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1. Full Text Longitudinal Associations Between Binge Eating and Overeating and Adverse Outcomes Among Adolescents and Young Adults: Does Loss of Control Matter?
by Sonneville, Kendrin R and Horton, Nicholas J and Micali, Nadia and Crosby, Ross D and Swanson, Sonja A and Solmi, Francesca and Field, Alison E
JAMA pediatrics, ISSN 2168-6203, 02/2013, Volume 167, Issue 2, pp. 149 - 155
Life Sciences & Biomedicine | Pediatrics | Science & Technology | Binge-Eating Disorder - complications | United States - epidemiology | Prevalence | Prospective Studies | Humans | Male | Hyperphagia - complications | Binge Drinking - epidemiology | Incidence | Depression - etiology | Young Adult | Binge-Eating Disorder - psychology | Overweight - epidemiology | Substance-Related Disorders - epidemiology | Female | Obesity - etiology | Surveys and Questionnaires | Odds Ratio | Severity of Illness Index | Substance-Related Disorders - etiology | Depression - epidemiology | Risk Factors | Binge-Eating Disorder - epidemiology | Models, Statistical | Health Surveys | Overweight - etiology | Obesity - epidemiology | Adolescent | Binge Drinking - etiology | Hyperphagia - epidemiology | Hyperphagia - psychology | Longitudinal Studies | Control | Young adults | Compulsive eating | Youth | Teenagers | Health aspects | Risk factors | Hyperphagia | Index Medicus | Abridged Index Medicus
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2. Full Text Obesity-programmed mice are rescued by early genetic intervention
by Bumaschny, Viviana F and Yamashita, Miho and Casas-Cordero, Rodrigo and Otero-Corchón, Verónica and De Souza, Flav́io S. J and Rubinstein, Marcelo and Low, Malcolm J
The Journal of clinical investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4203 - 4212
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Neurons - pathology | Adipose Tissue - physiopathology | Obesity - genetics | Adipose Tissue - metabolism | Adiposity | Neurons - metabolism | Pro-Opiomelanocortin - genetics | Disease Models, Animal | Eating | Adipose Tissue - pathology | Obesity - physiopathology | Hypothalamus - pathology | Hyperphagia - metabolism | Hypothalamus - physiopathology | Mice, Knockout | Obesity - metabolism | Hyperphagia - genetics | Hyperphagia - pathology | Pro-Opiomelanocortin - metabolism | Animals | Hyperphagia - prevention & control | Hypothalamus - metabolism | Hyperphagia - physiopathology | Obesity - prevention & control | Mice | Prevention | Measurement | Obesity | Neurons | Body weight | Genetic aspects | Research | Gene expression | Risk factors | Index Medicus | Abridged Index Medicus | Hyperinsulinemia | Animal models | Adipose tissue | Liver | Body weight loss | Proopiomelanocortin | Hypothalamus | Body weight gain | Hyperglycemia | Fatty liver | Food intake | steatosis | Age | Hyperphagia | Locomotor activity | Metabolic disorders | Neurotransmission
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3. Full Text Reduced reward-driven eating accounts for the impact of a mindfulness-based diet and exercise intervention on weight loss: Data from the SHINE randomized controlled trial
by Mason, Ashley E and Epel, Elissa S and Aschbacher, Kirstin and Lustig, Robert H and Acree, Michael and Kristeller, Jean and Cohn, Michael and Dallman, Mary and Moran, Patricia J and Bacchetti, Peter and Laraia, Barbara and Hecht, Frederick M and Daubenmier, Jennifer
Appetite, ISSN 0195-6663, 05/2016, Volume 100, pp. 86 - 93
Obesity | Mindful eating | Reward-driven eating | Weight loss | Behavioral intervention | Life Sciences & Biomedicine | Behavioral Sciences | Nutrition & Dietetics | Science & Technology | Obesity - diet therapy | Obesity - psychology | Humans | Middle Aged | Male | Diet, Reducing | Appetite Regulation | Exercise | Obesity, Morbid - physiopathology | Adult | Female | Mindfulness - education | Obesity, Morbid - therapy | Patient Education as Topic | Body Mass Index | Stress, Psychological - therapy | Hyperphagia - diet therapy | Feeding Behavior | Obesity - physiopathology | Combined Modality Therapy | Group Processes | San Francisco | Obesity, Morbid - psychology | Weight Loss | Hyperphagia - physiopathology | Obesity - therapy | Obesity, Morbid - diet therapy | Hyperphagia - psychology | Patient Compliance | Reward | Hyperphagia - therapy | Stress, Psychological - etiology | Stress (Psychology) | Mindfulness meditation | Analysis | Body weight | Exercise therapy | Index Medicus | weight loss | mindful eating | behavioral intervention | obesity
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4. Full Text Emotional eating as a mediator between depression and weight gain
by van Strien, Tatjana and Konttinen, Hanna and Homberg, Judith R and Engels, Rutger C.M.E and Winkens, Laura H.H
Appetite, ISSN 0195-6663, 05/2016, Volume 100, pp. 216 - 224
Emotional eating | Longitudinal | Gender | Depressive feelings | Weight gain | Mediation | Life Sciences & Biomedicine | Behavioral Sciences | Nutrition & Dietetics | Science & Technology | Anorexia - epidemiology | Depression - physiopathology | Humans | Netherlands - epidemiology | Male | Anorexia - genetics | Stress, Psychological - psychology | Psychiatric Status Rating Scales | Serotonin Plasma Membrane Transport Proteins - genetics | Anorexia - psychology | Overweight - epidemiology | Adult | Female | Weight Gain | Body Mass Index | Genetic Predisposition to Disease | Hyperphagia - etiology | Genetic Association Studies | Self Report | Models, Psychological | Polymorphism, Genetic | Hyperphagia - genetics | Overweight - etiology | Anorexia - etiology | Sex Factors | Depression - psychology | Hyperphagia - epidemiology | Hyperphagia - psychology | Overweight - psychology | Longitudinal Studies | Stress, Psychological - physiopathology | Obesity | Neurosciences | Research institutes | Analysis | Depression, Mental | Food habits | Index Medicus
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5. Full Text Chronic Activation of γ2 AMPK Induces Obesity and Reduces β Cell Function
by Yavari, Arash and Stocker, Claire J and Ghaffari, Sahar and Wargent, Edward T and Steeples, Violetta and Czibik, Gabor and Pinter, Katalin and Bellahcene, Mohamed and Woods, Angela and Martínez de Morentin, Pablo B and Cansell, Céline and Lam, Brian Y.H and Chuster, André and Petkevicius, Kasparas and Nguyen-Tu, Marie-Sophie and Martinez-Sanchez, Aida and Pullen, Timothy J and Oliver, Peter L and Stockenhuber, Alexander and Nguyen, Chinh and Lazdam, Merzaka and O’Dowd, Jacqueline F and Harikumar, Parvathy and Tóth, Mónika and Beall, Craig and Kyriakou, Theodosios and Parnis, Julia and Sarma, Dhruv and Katritsis, George and Wortmann, Diana D.J and Harper, Andrew R and Brown, Laurence A and Willows, Robin and Gandra, Silvia and Poncio, Victor and de Oliveira Figueiredo, Márcio J and Qi, Nathan R and Peirson, Stuart N and McCrimmon, Rory J and Gereben, Balázs and Tretter, László and Fekete, Csaba and Redwood, Charles and Yeo, Giles S.H and Heisler, Lora K and Rutter, Guy A and Smith, Mark A and Withers, Dominic J and Carling, David and Sternick, Eduardo B and Arch, Jonathan R.S and Cawthorne, Michael A and Watkins, Hugh and Ashrafian, Houman
Cell metabolism, ISSN 1550-4131, 05/2016, Volume 23, Issue 5, pp. 821 - 836
Index Medicus
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6. Full Text Cinnamaldehyde supplementation prevents fasting‐induced hyperphagia, lipid accumulation, and inflammation in high‐fat diet‐fed mice
by Khare, Pragyanshu and Jagtap, Sneha and Jain, Yachna and Baboota, Ritesh K and Mangal, Priyanka and Boparai, Ravneet K and Bhutani, Kamlesh K and Sharma, Shyam S and Premkumar, Louis S and Kondepudi, Kanthi K and Chopra, Kanwaljit and Bishnoi, Mahendra
BioFactors (Oxford), ISSN 0951-6433, 03/2016, Volume 42, Issue 2, pp. 201 - 211
adipose tissue | hyperphagia ghrelin | diet‐induced obesity | cinnamaldehyde | lipolysis | leptin | gut microbiota | Gut microbiota | Cinnamaldehyde | Adipose tissue | Diet-induced obesity | Hyperphagia ghrelin | Leptin | Lipolysis | Biochemistry & Molecular Biology | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Acrolein - analogs & derivatives | Inflammation - pathology | Lipolysis - drug effects | Acrolein - administration & dosage | Fasting - adverse effects | Humans | Adipose Tissue - pathology | Hyperphagia - drug therapy | 3T3-L1 Cells | Hyperphagia - metabolism | Inflammation - blood | Interleukin-1beta - blood | Gene Expression Regulation - drug effects | Hyperphagia - pathology | Animals | Inflammation - drug therapy | Diet, High-Fat | Lipid Metabolism - drug effects | Inflammation - genetics | Weight Gain - drug effects | Mice | Adipose Tissue - drug effects | Dietary Supplements | Prevention | Fasting | Body weight | Inflammation | Gene expression | Aldehydes | Hyperphagia | Index Medicus
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7. Full Text Video: HEALTHY OVEREATING
01/2016
Hyperphagia
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8. Full Text Short-term feeding at the wrong time is sufficient to desynchronize peripheral clocks and induce obesity with hyperphagia, physical inactivity and metabolic disorders in mice
by Yasumoto, Yuki and Hashimoto, Chiaki and Nakao, Reiko and Yamazaki, Haruka and Hiroyama, Hanako and Nemoto, Tadashi and Yamamoto, Saori and Sakurai, Mutsumi and Oike, Hideaki and Wada, Naoyuki and Yoshida-Noro, Chikako and Oishi, Katsutaka
Metabolism, clinical and experimental, ISSN 0026-0495, 2016, Volume 65, Issue 5, pp. 714 - 727
Endocrinology & Metabolism | Lipogenesis | Peripheral clock | Feeding rhythm | Leptin resistance | Life Sciences & Biomedicine | Science & Technology | Liver - pathology | Liver - enzymology | Adipose Tissue, White - metabolism | Male | Muscle, Skeletal - metabolism | Motor Activity | Behavior, Animal | Appetite Regulation | Adiposity | Obesity - etiology | Adipose Tissue, White - pathology | Feeding Methods - adverse effects | Muscle, Skeletal - enzymology | Hyperphagia - etiology | Liver - metabolism | Mice, Inbred C57BL | Body Temperature Regulation | Gene Expression Regulation | Lipid Metabolism | Hyperphagia - metabolism | Energy Intake | Animals | Hypothalamus - metabolism | Energy Metabolism | Metabolic Diseases - etiology | Hyperphagia - physiopathology | Adipose Tissue, White - enzymology | Circadian Clocks | Muscle, Skeletal - pathology | Fatty Liver - etiology | Obesity | Neuropeptide Y | Leptin | Analysis | Body weight | Physiological aspects | Hyperphagia | Index Medicus
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9. Full Text Food addiction and obesity: Unnecessary medicalization of hedonic overeating
by Finlayson, Graham
Nature reviews. Endocrinology, ISSN 1759-5029, 08/2017, Volume 13, Issue 8, pp. 493 - 498
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Medicalization - trends | Obesity - psychology | Humans | Behavior, Addictive - therapy | Animals | Behavior, Addictive - diagnosis | Obesity - therapy | Obesity - diagnosis | Feeding Behavior - psychology | Hyperphagia - diagnosis | Hyperphagia - psychology | Behavior, Addictive - psychology | Hyperphagia - therapy | Philosophy | Obesity | Complications and side effects | Health aspects | Food habits | Risk factors | Hyperphagia | Phenotypes | Motivation | Addiction | Hedonic response | Reinforcement | Addictions | Food | Index Medicus
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