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PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e49023
Intrauterine infection and inflammation are major reasons for preterm birth. The switch from placenta-mediated to lung-mediated oxygen supply during birth is... 
INDUCED CELL-DEATH | ISCHEMIC TOLERANCE | MICROGLIAL TOXICITY | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | PERIVENTRICULAR LEUKOMALACIA | DEVELOPING OLIGODENDROCYTES | NECROSIS-FACTOR-ALPHA | EXCITOTOXIC INJURY | INDUCED BRAIN-INJURY | BACTERIAL-ENDOTOXIN SENSITIZES | Inflammation - chemically induced | Inflammation - pathology | Microglia - metabolism | Oligodendroglia - metabolism | Rats, Wistar | Apoptosis - drug effects | Caspase 3 - metabolism | Brain - metabolism | Inflammation - metabolism | Oligodendroglia - drug effects | Nerve Fibers, Myelinated - pathology | Hyperoxia - metabolism | Microglia - pathology | Animals, Newborn | Nerve Fibers, Myelinated - metabolism | Leukoencephalopathies - pathology | Microglia - drug effects | Nerve Fibers, Myelinated - drug effects | Cells, Cultured | Rats | Leukoencephalopathies - metabolism | Rats, Sprague-Dawley | Brain - drug effects | Hyperoxia - pathology | Oligodendroglia - pathology | Animals | Lipopolysaccharides - pharmacology | Brain - pathology | Apoptosis - physiology | Immunohistochemistry | Brain | Cell death | Infants (Premature) | Inflammation | Research | Mitogens | Health aspects | Injuries | Neuroimaging | Neonates | Cell culture | Pediatrics | Transcription factors | Traumatic brain injury | Premature birth | Interleukin | Superoxide dismutase | Infants | Infections | Birth | Western blotting | Lipopolysaccharides | Head injuries | Newborn babies | Ischemia | Rodents | Oligodendrocytes | Oxygen | Pain perception | Cytokines | Maturation | Incubation | Mortality | Superoxide | Gene expression | Substantia alba | Microglia | Hospitals | Placenta | Magnetic resonance imaging | Lungs | Magnetic permeability | Hyperoxia | Interleukin 10 | Brain damage | Hypoxia | Oxygen tension | Brain injury | Apoptosis | pathology | Leukoencephalopathies | Sprague-Dawley | pharmacology | Caspase 3 | Cultured | Basic Medicine | chemically induced | Myelinated | physiology | drug effects | Cells | Newborn | Wistar | Medicinska grundvetenskaper | Oligodendroglia | metabolism | Nerve Fibers
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2015, Volume 52, pp. 106 - 119
Highlights • Fingolimod (FTY720) improves long-term cognitive deficits after neonatal hyperoxia. • Hyperoxia-related long-term microstructural abnormalities... 
Psychiatry | Allergy and Immunology | White matter development | Oligodendrocyte | Fingolimod | Hyperoxia | Neonatal brain injury | SURVIVAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | RAT | IMMUNOLOGY | NEUROSCIENCES | CELL-DEATH | MULTIPLE-SCLEROSIS | OLIGODENDROCYTE PROGENITORS | FTY720 | INFANTS | ISCHEMIC BRAIN-INJURY | MODULATION | Lysophospholipids - metabolism | Microglia - metabolism | Oligodendroglia - metabolism | Receptors, Lysosphingolipid - antagonists & inhibitors | Rats, Wistar | White Matter - metabolism | Cognition Disorders - metabolism | Male | Brain - metabolism | Cognition Disorders - prevention & control | Oligodendroglia - drug effects | Microglia - pathology | Female | Sphingosine - metabolism | Receptors, Lysosphingolipid - metabolism | Fingolimod Hydrochloride - therapeutic use | Animals, Newborn | Microglia - drug effects | Nerve Fibers, Myelinated - drug effects | Cognition Disorders - pathology | Rats | White Matter - drug effects | Random Allocation | White Matter - pathology | Hyperoxia - pathology | Oligodendroglia - pathology | Pregnancy | Oxygen - administration & dosage | Sphingosine - analogs & derivatives | Animals | Diffusion Magnetic Resonance Imaging | Hyperoxia - drug therapy | Infants (Newborn) | Brain | Multiple sclerosis | Analysis | Injuries | Sphingosine | Index Medicus
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 05/2012, Volume 302, Issue 9, pp. L829 - L837
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 12/2014, Volume 307, Issue 12, pp. L936 - L947
Journal Article
Journal of Neurotrauma, ISSN 0897-7151, 04/2010, Volume 27, Issue 4, pp. 753 - 762
In this study we tested the hypothesis that long-term neuropathological outcome is worsened by hyperoxic compared to normoxic reperfusion in a rat global... 
Morris water maze | Astrocyte | Hippocampus | Resuscitation | Cardiac arrest | Microglia | hippocampus | BRAIN-INJURY | 100-PERCENT OXYGEN | CARDIOPULMONARY | NORMOBARIC OXYGEN | RATS | FOREBRAIN ISCHEMIA | cardiac arrest | NEUROSCIENCES | CLINICAL NEUROLOGY | STROKE | CARDIAC-ARREST | resuscitation | astrocyte | microglia | CRITICAL CARE MEDICINE | Neurons - pathology | Hyperoxia - physiopathology | Rats, Inbred F344 | Nerve Degeneration - physiopathology | Learning Disorders - physiopathology | Male | Gliosis - physiopathology | Time | Learning Disorders - etiology | Oxygen Consumption - physiology | Oxygen - metabolism | Gliosis - pathology | Learning Disorders - pathology | Encephalitis - physiopathology | Cognition Disorders - etiology | Hyperoxia - complications | Neurons - physiology | Reperfusion Injury - complications | Cell Survival - physiology | Gliosis - etiology | Resuscitation - adverse effects | Disease Models, Animal | Brain Ischemia - complications | Cognition Disorders - physiopathology | Oxygen - toxicity | Reperfusion Injury - pathology | Cognition Disorders - pathology | Encephalitis - pathology | Rats | Brain Ischemia - physiopathology | Hippocampus - pathology | Nerve Degeneration - pathology | Hyperoxia - pathology | Animals | Brain Ischemia - pathology | Reperfusion Injury - physiopathology | Nerve Degeneration - etiology | Hippocampus - physiopathology | Encephalitis - etiology | Cerebral ischemia | Care and treatment | Neurons | Patient outcomes | Physiological aspects | Reperfusion (Physiology) | Research | Hippocampus (Brain) | Stroke | Ischemia | Hyperoxia | Rodents | Apoptosis | Original
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 03/2013, Volume 48, Issue 3, pp. 280 - 287
Mechanical ventilation with supraphysiological concentrations of oxygen (hyperoxia) is routinely used to treat patients with respiratory distress. However, a... 
Bacterial infection | Macrophage function | HMGB1 | Hyperoxia | IMMUNE-RESPONSE | hyperoxia | VENTILATOR-ASSOCIATED PNEUMONIA | BIOCHEMISTRY & MOLECULAR BIOLOGY | INTENSIVE-CARE UNITS | MECHANICAL VENTILATION | bacterial infection | CELL BIOLOGY | ANTIBACTERIAL FUNCTION | BACTERIAL CLEARANCE | macrophage function | RESPIRATORY SYSTEM | HOST-DEFENSE | ALVEOLAR MACROPHAGES | INFECTION-RATES | CYSTIC-FIBROSIS | Pseudomonas aeruginosa - immunology | Leukocytes - pathology | Lung Injury - pathology | Macrophages, Alveolar - pathology | Bronchoalveolar Lavage Fluid - immunology | Male | HMGB1 Protein - immunology | Pneumonia, Bacterial - metabolism | Hyperoxia - immunology | Bronchoalveolar Lavage Fluid - microbiology | Leukocytes - immunology | Pseudomonas Infections - metabolism | Respiration, Artificial - adverse effects | Pseudomonas aeruginosa - metabolism | HMGB1 Protein - metabolism | Hyperoxia - metabolism | Lung Injury - metabolism | Pneumonia, Ventilator-Associated - metabolism | Lung - metabolism | Neutrophils - metabolism | Pneumonia, Bacterial - pathology | Antibodies, Monoclonal - immunology | Neutrophils - pathology | Lung - pathology | Macrophages, Alveolar - microbiology | Pneumonia, Bacterial - microbiology | Mice, Inbred C57BL | Neutrophils - immunology | Phagocytosis - immunology | Phagocytosis - physiology | Pseudomonas Infections - microbiology | Pseudomonas Infections - immunology | Hyperoxia - pathology | Immunity, Innate - immunology | Animals | Lung Injury - microbiology | Pneumonia, Ventilator-Associated - microbiology | Pseudomonas Infections - pathology | Pneumonia, Bacterial - immunology | Mice | Macrophages, Alveolar - metabolism | Pneumonia, Ventilator-Associated - pathology | Leukocytes - metabolism | Lung Injury - immunology | Lung - immunology | Pneumonia, Ventilator-Associated - immunology
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2013, Volume 33, Issue 8, pp. 1881 - 1891
OBJECTIVE—Proinflammatory cytokines contribute to the development of retinal vasculopathies. However, the role of these factors and the mechanisms by which... 
Diabetic retinopathy | Retinopathy of prematurity | Semaphorin-3A | Interleukin-1β | Vaso-obliteration | Microglia | Microglia - metabolism | Retinal Diseases - immunology | Apoptosis - drug effects | NLR Family, Pyrin Domain-Containing 3 Protein | Oxidative Stress - immunology | Semaphorin-3A - metabolism | Retinal Ganglion Cells - metabolism | Hyperoxia - immunology | Gene Knockdown Techniques | Retinal Ganglion Cells - pathology | Microcirculation - physiology | Retinitis - immunology | Receptors, Cytoplasmic and Nuclear - immunology | Microglia - immunology | Ischemia - immunology | Interleukin-1beta - metabolism | Hyperoxia - metabolism | Microglia - pathology | Retinal Diseases - pathology | Antirheumatic Agents - pharmacology | Interleukin 1 Receptor Antagonist Protein - pharmacology | Ischemia - pathology | Carrier Proteins | Interleukin-1beta - antagonists & inhibitors | Semaphorin-3A - genetics | Retinitis - pathology | Endothelial Cells - metabolism | Interleukin-1beta - immunology | Rats | Retinal Ganglion Cells - immunology | Rats, Sprague-Dawley | Peptides - pharmacology | Endothelial Cells - immunology | Hyperoxia - pathology | Animals | Apoptosis - immunology | Ischemia - drug therapy | Retinitis - drug therapy | Semaphorin-3A - immunology | Oxidative Stress - drug effects | Primary Cell Culture | Retinal Diseases - drug therapy | Endothelial Cells - pathology | Receptors, Cytoplasmic and Nuclear - metabolism
Journal Article
Brain Research, ISSN 0006-8993, 12/2017, Volume 1676, pp. 57 - 68
Journal Article
Atherosclerosis, ISSN 0021-9150, 2012, Volume 225, Issue 2, pp. 296 - 303
Abstract Objective Notch signaling plays pivotal roles in the pathogenesis of vascular disease. However, little is known about its role in atherosclerosis. We... 
Cardiovascular | Endothelial cells (EC) | Vascular inflammation | Notch | Atherosclerosis | EC senescence | ANGIOGENESIS | MOBILIZATION | VEGF | INJURY | HYPEROXIA | VASCULAR MORPHOGENESIS | DISEASE | PERIPHERAL VASCULAR DISEASE | TELOMERES | Inflammation - pathology | NIH 3T3 Cells | Up-Regulation | Age Factors | Apolipoproteins E - deficiency | Receptors, Notch - metabolism | Atherosclerosis - genetics | Humans | Receptors, Notch - genetics | Gene Expression Profiling | Aorta - metabolism | Aortic Diseases - metabolism | Coronary Vessels - metabolism | Inflammation - metabolism | Transfection | Time Factors | Transendothelial and Transepithelial Migration | Cellular Senescence | Coronary Artery Disease - pathology | HEK293 Cells | Inflammation Mediators - metabolism | Polymerase Chain Reaction | Aortic Diseases - pathology | Disease Models, Animal | Atherosclerosis - pathology | Coronary Vessels - pathology | Cytokines - metabolism | Endothelial Cells - metabolism | Coronary Artery Disease - metabolism | Mice, Inbred C57BL | Signal Transduction - genetics | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Blotting, Western | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Animals | Apolipoproteins E - genetics | Coronary Artery Disease - genetics | Inflammation - genetics | Protein Array Analysis | Mice | Polymorphism, Single Nucleotide | Endothelial Cells - pathology | Leukocytes - metabolism | Blood circulation disorders | Endothelium | EC Senescence
Journal Article
Journal Article