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1. Full Text Longitudinal Associations Between Binge Eating and Overeating and Adverse Outcomes Among Adolescents and Young Adults: Does Loss of Control Matter?
by Sonneville, Kendrin R and Horton, Nicholas J and Micali, Nadia and Crosby, Ross D and Swanson, Sonja A and Solmi, Francesca and Field, Alison E
JAMA Pediatrics, ISSN 2168-6203, 02/2013, Volume 167, Issue 2, pp. 149 - 155
OBJECTIVE To investigate the association between overeating (without loss of control) and binge eating (overeating with loss of control) and adverse outcomes.... 
SAMPLE | SELF-REPORTED WEIGHT | FEMALE ADOLESCENTS | PROTECTIVE FACTORS | OBESITY | RISK-FACTORS | DISORDER EXAMINATION | OVERWEIGHT CHILDREN | PEDIATRICS | BULIMIC BEHAVIORS | GIRLS | Binge-Eating Disorder - complications | United States - epidemiology | Prevalence | Prospective Studies | Humans | Male | Hyperphagia - complications | Binge Drinking - epidemiology | Incidence | Depression - etiology | Young Adult | Binge-Eating Disorder - psychology | Overweight - epidemiology | Substance-Related Disorders - epidemiology | Female | Obesity - etiology | Surveys and Questionnaires | Odds Ratio | Severity of Illness Index | Substance-Related Disorders - etiology | Depression - epidemiology | Risk Factors | Binge-Eating Disorder - epidemiology | Models, Statistical | Health Surveys | Overweight - etiology | Obesity - epidemiology | Adolescent | Binge Drinking - etiology | Hyperphagia - epidemiology | Hyperphagia - psychology | Longitudinal Studies | Control | Young adults | Compulsive eating | Youth | Teenagers | Health aspects | Risk factors | Hyperphagia
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2. Full Text Selective stimulation of central GABAAα2,3,5 receptors increases intake and motivation to consume sucrose solution in rats
by Nelson, Tyler S and Holstein, Sarah E and Baird, John-Paul and Pittman, David W
Neuroscience, ISSN 0306-4522, 06/2019, Volume 409, pp. 111 - 119
Benzodiazepines are one of the most commonly prescribed anxiolytic drugs in America, and between 2006 and 2015 prescription rates increased by an estimated... 
hyperphagia | L-838417 | GABA-A | benzodiazepine
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3. Full Text Selective stimulation of central GABA A α2,3,5 receptors increases intake and motivation to consume sucrose solution in rats
by Nelson, Tyler S and Holstein, Sarah E and Baird, John-Paul and Pittman, David W
Neuroscience, ISSN 0306-4522, 06/2019, Volume 409, pp. 111 - 119
Benzodiazepines are one of the most commonly prescribed anxiolytic drugs in America, and between 2006 and 2015 prescription rates increased by an estimated... 
hyperphagia | L-838417 | GABA-A | benzodiazepine
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4. Full Text Obesity-programmed mice are rescued by early genetic intervention
by Bumaschny, Viviana F and Yamashita, Miho and Casas-Cordero, Rodrigo and Otero-Corchón, Verónica and De Souza, Flav́io S. J and Rubinstein, Marcelo and Low, Malcolm J
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4203 - 4212
Obesity is a chronic metabolic disorder affecting half a billion people worldwide. Major difficulties in managing obesity are the cessation of continued weight... 
POMC NEURONS | MEDICINE, RESEARCH & EXPERIMENTAL | BODY-WEIGHT | ENERGY-EXPENDITURE | FOOD-INTAKE | MOUSE | PROOPIOMELANOCORTIN GENE | RATS | EARLY-ONSET OBESITY | MELANOCORTIN | CENTRAL LEPTIN | Neurons - pathology | Adipose Tissue - physiopathology | Obesity - genetics | Adipose Tissue - metabolism | Adiposity | Neurons - metabolism | Pro-Opiomelanocortin - genetics | Disease Models, Animal | Eating | Adipose Tissue - pathology | Obesity - physiopathology | Hypothalamus - pathology | Hyperphagia - metabolism | Hypothalamus - physiopathology | Mice, Knockout | Obesity - metabolism | Hyperphagia - genetics | Hyperphagia - pathology | Pro-Opiomelanocortin - metabolism | Animals | Hyperphagia - prevention & control | Hypothalamus - metabolism | Hyperphagia - physiopathology | Obesity - prevention & control | Mice | Prevention | Measurement | Obesity | Neurons | Body weight | Genetic aspects | Research | Gene expression | Risk factors
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5. 10 clinical skills to overcoming overeating
: 360 degrees of mindful eating
by Somov, Pavel G
2011, Video journal of counseling and therapy
There is more to mindful/conscious eating than just slowing down, paying attention and chewing ten times. Watch this seminar recording and take home a highly... 
Treatment | Food habits | Hyperphagia
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6. Food junkies
: the truth about food addiction
by Tarman, Vera Ingrid and Werdell, Philip R and Canadian Electronic Library (Firm)
2014, ISBN 1459728599
Overeating, binge eating, obesity, anorexia, and bulimia: Food Junkies Built around the experiences of people suffering and recovering from food addictions,... 
Eating disorders | Compulsive eating | Hyperphagia
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7. Full Text Emotional eating as a mediator between depression and weight gain
by van Strien, Tatjana and Konttinen, Hanna and Homberg, Judith R and Engels, Rutger C.M.E and Winkens, Laura H.H
Appetite, ISSN 0195-6663, 05/2016, Volume 100, pp. 216 - 224
Depression is often associated with weight gain but underlying mechanisms are unclear. This study assessed whether three psychological eating styles (emotional... 
Emotional eating | Longitudinal | Gender | Depressive feelings | Weight gain | Mediation | POPULATION | 5-HTTLPR | FOOD-INTAKE | FOLLOW-UP | POLYMORPHISM | OBESITY | NUTRITION & DIETETICS | GENE | DIETARY RESTRAINT | BODY-MASS INDEX | BEHAVIORAL SCIENCES | STRESS | Anorexia - epidemiology | Depression - physiopathology | Humans | Netherlands - epidemiology | Male | Anorexia - genetics | Stress, Psychological - psychology | Psychiatric Status Rating Scales | Serotonin Plasma Membrane Transport Proteins - genetics | Anorexia - psychology | Overweight - epidemiology | Adult | Female | Weight Gain | Body Mass Index | Genetic Predisposition to Disease | Hyperphagia - etiology | Genetic Association Studies | Self Report | Models, Psychological | Polymorphism, Genetic | Hyperphagia - genetics | Overweight - etiology | Anorexia - etiology | Sex Factors | Depression - psychology | Hyperphagia - epidemiology | Hyperphagia - psychology | Overweight - psychology | Longitudinal Studies | Stress, Psychological - physiopathology | Obesity | Neurosciences | Research institutes | Analysis | Depression, Mental | Food habits
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8. Full Text Hypothalamic mTOR pathway mediates thyroid hormone‐induced hyperphagia in hyperthyroidism
by Varela, Luis and Martínez‐Sánchez, Noelia and Gallego, Rosalía and Vázquez, María J and Roa, Juan and Gándara, Marina and Schoenmakers, Erik and Nogueiras, Rubén and Chatterjee, Krishna and Tena‐Sempere, Manuel and Diéguez, Carlos and López, Miguel
The Journal of Pathology, ISSN 0022-3417, 06/2012, Volume 227, Issue 2, pp. 209 - 222
Hyperthyroidism is characterized in rats by increased energy expenditure and marked hyperphagia. Alterations of thermogenesis linked to hyperthyroidism are... 
mTOR | AMPK | energy balance | hyperthyroidism | hypothalamus | food intake | RAT SKELETAL-MUSCLE | MAMMALIAN TARGET | NERVOUS-SYSTEM | ACTIVATED PROTEIN-KINASE | FATTY-ACID-METABOLISM | CELL-GROWTH | PATHOLOGY | MESSENGER-RNA LEVELS | RAPAMYCIN MTOR | ONCOLOGY | ENERGY HOMEOSTASIS | REGULATES FOOD-INTAKE | Neural Pathways - enzymology | AMP-Activated Protein Kinases - metabolism | Phosphorylation | Neural Pathways - drug effects | Hyperphagia - enzymology | TOR Serine-Threonine Kinases - metabolism | Male | Neuropeptide Y - genetics | RNA, Messenger - metabolism | Hyperthyroidism - chemically induced | TOR Serine-Threonine Kinases - antagonists & inhibitors | Time Factors | Triiodothyronine | Hypothalamus - drug effects | Pro-Opiomelanocortin - genetics | Disease Models, Animal | Hyperthyroidism - enzymology | Hyperphagia - etiology | Rats | Hyperthyroidism - physiopathology | Hypothalamus - physiopathology | Rats, Sprague-Dawley | Sirolimus - pharmacology | Feeding Behavior - drug effects | Hyperphagia - genetics | Eating - drug effects | Animals | Hyperphagia - prevention & control | Signal Transduction - drug effects | Weight Loss | Agouti-Related Protein - genetics | Hyperphagia - physiopathology | Hyperthyroidism - genetics | Hypothalamus - enzymology | Hyperthyroidism - complications | Thyroid Hormone Receptors alpha - metabolism | Protein Kinase Inhibitors - pharmacology
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9. Full Text Cinnamaldehyde supplementation prevents fasting‐induced hyperphagia, lipid accumulation, and inflammation in high‐fat diet‐fed mice
by Khare, Pragyanshu and Jagtap, Sneha and Jain, Yachna and Baboota, Ritesh K and Mangal, Priyanka and Boparai, Ravneet K and Bhutani, Kamlesh K and Sharma, Shyam S and Premkumar, Louis S and Kondepudi, Kanthi K and Chopra, Kanwaljit and Bishnoi, Mahendra
BioFactors, ISSN 0951-6433, 03/2016, Volume 42, Issue 2, pp. 201 - 211
Cinnamaldehyde, a bioactive component of cinnamon, is increasingly gaining interest for its preventive and therapeutic effects against metabolic complications... 
adipose tissue | hyperphagia ghrelin | diet‐induced obesity | cinnamaldehyde | lipolysis | leptin | gut microbiota | Gut microbiota | Cinnamaldehyde | Adipose tissue | Diet-induced obesity | Hyperphagia ghrelin | Leptin | Lipolysis | 3T3-L1 ADIPOCYTES | ACTIVATED PROTEIN-KINASE | FOOD-INTAKE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RATS | BROWN ADIPOSE-TISSUE | diet-induced obesity | BODY-WEIGHT | OBESITY | GHRELIN SECRETION | ENDOCRINOLOGY & METABOLISM | CARCASS FAT | BASE-LINE LEPTIN | Acrolein - analogs & derivatives | Inflammation - pathology | Lipolysis - drug effects | Acrolein - administration & dosage | Fasting - adverse effects | Humans | Adipose Tissue - pathology | Hyperphagia - drug therapy | 3T3-L1 Cells | Hyperphagia - metabolism | Inflammation - blood | Interleukin-1beta - blood | Gene Expression Regulation - drug effects | Hyperphagia - pathology | Animals | Inflammation - drug therapy | Diet, High-Fat | Lipid Metabolism - drug effects | Inflammation - genetics | Weight Gain - drug effects | Mice | Adipose Tissue - drug effects | Dietary Supplements | Prevention | Fasting | Body weight | Inflammation | Gene expression | Aldehydes | Hyperphagia | Index Medicus
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10. Full Text Probiotic supplementation prevents high-fat, overfeeding-induced insulin resistance in human subjects
by Hulston, Carl J and Churnside, Amelia A and Venables, Michelle C
British Journal of Nutrition, ISSN 0007-1145, 02/2015, Volume 113, Issue 4, pp. 596 - 602
The purpose of the present study was to determine whether probiotic supplementation (Lactobacillus casei Shirota (LcS)) prevents diet-induced insulin... 
Probiotics | Insulin resistance | High-fat diets | HIGH-CARBOHYDRATE | LIPID-METABOLISM | FERMENTED MILK | INDUCED INFLAMMATION | GUT MICROBIOTA | DIET-INDUCED OBESITY | CASEI STRAIN SHIROTA | SKELETAL-MUSCLE | NUTRITION & DIETETICS | LACTOBACILLUS-CASEI | GLUCOSE-TOLERANCE | Lactobacillus casei | Humans | Diet, High-Fat - adverse effects | England | Male | Insulin - blood | Hyperphagia - blood | Young Adult | Adult | Female | Weight Gain | Hyperglycemia - prevention & control | Cultured Milk Products - microbiology | Glucose Tolerance Test | Blood Glucose - analysis | Hyperphagia - diet therapy | Insulin Resistance | Hyperphagia - metabolism | Energy Intake | Overweight - prevention & control | Animals | Overweight - etiology | Hyperphagia - physiopathology | Probiotics - therapeutic use | Hyperglycemia - etiology | Prevention | Metabolic disorders
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11. Full Text Chronic Activation of γ2 AMPK Induces Obesity and Reduces β Cell Function
by Yavari, Arash and Stocker, Claire J and Ghaffari, Sahar and Wargent, Edward T and Steeples, Violetta and Czibik, Gabor and Pinter, Katalin and Bellahcene, Mohamed and Woods, Angela and Martínez de Morentin, Pablo B and Cansell, Céline and Lam, Brian Y.H and Chuster, André and Petkevicius, Kasparas and Nguyen-Tu, Marie-Sophie and Martinez-Sanchez, Aida and Pullen, Timothy J and Oliver, Peter L and Stockenhuber, Alexander and Nguyen, Chinh and Lazdam, Merzaka and O’Dowd, Jacqueline F and Harikumar, Parvathy and Tóth, Mónika and Beall, Craig and Kyriakou, Theodosios and Parnis, Julia and Sarma, Dhruv and Katritsis, George and Wortmann, Diana D.J and Harper, Andrew R and Brown, Laurence A and Willows, Robin and Gandra, Silvia and Poncio, Victor and de Oliveira Figueiredo, Márcio J and Qi, Nathan R and Peirson, Stuart N and McCrimmon, Rory J and Gereben, Balázs and Tretter, László and Fekete, Csaba and Redwood, Charles and Yeo, Giles S.H and Heisler, Lora K and Rutter, Guy A and Smith, Mark A and Withers, Dominic J and Carling, David and Sternick, Eduardo B and Arch, Jonathan R.S and Cawthorne, Michael A and Watkins, Hugh and Ashrafian, Houman
Cell Metabolism, ISSN 1550-4131, 05/2016, Volume 23, Issue 5, pp. 821 - 836
Despite significant advances in our understanding of the biology determining systemic energy homeostasis, the treatment of obesity remains a medical challenge.... 
AMP-Activated Protein Kinases - metabolism | Hyperphagia - enzymology | Humans | Ribosomes - metabolism | Male | Hyperphagia - complications | Obesity - blood | Receptors, Ghrelin - metabolism | Adult | Female | Neurons - metabolism | Energy Metabolism - genetics | Obesity - complications | Arcuate Nucleus of Hypothalamus - metabolism | Oxidative Phosphorylation | Feeding Behavior | Up-Regulation - genetics | Mitochondria - metabolism | Signal Transduction - genetics | Transcriptome - genetics | Mutation - genetics | Adiposity - genetics | Agouti-Related Protein - metabolism | Aging - pathology | Obesity - pathology | Hyperphagia - genetics | Hyperphagia - pathology | Insulin - metabolism | Animals | Hypothalamus - metabolism | Heterozygote | Mice | Obesity - enzymology | Enzyme Activation | Insulin-Secreting Cells - pathology | Insulin-Secreting Cells - enzymology
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12. Full Text Proopiomelanocortin Deficiency Treated with a Melanocortin-4 Receptor Agonist
by Kühnen, Peter and Clément, Karine and Wiegand, Susanna and Blankenstein, Oliver and Gottesdiener, Keith and Martini, Lea L and Mai, Knut and Blume-Peytavi, Ulrike and Grüters, Annette and Krude, Heiko
The New England Journal of Medicine, ISSN 0028-4793, 07/2016, Volume 375, Issue 3, pp. 240 - 246
Absence of proopiomelanocortin results in early-onset obesity, hyperphagia, hypopigmentation, and hypocortisolism. Two affected patients received... 
CONGENITAL LEPTIN DEFICIENCY | MEDICINE, GENERAL & INTERNAL | GENE | MUTATION | HUMANS | EARLY-ONSET OBESITY | DYSFUNCTION | PIGMENTATION | Blood Pressure | Obesity - drug therapy | Humans | Hyperphagia - drug therapy | alpha-MSH - adverse effects | Receptor, Melanocortin, Type 4 - agonists | alpha-MSH - therapeutic use | Metabolism, Inborn Errors - genetics | Obesity - genetics | Hyperphagia - genetics | Young Adult | Phenotype | Pilot Projects | Adult | Female | Pro-Opiomelanocortin - deficiency | Metabolism, Inborn Errors - drug therapy | Pro-Opiomelanocortin - genetics | alpha-MSH - analogs & derivatives | Hair | Obesity | Melanocyte-stimulating hormone | Nutrition | Melanocortin | Proopiomelanocortin | Body weight loss | Hydrocortisone | Patients | Pigmentation | Quality of life | Defects | Weight control | Hunger | Insulin resistance | Gastrointestinal surgery | Diabetes | Mutation | Endocrinology | Age | Hyperphagia | Pharmaceuticals
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13. Full Text Postnatal overfeeding in rodents by litter size reduction induces major short- and long-term pathophysiological consequences
by Habbout, Ahmed and Li, Na and Rochette, Luc and Vergely, Catherine
Journal of Nutrition, ISSN 0022-3166, 05/2013, Volume 143, Issue 5, pp. 553 - 562
Numerous studies have demonstrated that the early postnatal environment can influence body weight and energy homeostasis into adulthood. Rodents raised in... 
DIABETES-MELLITUS | METABOLIC SYNDROME | NUTRITION & DIETETICS | HYPOTHALAMIC ARCUATE NEURONS | INSULIN-RESISTANCE | GLUCOCORTICOID METABOLISM | ADULT-RATS | DIET-INDUCED OBESITY | CARDIOVASCULAR RISK | NEUROPEPTIDE-Y | EARLY OVERNUTRITION | Appetite | Litter Size | Cardiovascular Diseases - etiology | Infant Nutritional Physiological Phenomena | Overnutrition - physiopathology | Humans | Insulin Resistance | Infant | Overnutrition - metabolism | Hyperphagia - metabolism | Hyperphagia - complications | Hypothalamus - physiopathology | Overnutrition - complications | Energy Intake - physiology | Adipose Tissue - metabolism | Animals | Hypothalamus - metabolism | Hormones - metabolism | Hyperphagia - physiopathology | Obesity - etiology | Development and progression | Infants | Food and nutrition | Hyperphagia
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14. Full Text Stress as a Common Risk Factor for Obesity and Addiction
by Sinha, Rajita and Jastreboff, Ania M
Biological Psychiatry, ISSN 0006-3223, 2013, Volume 73, Issue 9, pp. 827 - 835
Stress is associated with obesity, and the neurobiology of stress overlaps significantly with that of appetite and energy regulation. This review will discuss... 
Psychiatry | reward | stress | metabolism | neuroendocrine | Addiction | obesity | WEIGHT-LOSS MAINTENANCE | EATING BEHAVIOR | NERVOUS-SYSTEM | PSYCHOLOGICAL STRESS | METABOLIC SYNDROME | PSYCHIATRY | FOOD-INTAKE | NEUROSCIENCES | ABDOMINAL OBESITY | INSULIN-RESISTANCE | BODY-MASS INDEX | PITUITARY-ADRENAL AXIS | Cues | Hyperphagia - etiology | Obesity - psychology | Brain - physiopathology | Humans | Risk Factors | Behavior, Addictive - etiology | Obesity - physiopathology | Stress, Psychological - psychology | Feeding Behavior - physiology | Motivation | Hyperphagia - physiopathology | Obesity - etiology | Feeding Behavior - psychology | Hyperphagia - psychology | Behavior, Addictive - psychology | Behavior, Addictive - physiopathology | Stress, Psychological - complications | Stress, Psychological - physiopathology | Obesity | Glucose metabolism | Glucose | Food habits | Risk factors | Dextrose | Reward | Metabolism | Neuroendocrine | Stress
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