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Journal of Biological Chemistry, ISSN 0021-9258, 09/2009, Volume 284, Issue 38, pp. 25593 - 25601
Glucocorticoids are important regulators of lipid homeostasis, and chronically elevated glucocorticoid levels induce hypertriglyceridemia, hepatic steatosis,... 
OBESITY | PLASMA | GENE | CHROMATIN | ANGIOGENESIS | DIET | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYPERLIPIDEMIA | MICE | TRANSCRIPTIONAL REGULATION | PROTEIN-4 | Transcription, Genetic - drug effects | Hypertriglyceridemia - genetics | Humans | Dexamethasone - adverse effects | Receptors, Glucocorticoid - metabolism | Hepatocytes - metabolism | Fatty Liver - chemically induced | Adipose Tissue - metabolism | Angiopoietin-like 4 Protein | Dexamethasone - pharmacology | Glucocorticoids - genetics | Glucocorticoids - metabolism | Response Elements - genetics | Lipoprotein Lipase - metabolism | Glucocorticoids - adverse effects | Fatty Liver - genetics | Angiopoietins - metabolism | Fatty Liver - metabolism | Lipoprotein Lipase - genetics | Liver - metabolism | Rats | Hypertriglyceridemia - metabolism | Mice, Knockout | Triglycerides - metabolism | Adipose Tissue, White | Hypertriglyceridemia - chemically induced | Animals | Receptors, Glucocorticoid - genetics | Cell Line, Tumor | Dexamethasone - metabolism | Glucocorticoids - pharmacology | Mice | Transcription, Genetic - genetics | Triglycerides - genetics | Angiopoietins - genetics | Index Medicus | Lipids and Lipoproteins | Metabolism, Regulation, and Signaling | Transcription | Dermatologi och venereologi | Dermatology and Venereal Diseases | Dexamethasone/adverse effects/metabolism/pharmacology | Hypertriglyceridemia/chemically induced/genetics/metabolism | Receptors | Adipose Tissue/metabolism | Liver/metabolism | Tumor | Adipose Tissue | Cell Line | Fatty Liver/chemically induced/genetics/metabolism | Triglycerides/genetics/ metabolism | Response Elements/genetics | Angiopoietins/genetics/ metabolism | Knockout | Lipoprotein Lipase/genetics/metabolism | White | Hepatocytes/metabolism | Genetic/drug effects/genetics | Glucocorticoids/adverse effects/genetics/ metabolism/pharmacology | Glucocorticoid/genetics/ metabolism
Journal Article
Cell Metabolism, ISSN 1550-4131, 04/2005, Volume 1, Issue 4, pp. 245 - 258
Obesity is typically associated with elevated levels of free fatty acids (FFAs) and is linked to glucose intolerance and type 2 diabetes. FFAs exert divergent... 
STIMULATED INSULIN-SECRETION | PROTEIN-COUPLED RECEPTOR | FREE FATTY-ACIDS | ENDOCRINOLOGY & METABOLISM | LONG-TERM EXPOSURE | GENE-EXPRESSION | PANCREATIC BETA-CELLS | PPAR-GAMMA | UNCOUPLING PROTEIN-2 | DIET-INDUCED OBESITY | ADIPOSE-TISSUE | CELL BIOLOGY | Glucose Intolerance - metabolism | Hyperinsulinism - metabolism | Glucose Intolerance - genetics | Fatty Liver - metabolism | Hypertriglyceridemia - genetics | Receptors, G-Protein-Coupled - metabolism | Diabetes Mellitus, Type 2 - genetics | Hypertriglyceridemia - metabolism | Diabetes Mellitus, Type 2 - metabolism | Insulin - metabolism | Animals | Diabetes Mellitus, Type 2 - physiopathology | Receptors, G-Protein-Coupled - deficiency | Glucose - physiology | Islets of Langerhans - metabolism | Homeostasis - physiology | Mice | Receptors, G-Protein-Coupled - genetics | Insulin Secretion | Type 2 diabetes | Obesity | Hyperglycemia | Homeostasis | Glucose | Fatty acids | Insulin | Dextrose | Membrane proteins | Fatty Liver/metabolism | Glucose Intolerance/genetics/metabolism | Hypertriglyceridemia/genetics/metabolism | Receptors; G-Protein-Coupled/deficiency/genetics/metabolism | Homeostasis/physiology | Insulin/secretion | Hyperinsulinism/metabolism | Diabetes Mellitus; Type 2/genetics/metabolism/physiopathology | Islets of Langerhans/metabolism | Glucose/physiology
Journal Article
Journal Article
Nature, ISSN 0028-0836, 03/2013, Volume 495, Issue 7442, pp. 524 - 528
Macrophages consist of at least two subgroups, M1 and M2 (refs 1-3). Whereas M1 macrophages are proinflammatory and have a central role in host defence against... 
GLUCOSE-HOMEOSTASIS | OBESITY | TRIBBLES HOMOLOG | INSULIN-RESISTANCE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MYELOID LEUKEMOGENESIS | POLARIZATION | ASSOCIATION | ALTERNATIVE ACTIVATION | ADIPOSE-TISSUE | Neutrophils - cytology | Protein-Serine-Threonine Kinases - deficiency | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Eosinophils - metabolism | Hypertriglyceridemia - genetics | Cell Count | Ubiquitin - metabolism | Adipose Tissue - cytology | Diet, High-Fat - adverse effects | Male | Intracellular Signaling Peptides and Proteins - metabolism | Lung - cytology | Intracellular Signaling Peptides and Proteins - deficiency | Adipose Tissue - metabolism | Inflammation Mediators - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cell Cycle Proteins - genetics | Female | Cell Differentiation | Cytokines - genetics | Intracellular Signaling Peptides and Proteins - genetics | Neutrophils - metabolism | Protein-Serine-Threonine Kinases - metabolism | Protein Structure, Tertiary | Macrophages - classification | Bone Marrow Cells - cytology | Adipose Tissue - pathology | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Cell Cycle Proteins - deficiency | Lipodystrophy - metabolism | Spleen - cytology | Macrophages - cytology | Organ Specificity | Lipolysis | Hypertriglyceridemia - chemically induced | Macrophages - metabolism | Animals | Intracellular Signaling Peptides and Proteins - chemistry | Insulin Resistance - genetics | Lipodystrophy - pathology | Lipodystrophy - chemically induced | Mice | Protein-Serine-Threonine Kinases - chemistry | Eosinophils - cytology | Bone Marrow Cells - metabolism | Proteins | Care and treatment | Metabolic diseases | Cellular signal transduction | Research | Macrophages | Properties | Identification and classification | Testing | Studies | Spleen | Heart attacks | Transplants & implants | Bone marrow | Binding sites | Metabolic disorders | Defects
Journal Article
Endocrinology, ISSN 0013-7227, 05/2011, Volume 152, Issue 5, pp. 1848 - 1859
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 2011, Volume 251, Issue 1, pp. 32 - 40
Consumption of beverages that contain fructose favors the increasing prevalence of metabolic syndrome alterations in humans, including non-alcoholic fatty... 
Fatty acid oxidation | NAFLD | ChREBP | NFκB | NFΚB | PPAR-ALPHA | KAPPA-B ACTIVATION | LIPID-METABOLISM | ELEMENT-BINDING PROTEIN | HYPERTRIGLYCERIDEMIA | DISEASE | FATTY LIVER | PHARMACOLOGY & PHARMACY | TOXICOLOGY | TRANSCRIPTION FACTOR | UP-REGULATION | NF kappa B | CARDIOVASCULAR RISK | Hypertriglyceridemia - drug therapy | Metallothionein - metabolism | Liver - pathology | Phosphorylation | Hepatitis - enzymology | Liver - enzymology | Fatty Liver - pathology | Male | NF-kappa B - metabolism | I-kappa B Proteins - metabolism | Dietary Carbohydrates - metabolism | Fructose - metabolism | Necrosis | Hepatitis - prevention & control | Liver - drug effects | Hepatitis - genetics | Fatty Liver - enzymology | Inflammation Mediators - metabolism | Non-alcoholic Fatty Liver Disease | Lipid Metabolism - genetics | Fatty Acids - metabolism | Heptanoic Acids - pharmacology | Disease Models, Animal | Cyclic AMP-Dependent Protein Kinases - metabolism | Fatty Liver - genetics | Hepatitis - etiology | Oxidation-Reduction | Down-Regulation | Fatty Liver - prevention & control | Rats | Rats, Sprague-Dawley | Fructokinases - metabolism | Triglycerides - metabolism | Gene Expression Regulation - drug effects | Atorvastatin Calcium | Hypertriglyceridemia - enzymology | Pyrroles - pharmacology | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Hypertriglyceridemia - etiology | Lipid Metabolism - drug effects | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Fatty Liver - etiology | Liver diseases | Synthesis | Liver | Physiological aspects | Triglycerides | Beverages | Fatty acids | Fructose | Metallothionein | Antilipemic agents | Protein binding | Index Medicus | Enzymes | Transcription factors | Inflammation | Regulatory sequences | Metabolism | Dietary restrictions | reductase | Fatty liver | Molecular modelling | Hypertriglyceridemia | Atorvastatin | Fructokinase | Supplementation | steatosis | Metabolic disorders | PATIENTS | METALLOTHIONEIN | DIGESTIVE SYSTEM | RATS | TRIGLYCERIDES | HISTOLOGY | 60 APPLIED LIFE SCIENCES | FRUCTOSE | GLANDS | METABOLISM | ENZYMES | GENES | ESTERS | CHEMICAL REACTIONS | VERTEBRATES | LIPIDS | MAMMALS | HUMAN POPULATIONS | OXIDATION | TRANSCRIPTION FACTORS | ANIMALS | SACCHARIDES | CATABOLISM | RODENTS | ORGANIC COMPOUNDS | METALLOPROTEINS | INTAKE | ORGANIC ACIDS | POPULATIONS | SYMPTOMS | HEXOSES | KETONES | ORGANS | MONOSACCHARIDES | DISEASES | INGESTION | INFLAMMATION | LIVER | SYNTHESIS | CARBOXYLIC ACIDS | BEVERAGES | PROTEINS | BODY | PATHOLOGICAL CHANGES | CARBOHYDRATES | CONTROL | FOOD
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 10/2016, Volume 5, Issue 10, p. n/a
Background Overactivation of the aldosterone and mineralocorticoid receptor (MR) pathway is associated with hyperglycemia and dyslipidemia. Caveolin 1 (cav‐1)... 
caveolin 1 | mineralocorticoid receptor | dyslipidemia | insulin resistance | aldosterone | eplerenone | Insulin resistance | Caveolin 1 | Eplerenone | Aldosterone | Mineralocorticoid receptor | Dyslipidemia | METABOLIC SYNDROME | OXIDATIVE STRESS | CAVEOLIN-1-DEFICIENT MICE | CARDIAC & CARDIOVASCULAR SYSTEMS | OXIDANT STRESS | VASCULAR RELAXATION | MINERALOCORTICOID RECEPTOR BLOCKADE | DIETARY-SODIUM | INSULIN-RESISTANCE | ADIPOCYTE DYSFUNCTION | ALDOSE REDUCTASE | Dyslipidemias - genetics | Aldehyde Reductase - genetics | Hypertriglyceridemia - genetics | Resistin - genetics | Receptors, Mineralocorticoid - metabolism | Humans | Middle Aged | Lipoproteins, HDL - metabolism | Homeostasis | Male | RNA, Messenger - metabolism | Young Adult | Adipose Tissue - metabolism | Aldosterone - metabolism | Spironolactone - pharmacology | Mineralocorticoid Receptor Antagonists - pharmacology | Adult | Aldehyde Reductase - metabolism | Female | Lipid Metabolism - genetics | Retinol-Binding Proteins, Plasma - metabolism | Spironolactone - analogs & derivatives | Resistin - metabolism | NADPH Oxidase 4 - genetics | Signal Transduction | Gene Frequency | Liver - metabolism | Caveolin 1 - genetics | Hypertriglyceridemia - metabolism | Cholesterol - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Triglycerides - metabolism | Blood Glucose - drug effects | Insulin - metabolism | NADPH Oxidase 4 - metabolism | Animals | Adolescent | Insulin Resistance - genetics | Dyslipidemias - metabolism | Glucose - metabolism | Aged | Mice | Polymorphism, Single Nucleotide | Retinol-Binding Proteins, Plasma - genetics
Journal Article
British Journal of Nutrition, ISSN 0007-1145, 9/2007, Volume 98, Issue 3, pp. 458 - 473
Most of diurnal time is spent in a postprandial state due to successive meal intakes during the day. As long as the meals contain enough fat, a transient... 
Lifestyle conditions | Diet | Coronary heart disease | Gene polymorphism | Postprandial lipaemia | Physiological and pathological factors | FATTY-ACID-BINDING | CORONARY-ARTERY DISEASE | RECEPTOR-RELATED PROTEIN | lifestyle conditions | LOW-DENSITY-LIPOPROTEIN | EUROPEAN ATHEROSCLEROSIS RESEARCH | gene polymorphism | TRIGLYCERIDE-RICH LIPOPROTEINS | physiological and pathological factors | HEALTHY-YOUNG MEN | postprandial lipaemia | APOLIPOPROTEIN-A-IV | NUTRITION & DIETETICS | MIDDLE-AGED MEN | FAMILIAL COMBINED HYPERLIPIDEMIA | coronary heart disease | diet | Dietary Fats - metabolism | Hyperlipidemias - genetics | Life Style | Hypertriglyceridemia - genetics | Atherosclerosis - genetics | Humans | Dietary Fiber - administration & dosage | Dietary Carbohydrates - metabolism | Obesity - genetics | Postprandial Period - physiology | Dietary Proteins - metabolism | Coronary Disease - metabolism | Apolipoproteins - genetics | Dietary Fats - administration & dosage | Lipid Metabolism - genetics | Dietary Carbohydrates - administration & dosage | Hyperlipidemias - metabolism | Apolipoproteins - metabolism | Dietary Fiber - metabolism | Hypertriglyceridemia - metabolism | Atherosclerosis - metabolism | Obesity - metabolism | Polymorphism, Genetic | Coronary Disease - genetics | Postprandial Period - genetics | Lipid Metabolism - physiology
Journal Article
Journal Article