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American Journal of Surgical Pathology, ISSN 0147-5185, 07/2006, Volume 30, Issue 7, pp. 850 - 857
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2016, Volume 11, Issue 3, pp. e0152203 - e0152203
Background and aim Glomerulonephritis (GN) is a leading cause of end-stage renal disease (ESRD) in Africa. Data on epidemiology and outcomes of glomerular... 
SOUTH-AFRICA | NIGERIA | SEGMENTAL GLOMERULOSCLEROSIS | PROFILE | MULTIDISCIPLINARY SCIENCES | GLOMERULAR-DISEASES | SINGLE-CENTER | NEPHROTIC SYNDROME | CHRONIC-RENAL-FAILURE | CHILDREN | FEATURES | Africa - epidemiology | Kidney - pathology | Glomerulosclerosis, Focal Segmental - epidemiology | Humans | Hepatitis B - pathology | Nephrotic Syndrome - pathology | Hepatitis B - epidemiology | Male | Kidney Failure, Chronic - epidemiology | Biopsy | Kidney Failure, Chronic - pathology | Glomerulonephritis - pathology | Nephrotic Syndrome - epidemiology | Female | Glomerulonephritis - epidemiology | Glomerulosclerosis, Focal Segmental - pathology | Glomerulonephritis | Research | Demographic aspects | Risk factors | Prevalence studies (Epidemiology) | End-stage renal disease | Nephrology | Medical services | Systematic review | Immunoglobulin A | Epidemiology | Developing countries--LDCs | Data bases | Hepatitis | IgA nephropathy | Systemic lupus erythematosus | Acquired immune deficiency syndrome--AIDS | Hepatitis B virus | Hypertension | Medical treatment | Industrialized nations | Nephrotic syndrome | Diseases | Studies | Chronic conditions | Observatories | Hospitals | Nursing | Reviews | Diabetes | Kidney diseases | Autoimmune diseases | Hepatitis B | Index Medicus | Life Sciences | Human health and pathology | Developing countries | Acquired immune deficiency syndrome | AIDS | LDCs
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 142, Issue 4, pp. 967 - 977.e2
Background & Aims Notch signaling mediates embryonic vascular development and normal vascular remodeling; Notch1 knockout mice develop nodular regenerative... 
Gastroenterology and Hepatology | Ephrin Signaling | Liver Cancer | Sinusoidal Capillarization | Vascular Tumor | RAT-LIVER | MODEL | INACTIVATION | NODULAR REGENERATIVE HYPERPLASIA | ALAGILLE-SYNDROME | EPHRIN B2/EPHB4 | PATHWAY | ENDOTHELIAL-CELLS | GROWTH-FACTOR | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Hypertension, Portal - genetics | Hemangiosarcoma - metabolism | Liver - pathology | Cell Proliferation | Hypertension, Portal - physiopathology | Humans | Cell Dedifferentiation | Hepatocytes - pathology | Hemangiosarcoma - genetics | Hepatocytes - metabolism | Neovascularization, Pathologic - pathology | Receptor, Notch1 - deficiency | Time Factors | Cell Transformation, Neoplastic - genetics | Liver Neoplasms - pathology | Hemangiosarcoma - pathology | Microscopy, Electron, Scanning | Liver Neoplasms - genetics | Signal Transduction | Endothelial Cells - metabolism | Focal Nodular Hyperplasia - pathology | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Genotype | Focal Nodular Hyperplasia - metabolism | Receptor, Notch1 - metabolism | Cell Transformation, Neoplastic - metabolism | Ephrin-B2 - metabolism | Receptor, EphB4 - metabolism | Mice, Knockout | Liver - blood supply | Phenotype | Animals | Portal Pressure | Hypertension, Portal - metabolism | Focal Nodular Hyperplasia - genetics | Liver Neoplasms - metabolism | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | Cell Transformation, Neoplastic - pathology | Endothelial Cells - pathology | Receptor, Notch1 - genetics | Embryonic development | Liver cancer | Endothelial growth factors | Oncology, Experimental | Research | Universities and colleges | Glycine | Vascular endothelial growth factor | Cancer | Endothelium | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2015, Volume 10, Issue 2, pp. e0118655 - e0118655
We tested the hypothesis that phenotypically modulated smooth muscle cells (SMCs) and related inflammation are associated with the progression of experimental... 
VASCULAR DEVELOPMENT | CHRONIC HYPOXIA | MULTIDISCIPLINARY SCIENCES | HEAVY-CHAIN EXPRESSION | ARTERIAL-HYPERTENSION | PLEXIFORM LESIONS | DISEASE | ATHEROSCLEROSIS | MICE | PATHOLOGY | CORONARY ANGIOPLASTY | Inflammation - pathology | Muscle, Smooth, Vascular - metabolism | Hypertension, Pulmonary - physiopathology | Male | Arterial Occlusive Diseases - genetics | Hypoxia - metabolism | Inflammation - metabolism | Arterial Occlusive Diseases - pathology | T-Lymphocytes - pathology | Arterial Occlusive Diseases - physiopathology | Myocytes, Smooth Muscle - metabolism | Macrophages - immunology | Disease Models, Animal | Mast Cells - immunology | Gene Expression | Macrophages - pathology | Rats | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Inflammation - immunology | Tunica Intima - metabolism | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Tunica Intima - pathology | Arterial Occlusive Diseases - metabolism | Mast Cells - pathology | Fibrosis | Inflammation - genetics | T-Lymphocytes - immunology | Hemodynamics | Hypertension, Pulmonary - pathology | Heart enlargement | Smooth muscle | Inflammation | Muscle proteins | Gene expression | Intermediate filament proteins | Vascular endothelial growth factor | Pulmonary hypertension | Vimentin | Heart | Pediatrics | RANTES | Critical care | Kinases | Macrophages | Experiments | Interleukin 6 | Vascular diseases | Actin | Rodents | Protein-tyrosine kinase receptors | Lesions | Protein-tyrosine kinase | University graduates | Thickening | Hypertension | Tyrosine | Cytokines | Pulmonary arteries | Lung diseases | Muscles | Molecular chains | Gelatinase B | Medicine | Pathology | Hypotheses | Lungs | Obstructive lung disease | Monolayers | Hypoxia | Anesthesiology | Ventricle | Laboratory animals | Hypertrophy | Apoptosis | Index Medicus
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