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Free Radical Biology and Medicine, ISSN 0891-5849, 02/2014, Volume 67, pp. 221 - 234
Caffeine administered to preterm infants has been shown to reduce rates of cerebral palsy and cognitive delay, compared to placebo. We investigated the... 
Methylxanthine | Oxidative stress | Postnatal neurogenesis | Developmental brain | Free radicals | Hyperoxia | Preterm infants | PROGENITOR CELLS | A(2A) RECEPTOR ANTAGONISTS | FUNCTIONAL IMPLICATIONS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ADULT HIPPOCAMPAL NEUROGENESIS | TNF-ALPHA PRODUCTION | NEURAL STEM-CELLS | INTENSIVE-CARE-UNIT | ENDOCRINOLOGY & METABOLISM | DENTATE GYRUS | EXPRESSION | Cell Proliferation | Homeodomain Proteins - metabolism | Cerebral Cortex - pathology | Ki-67 Antigen - metabolism | SOXB1 Transcription Factors - metabolism | Dentate Gyrus - drug effects | SOXB1 Transcription Factors - genetics | Hyperoxia - metabolism | Caffeine - pharmacology | Neurons - metabolism | Cerebral Cortex - drug effects | Repressor Proteins - metabolism | Animals, Newborn | Biomarkers - metabolism | Gene Expression | Tumor Suppressor Proteins - metabolism | Dentate Gyrus - metabolism | Rats | Repressor Proteins - genetics | Neuropeptides - metabolism | T-Box Domain Proteins - metabolism | Hyperoxia - pathology | Eye Proteins - metabolism | Ki-67 Antigen - genetics | Dentate Gyrus - pathology | Paired Box Transcription Factors - genetics | Neurons - pathology | Microtubule-Associated Proteins - genetics | Rats, Wistar | Antigens, Nuclear - metabolism | Microtubule-Associated Proteins - metabolism | Gray Matter - drug effects | Cerebral Cortex - metabolism | Gray Matter - metabolism | Tumor Suppressor Proteins - genetics | Nestin - genetics | Eye Proteins - genetics | Neuropeptides - genetics | Neurons - drug effects | PAX6 Transcription Factor | Hyperoxia - prevention & control | Nerve Tissue Proteins - genetics | T-Box Domain Proteins - genetics | Homeodomain Proteins - genetics | Nerve Tissue Proteins - metabolism | Nestin - metabolism | Animals | Antigens, Nuclear - genetics | Gray Matter - pathology | Paired Box Transcription Factors - metabolism | Antioxidants | Neurons | Analysis | Caffeine | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, pp. e49023 - e49023
Intrauterine infection and inflammation are major reasons for preterm birth. The switch from placenta-mediated to lung-mediated oxygen supply during birth is... 
INDUCED CELL-DEATH | ISCHEMIC TOLERANCE | MICROGLIAL TOXICITY | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | PERIVENTRICULAR LEUKOMALACIA | DEVELOPING OLIGODENDROCYTES | NECROSIS-FACTOR-ALPHA | EXCITOTOXIC INJURY | INDUCED BRAIN-INJURY | BACTERIAL-ENDOTOXIN SENSITIZES | Inflammation - chemically induced | Inflammation - pathology | Microglia - metabolism | Oligodendroglia - metabolism | Rats, Wistar | Apoptosis - drug effects | Caspase 3 - metabolism | Brain - metabolism | Inflammation - metabolism | Oligodendroglia - drug effects | Nerve Fibers, Myelinated - pathology | Hyperoxia - metabolism | Microglia - pathology | Animals, Newborn | Nerve Fibers, Myelinated - metabolism | Leukoencephalopathies - pathology | Microglia - drug effects | Nerve Fibers, Myelinated - drug effects | Cells, Cultured | Rats | Leukoencephalopathies - metabolism | Rats, Sprague-Dawley | Brain - drug effects | Hyperoxia - pathology | Oligodendroglia - pathology | Animals | Lipopolysaccharides - pharmacology | Brain - pathology | Apoptosis - physiology | Immunohistochemistry | Brain | Cell death | Infants (Premature) | Inflammation | Research | Mitogens | Health aspects | Injuries | Neuroimaging | Neonates | Cell culture | Pediatrics | Transcription factors | Traumatic brain injury | Lung | Premature birth | Interleukin | Superoxide dismutase | Infants | Infections | Birth | Western blotting | Lipopolysaccharides | Head injuries | Newborn babies | Ischemia | Rodents | Oligodendrocytes | Oxygen | Pain perception | Cytokines | Maturation | Incubation | Mortality | Superoxide | Gene expression | Substantia alba | Microglia | Hospitals | Placenta | Magnetic resonance imaging | Magnetic permeability | Hyperoxia | Interleukin 10 | Brain damage | Hypoxia | Oxygen tension | Brain injury | Apoptosis | Index Medicus | pathology | Leukoencephalopathies | Sprague-Dawley | pharmacology | Caspase 3 | Cultured | Basic Medicine | chemically induced | Myelinated | physiology | drug effects | Cells | Newborn | Wistar | Medicinska grundvetenskaper | Oligodendroglia | metabolism | Nerve Fibers
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2015, Volume 52, pp. 106 - 119
Highlights • Fingolimod (FTY720) improves long-term cognitive deficits after neonatal hyperoxia. • Hyperoxia-related long-term microstructural abnormalities... 
Psychiatry | Allergy and Immunology | White matter development | Oligodendrocyte | Fingolimod | Hyperoxia | Neonatal brain injury | SURVIVAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | RAT | IMMUNOLOGY | NEUROSCIENCES | CELL-DEATH | MULTIPLE-SCLEROSIS | OLIGODENDROCYTE PROGENITORS | FTY720 | INFANTS | ISCHEMIC BRAIN-INJURY | MODULATION | Lysophospholipids - metabolism | Microglia - metabolism | Oligodendroglia - metabolism | Receptors, Lysosphingolipid - antagonists & inhibitors | Rats, Wistar | White Matter - metabolism | Cognition Disorders - metabolism | Male | Brain - metabolism | Cognition Disorders - prevention & control | Oligodendroglia - drug effects | Microglia - pathology | Female | Sphingosine - metabolism | Receptors, Lysosphingolipid - metabolism | Fingolimod Hydrochloride - therapeutic use | Animals, Newborn | Microglia - drug effects | Nerve Fibers, Myelinated - drug effects | Cognition Disorders - pathology | Rats | White Matter - drug effects | Random Allocation | White Matter - pathology | Hyperoxia - pathology | Oligodendroglia - pathology | Pregnancy | Oxygen - administration & dosage | Sphingosine - analogs & derivatives | Animals | Diffusion Magnetic Resonance Imaging | Hyperoxia - drug therapy | Infants (Newborn) | Brain | Multiple sclerosis | Analysis | Injuries | Sphingosine | Index Medicus
Journal Article
Shock, ISSN 1073-2322, 12/2012, Volume 38, Issue 6, pp. 656 - 663
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, pp. e0166886 - e0166886
Bronchopulmonary dysplasia (BPD) is a major cause of neonatal morbidity in premature infants, occurring as a result of arrested lung development combined with... 
BRONCHOPULMONARY DYSPLASIA | OXIDATIVE STRESS | NAD(P)H OXIDASE | PULMONARY-HYPERTENSION | PLEXIFORM LAYERS | OXYGEN-INDUCED RETINOPATHY | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | LUNG DEVELOPMENT | PRIMARY PREVENTION | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Retina - metabolism | Membrane Glycoproteins - metabolism | Vitreoretinopathy, Proliferative - etiology | Humans | NADPH Oxidases - metabolism | Retinal Neovascularization - metabolism | Vascular Endothelial Growth Factor A - metabolism | Retinal Ganglion Cells - metabolism | Oxygen - metabolism | Retinal Vessels - pathology | Retinitis - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Hyperoxia - metabolism | NADPH Oxidases - genetics | Disease Models, Animal | Retinitis - pathology | Gene Expression | Retinal Vessels - metabolism | Vitreoretinopathy, Proliferative - pathology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Vitreoretinopathy, Proliferative - metabolism | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Collagen - metabolism | Animals | Mice | Retina - pathology | Retinal Neovascularization - pathology | Retinitis - metabolism | Proteins | Dysplasia | Infants (Premature) | Analysis | Inflammation | Vascular endothelial growth factor | Retinal diseases | Neonates | Pediatrics | GTP-binding protein | Animal models | Retinopathy | Lung | Retina | Infants | Macrophages | Angiogenesis | Signal transduction | Newborn babies | Lymphocytes | Rodents | CYBB protein | Fibrinogen | Bioindicators | Age | Phenotypes | Oxygen | Wound healing | Lung diseases | Blood vessels | Morbidity | Medicine | Cyclin-dependent kinase inhibitor p21 | Ostomy | Detachment | Hyperoxia | Laboratory animals | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 07/2004, Volume 430, Issue 6997, pp. 317 - 322
Journal Article
Journal Article
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 05/2015, Volume 52, Issue 5, pp. 611 - 621
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 06/2011, Volume 50, Issue 12, pp. 1749 - 1759
Reactive oxygen species (ROS) generated by vascular endothelial and smooth muscle cells contribute to the development and progression of vascular diseases. We... 
Nox4 | Free radicals | ARE | Nrf2 | ROS | Endothelium | PULMONARY EPITHELIAL-CELLS | NADPH OXIDASE | OXIDATIVE STRESS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | OXYGEN SPECIES GENERATION | INJURY | NAD(P)H OXIDASE | ENDOCRINOLOGY & METABOLISM