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1. Full Text The genetics of asthma and allergic disease: a 21st century perspective
by Ober, Carole and Yao, Tsung‐Chieh
Immunological Reviews, ISSN 0105-2896, 07/2011, Volume 242, Issue 1, pp. 10 - 30
Asthma and allergy are common conditions with complex etiologies involving both genetic and environmental contributions. Recent genome‐wide association studies... 
genome‐wide association studies | atopy | linkage studies | asthma | association studies | allergic disease | Association studies | Genome-wide association studies | Linkage studies | Allergic disease | Asthma | Atopy | ALOX5 PROMOTER GENOTYPE | DAY-CARE ATTENDANCE | ENVIRONMENTAL TOBACCO-SMOKE | IMMUNOLOGY | COPY-NUMBER VARIATIONS | HOUSE-DUST MITE | CHILDHOOD ASTHMA | TOTAL SERUM IGE | 3RD NATIONAL-HEALTH | RESPIRATORY-SYNCYTIAL-VIRUS | genome-wide association studies | GENOME-WIDE ASSOCIATION | Interleukin-1 Receptor-Like 1 Protein | Asthma - metabolism | Meta-Analysis as Topic | Humans | Genome-Wide Association Study - trends | Infant | Th2 Cells - immunology | Genetic Loci | Neoplasm Proteins - metabolism | Interleukins - metabolism | Genetic Variation | Interleukins - genetics | Asthma - immunology | Hypersensitivity, Immediate - immunology | Adult | Membrane Proteins - metabolism | Neoplasm Proteins - genetics | Child | Cytokines - genetics | Asthma - epidemiology | Genetic Linkage | Genetic Predisposition to Disease | Cytokines - metabolism | Gene Expression Regulation - immunology | Interleukin-33 | Lymphocyte Activation | Membrane Proteins - genetics | Hypersensitivity, Immediate - genetics | Receptors, Cell Surface - metabolism | Immunity, Innate | Th2 Cells - metabolism | Asthma - genetics | Chromosomes, Human, Pair 17 | Hypersensitivity, Immediate - metabolism | Hypersensitivity, Immediate - epidemiology | Receptors, Cell Surface - genetics | Allergy | Genetic aspects | Disease susceptibility | Asthma in children | Allergic reaction | Genomics | Immune response | Reviews | Thymic stromal lymphopoietin | Etiology | Hypersensitivity | Interleukin 1 | Allergic diseases | Environmental factors | Children | Differentiation | chromosome 17 | GWAS
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2. Full Text Genome-wide scan on total serum IgE levels identifies FCER1A as novel susceptibility locus
by Weidinger, Stephan and Gieger, Christian and Rodriguez, Elke and Baurecht, Hansjörg and Mempel, Martin and Klopp, Norman and Gohlke, Henning and Wagenpfeil, Stefan and Ollert, Markus and Ring, Johannes and Behrendt, Heidrun and Heinrich, Joachim and Novak, Natalija and Bieber, Thomas and Krämer, Ursula and Berdel, Dietrich and Von Berg, Andrea and Bauer, Carl Peter and Herbarth, Olf and Koletzko, Sibylle and Prokisch, Holger and Mehta, Divya and Meitinger, Thomas and Depner, Martin and Von Mutius, Erika and Liang, Liming and Moffatt, Miriam and Cookson, William and Kabesch, Michael and Wichmann, H.-Erich and Illig, Thomas
PLoS Genetics, ISSN 1553-7390, 08/2008, Volume 4, Issue 8, p. e1000166
High levels of serum IgE are considered markers of parasite and helminth exposure. In addition, they are associated with allergic disorders, play a key role in... 
TH2 CYTOKINE LOCUS | CONTROL REGION | ATOPIC-DERMATITIS | IMMUNOGLOBULIN-E CONCENTRATIONS | GENE | JAPANESE POPULATION | DISEASE | GENETICS & HEREDITY | ASTHMA | ASSOCIATION | EPSILON-RI-BETA | Genetic Predisposition to Disease | Multigene Family | Humans | Middle Aged | DNA Repair Enzymes - genetics | Hypersensitivity, Immediate - blood | Hypersensitivity, Immediate - genetics | Male | STAT6 Transcription Factor - genetics | Receptors, IgE - genetics | DNA-Binding Proteins - genetics | Immunoglobulin E - blood | Linkage Disequilibrium | Immunoglobulin E - genetics | Hypersensitivity, Immediate - immunology | Adult | Female | Aged | Polymorphism, Single Nucleotide | Chromosomes, Human, Pair 1 - genetics | Germany | Chromosomes, Human, Pair 5 - genetics | Cohort Studies | Studies | Health sciences | Cytokines | Education | Genes | Eczema | Genomes | Parasites | Autoimmune diseases | Chromosomes | Allergies | Asthma
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3. Full Text Identification of IL6R and chromosome 11q13.5 as risk loci for asthma
by Ferreira, Manuel AR, Dr and Matheson, Melanie C, PhD and Duffy, David L, PhD and Marks, Guy B, Prof and Hui, Jennie, PhD and Le Souëf, Peter, Prof and Danoy, Patrick, PhD and Baltic, Svetlana, PhD and Nyholt, Dale R, PhD and Jenkins, Mark, PhD and Hayden, Catherine, PhD and Willemsen, Gonneke, PhD and Ang, Wei, MSc and Kuokkanen, Mikko, PhD and Beilby, John, PhD and Cheah, Faang, BSc and de Geus, Eco JC, PhD and Ramasamy, Adaikalavan, DPhil and Vedantam, Sailaja, PhD and Salomaa, Veikko, PhD and Madden, Pamela A, PhD and Heath, Andrew C, Prof and Hopper, John L, Prof and Visscher, Peter M, PhD and Musk, Bill, Prof and Leeder, Stephen R, Prof and Jarvelin, Marjo-Riitta, Prof and Pennell, Craig, PhD and Boomsma, Dorret I, Prof and Hirschhorn, Joel N, Prof and Walters, Haydn, Prof and Martin, Nicholas G, Prof and James, Alan, FRACP and Jones, Graham, PhD and Abramson, Michael J, Prof and Robertson, Colin F, FRACP and Dharmage, Shyamali C, PhD and Brown, Matthew A, Prof and Montgomery, Grant W, PhD and Thompson, Philip J, Prof and Australian Asthma Genetics Consort and Australian Asthma Genetics Consortium
Lancet, The, ISSN 0140-6736, 2011, Volume 378, Issue 9795, pp. 1006 - 1014
Summary Background We aimed to identify novel genetic variants affecting asthma risk, since these might provide novel insights into molecular mechanisms... 
Internal Medicine | MEDICINE, GENERAL & INTERNAL | METAANALYSIS | VARIANTS | GENETIC RISK | ECZEMA | DISEASE | EXPRESSION | CHILDHOOD | HERITABILITY | GENOME-WIDE ASSOCIATION | PREDICTION | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Membrane Proteins - genetics | Receptors, Interleukin-6 - genetics | Humans | Middle Aged | Genetic Loci - genetics | Hypersensitivity, Immediate - genetics | Child, Preschool | Male | Chromosomes, Human, Pair 11 - genetics | Asthma - genetics | Linkage Disequilibrium | Young Adult | Adolescent | Asthma - immunology | Aged, 80 and over | Adult | Female | Aged | Polymorphism, Single Nucleotide | Child | Genetic aspects | Research | Risk factors | Asthma | Studies | Genealogy | Disease | Quality control | Genomes | Independent sample | Interleukin 6 | Molecular modelling | Genetic load | Interleukin 6 receptors | Replication | chromosome 11 | Histocompatibility antigen HLA | Atopy
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4. Full Text Prenatal and early-life exposures alter expression of innate immunity genes: The PASTURE cohort study
by Loss, Georg, MSc and Bitter, Sondhja, MD and Wohlgensinger, Johanna, PhD and Frei, Remo, PhD and Roduit, Caroline, MD, MPH and Genuneit, Jon, MD and Pekkanen, Juha, MD and Roponen, Marjut, PhD and Hirvonen, Maija-Riitta, PhD and Dalphin, Jean-Charles, MD, PhD and Dalphin, Marie-Laure, MD and Riedler, Josef, MD and von Mutius, Erika, MD, MSc and Weber, Juliane, MD and Kabesch, Michael, MD and Michel, Sven, MSc and Braun-Fahrländer, Charlotte, MD and Lauener, Roger, MD and PASTURE Study Grp and PASTURE study group
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2012, Volume 130, Issue 2, pp. 523 - 530.e9
Background There is evidence that gene expression of innate immunity receptors is upregulated by farming-related exposures. Objective We sought to determine... 
Allergy and Immunology | nutrition | childhood | prenatal | farming | farm milk | Toll-like receptors | Innate immunity | CD14 | FARM MILK CONSUMPTION | TIME QUANTITATIVE PCR | POLYMORPHISM | IMMUNOLOGY | CHILDHOOD ASTHMA | CHILDREN | PROTECT | TOLL-LIKE RECEPTOR-2 | ALLERGY | HEALTH | ALLERGIC DISEASES | ATOPIC SENSITIZATION | RNA, Messenger - immunology | Prospective Studies | Humans | Immunity, Innate - genetics | Prenatal Exposure Delayed Effects - prevention & control | Infant | Male | Maternal Exposure | RNA, Messenger - biosynthesis | Rural Population | Fetal Blood - chemistry | Gene Expression - immunology | Hypersensitivity, Immediate - immunology | Adult | Female | Surveys and Questionnaires | Agriculture | Milk - immunology | Polymorphism, Single Nucleotide - immunology | Fetal Blood - immunology | Cats | Toll-Like Receptors - immunology | Hypersensitivity, Immediate - genetics | Lipopolysaccharide Receptors - immunology | Hypersensitivity, Immediate - prevention & control | Pregnancy | Prenatal Exposure Delayed Effects - genetics | Immunity, Innate - immunology | Animals | Prenatal Exposure Delayed Effects - immunology | Toll-Like Receptors - genetics | Lipopolysaccharide Receptors - genetics | Polymorphism, Single Nucleotide - genetics | Allergy | Medical colleges | Pregnant women | Analysis | Genes | Environmental health | Genetic research | Respiratory tract diseases | Gene expression | Allergic reaction | Biometry | Public health | Disease | Rural areas | Farm buildings | Allergies | Asthma | Hay fever | Womens health | Questionnaires | Age | Immune system | Farming | Neonates | Inventories | TLR7 protein | TLR5 protein | Hypersensitivity | Single-nucleotide polymorphism | Regression analysis | TLR4 protein | CD14 antigen | Gene polymorphism | Immunity | Rural environments | TLR9 protein | Cord blood | Farms | Pasture | Prenatal experience | Food | Milk | Prenatal Exposure Delayed Effects | Antigens, CD14 | Hypersensitivity, Immediate | Life Sciences | Toll-Like Receptors | Pulmonology and respiratory tract | Gene Expression | Immunity, Innate | Human health and pathology | Polymorphism, Single Nucleotide | RNA, Messenger | Fetal Blood
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5. Full Text Th9 cell development requires a BATF-regulated transcriptional network
by Jabeen, Rukhsana and Goswami, Ritobrata and Awe, Olufolakemi and Kulkarni, Aishwarya and Nguyen, Evelyn T and Attenasio, Andrea and Walsh, Daniel and Olson, Matthew R and Kim, Myung H and Tepper, Robert S and Sun, Jie and Kim, Chang H and Taparowsky, Elizabeth J and Zhou, Baohua and Kaplan, Mark H
Journal of Clinical Investigation, ISSN 0021-9738, 11/2013, Volume 123, Issue 11, pp. 4641 - 4653
T helper 9 (Th9) cells are specialized for the production of IL-9, promote allergic inflammation in mice, and are associated with allergic disease in humans.... 
LINEAGE COMMITMENT | MEDICINE, RESEARCH & EXPERIMENTAL | HOMEOSTASIS | ACTIVATOR | DIFFERENTIATION | INDUCTION | RECEPTORS | PU.1 | EXPRESSION | T-HELPER-CELLS | PLASTICITY | T-Lymphocyte Subsets - immunology | Basic-Leucine Zipper Transcription Factors - metabolism | Oligonucleotide Array Sequence Analysis | Humans | Interferon Regulatory Factors - metabolism | Hypersensitivity - immunology | Infant | Adoptive Transfer | Interferon Regulatory Factors - deficiency | Interleukin-9 - biosynthesis | Inflammation - metabolism | Hypersensitivity - metabolism | Basic-Leucine Zipper Transcription Factors - deficiency | Hypersensitivity - etiology | T-Lymphocytes, Helper-Inducer - immunology | Hypersensitivity, Immediate - immunology | T-Lymphocyte Subsets - classification | Transcription, Genetic | Cell Lineage - genetics | T-Lymphocytes, Helper-Inducer - metabolism | T-Lymphocytes, Helper-Inducer - classification | Mice, Inbred C57BL | Hypersensitivity, Immediate - genetics | Interferon Regulatory Factors - genetics | Mice, Transgenic | Inflammation - immunology | Basic-Leucine Zipper Transcription Factors - genetics | Inflammation - etiology | Mice, Knockout | Animals | Hypersensitivity, Immediate - metabolism | Interleukin-9 - genetics | T-Lymphocyte Subsets - metabolism | Mice | Mice, Inbred BALB C | Cell Lineage - immunology | Transcription factors | Physiological aspects | Development and progression | Inflammation | Genetic aspects | Research | T cells | Genotype & phenotype | Gene expression | Lymphocytes | Cell adhesion & migration
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6. Full Text The burden of disease associated with filaggrin mutations: A population-based, longitudinal birth cohort study
by Henderson, John, MD and Northstone, Kate, MSc and Lee, Simon P., MSc and Liao, Haihui, MD and Zhao, Yiwei, MD and Pembrey, Marcus, MD and Mukhopadhyay, Somnath, MD and Smith, George Davey, MD, DSc and Palmer, Colin N.A., PhD and McLean, W. H. Irwin, PhD, DSc and Irvine, Alan D., MD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2008, Volume 121, Issue 4, pp. 872 - 877.e9
Background Atopic disease is a major health problem. Mutations in the filaggrin gene (FLG) confer major susceptibility to eczema and related asthma. Objective... 
Allergy and Immunology | skin barrier | birth cohort | Eczema | atopy | atopic dermatitis | asthma | filaggrin | BRONCHIAL RESPONSIVENESS | BARRIER | PHENOTYPES | ALSPAC | IMMUNOLOGY | CHILDREN | ATOPIC-DERMATITIS | METHODOLOGY | GENE | ALLERGY | eczema | Respiratory Sounds - immunology | Eczema - diagnosis | Dermatitis, Atopic - genetics | Eczema - genetics | Eczema - immunology | Humans | Child, Preschool | Infant | Male | Dermatitis, Atopic - diagnosis | Asthma - immunology | Dermatitis, Atopic - immunology | Intermediate Filament Proteins - genetics | Hypersensitivity, Immediate - immunology | Female | Child | Hypersensitivity, Immediate - diagnosis | Asthma - diagnosis | Respiratory Sounds - genetics | Genetic Predisposition to Disease | Respiratory Sounds - diagnosis | Hypersensitivity, Immediate - genetics | Gestational Age | Asthma - genetics | Intermediate Filament Proteins - deficiency | Alleles | Mutation | Longitudinal Studies | Cohort Studies | Medical research | Analysis | Genetic research | Medicine, Experimental | Genetic aspects | Children | Health aspects | Prevalence studies (Epidemiology) | Tobacco smoke | Dermatitis | Allergies | Children & youth
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7. Full Text IgG4 production is confined to human IL-10–producing regulatory B cells that suppress antigen-specific immune responses
by van de Veen, Willem, MSc and Stanic, Barbara, PhD and Yaman, Görkem, MD and Wawrzyniak, Marcin, MSc and Söllner, Stefan, Sci Tec and Akdis, Deniz G and Rückert, Beate, Sci Tec and Akdis, Cezmi A., MD and Akdis, Mübeccel, MD, PhD
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2013, Volume 131, Issue 4, pp. 1204 - 1212
Background IL-10–producing regulatory B cells suppress immune responses, and lack of these cells leads to exacerbated symptoms in mouse models of chronic... 
Allergy and Immunology | IgG4 | Immune tolerance | regulatory B cells | IL-10 | IgG | Antigens, CD - immunology | Cell Proliferation | Beekeeping | Humans | Middle Aged | Bee Venoms - immunology | CD4-Positive T-Lymphocytes - pathology | Antigens, CD - genetics | B-Lymphocytes, Regulatory - pathology | CD4-Positive T-Lymphocytes - immunology | Peripheral Tolerance | Immunoglobulin G - biosynthesis | Immunoglobulin G - immunology | Hypersensitivity, Immediate - immunology | Adult | Hypersensitivity, Immediate - genetics | Phospholipases A2 - immunology | B-Lymphocytes, Regulatory - immunology | Hypersensitivity, Immediate - pathology | Adolescent | Antigens - immunology | Interleukin-10 - biosynthesis | Aged | Interleukin-10 - immunology | Desensitization, Immunologic
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8. Full Text Impaired innate interferon induction in severe therapy resistant atopic asthmatic children
by Edwards, M.R and Regamey, N and Vareille, M and Kieninger, E and Gupta, A and Shoemark, A and Saglani, S and Sykes, A and MacIntyre, J and Davies, J and Bossley, C and Bush, A and Johnston, S.L
Mucosal Immunology, ISSN 1933-0219, 07/2013, Volume 6, Issue 4, pp. 797 - 806
Deficient type I interferon-beta and type III interferon-lambda induction by rhinoviruses has previously been reported in mild/moderate atopic asthmatic... 
IN-VITRO | ANTIVIRAL RESPONSES | BRONCHIAL EPITHELIAL-CELLS | DEFICIENT | EXACERBATIONS | BUDESONIDE | ALPHA | RHINOVIRUS INFECTION | IMMUNOLOGY | EXPRESSION | BLOOD MONONUCLEAR-CELLS | Asthma - metabolism | Respiratory Mucosa - drug effects | Poly I-C - administration & dosage | Humans | Immunity, Innate - genetics | Child, Preschool | Male | Respiratory Mucosa - virology | Interferon-Induced Helicase, IFIH1 | Lung - virology | Immunoglobulin E - immunology | Time Factors | Respiratory Mucosa - immunology | Asthma - immunology | Hypersensitivity, Immediate - immunology | Interferon-beta - genetics | Female | Lung - metabolism | Picornaviridae Infections - genetics | Poly I-C - immunology | Toll-Like Receptor 3 - genetics | Interferon-gamma - genetics | Interferons - genetics | Rhinovirus - immunology | DEAD-box RNA Helicases - metabolism | Child | DEAD Box Protein 58 | Picornaviridae Infections - immunology | Interleukin-8 - biosynthesis | RNA, Messenger - genetics | Gene Expression Regulation | Hypersensitivity, Immediate - genetics | Toll-Like Receptor 3 - metabolism | Asthma - genetics | DEAD-box RNA Helicases - genetics | Hypersensitivity, Immediate - metabolism | Adolescent | Respiratory Mucosa - metabolism | Lung - immunology
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9. Full Text Filaggrin loss-of-function mutations predispose to phenotypes involved in the atopic march
by Marenholz, Ingo and Nickel, Renate and Rüschendorf, Franz and Schulz, Florian and Esparza-Gordillo, Jorge and Kerscher, Tamara and Grüber, Christoph and Lau, Susanne and Worm, Margitta and Keil, Thomas and Kurek, Michael and Zaluga, Elisabetha and Wahn, Ulrich and Lee, Young-Ae
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 2006, Volume 118, Issue 4, pp. 866 - 871
Childhood eczema often precedes the development of asthma and allergic rhinitis in the so-called . Recently, 2 loss-of-function mutations in the gene encoding... 
skin barrier | Eczema | atopic march | atopy | genetic association | asthma | allergic rhinitis | filaggrin | DERMATITIS | IMMUNOLOGY | CORNIFIED ENVELOPE | ALLERGY | SENSITIZATION | eczema | LINKAGE | ASSOCIATION | EXPOSURE | AGE | Eczema - complications | Eczema - genetics | Genetic Predisposition to Disease | Humans | Risk Factors | Hypersensitivity, Immediate - complications | Hypersensitivity, Immediate - genetics | Male | Asthma - genetics | Phenotype | Asthma - complications | Pedigree | Intermediate Filament Proteins - genetics | Polymerase Chain Reaction | Family | Female | Heterozygote | Mutation | Child | Genetic research | Genetic aspects | Atopic dermatitis |