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Circulation, ISSN 0009-7322, 02/2014, Volume 129, Issue 8, pp. 864 - 874
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 07/2016, Volume 55, Issue 1, pp. 47 - 57
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 07/2018, Volume 59, Issue 1, pp. 127 - 129
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2016, Volume 126, Issue 9, pp. 3313 - 3335
Journal Article
Nature Medicine, ISSN 1078-8956, 01/2013, Volume 19, Issue 1, pp. 74 - 82
Pulmonary arterial hypertension (PAH) is characterized by vascular remodeling associated with obliteration of pulmonary arterioles and formation of plexiform... 
MEDICINE, RESEARCH & EXPERIMENTAL | ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL PROLIFERATION | MONOCROTALINE | CELL BIOLOGY | CHRONIC HYPOXIA | SMOOTH-MUSCLE HYPERPLASIA | INFLAMMATION | GROWTH | APJ | RECEPTOR-2 | EXPRESSION | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Humans | Culture Media, Conditioned - pharmacology | MicroRNAs - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Pulmonary Artery - metabolism | RNA Interference | Fibroblast Growth Factor 2 - metabolism | Myocytes, Smooth Muscle - metabolism | Muscle, Smooth, Vascular - physiology | Apelin | Signal Transduction | Endothelial Cells - metabolism | Down-Regulation | Mice, Inbred C57BL | Cells, Cultured | Fibroblast Growth Factor 2 - biosynthesis | Rats | Familial Primary Pulmonary Hypertension | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Mice, Knockout | Animals | Mice | MicroRNAs - genetics | RNA, Small Interfering | Vascular Diseases - metabolism | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Receptor, Fibroblast Growth Factor, Type 1 - biosynthesis | Cell Movement | MicroRNA | Peptides | Development and progression | Fibroblast growth factors | Genetic aspects | Health aspects | Pulmonary hypertension | Endothelium | Hypertension | Vein & artery diseases | Pulmonary arteries | Index Medicus
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 02/2012, Volume 185, Issue 3, pp. 311 - 321
Journal Article
Circulation Research, ISSN 0009-7330, 10/2015, Volume 117, Issue 10, pp. 870 - 883
RATIONALE:The pathogenesis of pulmonary arterial hypertension (PAH) remains unclear. The 4 microRNAs representing the miR-143 and miR-145 stem loops are... 
microRNAs | hypertension, pulmonary | endothelium | myocytes, smooth muscle | cell movement | exosomes | MIGRATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MECHANISM | smooth muscle | PHENOTYPE | pulmonary | CANCER | myocytes | MIR-145 | GROWTH | DISEASE | PERIPHERAL VASCULAR DISEASE | CLUSTER | HEMATOLOGY | hypertension | EXPRESSION | Exosomes - metabolism | Vascular Remodeling | Muscle, Smooth, Vascular - metabolism | Humans | Myocytes, Smooth Muscle - pathology | Hypertension, Pulmonary - physiopathology | Male | MicroRNAs - metabolism | Muscle, Smooth, Vascular - physiopathology | Case-Control Studies | Hypertension, Pulmonary - prevention & control | Pulmonary Artery - metabolism | Arterial Pressure | Transfection | Time Factors | Cattle | Female | Binding Sites | Myocytes, Smooth Muscle - metabolism | Promoter Regions, Genetic | Signal Transduction | Endothelial Cells - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Cell Communication | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Mice, Knockout | Transcription Factors - metabolism | Muscle, Smooth, Vascular - pathology | Animals | MicroRNAs - genetics | Ventricular Pressure | HeLa Cells | Endothelial Cells - pathology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Ventricular Function, Right | Cell Movement | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 06/2010, Volume 121, Issue 24, pp. 2661 - 2671
Background-Excessive proliferation and impaired apoptosis of pulmonary artery (PA) smooth muscle cells (PASMCs) contribute to vascular obstruction in patients... 
genetic | mitochondrial diseases | epigenesis | hypoxia-inducible factor-1 | gene silencing | pulmonary | oxidation-reduction | subunit | hypertension | superoxide dismutase 2 | MULTIPLE-MYELOMA | APOPTOSIS | METHYLATION | CARDIAC & CARDIOVASCULAR SYSTEMS | hypertension, pulmonary | epigenesis, genetic | HYPOXIA | CANCER | OVEREXPRESSION | GENE | hypoxia-inducible factor-1, alpha-subunit | PATHWAY | DNA METHYLTRANSFERASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | SOD2 EXPRESSION | Superoxide Dismutase - genetics | Cell Proliferation | Mitochondria, Muscle - enzymology | Humans | Myocytes, Smooth Muscle - pathology | Rats | Hypertension, Pulmonary - metabolism | Rats, Sprague-Dawley | Hydrogen Peroxide - metabolism | Epigenesis, Genetic - physiology | Phenotype | Animals | Biomimetics | Epigenesis, Genetic - genetics | Rats, Mutant Strains | Adenoviridae - genetics | Hypertension, Pulmonary - drug therapy | Hypertension, Pulmonary - pathology | Myocytes, Smooth Muscle - metabolism | Superoxide Dismutase - metabolism | Apoptosis | Disease Models, Animal | Cell proliferation | Physiological aspects | Development and progression | Superoxide dismutase | Research | Vascular smooth muscle | Pulmonary hypertension | Index Medicus | Abridged Index Medicus | Epigenetic gene methylation | DNA methyltransferase | Pulmonary arterial hypertension | Hypoxia-inducible factor-1α (HIF-1α) | Voltage-gated potassium channels (Kv1.5)
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2013, Volume 123, Issue 8, pp. 3600 - 3613
Dysfunctional bone morphogenetic protein receptor-2 (BMPR2) signaling is implicated in the pathogenesis of pulmonary arterial hypertension (PAH). We used a... 
MEDICINE, RESEARCH & EXPERIMENTAL | GENE | BONE MORPHOGENETIC PROTEIN | ARTERIAL-HYPERTENSION | HEART-FAILURE | IN-VIVO | APELIN | SMOOTH-MUSCLE-CELLS | TGF-BETA RECEPTOR | EXPRESSION | T-CELLS | Cell Proliferation | Humans | Microvessels - pathology | Male | Cell Hypoxia | Tacrolimus - pharmacology | Hypertension, Pulmonary - drug therapy | Endothelial Cells - physiology | Bone Morphogenetic Protein Receptors, Type II - genetics | Signal Transduction | Bone Morphogenetic Protein 4 - physiology | Endothelium, Vascular - physiopathology | Rats | Hypertension, Pulmonary - metabolism | Inhibitor of Differentiation Protein 1 - metabolism | Neointima - metabolism | Rats, Sprague-Dawley | Bone Morphogenetic Protein Receptors, Type II - metabolism | Mice, Knockout | Animals | High-Throughput Screening Assays | Neointima - drug therapy | Neointima - pathology | Tacrolimus Binding Protein 1A - metabolism | Cell Line, Tumor | Inhibitor of Differentiation Protein 1 - genetics | Endothelium, Vascular - pathology | Mice | Hypertension, Pulmonary - pathology | Smad Proteins - metabolism | Pulmonary Artery - pathology | Apoptosis | Endothelial Cells - drug effects | Hypertension | Cell receptors | Physiological aspects | Cellular signal transduction | Research | Properties | Endothelium | Proteins | Medical research | Scholarships & fellowships | Research & development--R&D | Pulmonary arteries | Rodents | Hypoxia | Ligands | Mutation | Drug dosages | Index Medicus | Abridged Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 07/2012, Volume 126, Issue 4, pp. 455 - 467
Background-Epigenetic programming, dynamically regulated by histone acetylation, is a key mechanism regulating cell proliferation and survival. Little is known... 
genetic | epigenesis | vorinostat | histone deacetylation | pulmonary | valproic acid | hypertension | TARGET | CELLS | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | ACETYLATION | hypertension, pulmonary | epigenesis, genetic | PROLIFERATION | EMERGENCE | ARTERIAL-HYPERTENSION | PROSTATE-CANCER | IN-VIVO | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Muscle, Smooth, Vascular - metabolism | Humans | Male | Valproic Acid - pharmacology | Pulmonary Artery - metabolism | Histone Deacetylase 1 - antagonists & inhibitors | Valproic Acid - therapeutic use | Hypertension, Pulmonary - drug therapy | Lung - metabolism | Hydroxamic Acids - pharmacology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Lung - pathology | Cells, Cultured | Rats | Histone Deacetylases - metabolism | Hypertension, Pulmonary - metabolism | Hypoxia - complications | Platelet-Derived Growth Factor - pharmacology | Pulmonary Artery - drug effects | Rats, Sprague-Dawley | Muscle, Smooth, Vascular - pathology | Animals | Lung - drug effects | Histone Deacetylase Inhibitors - pharmacology | Histone Deacetylases - drug effects | Hydroxamic Acids - therapeutic use | Cell Proliferation - drug effects | Histone Deacetylase Inhibitors - therapeutic use | Hypertension, Pulmonary - etiology | Pulmonary Artery - pathology | Histone Deacetylase 1 - metabolism | Cell proliferation | Divalproex | Care and treatment | Usage | Enzyme inhibitors | Physiological aspects | Research | Valproic acid | Health aspects | Pulmonary hypertension | Index Medicus | Abridged Index Medicus
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 02/2016, Volume 310, Issue 3, pp. L249 - L262
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2015, Volume 112, Issue 13, pp. E1614 - E1623
Hypoxic pulmonary vasoconstriction (HPV) optimizes pulmonary ventilation-perfusion matching in regional hypoxia, but promotes pulmonary hypertension in global... 
Ceramide | Transient receptor potential canonical 6 | Pulmonary hypertension | Neutral sphingomyelinase | AIRWAY EPITHELIAL-CELLS | pulmonary hypertension | MULTIDISCIPLINARY SCIENCES | EPOXIDE HYDROLASE | transient receptor potential canonical 6 | SPHINGOSINE 1-PHOSPHATE | TRANSMEMBRANE CONDUCTANCE REGULATOR | neutral sphingomyelinase | ceramide | SMOOTH-MUSCLE | ARTERIAL-HYPERTENSION | LUNG-DISEASE | CYSTIC-FIBROSIS | GAS-EXCHANGE | Calcium - metabolism | Humans | Type C Phospholipases - metabolism | Oxygen - metabolism | Coronary Vessels - metabolism | Pulmonary Artery - metabolism | rho-Associated Kinases - metabolism | Sphingomyelin Phosphodiesterase - metabolism | Lung - metabolism | Receptors, Lysosphingolipid - metabolism | Myocytes, Smooth Muscle - metabolism | Calcium Signaling | Mice, Inbred CFTR | Lung - pathology | TRPC6 Cation Channel | Signal Transduction | Mice, Inbred C57BL | Vasoconstriction | Ceramides - chemistry | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Hypertension, Pulmonary - metabolism | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Protein Transport | Animals | Hypoxia - pathology | TRPC Cation Channels - metabolism | Mice | Heart failure | Care and treatment | Physiological aspects | Muscle cells | Development and progression | Sphingolipids | Research | Smooth muscle | Hypoxia | Calcium | Rodents | Index Medicus | Biological Sciences | PNAS Plus
Journal Article