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International Immunology, ISSN 0953-8178, 6/2008, Volume 20, Issue 6, pp. 791 - 800
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 07/2012, Volume 303, Issue 1, pp. L54 - L63
Tsuchiya K, Siddiqui S, Risse P-A, Hirota N, Martin JG. The presence of LPS in OVA inhalations affects airway inflammation and AHR but not remodeling in a... 
Animal model | Ovalbumin | Brown norway rat | Endotoxin | Airway hyperresponsiveness | airway hyperresponsiveness | KAPPA-B ACTIVATION | PHYSIOLOGY | MUCIN EXPRESSION | Brown Norway rat | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | HYPERRESPONSIVENESS | RAT MODEL | SMOOTH-MUSCLE | EPITHELIAL-CELLS | RESPIRATORY SYSTEM | ovalbumin | ALLERGIC-ASTHMA | LIPOPOLYSACCHARIDE | animal model | endotoxin | Inflammation - pathology | Hyperplasia - immunology | Bronchial Hyperreactivity - immunology | Eosinophils - drug effects | Ovalbumin - immunology | Chemokine CCL2 - immunology | Bronchoalveolar Lavage Fluid - immunology | Male | NF-kappa B - metabolism | Th2 Cells - immunology | Interferon-gamma - metabolism | Eosinophils - immunology | Lipopolysaccharides - immunology | Th2 Cells - drug effects | Th1 Cells - metabolism | Muscle, Smooth - drug effects | Asthma - immunology | Eosinophils - pathology | Neutrophils - metabolism | Disease Models, Animal | Lymphocytes - metabolism | Th1 Cells - drug effects | Macrophages - pathology | Neutrophils - drug effects | Lymph Nodes - metabolism | Rats | Lymph Nodes - immunology | Interleukin-13 - metabolism | Chemokine CXCL1 - immunology | Bronchial Hyperreactivity - pathology | Lymphocytes - pathology | Macrophages - metabolism | Lipopolysaccharides - pharmacology | Asthma - pathology | Drug Contamination | Inflammation - chemically induced | Asthma - metabolism | Eosinophils - metabolism | NF-kappa B - immunology | Interleukin-13 - immunology | Muscle, Smooth - immunology | Th1 Cells - immunology | Airway Remodeling - immunology | Rats, Inbred BN | Receptor, Epidermal Growth Factor - metabolism | Lymphocytes - immunology | Macrophages - immunology | Neutrophils - pathology | Receptor, Epidermal Growth Factor - immunology | Neutrophils - immunology | Inflammation - immunology | Muscle, Smooth - metabolism | Ovalbumin - pharmacology | Th2 Cells - metabolism | Asthma - chemically induced | Hyperplasia - pathology | Animals | Interferon-gamma - immunology | Lymphocytes - drug effects | Macrophages - drug effects | Airway Remodeling - drug effects | Muscle, Smooth - pathology | Albumin | Physiological aspects | Inflammation | Research | Asthma | Lipopolysaccharides | Index Medicus
Journal Article
Immunology, ISSN 0019-2805, 06/2013, Volume 139, Issue 2, pp. 205 - 218
Allergen‐specific IgE has long been regarded as a major molecular component of allergic asthma. Additionally, there is increasing evidence of the important... 
interleukin‐33 | macrophages | asthma | IgE | airway remodelling | Airway remodelling | Macrophages | Interleukin-33 | Asthma | TH2 RESPONSES | CD4(+) T-LYMPHOCYTES | IMMUNOLOGY | interleukin-33 | MURINE MODEL | ALTERNATIVELY ACTIVATED MACROPHAGES | CYTOKINE PRODUCTION | IMMUNE-RESPONSES | ALLERGIC-ASTHMA | GOBLET CELL HYPERPLASIA | LUNG INFLAMMATION | LATE ASTHMATIC RESPONSE | Immunohistochemistry | Interleukin-1 Receptor-Like 1 Protein | Respiratory System - pathology | Asthma - metabolism | Ovalbumin - immunology | 2-Chloroadenosine - pharmacology | Male | Airway Remodeling - immunology | Interleukins - metabolism | CD4-Positive T-Lymphocytes - immunology | Receptors, Interleukin - metabolism | Inflammation - metabolism | Flow Cytometry | Immunoglobulin E - immunology | Interleukins - immunology | Asthma - immunology | Interleukins - pharmacology | Antibodies, Monoclonal - immunology | Macrophages, Alveolar - immunology | Respiratory System - immunology | CD4 Antigens - immunology | Receptors, Interleukin - immunology | Macrophage Activation - immunology | Antibodies, Monoclonal - pharmacology | CD4-Positive T-Lymphocytes - metabolism | Inflammation - immunology | 2-Chloroadenosine - immunology | Animals | Respiratory System - metabolism | Mice | Mice, Inbred BALB C | Airway Remodeling - drug effects | Macrophages, Alveolar - metabolism | Macrophage Activation - drug effects | CD4 Antigens - metabolism | Monoclonal antibodies | Immunoglobulin E | Inflammation | Interleukins | Index Medicus | Original
Journal Article
BMC Pulmonary Medicine, ISSN 1471-2466, 09/2015, Volume 15, Issue 1, pp. 111 - 111
Journal Article
The American Journal of Pathology, ISSN 0002-9440, 2006, Volume 169, Issue 3, pp. 954 - 966
Journal Article
Molecular Cell, ISSN 1097-2765, 09/2016, Volume 63, Issue 6, pp. 976 - 989
Prostate inflammation has been suggested as an etiology for benign prostatic hyperplasia (BPH). We show that decreased expression of the androgen receptor (AR)... 
microenvironment | androgen receptor | interleukin-1 | inflammation | macrophage | epithelial homeostasis | benign prostatic hyperplasia | INFLAMMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | FINASTERIDE | PROLIFERATION | MODEL | CANCER | EXPRESSION | TUMORIGENESIS | CELL BIOLOGY | Prostatic Hyperplasia - pathology | Cell Proliferation | Epithelial Cells - metabolism | Interleukin-1alpha - immunology | Prostatic Hyperplasia - immunology | Chemokine CCL2 - immunology | Stromal Cells - pathology | Humans | Male | Interleukin-1beta - genetics | Neutrophil Infiltration | Prostate - metabolism | Leukocyte Common Antigens - immunology | Prostate - pathology | Prostate - immunology | Chemokine CXCL10 - immunology | Interleukin-1alpha - genetics | Macrophages - immunology | Macrophages - pathology | Signal Transduction | Stromal Cells - metabolism | Gene Expression Regulation | Homeostasis - immunology | Interleukin-1beta - immunology | Chemokine CCL2 - genetics | Epithelial Cells - pathology | Stromal Cells - immunology | Inflammation | Prostatic Hyperplasia - genetics | Prostatic Hyperplasia - metabolism | Receptors, Androgen - immunology | Macrophages - metabolism | Animals | Receptors, Androgen - genetics | Chemokine CXCL10 - genetics | Epithelial Cells - immunology | Leukocyte Common Antigens - genetics | Mice | Homeostasis - genetics | Medical colleges | Development and progression | Analysis | Index Medicus | Androgen receptor
Journal Article
International Immunology, ISSN 0953-8178, 5/2014, Volume 26, Issue 5, pp. 257 - 267
Tyk2 regulates IL-22/IL-23-induced psoriasis Tyrosine kinase 2 (Tyk2), a member of the Jak kinase family, mediates signals triggered by various cytokines,... 
Psoriasis | Tyk2 | IL-23 | IL-22 | IL-17 | IFN-GAMMA | KERATINOCYTES | CYTOKINE | DELTA-T-CELLS | ANTIMICROBIAL PEPTIDES | IMMUNOLOGY | NUCLEAR TRANSLOCATION | psoriasis | INHIBITOR | EXPRESSION | TH17 CELLS | Inflammation - chemically induced | Hyperplasia - immunology | Defensins - genetics | Skin - metabolism | Humans | Interleukin-23 | Interleukin-17 - immunology | Calgranulin A - genetics | Interleukins - metabolism | TYK2 Kinase - immunology | Signal Transduction - immunology | RNA Interference | Interleukins - immunology | Gene Expression - immunology | Inflammation Mediators - metabolism | TYK2 Kinase - antagonists & inhibitors | Calgranulin A - immunology | Interleukins - pharmacology | Psoriasis - prevention & control | Skin - pathology | Cytokines - immunology | Skin - immunology | Inflammation Mediators - immunology | Psoriasis - immunology | Cell Line | Cytokines - metabolism | Enzyme Inhibitors - pharmacology | Inflammation - immunology | Tyrphostins - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Keratinocytes - immunology | Mice, Knockout | Interleukin-17 - metabolism | Defensins - immunology | Animals | Keratinocytes - drug effects | Signal Transduction - drug effects | Keratinocytes - metabolism | Psoriasis - chemically induced | Inflammation - prevention & control | Mice, Inbred BALB C | TYK2 Kinase - genetics | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2014, Volume 193, Issue 2, pp. 797 - 806
Journal Article
FASEB Journal, ISSN 0892-6638, 06/2015, Volume 29, Issue 6, pp. 2431 - 2438
The first clinical trial of tissue-engineered vascular grafts (TEVGs) identified stenosis as the primary cause of graft failure. In this study, we aimed to... 
Stenosis | Platelet | NK cell | Macrophage | ARTERIAL INJURY | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | NEOINTIMAL HYPERPLASIA | macrophage | AUTOGRAFTS | DEFICIENT MICE | stenosis | CELL BIOLOGY | INTIMAL HYPERPLASIA | BALLOON INJURY | BIOLOGY | P-SELECTIN | platelet | Immunohistochemistry | Blood Platelets - immunology | Blood Vessel Prosthesis - adverse effects | Constriction, Pathologic - etiology | Blood Vessel Prosthesis Implantation - standards | Constriction, Pathologic - immunology | Blood Vessel Prosthesis Implantation - adverse effects | T-Lymphocytes - metabolism | Gene Expression - immunology | Inflammation Mediators - metabolism | Immune System - immunology | Killer Cells, Natural - immunology | Antigens, Differentiation - metabolism | Female | Adaptive Immunity - immunology | Cytokines - genetics | B-Lymphocytes - metabolism | Macrophages - immunology | Cytokines - immunology | Inflammation Mediators - immunology | Immune System - cytology | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Immunity, Innate - immunology | Macrophages - metabolism | Animals | B-Lymphocytes - immunology | Blood Vessel Prosthesis - standards | Blood Platelets - metabolism | Immune System - metabolism | T-Lymphocytes - immunology | Antigens, Differentiation - genetics | Mice | Killer Cells, Natural - metabolism | Antigens, Differentiation - immunology | Index Medicus | Research Communication
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, pp. e85666 - e85666
SHARPIN is a key regulator of NFKB and integrin signaling. Mice lacking Sharpin develop a phenotype known as chronic proliferative dermatitis (CPDM), typified... 
ATOPIC-DERMATITIS | APOPTOSIS | CYTOKINES | INCREASED EXPRESSION | INFLAMMATION | CPDM/CPDM | MULTIDISCIPLINARY SCIENCES | SKIN-DISEASE | IL-4 | PHENOTYPE | COMPONENT | Autoimmunity | Inflammation - pathology | Dermatitis - immunology | Interleukin-13 - immunology | Male | Homeodomain Proteins - immunology | Lymphocyte Depletion | Gene Deletion | Female | Carrier Proteins - immunology | Skin - pathology | Skin - immunology | Signal Transduction | Dermatitis - genetics | Gene Expression Regulation | Inflammation - immunology | Receptors, Cell Surface - immunology | Homeodomain Proteins - genetics | Keratinocytes - immunology | Mice, Knockout | Carrier Proteins - genetics | Dermatitis - pathology | Interleukin-4 - immunology | Keratinocytes - pathology | Animals | B-Lymphocytes - immunology | Inflammation - genetics | T-Lymphocytes - immunology | Mice | Crosses, Genetic | Receptors, Cell Surface - genetics | Skin | Inflammation | T cells | Dermatitis | Integrins | Apoptosis | Chitinase | Veterinary colleges | Disease | Laboratories | Liver | Hyperplasia | Lymphocytes T | mRNA | Kinases | Veterinary medicine | Genotype & phenotype | Interleukin 4 | Interleukin 5 | Lymphocytes | Mineralization | Rodents | Interleukin 1 | Tumor necrosis factor-TNF | Age | Hypoplasia | NF-κB protein | Cytokines | Leukocytes (eosinophilic) | Organs | Keratinocytes | Interleukin 13 | Gene expression | Joint diseases | Eosinophilia | Signaling | Thymic stromal lymphopoietin | Lymphocytes B | Lungs | RAG1 protein | Mutation | Index Medicus
Journal Article