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1. Full Text Genetic determinants of plasma triglycerides
by Johansen, Christopher T and Kathiresan, Sekar and Hegele, Robert A
Journal of Lipid Research, ISSN 0022-2275, 02/2011, Volume 52, Issue 2, pp. 189 - 206
Plasma triglyceride (TG) concentration is reemerging as an important cardiovascular disease risk factor. More complete understanding of the genes and variants... 
Hypertriglyceridemia - drug therapy | Dyslipidemias - genetics | Fibric Acids - therapeutic use | Apolipoproteins A - genetics | Hypertriglyceridemia - genetics | Upstream Stimulatory Factors - genetics | Humans | Male | Risk | Apolipoprotein C-III - genetics | Dyslipidemias - blood | Niacin - therapeutic use | Angiopoietin-like Proteins | Female | Lipid Metabolism - genetics | Intracellular Signaling Peptides and Proteins - genetics | Fatty Acid Desaturases - genetics | Apolipoprotein A-V | Genome-Wide Association Study | Lipoprotein Lipase - genetics | Membrane Proteins - genetics | Protein-Serine-Threonine Kinases - genetics | European Continental Ancestry Group | Lipid Metabolism Disorders - ethnology | Apolipoproteins B - genetics | Carrier Proteins - genetics | Phospholipid Transfer Proteins - genetics | Adaptor Proteins, Signal Transducing - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Coronary Artery Disease - genetics | Triglycerides - blood | Receptors, Lipoprotein | Polymorphism, Single Nucleotide | Triglycerides - genetics | Angiopoietins - genetics | Thematic Review | hypotriglyceridemia | resequencing | hypertriglyceridemia | genetic variation | genomics | dyslipidemias | lipoproteins
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2. Full Text Angiopoietin-like 4 (ANGPTL4, Fasting-induced Adipose Factor) Is a Direct Glucocorticoid Receptor Target and Participates in Glucocorticoid-regulated Triglyceride Metabolism
by Suneil K. Koliwad and Taiyi Kuo and Lauren E. Shipp and Nora E. Gray and Fredrik Backhed and Alex Yick-Lun So and Robert V. Farese, Jr and Jen-Chywan Wang and Sahlgrenska akademin and Institute of Medicine, Department of Molecular and Clinical Medicine and Institutionen för medicin, avdelningen för molekylär och klinisk medicin and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy
Journal of Biological Chemistry, ISSN 0021-9258, 09/2009, Volume 284, Issue 38, pp. 25593 - 25601
Glucocorticoids are important regulators of lipid homeostasis, and chronically elevated glucocorticoid levels induce hypertriglyceridemia, hepatic steatosis,... 
OBESITY | PLASMA | GENE | CHROMATIN | ANGIOGENESIS | DIET | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYPERLIPIDEMIA | MICE | TRANSCRIPTIONAL REGULATION | PROTEIN-4 | Transcription, Genetic - drug effects | Hypertriglyceridemia - genetics | Humans | Dexamethasone - adverse effects | Receptors, Glucocorticoid - metabolism | Hepatocytes - metabolism | Fatty Liver - chemically induced | Adipose Tissue - metabolism | Angiopoietin-like 4 Protein | Dexamethasone - pharmacology | Glucocorticoids - genetics | Glucocorticoids - metabolism | Response Elements - genetics | Lipoprotein Lipase - metabolism | Glucocorticoids - adverse effects | Fatty Liver - genetics | Angiopoietins - metabolism | Fatty Liver - metabolism | Lipoprotein Lipase - genetics | Liver - metabolism | Rats | Hypertriglyceridemia - metabolism | Mice, Knockout | Triglycerides - metabolism | Adipose Tissue, White | Hypertriglyceridemia - chemically induced | Animals | Receptors, Glucocorticoid - genetics | Cell Line, Tumor | Dexamethasone - metabolism | Glucocorticoids - pharmacology | Mice | Transcription, Genetic - genetics | Triglycerides - genetics | Angiopoietins - genetics | Index Medicus | Lipids and Lipoproteins | Metabolism, Regulation, and Signaling | Transcription | Dermatologi och venereologi | Dermatology and Venereal Diseases | Dexamethasone/adverse effects/metabolism/pharmacology | Hypertriglyceridemia/chemically induced/genetics/metabolism | Receptors | Adipose Tissue/metabolism | Liver/metabolism | Tumor | Adipose Tissue | Cell Line | Fatty Liver/chemically induced/genetics/metabolism | Triglycerides/genetics/ metabolism | Response Elements/genetics | Angiopoietins/genetics/ metabolism | Knockout | Lipoprotein Lipase/genetics/metabolism | White | Hepatocytes/metabolism | Genetic/drug effects/genetics | Glucocorticoids/adverse effects/genetics/ metabolism/pharmacology | Glucocorticoid/genetics/ metabolism
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3. Full Text Triglyceride-Rich Lipoproteins and Atherosclerotic Cardiovascular Disease: New Insights From Epidemiology, Genetics, and Biology
by Nordestgaard, Børge G
Circulation Research, ISSN 0009-7330, 02/2016, Volume 118, Issue 4, pp. 547 - 563
Scientific interest in triglyceride-rich lipoproteins has fluctuated over the past many years, ranging from beliefs that these lipoproteins cause... 
atherosclerosis | lipids | risk factors | cardiovascular diseases | prevention & control | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | LOW-DENSITY-LIPOPROTEIN | ISCHEMIC-HEART-DISEASE | OF-FUNCTION MUTATIONS | MENDELIAN RANDOMIZATION | ALL-CAUSE MORTALITY | ACUTE-CORONARY-SYNDROME | FOAM CELL-FORMATION | CHOLESTEROL-FED RABBITS | PERIPHERAL VASCULAR DISEASE | JOINT TASK-FORCE | HEMATOLOGY | Hypertriglyceridemia - drug therapy | Hypertriglyceridemia - genetics | Prognosis | Lipoproteins - genetics | Atherosclerosis - genetics | Humans | Atherosclerosis - epidemiology | Hypertriglyceridemia - mortality | Atherosclerosis - diagnosis | Cholesterol, LDL - blood | Hypertriglyceridemia - diagnosis | Genetic Predisposition to Disease | Risk Assessment | Risk Factors | Atherosclerosis - mortality | Biomarkers - blood | Lipoproteins - blood | Cholesterol, HDL - genetics | Hypertriglyceridemia - blood | Atherosclerosis - blood | Cholesterol, LDL - genetics | Hypertriglyceridemia - epidemiology | Phenotype | Animals | Primary Prevention - methods | Triglycerides - blood | Cholesterol, HDL - blood | Hypolipidemic Agents - therapeutic use | Atherosclerosis - prevention & control
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4. Full Text Amerindian-specific regions under positive selection harbour new lipid variants in Latinos
by Ko, Arthur and Cantor, Rita M and Weissglas-Volkov, Daphna and Nikkola, Elina and Reddy, Prasad M. V. Linga and Sinsheimer, Janet S and Pasaniuc, Bogdan and Brown, Robert and Alvarez, Marcus and Rodriguez, Alejandra and Rodriguez-Guillen, Rosario and Bautista, Ivette C and Arellano-Campos, Olimpia and Muñoz-Hernández, Linda L and Salomaa, Veikko and Kaprio, Jaakko and Jula, Antti and Jauhiainen, Matti and Heliövaara, Markku and Raitakari, Olli and Lehtimäki, Terho and Eriksson, Johan G and Perola, Markus and Lohmueller, Kirk E and Matikainen, Niina and Taskinen, Marja-Riitta and Rodriguez-Torres, Maribel and Riba, Laura and Tusie-Luna, Teresa and Aguilar-Salinas, Carlos A and Pajukanta, Päivi
Nature Communications, ISSN 2041-1723, 06/2014, Volume 5, Issue 1, p. 3983
Dyslipidemia and obesity are especially prevalent in populations with Amerindian backgrounds, such as Mexican- Americans, which predispose these populations to... 
LOCAL ANCESTRY | APOLIPOPROTEIN A-V | METAANALYSIS | MULTIDISCIPLINARY SCIENCES | DISEASE | GENE-EXPRESSION | NATIONAL-HEALTH | TRIGLYCERIDES | POPULATIONS | GENOME-WIDE ASSOCIATION | CARDIOVASCULAR RISK-FACTORS | Haplotypes | Chromosomes, Human, Pair 8 - genetics | Dyslipidemias - genetics | Protein Kinases - genetics | Apolipoproteins A - genetics | Hypertriglyceridemia - genetics | Humans | Middle Aged | Male | Obesity - genetics | Case-Control Studies | Young Adult | Mexico - ethnology | Adult | Female | Apolipoprotein A-V | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Lipoprotein Lipase - genetics | Genotype | Logistic Models | Chromosomes, Human, Pair 11 - genetics | Indians, North American - genetics | Polymorphism, Single Nucleotide | Hypercholesterolemia - genetics | Nuclear Receptor Subfamily 1, Group F, Member 1 - genetics
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5. Full Text Genomic study in Mexicans identifies a new locus for triglycerides and refines European lipid loci
by Weissglas-Volkov, Daphna and Aguilar-Salinas, Carlos A and Nikkola, Elina and Deere, Kerry A and Cruz-Bautista, Ivette and Arellano-Campos, Olimpia and Muñoz-Hernandez, Linda Liliana and Gomez-Munguia, Lizeth and Ordoñez-Sánchez, Maria Luisa and Linga Reddy, Prasad M. V and Lusis, Aldons J and Matikainen, Niina and Taskinen, Marja-Riitta and Riba, Laura and Cantor, Rita M and Sinsheimer, Janet S and Tusie-Luna, Teresa and Pajukanta, Päivi
Journal of Medical Genetics, ISSN 0022-2593, 2013, Volume 50, Issue 5, pp. 298 - 308
Background The Mexican population and others with Amerindian heritage exhibit a substantial predisposition to dyslipidemias and coronary heart disease. Yet,... 
COMMON VARIANTS | POPULATION | HEART-DISEASE | METAANALYSIS | GENE | CHOLESTEROL | GENETICS & HEREDITY | RISK | WIDE ASSOCIATION | PREVALENCE | CORONARY-DISEASE | Apolipoprotein A-V | European Continental Ancestry Group - genetics | Genome-Wide Association Study | Apolipoproteins A - genetics | Hypertriglyceridemia - genetics | Membrane Proteins - genetics | Humans | Genetic Loci - genetics | Genotype | Apolipoproteins A - blood | Linkage Disequilibrium | Hypertriglyceridemia - ethnology | Indians, North American - genetics | Triglycerides - blood | Polymorphism, Single Nucleotide - genetics | Mexico | Triglycerides - genetics | Hypoalphalipoproteinemias - ethnology | Hypoalphalipoproteinemias - genetics | Physiological aspects | Genetic aspects | Triglycerides | Research | Mexicans
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6. Full Text A replication study of GWAS-derived lipid genes in asian indians: The chromosomal region 11q23.3 harbors loci contributing to triglycerides
by Braun, Timothy R and Been, Latonya F and Singhal, Akhil and Worsham, Jacob and Ralhan, Sarju and Wander, Gurpreet S and Chambers, John C and Kooner, Jaspal S and Aston, Christopher E and Sanghera, Dharambir K
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e37056
Recent genome-wide association scans (GWAS) and meta-analysis studies on European populations have identified many genes previously implicated in lipid... 
DIABETES-MELLITUS | DENSITY-LIPOPROTEIN CHOLESTEROL | VARIANTS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | DISEASE | RISK | TRAITS | APOA5 | GENOME-WIDE ASSOCIATION | Body Mass Index | European Continental Ancestry Group - genetics | Genome-Wide Association Study | Age Factors | United States | Humans | Logistic Models | Male | Chromosomes, Human, Pair 11 - genetics | India | Carrier Proteins - genetics | Haplotypes - genetics | Cholesterol Ester Transfer Proteins - genetics | Genetic Markers - genetics | Sex Factors | Triglycerides - blood | Polymorphism, Single Nucleotide - genetics | Female | Lipid Metabolism - genetics | Cohort Studies | Type 2 diabetes | Medical research | Analysis | Genes | Genomics | Genetic research | Medicine, Experimental | Triglycerides | Coronary heart disease | Health sciences | Pediatrics | Sikhs | Melatonin | Transcription factors | Populations | Dyslipidemia | Lipids | Cardiovascular disease | Population studies | Genomes | Single-nucleotide polymorphism | Apolipoprotein E | Chromosome 11 | Population | Cardiology | Heart diseases | Lipoproteins (high density) | Asians | Fasting | Diabetes mellitus | Coronary artery | Principal components analysis | Whites | Metabolism | Gene expression | Coronary artery disease | Loci | Cholesterol | Meta-analysis | Medicine | Studies | Hypertriglyceridemia | Insulin resistance | Replication | Diabetes
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7. Full Text The effects of paternal high-fat diet exposure on offspring metabolism with epigenetic changes in the mouse adiponectin and leptin gene promoters
by Masuyama, Hisashi and Mitsui, Takashi and Eguchi, Takeshi and Tamada, Shoko and Hiramatsu, Yuji
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 07/2016, Volume 311, Issue 1, pp. E236 - E245
Recent studies have demonstrated that epigenetic changes resulting from malnutrition might play important roles in transgenerational links with metabolic... 
Epigenetics | Metabolic syndrome | High-fat diet | Leptin | Adiponectin | adiponectin | IN-UTERO | PHYSIOLOGY | MATERNAL OBESITY | X RECEPTOR | FEMALE MICE | BIRTH-WEIGHT | GENERATIONS | leptin | epigenetics | metabolic syndrome | INSULIN-RESISTANCE | high-fat diet | ENDOCRINOLOGY & METABOLISM | BODY-MASS INDEX | HEALTH | EXPRESSION | Promoter Regions, Genetic | Gene Expression | Glucose Intolerance - genetics | Hypertriglyceridemia - genetics | Male | Adipokines - metabolism | Leptin - genetics | Paternal Exposure | RNA, Messenger - metabolism | Obesity - genetics | Prenatal Exposure Delayed Effects - metabolism | Pregnancy | Prenatal Exposure Delayed Effects - genetics | Animals | Chromatin Immunoprecipitation | Epigenesis, Genetic - genetics | Metabolic Syndrome - genetics | Adiponectin - genetics | Diet, High-Fat | Weight Gain - genetics | Female | Mice | Hypertension - genetics | Real-Time Polymerase Chain Reaction | Epigenetic inheritance | Genetic aspects | Metabolism | Health aspects | Ketogenic diet
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8. Full Text Genetic Variants in PNPLA3 and Risk of Non-Alcoholic Fatty Liver Disease in a Han Chinese Population
by Peng, Xian-E and Wu, Yun-Li and Lin, Shao-Wei and Lu, Qing-Qing and Hu, Zhi-Jian and Lin, Xu
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e50256
We investigated the possible association between genetic variants in the Patatin like phospholipase-3 (PNPLA3) gene and nonalcoholic fatty liver disease... 
INSULIN | ADIPOSE TRIGLYCERIDE LIPASE | GREEN TEA | LIFE-STYLE | MESSENGER-RNA EXPRESSION | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | POLYMORPHISM | GENOME-WIDE ASSOCIATION | ADIPONUTRIN | INDEXES | Haplotypes | Liver - pathology | Hypertriglyceridemia - genetics | Liver - enzymology | Humans | Middle Aged | Fatty Liver - complications | Male | Risk | Fatty Liver - ethnology | Obesity - genetics | Case-Control Studies | Fatty Liver - enzymology | Non-alcoholic Fatty Liver Disease | Adult | Female | Membrane Proteins - metabolism | Hypertension - enzymology | Hypertension - genetics | Fatty Liver - genetics | Genetic Predisposition to Disease | Membrane Proteins - genetics | Obesity - complications | Obesity - ethnology | Lipase - metabolism | Hypertriglyceridemia - enzymology | Hypertriglyceridemia - ethnology | Tea | Asian Continental Ancestry Group | Hypertriglyceridemia - complications | Hypertension - ethnology | Alleles | Hypertension - complications | Obesity - enzymology | Polymorphism, Single Nucleotide | Lipase - genetics | Hypertension | Fatty liver | Genes | Development and progression | Disease susceptibility | Genetic aspects | Single nucleotide polymorphisms | Health care | Laboratories | Liver | Association analysis | Genomes | Single-nucleotide polymorphism | Adipocytes | Epidemiology | Hepatitis | Ultrasonic imaging | Education | Rodents | Gastroenterology | Population | Genotypes | Public health | Obesity | Enzymes | Liver diseases | Statistical analysis | Drinking behavior | Health risks | Triglycerides | Regression analysis | Abdomen | Genetic variance | Phospholipase | Ethnicity | Hospitals | Hypertriglyceridemia | Tagging | Insulin resistance | Diabetes | Cancer
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9. Full Text Mutations in LPL, APOC2, APOA5, GPIHBP1 and LMF1 in patients with severe hypertriglyceridaemia
by Surenan, R. P and Visser, M. E and Heemelaar, S and Wang, J and Peter, J and Defesche, J. C and Kuivenhoven, J. A and Hosseini, M and Péterfy, M and Kastelein, J. J. P and Johansen, C. T and Hegele, R. A and Stroes, E. S. G and Dallinga-Thie, G. M
Journal of internal medicine, ISSN 0954-6820, 2012, Volume 272, Issue 2, pp. 185 - 196
. Surendran RP, Visser ME, Heemelaar S, Wang J, Peter J, Defesche JC, Kuivenhoven JA, Hosseini M, Peterfy M, Kastelein JJP, Johansen CT, Hegele RA, Stroes ESG,... 
triglycerides | APOA5 | APOC2 | lipoprotein lipase | GPIHBP1 | LMF1 | Lipoprotein lipase | Triglycerides | APOLIPOPROTEIN-C-II | ENDOTHELIAL-CELL TRANSPORTER | DIFFERENT ANCESTRIES | DEFICIENCY | MEDICINE, GENERAL & INTERNAL | CAUSES CHYLOMICRONEMIA | MISSENSE MUTATION | BINDING | EXPRESSION | LIPOPROTEIN-LIPASE GENE | PLASMA TRIGLYCERIDES | Severity of Illness Index | Apolipoprotein A-V | Apolipoproteins A - genetics | Genetic Testing | Hypertriglyceridemia - genetics | Prevalence | Lipoprotein Lipase - genetics | Membrane Proteins - genetics | Humans | Hypertriglyceridemia - physiopathology | Netherlands - epidemiology | Male | Mutation, Missense | Apolipoprotein C-II - genetics | Carrier Proteins - genetics | Hypertriglyceridemia - epidemiology | Molecular Epidemiology | Receptors, Lipoprotein | Adult | Female | Medical colleges | Care and treatment | Anopheles | Genes | Pancreatitis | Medical genetics | Genetic research | Genetic aspects | Protein binding
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10. Full Text A Genetic Variant at the Fatty Acid-Binding Protein aP2 Locus Reduces the Risk for Hypertriglyceridemia, Type 2 Diabetes, and Cardiovascular Disease
by G. Tuncman and E. Erbay and X. Hom and I. De Vivo and H. Campos and E. B. Rimm and G. S. Hotamisligil
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2006, Volume 103, Issue 18, pp. 6970 - 6975
Obesity and the associated pathologies including dyslipidemia, insulin resistance, type 2 diabetes, and cardiovascular disease constitute a major threat to... 
Adipose tissues | Obesity | DNA | Alleles | Medical genetics | Metabolic diseases | Promoter regions | Type 2 diabetes mellitus | Lipid metabolism | Human genetics | Adipocyte | FABP4 | Metabolic syndrome | Triglyceride | Macrophage | triglyceride | MULTIDISCIPLINARY SCIENCES | macrophage | APOLIPOPROTEIN-E | metabolic syndrome | WOMEN | OBESITY | INSULIN-RESISTANCE | METABOLIC-RESPONSES | adipocyte | P2 GENE | MICE | CORONARY-HEART-DISEASE | EXPRESSION | ADIPOSE-TISSUE | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Adipose Tissue - physiology | Hypertriglyceridemia - genetics | Prospective Studies | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Molecular Sequence Data | Fatty Acid-Binding Proteins - metabolism | Cardiovascular Diseases - genetics | Obesity - genetics | Protein Isoforms - metabolism | Base Sequence | Adult | Female | Transcription, Genetic | Odds Ratio | Promoter Regions, Genetic | Genetic Predisposition to Disease | Obesity - complications | Genotype | Fatty Acid-Binding Proteins - genetics | Polymorphism, Genetic | Phenotype | Animals | Obesity - epidemiology | Triglycerides - blood | Protein Binding | Fatty Acid-Binding Proteins - chemistry | Cohort Studies | Protein Isoforms - genetics | Fat cells | Triglycerides | Research | Metabolic syndrome X | Macrophages | Biological Sciences
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11. Full Text Genetic Aspects of Pancreatitis
by Whitcomb, DC
ANNUAL REVIEW OF MEDICINE, ISSN 0066-4219, 2010, Volume 61, Issue 1, pp. 413 - 424
Acute pancreatitis and chronic pancreatitis are complex inflammatory disorders of the pancreas with unpredictable severity, complications, and clinical... 
INFLAMMATORY RESPONSE SYNDROME | hyperlipidemia | chronic pancreatitis | HEREDITARY PANCREATITIS | hypertriglyceridemia | PROMOTER POLYMORPHISM | SPINK1 MUTATIONS | RECURRENT ACUTE | CATIONIC TRYPSINOGEN GENE | MEDICINE, GENERAL & INTERNAL | TUMOR-NECROSIS-FACTOR | cystic fibrosis | acute pancreatitis | CYSTIC-FIBROSIS | IDIOPATHIC CHRONIC-PANCREATITIS | SENSING-RECEPTOR GENE | Pancreatitis - genetics | Hypertriglyceridemia - genetics | Hypertriglyceridemia - complications | Humans | Risk Factors | Hypertriglyceridemia - metabolism | Pancreatitis - pathology | Trypsin - physiology | Pancreatitis - metabolism | Cystic fibrosis | Acute pancreatitis | Hyperlipidemia | Hypertriglyceridemia | Chronic pancreatitis | Chymotrypsin | Care and treatment | Usage | Trypsinogen | Linkage (Genetics) | Gene mutations | Analysis | Pancreatitis | Genetic aspects | Health aspects
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12. Full Text A polygenic basis for four classical Fredrickson hyperlipoproteinemia phenotypes that are characterized by hypertriglyceridemia
by Hegele, Robert A and Ban, Matthew R and Hsueh, Neil and Kennedy, Brooke A and Cao, Henian and Zou, Guang Yong and Anand, Sonia and Yusuf, Salim and Huff, Murray W and Wang, Jian
Human Molecular Genetics, ISSN 0964-6906, 11/2009, Volume 18, Issue 21, pp. 4189 - 4194
Numerous single nucleotide polymorphisms (SNPs) have been found in recent genome wide association studies (GWAS) to be associated with subtle plasma... 
GENETIC-VARIATION | POPULATION | CHOLESTEROL | PLASMA-LIPOPROTEINS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | CARDIOVASCULAR-DISEASE | RISK | LOCI | HYPERCHOLESTEROLEMIA | CORONARY-HEART-DISEASE | GENOME-WIDE ASSOCIATION | Multivariate Analysis | Apolipoproteins A - genetics | Hypertriglyceridemia - genetics | Humans | Middle Aged | Hyperlipoproteinemias - pathology | Hypertriglyceridemia - pathology | Male | GTP-Binding Proteins - genetics | Angiopoietin-like Proteins | N-Acetylgalactosaminyltransferases - genetics | Adult | Female | Intracellular Signaling Peptides and Proteins - genetics | Multifactorial Inheritance - genetics | Apolipoprotein A-V | Genome-Wide Association Study | Gene Frequency | Protein-Serine-Threonine Kinases - genetics | Genotype | Logistic Models | Calcium-Calmodulin-Dependent Protein Kinases | Hyperlipoproteinemias - blood | Hypertriglyceridemia - blood | Linkage Disequilibrium | Phenotype | Adaptor Proteins, Signal Transducing - genetics | Triglycerides - blood | Aged | Hyperlipoproteinemias - genetics | Polymorphism, Single Nucleotide | Angiopoietins - genetics | Association Studies
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