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American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 07/2013, Volume 305, Issue 2, pp. C182 - C189
Inspiration of a high concentration of oxygen, a therapy for acute lung injury (ALI), could unexpectedly lead to reactive oxygen species (ROS) production and... 
Reactive oxygen species | Inflammation | Hyperoxia | Injury | Lung | ACTIVATION | PHYSIOLOGY | hyperoxia | INTERLEUKIN-1-BETA | INDUCTION | RESPIRATORY-DISTRESS-SYNDROME | CELL BIOLOGY | lung | EPITHELIAL-CELLS | inflammation | reactive oxygen species | ENDOTHELIAL-CELLS | GENE-EXPRESSION | MACROPHAGE INFLAMMATORY PROTEIN-2 | II PNEUMOCYTES | injury | TNF-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Interleukin-6 - analysis | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Oxygen - therapeutic use | Tumor Necrosis Factor-alpha - genetics | Bronchoalveolar Lavage Fluid - immunology | Acute Lung Injury - genetics | Oxygen - adverse effects | Inflammasomes - adverse effects | Acute Lung Injury - prevention & control | DNA Fragmentation | Hyperoxia - complications | Hyperoxia - metabolism | Chemokine CXCL2 - genetics | Lung - metabolism | Chemokine CXCL2 - metabolism | Genetic Predisposition to Disease | Interleukin-1beta - analysis | Lung - pathology | Gene Expression Regulation - physiology | Mice, Knockout | Gene Expression Regulation - drug effects | Carrier Proteins - genetics | Oxygen - administration & dosage | Animals | Carrier Proteins - metabolism | Acute Lung Injury - chemically induced | Mice | Acute respiratory distress syndrome | Physiological aspects | Hypoxia | Health aspects
Journal Article
Journal Article
Pediatric Research, ISSN 0031-3998, 12/2015, Volume 78, Issue 6, pp. 634 - 640
BACKGROUND: Pulmonary hypertension (PH) worsens clinical outcomes in former preterm infants with bronchopulmonary dysplasia (BPD). Oxidant stress disrupts... 
OVEREXPRESSION | VEGF | INFANTS | NITRIC-OXIDE | PEDIATRICS | MICE | HYPEROXIA | EXTRACELLULAR-SUPEROXIDE DISMUTASE | RAT LUNG | ENDOTHELIAL GROWTH-FACTOR | LUNG STRUCTURE | Bronchopulmonary Dysplasia - enzymology | Phosphorylation | Superoxide Dismutase - genetics | Vascular Remodeling | Oxidative Stress | Ventricular Dysfunction, Right - genetics | Bronchopulmonary Dysplasia - pathology | Hypertension, Pulmonary - physiopathology | Bronchopulmonary Dysplasia - physiopathology | Vascular Endothelial Growth Factor A - metabolism | Ventricular Dysfunction, Right - enzymology | Hypertension, Pulmonary - chemically induced | Pulmonary Alveoli - enzymology | Bronchopulmonary Dysplasia - genetics | Hypertrophy, Right Ventricular - physiopathology | Bleomycin | Ventricular Dysfunction, Right - physiopathology | Nitric Oxide Synthase Type III - metabolism | Ventricular Dysfunction, Right - chemically induced | Hypertension, Pulmonary - enzymology | Hypertrophy, Right Ventricular - genetics | Pulmonary Alveoli - blood supply | Animals, Newborn | Genetic Predisposition to Disease | Pulmonary Alveoli - pathology | Signal Transduction | Mice, Inbred C57BL | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Hypertension, Pulmonary - genetics | Superoxide Dismutase - deficiency | Pulmonary Artery - physiopathology | Hypertrophy, Right Ventricular - enzymology | Pulmonary Artery - enzymology | Mice, Knockout | Phenotype | Animals | Bronchopulmonary Dysplasia - chemically induced | Hypertrophy, Right Ventricular - chemically induced | Ventricular Pressure | Endothelial Cells - pathology | Nitric Oxide - metabolism | Endothelial Cells - enzymology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Ventricular Function, Right
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 05/2012, Volume 302, Issue 9, pp. L829 - L837
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e49023
Intrauterine infection and inflammation are major reasons for preterm birth. The switch from placenta-mediated to lung-mediated oxygen supply during birth is... 
INDUCED CELL-DEATH | ISCHEMIC TOLERANCE | MICROGLIAL TOXICITY | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | PERIVENTRICULAR LEUKOMALACIA | DEVELOPING OLIGODENDROCYTES | NECROSIS-FACTOR-ALPHA | EXCITOTOXIC INJURY | INDUCED BRAIN-INJURY | BACTERIAL-ENDOTOXIN SENSITIZES | Inflammation - chemically induced | Inflammation - pathology | Microglia - metabolism | Oligodendroglia - metabolism | Rats, Wistar | Apoptosis - drug effects | Caspase 3 - metabolism | Brain - metabolism | Inflammation - metabolism | Oligodendroglia - drug effects | Nerve Fibers, Myelinated - pathology | Hyperoxia - metabolism | Microglia - pathology | Animals, Newborn | Nerve Fibers, Myelinated - metabolism | Leukoencephalopathies - pathology | Microglia - drug effects | Nerve Fibers, Myelinated - drug effects | Cells, Cultured | Rats | Leukoencephalopathies - metabolism | Rats, Sprague-Dawley | Brain - drug effects | Hyperoxia - pathology | Oligodendroglia - pathology | Animals | Lipopolysaccharides - pharmacology | Brain - pathology | Apoptosis - physiology | Immunohistochemistry | Brain | Cell death | Infants (Premature) | Inflammation | Research | Mitogens | Health aspects | Injuries | Neuroimaging | Neonates | Cell culture | Pediatrics | Transcription factors | Traumatic brain injury | Premature birth | Interleukin | Superoxide dismutase | Infants | Infections | Birth | Western blotting | Lipopolysaccharides | Head injuries | Newborn babies | Ischemia | Rodents | Oligodendrocytes | Oxygen | Pain perception | Cytokines | Maturation | Incubation | Mortality | Superoxide | Gene expression | Substantia alba | Microglia | Hospitals | Placenta | Magnetic resonance imaging | Lungs | Magnetic permeability | Hyperoxia | Interleukin 10 | Brain damage | Hypoxia | Oxygen tension | Brain injury | Apoptosis | pathology | Leukoencephalopathies | Sprague-Dawley | pharmacology | Caspase 3 | Cultured | Basic Medicine | chemically induced | Myelinated | physiology | drug effects | Cells | Newborn | Wistar | Medicinska grundvetenskaper | Oligodendroglia | metabolism | Nerve Fibers
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 2013, Volume 304, Issue 10, pp. L646 - L656
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2007, Volume 104, Issue 25, pp. 10589 - 10594
Vessel loss precipitates many diseases. In particular, vessel loss resulting in hypoxia induces retinal neovascularization in diabetic retinopathy and in... 
Diabetic angiopathies | Diabetic retinopathy | Oxygen | Messenger RNA | Blood vessels | Retina | Regrowth | Hypoxia | Mice | Infants | Angiogenesis | Retinopathy of prematurity | Insulin-like growth factor | Binding protein-3 | Insulin-like growth factor 1 | Stem cells | SURVIVAL | stem cells | FACTOR-BINDING PROTEIN-3 | angiogenesis | MULTIDISCIPLINARY SCIENCES | MOUSE | binding protein-3 | DNA-SYNTHESIS | retinopathy of prematurity | BREAST-CANCER | ENDOTHELIAL-CELLS | DISEASE | LINE | insulin-like growth factor | insulin-like growth factor 1 | PREMATURITY | GROWTH-FACTOR-I | Insulin-Like Growth Factor Binding Protein 3 - genetics | Retinopathy of Prematurity - classification | Humans | Insulin-Like Growth Factor Binding Protein 3 - blood | Insulin-Like Growth Factor Binding Protein 3 - deficiency | Insulin-Like Growth Factor Binding Protein 3 - pharmacology | Retinal Neovascularization - metabolism | RNA, Messenger - metabolism | Retinopathy of Prematurity - physiopathology | Dose-Response Relationship, Drug | Retina - cytology | Hyperoxia - complications | Infant, Newborn | Disease Models, Animal | Retinopathy of Prematurity - etiology | Gene Expression Regulation | Mice, Transgenic | Gestational Age | Mice, Knockout | Animals | Infant, Premature | Retinal Neovascularization - chemically induced | Insulin-Like Growth Factor Binding Protein 1 - blood | Retinopathy of Prematurity - metabolism | Retinopathy of Prematurity - pathology | Angiogenesis inhibitors | Research | Retrolental fibroplasia | Binding proteins | Biological Sciences | like growth factor 1 | insulin | like growth factor binding protein | Premature | Disease Models | RNA | Transgenic | Retinal Neovascularization/chemically induced/metabolism | Infant | MEDICIN OCH HÄLSOVETENSKAP | Insulin-Like Growth Factor Binding Protein 1/blood | Prematurity/classification/etiology/metabolism/pathology/physiopathology | Hyperoxia/complications | MEDICAL AND HEALTH SCIENCES | Drug | Insulin-Like Growth Factor Binding Protein | Retinopathy of | Knockout | Messenger/metabolism | Retina/cytology | 3/blood/deficiency/genetics/pharmacology | Newborn | Animal | Dose-Response Relationship
Journal Article
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 2007, Volume 221, Issue 3, pp. 339 - 348
Exposure of mice to single-walled carbon nanotubes (SWCNTs) induces an unusually robust pulmonary inflammatory response with an early onset of fibrosis, which... 
Antioxidants | Carbon nanotubes | Oxidative stress | Inflammatory cells | Vitamin E deficiency | Pulmonary inflammatory response | antioxidants | INJURY | ALPHA-TOCOPHEROL | SOD | HYPEROXIA | PLASMA | carbon nanotubes | pulmonary inflammatory response | ANTIOXIDANT | inflammatory cells | PHARMACOLOGY & PHARMACY | TOXICOLOGY | vitamin E deficiency | EXTRACELLULAR-SUPEROXIDE DISMUTASE | oxidative stress | MOUSE LUNG | EXPOSURE | ASBESTOS | Particulate Matter - toxicity | Lung Diseases - chemically induced | Lung Diseases - metabolism | Glutathione - metabolism | Antioxidants - metabolism | Oxidative Stress - immunology | Vitamin E Deficiency - immunology | Ascorbic Acid - metabolism | Nanotubes, Carbon - toxicity | Particulate Matter - immunology | Glutathione - drug effects | Foreign-Body Reaction - immunology | Vitamin E Deficiency - complications | Lipid Peroxidation - drug effects | Female | Superoxide Dismutase - metabolism | Cytokines - immunology | Cytokines - metabolism | Mice, Inbred C57BL | Foreign-Body Reaction - metabolism | Superoxide Dismutase - drug effects | Lung Diseases - immunology | Animals | Foreign-Body Reaction - chemically induced | Lung Diseases - complications | Mice | Oxidative Stress - drug effects | Lipid Peroxidation - immunology | Nutritional aspects | Physiological aspects | Vitamin E | Nanotechnology | Nanotubes | FIBROSIS | OXIDATION | CARBON | COLLAGEN | SUPEROXIDE DISMUTASE | ANTIOXIDANTS | LUNGS | SENSITIVITY | 60 APPLIED LIFE SCIENCES | BIOLOGICAL STRESS | LEUKOCYTES | DIET | LYMPHOKINES | INFLAMMATION | NANOTUBES | MICE | OPTIMIZATION | LIPIDS | VITAMIN E | Carbon Nanotubes | Pulmonary Inflammatory Response | Vitamin E Deficiency | Oxidative Stress
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2009, Volume 4, Issue 11, pp. e8013 - e8013
Many common diseases of the gas exchange surface of the lung have no specific treatment but cause serious morbidity and mortality. Idiopathic Pulmonary... 
PROGENITOR CELLS | ENGRAFTMENT | MULTIPLE MECHANISMS | IN-VIVO | BIOLOGY | INDUCED LUNG INJURY | GENE-EXPRESSION | II CELLS | MICE | MODEL | EPITHELIAL PROLIFERATION | Bone Marrow Cells - cytology | Humans | Antibiotics, Antineoplastic - adverse effects | Stem Cells - cytology | Bleomycin - adverse effects | Fibroblast Growth Factor 7 - biosynthesis | Immunohistochemistry - methods | Lentivirus - metabolism | Animals | Models, Biological | Fibrosis | Lung Injury - metabolism | Pulmonary Fibrosis - chemically induced | Cell Differentiation | Mice | Pulmonary Fibrosis - metabolism | Immunohistochemistry | Bleomycin | Pulmonary fibrosis | Collagen | Respiratory tract diseases | Fibroblast growth factors | Inflammation | B cells | Gene therapy | Health aspects | Hematopoietic stem cells | Cell proliferation | Fibroblast growth factor | Disease | Mesenchyme | Laboratories | Syngeneic grafts | Epithelial cells | Lung transplantation | Bone marrow transplantation | Parenchyma | Transplantation | Metastasis | Cancer therapies | Accumulation | Histopathology | Rodents | Hemophilia | Bone marrow | Fibroblasts | Repair | Damage | Growth factors | Medical research | Cytokines | Lung diseases | Keratinocyte growth factor | Fibroblast growth factor 7 | Morbidity | Cell injury | Lungs | Stromal cells | Hyperoxia | Stem cells | Bone | Gas exchange | Tumors | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e72135