etiology | Cardiovascular Diseases - etiology | Graft Rejection - drug therapy | Cardiovascular Diseases - physiopathology | Pneumonia - etiology | Hyperkalemia - physiopathology | Mycophenolic Acid - pharmacokinetics | Hyperuricemia - physiopathology | Bone Diseases - physiopathology | Sirolimus - pharmacokinetics | Hyperuricemia - etiology | Renal Insufficiency, Chronic - physiopathology | Adrenal Cortex Hormones - adverse effects | Mycophenolic Acid - adverse effects | Diabetes Mellitus, Type 2 - physiopathology | Hematologic Diseases - physiopathology | Hematologic Diseases - etiology | Nervous System Diseases - etiology | Immunosuppressive Agents - adverse effects | Adrenal Cortex Hormones - pharmacokinetics | Graft Survival - drug effects | Index Medicus
Journal Article
American Journal of Kidney Diseases, ISSN 0272-6386, 2014, Volume 64, Issue 2, pp. 230 - 238
Journal Article
European Heart Journal, ISSN 0195-668X, 11/2012, Volume 33, Issue 22, pp. 2782 - U18
Mineralocorticoid receptor antagonists (MRAs) improve survival and reduce morbidity in patients with heart failure, reduced ejection fraction (HFREF), and... 
Heart failure | Aldosterone antagonist spironolactone | Mineralocorticoid receptors | SERUM POTASSIUM LEVELS | CARDIAC & CARDIOVASCULAR SYSTEMS | POSTMYOCARDIAL INFARCTION | ANGIOTENSIN-ALDOSTERONE SYSTEM | ENDOTHELIAL FUNCTION | ATRIAL-FIBRILLATION | LEFT-VENTRICULAR DYSFUNCTION | ACUTE MYOCARDIAL-INFARCTION | SUDDEN CARDIAC DEATH | CHRONIC KIDNEY-DISEASE | RANDOMIZED ALDACTONE EVALUATION | Endomyocardial Fibrosis - etiology | Evidence-Based Practice | Glycated Hemoglobin A - metabolism | Humans | Heart Failure - physiopathology | Hydrocortisone - metabolism | Arrhythmias, Cardiac - physiopathology | Dose-Response Relationship, Drug | Kidney Diseases - chemically induced | Mineralocorticoid Receptor Antagonists - pharmacology | Risk Factors | Kaplan-Meier Estimate | Rats | Stroke Volume - drug effects | Treatment Outcome | Arrhythmias, Cardiac - etiology | Ventricular Remodeling - physiology | Ventricular Dysfunction, Left - physiopathology | Heart Failure - drug therapy | Mineralocorticoid Receptor Antagonists - therapeutic use | Randomized Controlled Trials as Topic | Myocardial Infarction - complications | Animals | Ventricular Dysfunction, Left - drug therapy | Dogs | Hyperkalemia - chemically induced | Endomyocardial Fibrosis - physiopathology | Mice | Practice Guidelines as Topic | Klinisk medicin | Clinical Medicine
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 03/2015, Volume 116, Issue 6, pp. 960 - 975
Blockers of the renin–angiotensin–aldosterone system (RAAS), that is, renin inhibitors, angiotensin (Ang)-converting enzyme (ACE) inhibitors, Ang II type 1... 
remodeling | gender | aldosterone | AT2 receptor | hyperkalemia | resistant hypertension | receptor | renin | primary aldosteronism | reactive oxygen species | angiotensin | angiotensin-(1-7) | prorenin | angiotensin-(1–7) | CARDIAC & CARDIOVASCULAR SYSTEMS | BLOOD-PRESSURE RESPONSES | PLASMA-RENIN | CONVERTING ENZYME-INHIBITORS | CORONARY VASCULAR BED | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | II TYPE-2 RECEPTOR | HEMATOLOGY | MYOCARDIAL-INFARCTION | AT 2 receptor | CELL MINERALOCORTICOID RECEPTORS | SEX-DIFFERENCES | CONGESTIVE-HEART-FAILURE | Antihypertensive Agents - pharmacology | Drugs, Investigational - pharmacology | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Humans | Drugs, Investigational - therapeutic use | Endothelium, Vascular - drug effects | Hypertension - drug therapy | Male | Ion Channels - physiology | Sex Chromosomes | Molecular Targeted Therapy | Gonadal Steroid Hormones - physiology | Vasoconstriction - physiology | Drug Interactions | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Hyperaldosteronism - physiopathology | Female | Hypertension - genetics | Drug Evaluation, Preclinical | Drug Resistance | Precision Medicine | Aldosterone - physiology | Endothelium, Vascular - physiopathology | Hyperaldosteronism - genetics | Mice, Transgenic | Renin-Angiotensin System - genetics | Renin-Angiotensin System - physiology | Clinical Trials as Topic | Sex Characteristics | Antihypertensive Agents - therapeutic use | Vasoconstriction - drug effects | Hyperaldosteronism - drug therapy | Hypertension - physiopathology | Mice, Knockout | Mineralocorticoid Receptor Antagonists - therapeutic use | Animals | Renin-Angiotensin System - drug effects | Mice | Therapeutic Equivalency
Journal Article
Journal of Applied Physiology, ISSN 8750-7587, 07/2009, Volume 107, Issue 1, pp. 275 - 282
Journal Article