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PLoS genetics, ISSN 1553-7404, 08/2008, Volume 4, Issue 8, pp. e1000166 - e1000166
Journal Article
PLoS genetics, ISSN 1553-7390, 09/2010, Volume 6, Issue 9, pp. e1001113 - e1001113
Journal Article
PloS one, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, pp. e9258 - e9258
Background: The Notch pathway is essential for proper epidermal differentiation during embryonic skin development. Moreover, skin specific loss of Notch... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Dermatitis, Atopic - genetics | Skin - metabolism | Humans | Receptor, Notch2 - genetics | Receptors, Notch - genetics | Myeloproliferative Disorders - genetics | Granulocyte Colony-Stimulating Factor - genetics | Dermatitis, Atopic - mortality | Flow Cytometry | Receptors, Cytokine - metabolism | Receptors, Cytokine - genetics | Granulocyte Colony-Stimulating Factor - metabolism | Skin - pathology | Immunoglobulins | Cytokines - metabolism | Mice, Inbred C57BL | Receptors, Notch - physiology | Mice, Transgenic | Survival Rate | Signal Transduction - genetics | Dermatitis, Atopic - physiopathology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Receptor, Notch2 - physiology | Animals | Mice, Nude | Models, Biological | Survival Analysis | Myeloproliferative Disorders - physiopathology | Signal Transduction - physiology | Mice | Skin - physiopathology | Receptor, Notch1 - genetics | Myeloproliferative Disorders - mortality | Receptor, Notch1 - physiology | Embryonic development | Psoriasis | Atopic dermatitis | Bone marrow | Transplantation | Skin | B cells | Syngeneic grafts | Pathogenesis | Bone marrow transplantation | Homeostasis | Lichen planus | Inactivation | Dermatitis | Thymus | Signal transduction | Embryogenesis | Biomedical materials | Immunology | Granulocytes | Granulocyte colony-stimulating factor | Lymphocytes | Rodents | Biocompatibility | Drug dosages | Osteopenia | Neutropenia | Spleen | Repressing | Deactivation | Cytokines | Developmental biology | Dermatology | Leukocytes (eosinophilic) | Neutrophils | Keratinocytes | Breast cancer | Gene expression | Myeloproliferative diseases | Hemopoiesis | Signaling | Thymic stromal lymphopoietin | Stem cells | Mast cells | Infiltration | Notch protein | Hematopoietic system | Mutation | Eosinophils | Index Medicus
Journal Article
Immunological reviews, ISSN 0105-2896, 07/2011, Volume 242, Issue 1, pp. 10 - 30
Asthma and allergy are common conditions with complex etiologies involving both genetic and environmental contributions. Recent genome... 
genome‐wide association studies | atopy | linkage studies | asthma | association studies | allergic disease | Association studies | Genome-wide association studies | Linkage studies | Allergic disease | Asthma | Atopy | Life Sciences & Biomedicine | Immunology | Science & Technology | Interleukin-1 Receptor-Like 1 Protein | Asthma - metabolism | Meta-Analysis as Topic | Humans | Genome-Wide Association Study - trends | Infant | Th2 Cells - immunology | Genetic Loci | Neoplasm Proteins - metabolism | Interleukins - metabolism | Genetic Variation | Interleukins - genetics | Asthma - immunology | Hypersensitivity, Immediate - immunology | Adult | Membrane Proteins - metabolism | Neoplasm Proteins - genetics | Child | Cytokines - genetics | Asthma - epidemiology | Genetic Linkage | Genetic Predisposition to Disease | Cytokines - metabolism | Gene Expression Regulation - immunology | Interleukin-33 | Lymphocyte Activation | Membrane Proteins - genetics | Hypersensitivity, Immediate - genetics | Receptors, Cell Surface - metabolism | Immunity, Innate | Th2 Cells - metabolism | Asthma - genetics | Chromosomes, Human, Pair 17 | Hypersensitivity, Immediate - metabolism | Hypersensitivity, Immediate - epidemiology | Receptors, Cell Surface - genetics | Allergy | Genetic aspects | Disease susceptibility | Asthma in children | Allergic reaction | Genomics | Index Medicus | Immune response | Reviews | Thymic stromal lymphopoietin | Etiology | Hypersensitivity | Interleukin 1 | Allergic diseases | Environmental factors | Children | Differentiation | chromosome 17 | GWAS
Journal Article