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Journal Article
Cell, ISSN 0092-8674, 10/2011, Volume 147, Issue 2, pp. 293 - 305
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2016, Volume 36, Issue 10, pp. 2078 - 2087
Journal Article
Circulation, ISSN 0009-7322, 03/2015, Volume 131, Issue 11, pp. 1006 - 1018
BACKGROUND—The vascular remodeling responsible for pulmonary arterial hypertension (PAH) involves predominantly the accumulation of α-smooth muscle... 
Human | Hypertension | Vimentin | Sirolimus | Twist1 protein | Vascular remodeling | Epithelial-mesenchymal transition | Neointima | Animal | Type ii | Models | Bone morphogenetic protein receptors | Cardiovascular diseases | Pulmonary | RAPAMYCIN | MIGRATION | FIBROSIS | models, animal | CARDIAC & CARDIOVASCULAR SYSTEMS | neointima | hypertension, pulmonary | vimentin | cardiovascular diseases | vascular remodeling | DISCOVERY | bone morphogenetic protein receptors, type II | IN-VITRO | sirolimus | TWIST1 protein, human | ARTERIAL-HYPERTENSION | PERIPHERAL VASCULAR DISEASE | epithelial-mesenchymal transition | SMOOTH-MUSCLE-CELLS | KNOCKOUT RATS | TRANSDIFFERENTIATION | Vascular Remodeling | Humans | Gene Expression Profiling | Hypertension, Pulmonary - chemically induced | Actins - genetics | RNA, Messenger - biosynthesis | Vimentin - genetics | Cell Transdifferentiation | Lung - metabolism | Disease Models, Animal | Lung - pathology | Actins - biosynthesis | Bone Morphogenetic Protein Receptors, Type II - genetics | Vimentin - biosynthesis | Cells, Cultured | Rats | Hypertension, Pulmonary - genetics | Hypoxia - complications | Monocrotaline - toxicity | Sirolimus - pharmacology | Animals | Biomarkers | Mutation | Endothelial Cells - pathology | Lung - blood supply | Hypertension, Pulmonary - pathology | Mesoderm - pathology | Bone Morphogenetic Protein Receptors, Type II - biosynthesis | Cell Movement | Index Medicus | Abridged Index Medicus | Life Sciences
Journal Article
Circulation, ISSN 0009-7322, 04/2018, Volume 137, Issue 17, pp. 1796 - 1810
BACKGROUND:We hypothesized that pulmonary venous hypertension in heart failure (HF) leads to predominate remodeling of pulmonary veins and that the severity of... 
heart failure | CARDIAC & CARDIOVASCULAR SYSTEMS | SILDENAFIL | MITRAL-STENOSIS | VENOOCCLUSIVE DISEASE | pulmonary hypertension | pulmonary function | EXERCISE CAPACITY | PATHOLOGY | CLINICAL STATUS | HEMODYNAMICS | diffusing capacity of the lungs for carbon monoxide | heart failure with preserved ejection fraction | right ventricle | COMMUNITY | ARTERIAL-HYPERTENSION | PERIPHERAL VASCULAR DISEASE | PHOSPHODIESTERASE-5 INHIBITION | Pulmonary Veins - pathology | Vascular Remodeling | Humans | Middle Aged | Heart Failure - physiopathology | Hypertension, Pulmonary - diagnostic imaging | Hypertension, Pulmonary - physiopathology | Male | Pulmonary Artery - diagnostic imaging | Case-Control Studies | Arterial Pressure | Autopsy | Aged, 80 and over | Female | Registries | Ventricular Dysfunction, Right - physiopathology | Heart Failure - diagnostic imaging | Severity of Illness Index | Pulmonary Veins - physiopathology | Lung - physiopathology | Pulmonary Artery - physiopathology | Pulmonary Veins - diagnostic imaging | Heart Failure - pathology | Stroke Volume | Pulmonary Diffusing Capacity | Venous Pressure | Aged | Lung - blood supply | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Ventricular Dysfunction, Right - pathology | Ventricular Function, Right | Ventricular Dysfunction, Right - diagnostic imaging | Heart failure | Pulmonary veins | Cardiac output | Research | Pulmonary hypertension | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2013, Volume 123, Issue 8, pp. 3600 - 3613
Dysfunctional bone morphogenetic protein receptor-2 (BMPR2) signaling is implicated in the pathogenesis of pulmonary arterial hypertension (PAH). We used a... 
MEDICINE, RESEARCH & EXPERIMENTAL | GENE | BONE MORPHOGENETIC PROTEIN | ARTERIAL-HYPERTENSION | HEART-FAILURE | IN-VIVO | APELIN | SMOOTH-MUSCLE-CELLS | TGF-BETA RECEPTOR | EXPRESSION | T-CELLS | Cell Proliferation | Humans | Microvessels - pathology | Male | Cell Hypoxia | Tacrolimus - pharmacology | Hypertension, Pulmonary - drug therapy | Endothelial Cells - physiology | Bone Morphogenetic Protein Receptors, Type II - genetics | Signal Transduction | Bone Morphogenetic Protein 4 - physiology | Endothelium, Vascular - physiopathology | Rats | Hypertension, Pulmonary - metabolism | Inhibitor of Differentiation Protein 1 - metabolism | Neointima - metabolism | Rats, Sprague-Dawley | Bone Morphogenetic Protein Receptors, Type II - metabolism | Mice, Knockout | Animals | High-Throughput Screening Assays | Neointima - drug therapy | Neointima - pathology | Tacrolimus Binding Protein 1A - metabolism | Cell Line, Tumor | Inhibitor of Differentiation Protein 1 - genetics | Endothelium, Vascular - pathology | Mice | Hypertension, Pulmonary - pathology | Smad Proteins - metabolism | Pulmonary Artery - pathology | Apoptosis | Endothelial Cells - drug effects | Hypertension | Cell receptors | Physiological aspects | Cellular signal transduction | Research | Properties | Endothelium | Proteins | Medical research | Scholarships & fellowships | Research & development--R&D | Pulmonary arteries | Rodents | Hypoxia | Ligands | Mutation | Drug dosages | Index Medicus | Abridged Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 01/2009, Volume 104, Issue 2, pp. 236 - 244
Inflammatory cytokine interleukin (IL)-6 is elevated in the serum and lungs of patients with pulmonary artery hypertension (PAH). Several animal models of PAH... 
Pulmonary artery hypertension | Proliferation | Interleukin-6 | CARDIAC & CARDIOVASCULAR SYSTEMS | pulmonary artery hypertension | proliferation | UP-REGULATES EXPRESSION | interleukin-6 | VASCULAR STRUCTURE | ARTERIAL-HYPERTENSION | PLEXIFORM LESIONS | ENDOTHELIAL-CELLS | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | INFLAMMATORY CYTOKINES | HEMATOLOGY | NEOINTIMAL FORMATION | TRANSGENIC MICE | Blood Pressure | Up-Regulation | Cell Proliferation | Microtubule-Associated Proteins - metabolism | Arterioles - immunology | Hyperplasia | Repressor Proteins | Hypertension, Pulmonary - physiopathology | Muscle, Smooth, Vascular - immunology | Vascular Endothelial Growth Factor A - metabolism | Muscle, Smooth, Vascular - physiopathology | Hypertrophy, Right Ventricular - physiopathology | Time Factors | Hypertrophy, Right Ventricular - pathology | Interleukin-6 - metabolism | Proto-Oncogene Proteins - metabolism | Vascular Resistance | Inhibitor of Apoptosis Proteins | Interleukin-6 - genetics | Mice, Inbred C57BL | Hypertension, Pulmonary - immunology | Mice, Transgenic | Hypoxia - complications | Pulmonary Artery - physiopathology | Proto-Oncogene Proteins c-myc - metabolism | Endothelial Cells - immunology | Arterioles - pathology | Muscle, Smooth, Vascular - pathology | Animals | Hypertrophy, Right Ventricular - immunology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Ventricular Pressure | Proto-Oncogene Proteins c-bcl-2 | Endothelial Cells - pathology | Chronic Disease | Hypertension, Pulmonary - pathology | Transforming Growth Factor beta - metabolism | Pulmonary Artery - pathology | Ventricular Function, Right | Apoptosis | Mitogen-Activated Protein Kinases - metabolism | Pulmonary Artery - immunology | Index Medicus
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2015, Volume 21, Issue 7, pp. 777 - 785
Journal Article
Circulation Research, ISSN 0009-7330, 10/2015, Volume 117, Issue 10, pp. 870 - 883
RATIONALE:The pathogenesis of pulmonary arterial hypertension (PAH) remains unclear. The 4 microRNAs representing the miR-143 and miR-145 stem loops are... 
microRNAs | hypertension, pulmonary | endothelium | myocytes, smooth muscle | cell movement | exosomes | MIGRATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MECHANISM | smooth muscle | PHENOTYPE | pulmonary | CANCER | myocytes | MIR-145 | GROWTH | DISEASE | PERIPHERAL VASCULAR DISEASE | CLUSTER | HEMATOLOGY | hypertension | EXPRESSION | Exosomes - metabolism | Vascular Remodeling | Muscle, Smooth, Vascular - metabolism | Humans | Myocytes, Smooth Muscle - pathology | Hypertension, Pulmonary - physiopathology | Male | MicroRNAs - metabolism | Muscle, Smooth, Vascular - physiopathology | Case-Control Studies | Hypertension, Pulmonary - prevention & control | Pulmonary Artery - metabolism | Arterial Pressure | Transfection | Time Factors | Cattle | Female | Binding Sites | Myocytes, Smooth Muscle - metabolism | Promoter Regions, Genetic | Signal Transduction | Endothelial Cells - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Cell Communication | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Mice, Knockout | Transcription Factors - metabolism | Muscle, Smooth, Vascular - pathology | Animals | MicroRNAs - genetics | Ventricular Pressure | HeLa Cells | Endothelial Cells - pathology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Ventricular Function, Right | Cell Movement | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 2012, Volume 126, Issue 22, pp. 2601 - 2611
Journal Article