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Human Pathology, ISSN 0046-8177, 2015, Volume 49, pp. 27 - 32
Summary Complex I deficiency causes Leigh syndrome, fatal infant lactic acidosis, and neonatal cardiomyopathy. Mutations in more than 100 nuclear DNA and... 
Pathology | Metabolic disease | Lethal neonatal lactic acidosis | Whole-exome sequencing | Mitochondriopathy | Complex I deficiency | ACAD9 defect | Mitochondrial hyperplasia | Multiorgan failure | Life Sciences & Biomedicine | Science & Technology | Immunohistochemistry | Fibroblasts - enzymology | Diaphragm - pathology | Mitochondria, Heart - pathology | Electron Transport Complex I - deficiency | Humans | Hyperplasia | Male | Cardiomyopathy, Hypertrophic - enzymology | Acyl-CoA Dehydrogenases - genetics | Leigh Disease - pathology | Muscle Weakness - genetics | Amino Acid Metabolism, Inborn Errors - genetics | Autopsy | DNA Mutational Analysis | Electron Transport Complex I - genetics | Fatal Outcome | Muscle Weakness - pathology | Acyl-CoA Dehydrogenases - deficiency | Amino Acid Metabolism, Inborn Errors - pathology | Kidney Tubules - pathology | Infant, Newborn | Multiple Organ Failure - pathology | Acyl-CoA Dehydrogenase - genetics | Cardiomyopathy, Hypertrophic - genetics | Genetic Predisposition to Disease | Mitochondria, Liver - pathology | Mitochondria, Heart - enzymology | Fibroblasts - pathology | Leigh Disease - genetics | Acidosis, Lactic - pathology | Cardiomyopathy, Hypertrophic - diagnosis | Mitochondrial Diseases - enzymology | Phenotype | Acyl-CoA Dehydrogenase - deficiency | Kidney Tubules - enzymology | Acidosis - diagnosis | Multiple Organ Failure - enzymology | Acidosis - genetics | Amino Acid Metabolism, Inborn Errors - enzymology | Mitochondrial Diseases - pathology | Muscle Weakness - diagnosis | Acidosis, Lactic - genetics | Cause of Death | Acidosis, Lactic - enzymology | Amino Acid Metabolism, Inborn Errors - diagnosis | DNA, Mitochondrial - genetics | Muscle Weakness - enzymology | Transfection | Mitochondrial Diseases - genetics | Mitochondria, Muscle - enzymology | Mitochondria, Muscle - pathology | Leigh Disease - enzymology | Cells, Cultured | Multiple Organ Failure - genetics | Codon, Nonsense | Mitochondria, Liver - enzymology | Multiple Organ Failure - diagnosis | Diaphragm - enzymology | Acidosis - pathology | Leigh Disease - diagnosis | Acidosis - enzymology | Mitochondrial Diseases - diagnosis | Acidosis, Lactic - diagnosis | Cardiomyopathy, Hypertrophic - pathology | Infants (Newborn) | Muscles | Genetic aspects | Mitochondrial DNA | Liver | Heart | Urine | Cytochrome | Antigens | Enzymes | Cytomegalovirus | Cardiomyopathy | Defects | Musculoskeletal system | Databases | Rodents | Fibroblasts | Oxidation | Mutation | Metabolic disorders | Deoxyribonucleic acid--DNA | Index Medicus
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 01/2005, Volume 115, Issue 2, pp. 247 - 257
Edema occurs in asthma and other inflammatory diseases when the rate of plasma leakage from blood vessels exceeds the drainage through lymphatic vessels and... 
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Bronchi - microbiology | Inflammation - pathology | Adenoviridae | Lymph Nodes - pathology | Macrophages, Alveolar - pathology | Airway Obstruction | Endothelial Growth Factors | Dendritic Cells - pathology | Hyperplasia - microbiology | Neovascularization, Pathologic - pathology | Respiratory Mucosa - pathology | Respiratory Mucosa - microbiology | Inflammation - metabolism | Lymphatic Vessels - metabolism | Bronchi - blood supply | Mycoplasma Infections - metabolism | Mycoplasma pulmonis | Pulmonary Edema - microbiology | Bronchi - metabolism | Pulmonary Edema - pathology | Vascular Endothelial Growth Factor D - genetics | Bronchi - pathology | Neutrophils - metabolism | Dendritic Cells - metabolism | Neovascularization, Pathologic - microbiology | Neutrophils - pathology | Pulmonary Edema - genetics | Inflammation - microbiology | Mycoplasma Infections - pathology | Gene Expression Regulation - genetics | Lymph Nodes - metabolism | Signal Transduction - genetics | Mice, Inbred C3H | Vascular Endothelial Growth Factor C - genetics | Hyperplasia - pathology | Vascular Endothelial Growth Factor D - metabolism | Animals | Vascular Endothelial Growth Factor C - metabolism | Lymphatic Vessels - pathology | Inflammation - genetics | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | Macrophages, Alveolar - metabolism | Respiratory Mucosa - metabolism | Chronic Disease | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
The American journal of pathology, ISSN 0002-9440, 08/2016, Volume 186, Issue 8, pp. 2193 - 2203
..., and has helped to unravel an unanticipated role of lymphatic vessels in a large number of pathologies, including cancer progression, inflammation, and cardiovascular disease... 
Pathology | Life Sciences & Biomedicine | Science & Technology | Lymphedema - pathology | Tail - pathology | Hyperplasia - pathology | Lymphocytes - pathology | Animals | Flow Cytometry | Lymphatic Vessels - pathology | Fluorescent Antibody Technique | Female | Mice | Myeloid Cells - pathology | Endothelial Cells - pathology | Disease Models, Animal | Index Medicus | Abridged Index Medicus
Journal Article
Nature (London), ISSN 1476-4687, 08/2018, Volume 560, Issue 7720, pp. 649 - 654
Barrier tissue dysfunction is a fundamental feature of chronic human inflammatory diseases(1). Specialized subsets of epithelial cells-including secretory and... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Stem Cells - immunology | Epithelial Cells - metabolism | Epigenesis, Genetic | Humans | Middle Aged | Transcriptome | Hypersensitivity - immunology | Interleukin-13 - immunology | Case-Control Studies | Respiratory Mucosa - pathology | Young Adult | Sinusitis - immunology | Respiratory Mucosa - immunology | Sinusitis - pathology | Adult | Transcription, Genetic | Single-Cell Analysis | Hyperplasia - metabolism | Nasal Polyps - immunology | Rhinitis - immunology | Nasal Polyps - pathology | Cells, Cultured | Epithelial Cells - pathology | Interleukin-4 Receptor alpha Subunit - antagonists & inhibitors | Interleukin-4 Receptor alpha Subunit - immunology | Hyperplasia - pathology | Interleukin-4 - immunology | Sequence Analysis, RNA | Hypersensitivity - pathology | Epithelial Cells - immunology | Adolescent | Stem Cells - pathology | Aged | Rhinitis - pathology | Allergy | Medical research | Immunologic memory | Respiratory organs | Epithelial cells | Physiological aspects | Medicine, Experimental | Cardiopulmonary system | Allergic reaction | Repositories | Disease | Rhinitis | Ecosystems | Rhinosinusitis | Genomics | Hyperplasia | Genomes | Inflammatory diseases | Respiratory tract | Interleukin 4 | Surgery | Bioinformatics | Cytokines | Hypersensitivity | Basal cells | Interleukin 13 | Gene expression | Ribonucleic acid--RNA | Progenitor cells | Stem cells | Cells (biology) | In vivo methods and tests | Polyps | Chemokines | Index Medicus
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, pp. e114885 - e114885
Journal Article