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Hypertension, ISSN 0194-911X, 02/2011, Volume 57, Issue 2, pp. 308 - 313
Journal Article
Hypertension, ISSN 0194-911X, 12/2011, Volume 58, Issue 6, pp. 1066 - 1072
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 08/2015, Volume 309, Issue 4, pp. F273 - F279
Obesity remains a prominent public health concern. Obesity not only contributes greatly to cardiovascular events but has also been identified to initiate and... 
Adipocyte | Obesity | Insulin resistance | Renin-angiotensin-aldosterone system | Renal artery stenosis | Microvascula-ture | OXIDATIVE STRESS | PHYSIOLOGY | RENAL-FUNCTION | WEIGHT-LOSS | BLOOD-PRESSURE | renin-angiotensin-aldosterone system | renal artery stenosis | INSULIN-RESISTANCE | adipocyte | UROLOGY & NEPHROLOGY | BODY-MASS INDEX | INTRAABDOMINAL PRESSURE | CHRONIC KIDNEY-DISEASE | insulin resistance | obesity | microvasculature | INITIATED METABOLIC SYNDROME | ADIPOSE-TISSUE | Hypertension, Renovascular - epidemiology | Prevalence | Prognosis | Humans | Renal Artery Obstruction - physiopathology | Hypertension, Renovascular - physiopathology | Hypertension, Renovascular - metabolism | Renal Insufficiency, Chronic - epidemiology | Renal Insufficiency, Chronic - metabolism | Microcirculation | Renal Insufficiency, Chronic - therapy | Renin-Angiotensin System | Risk Assessment | Signal Transduction | Risk Factors | Obesity - physiopathology | Hypertension, Renovascular - therapy | Renal Insufficiency, Chronic - physiopathology | Obesity - metabolism | Renal Artery Obstruction - epidemiology | Animals | Obesity - epidemiology | Renal Circulation | Obesity - therapy | Hemodynamics | Renal Artery Obstruction - metabolism | Renal Artery Obstruction - therapy | Angiogenic Proteins - metabolism | Fat cells | Angiotensin | Development and progression | Research | Health aspects | Reviews
Journal Article
Journal Article
American Journal of Hypertension, ISSN 0895-7061, 9/2015, Volume 28, Issue 9, pp. 1083 - 1090
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 03/2016, Volume 310, Issue 5, pp. F372 - F384
Renovascular hypertension (RVH) is a common cause of both cardiovascular and renal morbidity and mortality. In renal artery stenosis (RAS), atrophy in the... 
Hypertension | CCL2 | Macrophages | Parenchymal cells | Renal artery stenosis | CCR2 | parenchymal cells | GENETIC DEFICIENCY | PHYSIOLOGY | DB/DB MICE | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INJURY | SMAD3 PROTECTS | DIABETIC-NEPHROPATHY | CHEMOKINE RECEPTORS CCR2 | macrophages | renal artery stenosis | DISEASE | UROLOGY & NEPHROLOGY | ARTERY STENOSIS | KIDNEY | hypertension | Mannose-Binding Lectins - metabolism | Kidney - pathology | Arginase - metabolism | Male | Molecular Targeted Therapy | Hypertension, Renovascular - genetics | Hypertension, Renovascular - metabolism | Receptors, CCR2 - antagonists & inhibitors | Atrophy | Hypertension, Renovascular - drug therapy | Lectins, C-Type - metabolism | Kidney - metabolism | Nephritis, Interstitial - metabolism | Nephritis, Interstitial - pathology | Piperidines - pharmacology | Time Factors | Nephritis, Interstitial - prevention & control | Protective Agents - pharmacology | Renal Artery Obstruction - genetics | Antigens, Differentiation - metabolism | Chemokine CCL2 - metabolism | Cytoprotection | Disease Models, Animal | Renal Artery Obstruction - drug therapy | Kidney - drug effects | Macrophages - pathology | Mice, Inbred C57BL | Receptors, Cell Surface - metabolism | Chemokine CCL2 - genetics | Mice, Transgenic | Hypertension, Renovascular - pathology | Macrophages - metabolism | Receptors, CCR2 - metabolism | Animals | Signal Transduction - drug effects | Benzoxazines - pharmacology | Macrophages - drug effects | Renal Artery Obstruction - metabolism | Nitric Oxide Synthase Type II - metabolism | Renal Artery Obstruction - pathology | Complications and side effects | Chronic kidney failure | Renovascular hypertension | Development and progression | Health aspects | Chemokine receptors
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, p. e78464
Renovascular hypertension induced by 2 Kidney-1 Clip (2K-1C) is a renin-angiotensin-system (RAS)-dependent model, leading to renal vascular rarefaction and... 
ENDOTHELIAL PROGENITOR CELLS | ACUTE KIDNEY INJURY | OXIDATIVE STRESS | THERAPY | 2-KIDNEY | MULTIDISCIPLINARY SCIENCES | RATS | WATER CHANNELS | MECHANISMS | EXPRESSION | BLOOD-PRESSURE | Blood Pressure | Tumor Necrosis Factor-alpha - metabolism | Blood-Brain Barrier - cytology | Kidney - pathology | Rats, Wistar | Tumor Necrosis Factor-alpha - genetics | Male | Interleukin-1beta - genetics | Proteinuria - pathology | Hypertension, Renovascular - genetics | Hypertension, Renovascular - metabolism | Peptidyl-Dipeptidase A - genetics | Kidney - metabolism | Mesenchymal Stromal Cells - cytology | Receptor, Angiotensin, Type 1 - genetics | Interleukin-1beta - metabolism | Peptidyl-Dipeptidase A - metabolism | Mesenchymal Stromal Cells - physiology | Receptor, Angiotensin, Type 2 - genetics | Gene Expression | Proteinuria - metabolism | Rats | Renin-Angiotensin System - genetics | Treatment Outcome | Hypertension, Renovascular - therapy | Proteinuria - genetics | Blood-Brain Barrier - metabolism | Hypertension, Renovascular - pathology | Proteinuria - therapy | Animals | Receptor, Angiotensin, Type 2 - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Renal Artery - surgery | Fluorescent Dyes | Mesenchymal Stem Cell Transplantation | Development and progression | Transplantation | Renovascular hypertension | Stem cells | Angiotensin | Occlusion | Therapy | Oxidative stress | Flow cytometry | Renal function | Renal artery | Mesenchyme | Hyperactivity | Central nervous system | Stem cell transplantation | Nervous system | Rarefaction | Adipocytes | Glucose | Renin | Ischemia | Blood-brain barrier | Cerebral blood flow | Rodents | Angiotensinogen | Physiology | Blood pressure | Medulla oblongata | Angiotensin II | Growth factors | Hypertension | Kidneys | Cytokines | Aquaporins | Inflammation | IL-1β | Medicine | Cytometry | Morphology | Fibrosis | Renal failure | Microvasculature | Proteinuria
Journal Article
Journal Article
Journal of Comparative Neurology, ISSN 0021-9967, 03/2010, Volume 518, Issue 5, pp. 567 - 585
An imbalance of excitatory and inhibitory functions has been shown to contribute to numerous pathological disorders. Accumulating evidence supports the idea... 
plasticity | brainstem | immunohistochemistry | sympathetic | hypertension | synaptic | Hypertension | Immunohistochemistry | Synaptic | Sympathetic | Brainstem | Plasticity | GLUTAMIC-ACID DECARBOXYLASE | HEART-FAILURE | SPINAL-CORD | SYMPATHETIC VASOMOTOR TONE | ANGIOTENSIN-II | NEUROSCIENCES | BLOOD-PRESSURE | FLUORESCENT LATEX MICROSPHERES | ZOOLOGY | THYROTROPIN-RELEASING-HORMONE | RECEPTOR-MEDIATED INHIBITION | PRESYMPATHETIC PVN NEURONS | Synaptic Transmission - physiology | Rats, Wistar | gamma-Aminobutyric Acid - metabolism | Male | Neurons - cytology | Dendrites - ultrastructure | Hypertension, Renovascular - physiopathology | Hypertension, Renovascular - metabolism | Oxytocin - metabolism | Excitatory Postsynaptic Potentials - physiology | Paraventricular Hypothalamic Nucleus - cytology | Reticular Formation - metabolism | Cardiovascular System - physiopathology | Medulla Oblongata - cytology | Neurons - metabolism | Kidney - physiopathology | Disease Models, Animal | Biomarkers - metabolism | Dendrites - metabolism | Vesicular Glutamate Transport Protein 2 - metabolism | Medulla Oblongata - metabolism | Inhibitory Postsynaptic Potentials - physiology | Rats | Animals | Reticular Formation - cytology | Neural Pathways - cytology | Paraventricular Hypothalamic Nucleus - metabolism | Glutamate Decarboxylase - metabolism | Neural Pathways - metabolism | Glutamic Acid - metabolism
Journal Article