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Metabolism, clinical and experimental, ISSN 0026-0495, 1952
Journal
Diabetes, ISSN 0012-1797, 12/2009, Volume 58, Issue 12, pp. 2880 - 2892
Natriuretic Peptides/cGMP/cGMP-Dependent Protein Kinase Cascades Promote Muscle Mitochondrial Biogenesis and Prevent Obesity Kazutoshi Miyashita 1 , Hiroshi... 
SKELETAL-MUSCLE | METABOLIC SYNDROME | PEPTIDE LEVELS | NITRIC-OXIDE SYNTHASE | INSULIN-RESISTANCE | LIPID MOBILIZATION | ENDOCRINOLOGY & METABOLISM | CONGESTIVE-HEART-FAILURE | HYPERTENSION | MICE LACKING | HUMAN ADIPOCYTES | Glucose Intolerance - metabolism | Protein Kinases - metabolism | Dietary Fats - metabolism | Up-Regulation | Natriuretic Peptide, Brain - metabolism | Molecular Sequence Data | Muscle, Skeletal - metabolism | PPAR gamma - metabolism | Glucose Intolerance - etiology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Dietary Fats - administration & dosage | Obesity - etiology | Dietary Fats - adverse effects | Natriuretic Peptides - metabolism | Receptors, Atrial Natriuretic Factor - metabolism | Down-Regulation | Cells, Cultured | Insulin Resistance | Muscle Cells - metabolism | Oxygen Consumption | Mitochondria - metabolism | Mice, Knockout | Obesity - metabolism | Animals | Genetic Engineering | Cyclic GMP - metabolism | Obesity - prevention & control | Trans-Activators - metabolism | Mice | Transcription Factors | PPAR delta - metabolism | Blood Glucose - metabolism | Lipid Peroxidation | Physiological aspects | Mitochondrial DNA | Genetic aspects | Research | Guanylate cyclase | Cyclic guanylic acid | Natriuretic peptides | Index Medicus | Abridged Index Medicus | Original
Journal Article
Gastroenterology, ISSN 0016-5085, 04/2018, Volume 154, Issue 5, pp. 1465 - 1479.e13
Cirrhosis results from accumulation of myofibroblasts derived from quiescent hepatic stellate cells (Q-HSCs); it regresses when myofibroblastic HSCs are... 
Metabolic Reprogramming | Liver Diseases | Hippo Pathway | Fibrogenesis | FIBROSIS | TRANSCRIPTIONAL ACTIVITY | PULMONARY-HYPERTENSION | FATTY LIVER-DISEASE | MECHANISMS | CANCER | METABOLISM | GLUCOSE | GROWTH | Fibro-genesis | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Liver - pathology | Cell Proliferation | Mitochondria, Liver - metabolism | Humans | Glutamine - metabolism | Hedgehog Proteins - metabolism | Hepatic Stellate Cells - metabolism | Smoothened Receptor - metabolism | Phosphoproteins - metabolism | Smoothened Receptor - genetics | Case-Control Studies | Cellular Reprogramming | Myofibroblasts - metabolism | Transfection | Hedgehog Proteins - genetics | RNA Interference | Time Factors | Liver Cirrhosis - metabolism | Liver Cirrhosis, Experimental - pathology | Cell Transdifferentiation | Ketoglutaric Acids - metabolism | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Myofibroblasts - pathology | Mitochondria, Liver - pathology | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Rats | Glutaminase - metabolism | Liver Cirrhosis, Experimental - genetics | Phosphoproteins - genetics | Mice, Knockout | Phenotype | Animals | Energy Metabolism | Adaptor Proteins, Signal Transducing - genetics | Liver Cirrhosis - pathology | Adaptor Proteins, Signal Transducing - metabolism | Liver Cirrhosis, Experimental - metabolism | Glucose metabolism | Metabolites | RNA | Analysis | Liver | Physiological aspects | Genetic research | Verteporfin | Development and progression | Mice | Liver cirrhosis | Glutamine | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 12/2017, Volume 136, Issue 25, pp. 2451 - 2467
BACKGROUND: Pulmonary arterial hypertension (PAH) is characterized by abnormal growth and enhanced glycolysis of pulmonary artery endothelial cells. However,... 
microRNAs | hypertension, pulmonary | endothelial cells | metabolism | endothelial progenitor cells | glycolysis | APOPTOSIS | COACTIVATOR | CARDIAC & CARDIOVASCULAR SYSTEMS | hypertension pulmonary | TUMOR | INHIBITION | MIR-124 | GROWTH | PERIPHERAL VASCULAR DISEASE | EXPRESSION | Cell Proliferation | MicroRNAs - antagonists & inhibitors | Humans | MicroRNAs - metabolism | Polypyrimidine Tract-Binding Protein - antagonists & inhibitors | Familial Primary Pulmonary Hypertension - metabolism | Lim Kinases - metabolism | Monocarboxylic Acid Transporters - metabolism | RNA Interference | Smad5 Protein - metabolism | Bone Morphogenetic Protein Receptors, Type II - antagonists & inhibitors | Disease Models, Animal | Familial Primary Pulmonary Hypertension - pathology | Bone Morphogenetic Protein Receptors, Type II - genetics | Pyruvate Kinase - metabolism | Endothelial Cells - metabolism | Cells, Cultured | Heterogeneous-Nuclear Ribonucleoproteins - metabolism | Rats | Familial Primary Pulmonary Hypertension - genetics | Heterogeneous-Nuclear Ribonucleoproteins - antagonists & inhibitors | Polypyrimidine Tract-Binding Protein - metabolism | Bone Morphogenetic Protein Receptors, Type II - metabolism | Symporters - metabolism | Heterogeneous-Nuclear Ribonucleoproteins - genetics | Animals | Endothelial Cells - cytology | Glycolysis | Smad1 Protein - metabolism | MicroRNAs - genetics | Antagomirs - metabolism | Pyruvate Kinase - genetics | Polypyrimidine Tract-Binding Protein - genetics | RNA, Small Interfering - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 01/2014, Volume 20, Issue 2, pp. 281 - 294
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2013, Volume 123, Issue 5, pp. 2011 - 2023
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, pp. e62068 - e62068
The NF-kappa B pathway plays an important role in chronic inflammatory and autoimmune diseases. Recently, NF-kappa B has also been suggested as an important... 
METABOLIC SYNDROME | OXIDATIVE STRESS | ACTIVATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | MESANGIAL CELLS | INDUCED INFLAMMATION | DIABETIC-NEPHROPATHY | BINDING-ACTIVITY | TRANSCRIPTION FACTOR | ADIPOSE-TISSUE | Albuminuria - complications | Diabetes Mellitus, Experimental - drug therapy | Liver - pathology | Triterpenes - pharmacology | Kidney - pathology | Glycated Hemoglobin A - metabolism | Triterpenes - therapeutic use | Kidney Glomerulus - physiopathology | Male | NF-kappa B - metabolism | Kidney Function Tests | Diabetes Mellitus, Experimental - blood | Adipose Tissue - metabolism | Albuminuria - physiopathology | Kidney - metabolism | Liver - drug effects | Lipids - blood | Inflammation Mediators - metabolism | Lipid Peroxidation - drug effects | Kidney Glomerulus - metabolism | Albuminuria - metabolism | Diabetes Mellitus, Experimental - metabolism | Fatty Acids - metabolism | Kidney - physiopathology | Diabetes Mellitus, Experimental - physiopathology | Albuminuria - pathology | NF-kappa B - antagonists & inhibitors | Kidney - drug effects | Podocytes - metabolism | Cytokines - metabolism | Kidney Glomerulus - drug effects | Liver - metabolism | Adipose Tissue - pathology | Cells, Cultured | Insulin Resistance | Kidney Glomerulus - pathology | Podocytes - pathology | Animals | Podocytes - drug effects | Mice | Oxidative Stress - drug effects | Adipose Tissue - drug effects | Transcription factors | Nephrology | Adipose tissue | Disease | Syngeneic grafts | Oxidative metabolism | Liver | Body weight | Neuropeptides | Adipocytes | Glucose | Animal tissues | Lipid metabolism | Stresses | Obesity | Medical research | NF-κB protein | Cytokines | Internal medicine | Mortality | Abnormalities | Polyamide-imides | Metabolism | Insulin | Fatty acids | Inhibitors | Nephropathy | Weight reduction | Metabolic disorders | Structure-function relationships | Oxidative stress | Animal models | Homeostasis | Adiponectin | Oxidation resistance | Rodents | Collagen (type IV) | Creatinine | Urine | Hypertension | Excretion | Fasting | Kidneys | Diabetes mellitus | Organs | Inflammation | Stress | Medicine | Insulin resistance | Diabetes | Autoimmune diseases | Index Medicus
Journal Article
Antioxidants and Redox Signaling, ISSN 1523-0864, 10/2010, Volume 13, Issue 7, pp. 1011 - 1022
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, pp. e87936 - e87936
The growing incidence of chronic kidney disease remains a global health problem. Obesity is a major risk factor for type-2 diabetes and renal impairment.... 
CHRONIC HEART-FAILURE | TISSUE INFLAMMATION | ATRIAL-NATRIURETIC-PEPTIDE | WAIST CIRCUMFERENCE | GLUCOSE-METABOLISM | MULTIDISCIPLINARY SCIENCES | IN-VIVO | ENHANCES INSULIN SENSITIVITY | FACTOR-KAPPA-B | CHRONIC KIDNEY-DISEASE | HYPERTENSIVE-RATS | Tumor Necrosis Factor-alpha - metabolism | Atrial Natriuretic Factor - metabolism | Endothelin-1 - metabolism | Diabetic Nephropathies - metabolism | Rats | Male | Dinoprost - metabolism | Interleukins - metabolism | Hemin - metabolism | Rats, Zucker | Inflammation - metabolism | Kidney - metabolism | Macrophages - metabolism | Animals | Diabetic Nephropathies - physiopathology | Chemokine CCL3 - metabolism | Adiposity - physiology | Dinoprost - analogs & derivatives | Blood Glucose - metabolism | Heme Oxygenase (Decyclizing) - metabolism | Extracellular Matrix Proteins - metabolism | Kidney - physiopathology | Inflammation - physiopathology | Type 2 diabetes | Adipose tissues | Fibronectins | Obesity | Endothelin | Cytokines | Diabetic nephropathies | Macrophages | Risk factors | Glucose metabolism | Chronic kidney failure | Collagen | Heme | Natriuretic peptides | Adipose tissue | Syngeneic grafts | Selectivity | Adipocytes | Fibronectin | Proteins | Genotype & phenotype | Cyclic GMP | Fibroblasts | Physiology | Carbon monoxide | Public health | Pulmonary arteries | Endothelin 1 | Tumor necrosis factor-α | Metabolism | Gene expression | Studies | Oxygenase | Hemin | Nephropathy | Fibrosis | Interleukin 10 | Infiltration | Renal function | Paracrine signalling | Adiponectin | Interleukin 6 | Rodents | Diaphragm (anatomy) | Extracellular matrix | Tumor necrosis factor-TNF | Lesions | Inducers | Creatinine | Hypertension | Excretion | Kidneys | Diabetes mellitus | Markers | Health risks | Inflammation | Morbidity | Scaffolding | Insulin resistance | Diaphragm | Diabetes | Proteinuria | Index Medicus
Journal Article
Plos one, ISSN 1932-6203, 09/2014, Volume 9, Issue 9, pp. e107392 - e107392
Studies with dietary supplementation of various types of fibers have shown beneficial effects on symptoms of the metabolic syndrome. Short-chain fatty acids... 
IMPROVES INSULIN SENSITIVITY | GERM-FREE-MICE | MULTIDISCIPLINARY SCIENCES | PERFECT ADAPTATION | RATS | SMALL-INTESTINE | MICROBIOTA | BUTYRATE | PROPIONIC-ACID | ENERGY-METABOLISM | INDUCED OBESITY | Biomarkers - metabolism | Plant Gums - metabolism | Galactans - metabolism | Liver - metabolism | Mice, Inbred C57BL | Dietary Fiber - metabolism | Male | Metabolic Syndrome - metabolism | Insulin - blood | Fatty Acids, Volatile - metabolism | Metabolic Syndrome - blood | Animals | Cecum - metabolism | Mannans - metabolism | Triglycerides - blood | Mice | Blood Glucose - metabolism | Body Weight - physiology | Diet, High-Fat - methods | Dietary Supplements | Synthesis | RNA | Body weight | Dietary supplements | Physiological aspects | Triglycerides | Glucose | Fatty acids | Guar | Dextrose | Pediatrics | Laboratories | Liver | Chains | Biology | Metabolic syndrome | High fat diet | Fibers | Consortia | Infusion | Ethics | Microbiota | Hyperglycemia | Energy | Intestine | Rodents | Guar gum | Aging | Bioindicators | Isotopic enrichment | Feces | Supplementation | Gluconeogenesis | Adaptation | Hypertension | Obesity | Nutrition | Propionic acid | Cecum | Fluxes | Gene expression | Fermentation | Metabolism | Insulin | Diet | Biomarkers | Insulin resistance | In vivo methods and tests | Diabetes | Dietary fiber | Acetic acid | Steady state models | Metabolic disorders | Index Medicus
Journal Article