X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (487) 487
male (358) 358
index medicus (305) 305
hyperoxaluria (263) 263
urology & nephrology (250) 250
female (232) 232
animals (223) 223
primary hyperoxaluria (154) 154
oxalic acid (152) 152
adult (145) 145
oxalates (145) 145
calcium oxalate (138) 138
rats (136) 136
kidney - pathology (122) 122
nephrolithiasis (122) 122
calcium oxalate - metabolism (112) 112
kidney stones (110) 110
hyperoxaluria, primary - complications (102) 102
middle aged (99) 99
hyperoxaluria, primary - pathology (95) 95
transplantation (93) 93
hyperoxaluria - pathology (92) 92
oxalate (91) 91
nephrology (88) 88
urolithiasis (88) 88
crystallization (84) 84
kidney transplantation (80) 80
oxalosis (79) 79
biopsy (78) 78
hyperoxaluria - complications (77) 77
disease models, animal (75) 75
hyperoxaluria, primary - diagnosis (73) 73
kidneys (69) 69
kidney - metabolism (68) 68
medicine & public health (68) 68
child (67) 67
hyperoxaluria - metabolism (65) 65
diagnosis (62) 62
oxalates - metabolism (60) 60
pathology (60) 60
rats, wistar (59) 59
urine (59) 59
adolescent (57) 57
disease (56) 56
hyperoxaluria, primary - genetics (56) 56
article (55) 55
child, preschool (55) 55
infant (55) 55
ethylene glycol (54) 54
kidney calculi - pathology (53) 53
oxidative stress (53) 53
rats, sprague-dawley (53) 53
hyperoxaluria - etiology (52) 52
kidney diseases (51) 51
calcium (49) 49
urology (48) 48
oxalates - urine (47) 47
kidney (46) 46
liver (46) 46
liver transplantation (46) 46
kidney failure, chronic - etiology (45) 45
kidney tubules - pathology (45) 45
pediatrics (45) 45
rodents (45) 45
risk factors (44) 44
hyperoxaluria, primary - metabolism (43) 43
aged (42) 42
hyperoxaluria - chemically induced (42) 42
primary hyperoxaluria type-1 (41) 41
kidney calculi - etiology (40) 40
mutation (40) 40
renal dialysis (40) 40
enzymes (39) 39
kidney - drug effects (38) 38
crystals (37) 37
analysis (36) 36
calcium oxalate - urine (36) 36
children (36) 36
surgery (36) 36
treatment outcome (36) 36
lipid-peroxidation (35) 35
metabolism (35) 35
care and treatment (34) 34
expression (34) 34
nephrocalcinosis (34) 34
epithelial-cells (33) 33
excretion (33) 33
kidney-stones (33) 33
osteopontin (33) 33
immunohistochemistry (32) 32
mice (32) 32
diagnosis, differential (31) 31
health aspects (31) 31
calcium oxalate - analysis (30) 30
calcium-oxalate (30) 30
hyperoxaluria - diagnosis (30) 30
liver - pathology (30) 30
medical biochemistry (30) 30
medicine (30) 30
oxaluria (30) 30
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (681) 681
French (26) 26
German (8) 8
Spanish (5) 5
Japanese (4) 4
Russian (4) 4
Italian (2) 2
Danish (1) 1
Hungarian (1) 1
Portuguese (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Journal Article
Kidney International, ISSN 0085-2538, 03/2018, Volume 93, Issue 3, pp. 656 - 669
Primary/secondary hyperoxalurias involve nephrocalcinosis-related chronic kidney disease (CKD) leading to end-stage kidney disease. Mechanistically, intrarenal... 
beta-hydroxybutyrate | necroinflammation | fibrosis | crystal nephropathy | NALP3 | OXALATE | REPAIR | DENDRITIC CELLS | RENAL INFLAMMATION | UROLOGY & NEPHROLOGY | MICROENVIRONMENTS | PROGRESSION | Cell Plasticity - drug effects | Inflammasomes - metabolism | Kidney - pathology | Nephrocalcinosis - immunology | Kidney - immunology | Hyperoxaluria - metabolism | Male | CARD Signaling Adaptor Proteins - genetics | Hyperoxaluria - drug therapy | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Renal Insufficiency, Chronic - metabolism | CARD Signaling Adaptor Proteins - metabolism | Kidney - metabolism | Nephrocalcinosis - metabolism | Butylene Glycols - pharmacology | Inflammasomes - drug effects | Interleukin-1 - immunology | Nephrocalcinosis - prevention & control | Female | Macrophages - immunology | Disease Models, Animal | Fibroblasts - metabolism | Interleukin-1 - metabolism | Renal Insufficiency, Chronic - immunology | Macrophages - pathology | Signal Transduction | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Mice, Inbred C57BL | Cells, Cultured | NLR Family, Pyrin Domain-Containing 3 Protein - antagonists & inhibitors | Fibroblasts - pathology | Inflammasomes - genetics | Hyperoxaluria - pathology | Mice, Knockout | Renal Insufficiency, Chronic - pathology | Renal Insufficiency, Chronic - prevention & control | Macrophages - metabolism | Nephrocalcinosis - pathology | Phenotype | Animals | NLR Family, Pyrin Domain-Containing 3 Protein - immunology | Receptors, Transforming Growth Factor beta - metabolism | Inflammasomes - immunology | Fibroblasts - immunology | Macrophages - drug effects | Hyperoxaluria - immunology | End-stage renal disease | Phenotypes | CD11b antigen | Kidneys | Interleukin 1 receptor antagonist | Therapeutic applications | CX3CR1 protein | Inflammation | Macrophages | CD45 antigen | Atrophy | Signal transduction | Calcium oxalate | Oxalic acid | Calcinosis | Interleukin 1 | Fibrosis | Fibroblasts | Hyperoxaluria | Kidney diseases
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/2010, Volume 120, Issue 3, pp. 706 - 712
Urolithiasis, a condition in which stones are present in the urinary system, including the kidneys and bladder, is a poorly understood yet common disorder... 
OXALATE | MEDICINE, RESEARCH & EXPERIMENTAL | SULFATE TRANSPORTERS | BRUSH-BORDER | ACETAMINOPHEN | MEMBRANE-VESICLES | RAT-KIDNEY | CALCIUM | ALPHA-L-IDURONIDASE | TRANSCRIPTIONAL REGULATION | GENE SAT1 | Acetaminophen - adverse effects | Liver - pathology | Analgesics, Non-Narcotic - pharmacology | Hyperoxaluria - metabolism | Intestines - metabolism | Urinary Bladder - pathology | Urolithiasis - pathology | Nephrocalcinosis - metabolism | Acetaminophen - pharmacology | Liver Failure, Acute - genetics | Analgesics, Non-Narcotic - adverse effects | Antiporters - genetics | Ion Transport | Kidney Tubules - pathology | Kidney Tubules - metabolism | Intestines - pathology | Sulfates - metabolism | Urinary Bladder - metabolism | Liver - metabolism | Antiporters - metabolism | Liver Failure, Acute - chemically induced | Liver Failure, Acute - metabolism | Anion Transport Proteins - metabolism | Hyperoxaluria - pathology | Mice, Knockout | Oxalates - metabolism | Nephrocalcinosis - pathology | Animals | Nephrocalcinosis - genetics | Urolithiasis - metabolism | Liver Failure, Acute - pathology | Mice | Hyperoxaluria - genetics | Urolithiasis - genetics | Anion Transport Proteins - genetics | Homeostasis - genetics | Usage | Calculi, Urinary | Oxalic acid | Models | Research | Health aspects | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Veterinary Pathology, ISSN 0300-9858, 11/2017, Volume 54, Issue 6, pp. 977 - 985
Journal Article
Hemodialysis International, ISSN 1492-7535, 10/2011, Volume 15, Issue 4, pp. 573 - 576
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2019, Volume 129, Issue 6, pp. 2571 - 2577
Journal Article
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 08/2011, Volume 6, Issue 8, pp. 1895 - 1902
Background and objectives Enteric overabsorption of oxalate may lead to hyperoxaluria and subsequent acute oxalate nephritis (AON). AON related to chronic... 
SURVIVAL | JEJUNOILEAL BYPASS | KIDNEY-TRANSPLANTATION | SECONDARY OXALOSIS | SHORT-BOWEL SYNDROME | DISEASE | UROLOGY & NEPHROLOGY | ACUTE-RENAL-FAILURE | DYSFUNCTION | EXCRETION | ENTERIC HYPEROXALURIA | Hyperoxaluria - therapy | Humans | Middle Aged | Hyperoxaluria - etiology | Hyperoxaluria - metabolism | Male | Pancreatitis, Chronic - physiopathology | Renal Insufficiency - etiology | Kidney - metabolism | Nephritis, Interstitial - metabolism | Nephritis, Interstitial - pathology | Renal Insufficiency - physiopathology | Time Factors | Kidney Failure, Chronic - pathology | Aged, 80 and over | Adult | Female | Retrospective Studies | France | Kidney - physiopathology | Kidney Failure, Chronic - physiopathology | Glomerular Filtration Rate | Nephritis, Interstitial - physiopathology | Treatment Outcome | Renal Insufficiency - metabolism | Calcium Oxalate - metabolism | Disease Progression | Hyperoxaluria - pathology | Kidney Failure, Chronic - metabolism | Kidney Failure, Chronic - therapy | Pancreatitis, Chronic - pathology | Renal Insufficiency - therapy | Pancreatitis, Chronic - complications | Renal Insufficiency - pathology | Pancreatitis, Chronic - therapy | Nephritis, Interstitial - etiology | Aged | Hyperoxaluria - physiopathology | Pancreatitis, Chronic - metabolism | Nephritis, Interstitial - therapy | Kidney Failure, Chronic - etiology | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e44174
Oxalate is a metabolic end product excreted by the kidney. Mild increases in urinary oxalate are most commonly associated with Nephrolithiasis. Chronically... 
KIM-1 | CRYSTAL-FORMATION | ACTIVATED PROTEIN-KINASE | LLC-PK1 CELLS | HK-2 CELLS | MULTIDISCIPLINARY SCIENCES | LINE | DNA-SYNTHESIS | CALCIUM-OXALATE | URINARY BIOMARKER | TREATED RATS | Cell Adhesion Molecules - chemistry | Immunohistochemistry | Cell Adhesion Molecules - genetics | Epithelial Cells - metabolism | Kidney - pathology | Membrane Glycoproteins - metabolism | Rats, Wistar | Cell Adhesion Molecules - urine | Receptors, Virus - metabolism | Epithelial Cells - drug effects | Humans | Male | Receptors, Virus - genetics | RNA, Messenger - metabolism | Kidney - metabolism | Kidney Tubules - pathology | Kidney Tubules - metabolism | Oxalates - pharmacology | Protein Structure, Tertiary | Cell Line | Kidney - drug effects | Kidney Tubules - drug effects | RNA, Messenger - genetics | Crystallization | Rats | Epithelial Cells - pathology | Hepatitis A Virus Cellular Receptor 1 | Cell Adhesion Molecules - metabolism | Hyperoxaluria - pathology | Membrane Glycoproteins - genetics | Up-Regulation - drug effects | Animals | Hyperoxaluria - urine | Hyperoxaluria - genetics | Oxalates | RNA | Oxalic acid | Kidney stones | Urine | Calcification (ectopic) | Cell culture | Kidneys | Epithelial cells | Tissue culture | Exposure | Nephrolithiasis | Kinases | Gene expression | Ribonucleic acid--RNA | Cell adhesion & migration | Injury prevention | Overexpression | Rodents | Transcription activation | Animal tissues | Renal failure | Calcification | Primary hyperoxaluria | Hyperoxaluria | Ribonucleic acid
Journal Article
Nutrition, ISSN 0899-9007, 2012, Volume 28, Issue 1, pp. 76 - 80
Abstract Objective Roux-en-Y gastric bypass (RYGB) surgery is the most common surgical intervention for long-term weight loss in morbidly obese patients. By... 
Gastroenterology and Hepatology | Interstitial nephritis | Morbid obesity | Oxalate | Gastric bypass | NUTRITION & DIETETICS | WEIGHT-LOSS | BARIATRIC SURGERY | MECHANISMS | HYPEROXALURIA | MODEL | KIDNEY | Leukocytes, Mononuclear - metabolism | Up-Regulation | Postoperative Complications - etiology | Obesity, Morbid - surgery | Postoperative Complications - metabolism | Diet, High-Fat - adverse effects | Osteopontin - metabolism | Glomerulonephritis - metabolism | Postoperative Complications - immunology | Nephritis, Interstitial - metabolism | Nephritis, Interstitial - pathology | Leukocytes, Mononuclear - immunology | Kidney Glomerulus - metabolism | Acute Kidney Injury - immunology | Glomerulonephritis - etiology | Kidney Tubules - pathology | Kidney Tubules - metabolism | Acute Kidney Injury - etiology | Disease Models, Animal | Postoperative Complications - pathology | Kidney Tubules - immunology | Severity of Illness Index | Acute Kidney Injury - pathology | Gastric Bypass - adverse effects | Rats | Glomerulonephritis - immunology | Kidney Glomerulus - pathology | Random Allocation | Rats, Sprague-Dawley | Leukocytes, Mononuclear - pathology | Pilot Projects | Animals | Weight Loss | Glomerulonephritis - pathology | Nephritis, Interstitial - etiology | Kidney Glomerulus - immunology | Acute Kidney Injury - metabolism | Nephritis, Interstitial - immunology | Hypertension | Medical colleges | Obesity | Diabetes | Kidney diseases | Weight control | Kidneys | Rodents | morbid obesity | oxalate | gastric bypass | interstitial nephritis
Journal Article
American Journal of Nephrology, ISSN 0250-8095, 09/2016, Volume 44, Issue 2, pp. 85 - 91
Background: Enteric hyperoxaluria (EH) occurs with a rate of 5-24% in patients with inflammatory bowel disease, ileal resection and modern bariatric surgery.... 
Original Report: Transplantation | Intestinal transplantation | Enteric hyperoxaluria | Secondary hyperoxaluria | Kidney transplantation | RENAL-FAILURE SECONDARY | MULTIVISCERAL TRANSPLANTATION | EXPANDING INDICATIONS | OXALOSIS | UROLOGY & NEPHROLOGY | OXALATE NEPHROPATHY | IMPROVING OUTCOMES | Recurrence | Postoperative Complications - etiology | Kidney - pathology | Renal Insufficiency, Chronic - etiology | Humans | Middle Aged | Calcium Oxalate - urine | Hyperoxaluria - complications | Intestinal Volvulus - surgery | Allografts - pathology | Anti-Bacterial Agents - therapeutic use | Hyperoxaluria - surgery | Cecum - surgery | Female | Kidney Tubular Necrosis, Acute - etiology | Membrane Transport Proteins - metabolism | Anti-Bacterial Agents - adverse effects | Parenteral Nutrition | Transplantation, Homologous - adverse effects | Vasoconstrictor Agents - therapeutic use | Blood Loss, Surgical - prevention & control | Renal Insufficiency, Chronic - surgery | Glomerular Filtration Rate | Intestines - pathology | Vasoconstrictor Agents - adverse effects | Gastric Bypass - adverse effects | Postoperative Complications - therapy | Delayed Graft Function - etiology | Intestinal Volvulus - etiology | Urinary Tract Infections - drug therapy | Hyperoxaluria - pathology | Renal Insufficiency, Chronic - pathology | Blood Component Transfusion | Calcium Oxalate - pharmacokinetics | Biopsy | Urinary Tract Infections - etiology | Creatinine - blood | Intestines - transplantation | Kidney Transplantation - adverse effects
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 04/2005, Volume 29, Issue 4, pp. 443 - 451
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 07/2008, Volume 359, Issue 1, pp. 100 - 102
Journal Article