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Human Pathology, ISSN 0046-8177, 2015, Volume 49, pp. 27 - 32
Summary Complex I deficiency causes Leigh syndrome, fatal infant lactic acidosis, and neonatal cardiomyopathy. Mutations in more than 100 nuclear DNA and... 
Pathology | Metabolic disease | Lethal neonatal lactic acidosis | Whole-exome sequencing | Mitochondriopathy | Complex I deficiency | ACAD9 defect | Mitochondrial hyperplasia | Multiorgan failure | DIAGNOSIS | CARDIOMYOPATHY | PATHOLOGY | GENE | DNA | EXPRESSION | Immunohistochemistry | Fibroblasts - enzymology | Diaphragm - pathology | Mitochondria, Heart - pathology | Electron Transport Complex I - deficiency | Humans | Hyperplasia | Male | Cardiomyopathy, Hypertrophic - enzymology | Acyl-CoA Dehydrogenases - genetics | Leigh Disease - pathology | Muscle Weakness - genetics | Amino Acid Metabolism, Inborn Errors - genetics | Autopsy | DNA Mutational Analysis | Electron Transport Complex I - genetics | Fatal Outcome | Muscle Weakness - pathology | Acyl-CoA Dehydrogenases - deficiency | Amino Acid Metabolism, Inborn Errors - pathology | Kidney Tubules - pathology | Infant, Newborn | Multiple Organ Failure - pathology | Acyl-CoA Dehydrogenase - genetics | Cardiomyopathy, Hypertrophic - genetics | Genetic Predisposition to Disease | Mitochondria, Liver - pathology | Mitochondria, Heart - enzymology | Fibroblasts - pathology | Leigh Disease - genetics | Acidosis, Lactic - pathology | Cardiomyopathy, Hypertrophic - diagnosis | Mitochondrial Diseases - enzymology | Phenotype | Acyl-CoA Dehydrogenase - deficiency | Kidney Tubules - enzymology | Acidosis - diagnosis | Multiple Organ Failure - enzymology | Acidosis - genetics | Amino Acid Metabolism, Inborn Errors - enzymology | Mitochondrial Diseases - pathology | Muscle Weakness - diagnosis | Acidosis, Lactic - genetics | Cause of Death | Acidosis, Lactic - enzymology | Amino Acid Metabolism, Inborn Errors - diagnosis | DNA, Mitochondrial - genetics | Muscle Weakness - enzymology | Transfection | Mitochondrial Diseases - genetics | Mitochondria, Muscle - enzymology | Mitochondria, Muscle - pathology | Leigh Disease - enzymology | Cells, Cultured | Multiple Organ Failure - genetics | Codon, Nonsense | Mitochondria, Liver - enzymology | Multiple Organ Failure - diagnosis | Diaphragm - enzymology | Acidosis - pathology | Leigh Disease - diagnosis | Acidosis - enzymology | Mitochondrial Diseases - diagnosis | Acidosis, Lactic - diagnosis | Cardiomyopathy, Hypertrophic - pathology | Infants (Newborn) | Muscles | Genetic aspects | Mitochondrial DNA | Liver | Heart | Urine | Cytochrome | Antigens | Enzymes | Cytomegalovirus | Cardiomyopathy | Data bases | Defects | Musculoskeletal system | Rodents | Fibroblasts | Oxidation | Mutation | Metabolic disorders | Deoxyribonucleic acid--DNA | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2005, Volume 115, Issue 2, pp. 247 - 257
Edema occurs in asthma and other inflammatory diseases when the rate of plasma leakage from blood vessels exceeds the drainage through lymphatic vessels and... 
MEDICINE, RESEARCH & EXPERIMENTAL | INHIBITION | DENDRITIC CELLS | FACTOR-C | GENE-EXPRESSION | LYMPHANGIOGENESIS | RECEPTOR | VEGF-C | TUMOR ANGIOGENESIS | C57BL/6N MICE | ENDOTHELIAL GROWTH-FACTOR
Journal Article
Journal of Oral Pathology & Medicine, ISSN 0904-2512, 10/2011, Volume 40, Issue 9, pp. 677 - 683
Journal Article
Journal Article
Journal Article
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 03/2007, Volume 31, Issue 3, pp. 417 - 426
This study was undertaken to determine the morphologic features and frequency of putative precursor lesions involved in the development of some pure forms of... 
Tubular carcinoma | Carcinoma in situ | Columnar cell lesions | Tubulolobular carcinoma | Cancer progression | Lobular carcinoma | Breast cancer | carcinoma in situ | SURGERY | tubulolobular carcinoma | breast cancer | PATHOLOGY | INTRAEPITHELIAL NEOPLASIA | tubular carcinoma | lobular carcinoma | PROMINENT APICAL SNOUTS | BREAST-CANCER | EVOLUTION | cancer progression | SECRETIONS | EPITHELIUM | columnar cell lesions | DISEASE | COMPARATIVE GENOMIC HYBRIDIZATION | PROGRESSION | GENETIC ALTERATIONS | Carcinoma, Lobular - pathology | Receptors, Estrogen - metabolism | Adenocarcinoma - pathology | Epithelial Cells - metabolism | Cadherins - metabolism | Humans | Middle Aged | Carcinoma, Intraductal, Noninfiltrating - metabolism | Precancerous Conditions - metabolism | Mammary Glands, Human - metabolism | Breast Neoplasms - metabolism | Adenocarcinoma - metabolism | Precancerous Conditions - classification | Biomarkers, Tumor - metabolism | Carcinoma, Ductal, Breast - pathology | Female | Precancerous Conditions - pathology | Carcinoma, Ductal, Breast - metabolism | Hyperplasia - metabolism | Epithelial Cells - classification | Epithelial Cells - pathology | Mammary Glands, Human - pathology | Hyperplasia - pathology | Carcinoma, Intraductal, Noninfiltrating - pathology | Breast Neoplasms - pathology | Carcinoma, Lobular - metabolism | Index Medicus
Journal Article
Journal Article