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1. Full Text Nutrition in cardiovascular disease: salt in hypertension and heart failure
by He, Feng J and Burnier, Michel and MacGregor, Graham A
European heart journal, ISSN 0195-668X, 12/2011, Volume 32, Issue 24, pp. 3073 - 3080
Heart failure | Cardiovascular disease | Blood pressure | Dietary salt | Left ventricular hypertrophy | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Heart | Diseases of the digestive system | Arterial hypertension. Arterial hypotension | Biological and medical sciences | Medical sciences | Heart failure, cardiogenic pulmonary edema, cardiac enlargement | Blood and lymphatic vessels | Radiotherapy. Instrumental treatment. Physiotherapy. Reeducation. Rehabilitation, orthophony, crenotherapy. Diet therapy and various other treatments (general aspects) | Life Style | Diet - adverse effects | Humans | Heart Failure - physiopathology | Health Promotion - economics | Hypertension - etiology | Dose-Response Relationship, Drug | Heart Failure - prevention & control | Hypertension - prevention & control | Blood Pressure - physiology | Heart Failure - etiology | Health Promotion - methods | Food-Drug Interactions | Hypertrophy, Left Ventricular - etiology | Risk Factors | Sodium Chloride, Dietary - adverse effects | Ventricular Dysfunction, Left - etiology | Antihypertensive Agents - therapeutic use | Hypertension - physiopathology | Ventricular Dysfunction, Left - physiopathology | Weight Loss | Cost-Benefit Analysis | Diet, Sodium-Restricted - methods | Obesity - prevention & control | Hypertrophy, Left Ventricular - physiopathology | Index Medicus
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2. Full Text Basigin Promotes Cardiac Fibrosis and Failure in Response to Chronic Pressure Overload in Mice
by Suzuki, Kota and Satoh, Kimio and Ikeda, Shohei and Sunamura, Shinichiro and Otsuki, Tomohiro and Satoh, Taijyu and Kikuchi, Nobuhiro and Omura, Junichi and Kurosawa, Ryo and Nogi, Masamichi and Numano, Kazuhiko and Sugimura, Koichiro and Aoki, Tatsuo and Tatebe, Shunsuke and Miyata, Satoshi and Mukherjee, Rupak and Spinale, Francis G and Kadomatsu, Kenji and Shimokawa, Hiroaki
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 04/2016, Volume 36, Issue 4, pp. 636 - 646
heart failure | cytokines | hypertrophy | inflammation | angiotensin II | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Oxidative Stress | Heart Failure - physiopathology | Male | Aortic Diseases - metabolism | Heart Failure - prevention & control | Hypertrophy, Left Ventricular - genetics | Time Factors | Ventricular Dysfunction, Left - genetics | Basigin - genetics | Myocardium - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Heart Failure - etiology | Disease Models, Animal | Fibroblasts - metabolism | Animals, Newborn | Angiotensin II - pharmacology | Hypertrophy, Left Ventricular - prevention & control | Rats | Heart Failure - genetics | Heart Failure - metabolism | Fibroblasts - pathology | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Aortic Diseases - genetics | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Fibrosis | Myocytes, Cardiac - metabolism | Hypertrophy, Left Ventricular - physiopathology | Rats, Wistar | Ventricular Function, Left | JNK Mitogen-Activated Protein Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Blood Proteins - metabolism | Hypertrophy, Left Ventricular - pathology | Inflammation Mediators - metabolism | Ventricular Dysfunction, Left - pathology | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - metabolism | Cells, Cultured | Ventricular Dysfunction, Left - etiology | Myocardium - pathology | Heart Failure - pathology | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Mechanotransduction, Cellular | Aortic Diseases - physiopathology | Animals | Aortic Diseases - complications | Basigin - metabolism | Matrix Metalloproteinases - metabolism | Index Medicus
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3. Full Text Prior starvation mitigates acute doxorubicin cardiotoxicity through restoration of autophagy in affected cardiomyocytes
by Kawaguchi, Tomonori and Takemura, Genzou and Kanamori, Hiromitsu and Takeyama, Toshiaki and Watanabe, Takatomo and Morishita, Kentaro and Ogino, Atsushi and Tsujimoto, Akiko and Goto, Kazuko and Maruyama, Rumi and Kawasaki, Masanori and Mikami, Atsushi and Fujiwara, Takako and Fujiwara, Hisayoshi and Minatoguchi, Shinya
Cardiovascular research, ISSN 0008-6363, 12/2012, Volume 96, Issue 3, pp. 456 - 465
Heart failure | Autophagy | Doxorubicin | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Heart | Biological and medical sciences | Medical sciences | Heart failure, cardiogenic pulmonary edema, cardiac enlargement | AMP-Activated Protein Kinases - metabolism | Microtubule-Associated Proteins - genetics | Ventricular Function, Left | Microtubule-Associated Proteins - metabolism | Heart Failure - physiopathology | Green Fluorescent Proteins - genetics | Ventricular Dysfunction, Left - chemically induced | Autophagy - drug effects | Autophagy-Related Protein-1 Homolog | Starvation - metabolism | Heart Failure - prevention & control | Hypertrophy, Left Ventricular - chemically induced | Time Factors | Hypertrophy, Left Ventricular - pathology | Adenosine Triphosphate - metabolism | Ventricular Dysfunction, Left - pathology | Protein-Serine-Threonine Kinases - metabolism | Green Fluorescent Proteins - metabolism | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Cells, Cultured | Rats | Mice, Transgenic | Heart Failure - metabolism | Antibiotics, Antineoplastic | Heart Failure - pathology | Cathepsin D - metabolism | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Stroke Volume | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Animals | Energy Metabolism | Myocytes, Cardiac - metabolism | Mice | Ventricular Pressure | Hypertrophy, Left Ventricular - physiopathology | Heart Failure - chemically induced | Starvation - complications | Index Medicus
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4. Full Text Does dapagliflozin regress left ventricular hypertrophy in patients with type 2 diabetes? A prospective, double-blind, randomised, placebo-controlled study
by Brown, Alexander J.M and Lang, Chim and McCrimmon, Rory and Struthers, Allan
BMC cardiovascular disorders, ISSN 1471-2261, 08/2017, Volume 17, Issue 1, pp. 229 - 229
Cardiac MRI | Diabetes | SGLT2 inhibitor | Left ventricular hypertrophy | Mechanistic trial | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Prospective Studies | Benzhydryl Compounds - administration & dosage | Diabetic Cardiomyopathies - etiology | Humans | Clinical Protocols | Scotland | Diabetic Cardiomyopathies - physiopathology | Benzhydryl Compounds - adverse effects | Hypoglycemic Agents - administration & dosage | Time Factors | Diabetic Cardiomyopathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Glucosides - adverse effects | Diabetic Cardiomyopathies - diagnostic imaging | Double-Blind Method | Administration, Oral | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - prevention & control | Risk Factors | Ventricular Function, Left - drug effects | Treatment Outcome | Disease Progression | Proof of Concept Study | Diabetes Mellitus, Type 2 - diagnosis | Magnetic Resonance Imaging | Glucosides - administration & dosage | Hypertrophy, Left Ventricular - physiopathology | Blood Pressure Monitoring, Ambulatory | Diabetes Mellitus, Type 2 - drug therapy | Hypoglycemic Agents - adverse effects | Research Design | Ventricular Remodeling - drug effects | Hypertrophy, Left Ventricular - diagnostic imaging | Type 2 diabetes | Complications and side effects | Heart enlargement | Dapagliflozin | Dosage and administration | Research | Drug therapy | Risk factors | Glucose transporter | Heart failure | Hypertension | Diabetes mellitus | Weight control | Magnetic resonance imaging | Sodium | Insulin resistance | Blood pressure | Ventricle | Full text | Cardiovascular diseases | Heart diseases | Hypertrophy | Index Medicus
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5. Full Text ELABELA-APJ axis protects from pressure overload heart failure and angiotensin II-induced cardiac damage
by Sato, Teruki and Sato, Chitose and Kadowaki, Ayumi and Watanabe, Hiroyuki and Ho, Lena and Ishida, Junji and Yamaguchi, Tomokazu and Kimura, Akinori and Fukamizu, Akiyoshi and Penninger, Josef M and Reversade, Bruno and Ito, Hiroshi and Imai, Yumiko and Kuba, Keiji
Cardiovascular research, ISSN 0008-6363, 2017, Volume 113, Issue 7, pp. 760 - 769
ACE2 | ACE | APJ | Elabela | Angiotensin | Apelin | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Humans | Heart Failure - physiopathology | Apelin Receptors - deficiency | Myocardial Contraction - drug effects | Male | Heart Failure - prevention & control | Ventricular Dysfunction, Left - genetics | Myocardium - metabolism | Peptidyl-Dipeptidase A - metabolism | Hypertension - genetics | Heart Failure - etiology | Disease Models, Animal | Constriction | Hypertrophy, Left Ventricular - prevention & control | Ventricular Function, Left - drug effects | Heart Failure - genetics | Cardiotonic Agents - pharmacology | Hypertension - metabolism | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Cardiotonic Agents - administration & dosage | Signal Transduction - drug effects | Aorta - surgery | Fibrosis | Forkhead Box Protein M1 - genetics | Ligands | Hypertrophy, Left Ventricular - physiopathology | Peptide Hormones - pharmacology | Peptidyl-Dipeptidase A - genetics | Arterial Pressure | Transfection | Apelin Receptors - genetics | HEK293 Cells | Hypertension - prevention & control | Aorta - physiopathology | Angiotensin II | Peptide Hormones - administration & dosage | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Myocardium - pathology | Hypertension - physiopathology | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Infusions, Subcutaneous | Apelin Receptors - metabolism | Animals | Forkhead Box Protein M1 - metabolism | Index Medicus
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6. Full Text MicroRNA-21 regulates peroxisome proliferator–activated receptor alpha, a molecular mechanism of cardiac pathology in Cardiorenal Syndrome Type 4
by Chuppa, Sandra and Liang, Mingyu and Liu, Pengyuan and Liu, Yong and Casati, Marc C and Cowley, Allen W and Patullo, Leah and Kriegel, Alison J
Kidney international, ISSN 0085-2538, 02/2018, Volume 93, Issue 2, pp. 375 - 389
cardiovascular disease | chronic kidney disease | inflammation | renal pathology | Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Cardio-Renal Syndrome - metabolism | Male | MicroRNAs - metabolism | Cardio-Renal Syndrome - pathology | Hypertrophy, Left Ventricular - genetics | Ventricular Dysfunction, Left - genetics | Hypertrophy, Left Ventricular - pathology | Ventricular Dysfunction, Left - pathology | Fatty Acids - metabolism | Clofibrate - pharmacology | Heart Ventricles - pathology | Disease Models, Animal | Signal Transduction | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Ventricular Function, Left - drug effects | Gene Expression Regulation | PPAR alpha - genetics | Cardio-Renal Syndrome - prevention & control | Rats, Sprague-Dawley | Ventricular Dysfunction, Left - prevention & control | Ventricular Dysfunction, Left - metabolism | Animals | Heart Ventricles - physiopathology | Fibrosis | PPAR alpha - agonists | Heart Ventricles - metabolism | MicroRNAs - genetics | PPAR alpha - metabolism | Cardio-Renal Syndrome - genetics | Ventricular Remodeling - drug effects | Heart Ventricles - drug effects | Animal models | Nephrectomy | Therapeutic applications | miRNA | Chromosome 5 | Inflammation | Lipid metabolism | Ventricle | Peroxisome proliferator-activated receptors | Gene expression | Heart diseases | Hypertrophy | Index Medicus
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7. Full Text Myocardial remodeling with aortic stenosis and after aortic valve replacement: Mechanisms and future prognostic implications
by Yarbrough, William M., MD and Mukherjee, Rupak, PhD and Ikonomidis, John S., MD, PhD and Zile, Michael R., MD and Spinale, Francis G., MD, PhD
The Journal of thoracic and cardiovascular surgery, ISSN 0022-5223, 2012, Volume 143, Issue 3, pp. 656 - 664
Cardiothoracic Surgery | Cardiac & Cardiovascular Systems | Respiratory System | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Surgery | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Medical sciences | Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy | Pneumology | Ventricular Function, Left | Humans | Heart Failure - physiopathology | Heart Failure - prevention & control | Ventricular Remodeling | Hypertrophy, Left Ventricular - pathology | Myocardium - metabolism | Ventricular Dysfunction, Left - pathology | Aortic Valve Stenosis - surgery | Extracellular Matrix Proteins - metabolism | Risk Assessment | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Risk Factors | Aortic Valve Stenosis - physiopathology | Myocardium - pathology | Treatment Outcome | Heart Failure - metabolism | Heart Failure - pathology | Disease Progression | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Ventricular Dysfunction, Left - metabolism | Animals | Aortic Valve Stenosis - pathology | Heart Valve Prosthesis Implantation - adverse effects | Fibrosis | Aortic Valve Stenosis - metabolism | Ventricular Pressure | Hypertrophy, Left Ventricular - physiopathology | Heart failure | Medical colleges | RNA | Peptides | Aortic valve stenosis | Stenosis | Transforming growth factors | Heart valve diseases | Messenger RNA | Collagen | Angiotensin | Cardiology | Health aspects | Natriuretic peptides | Index Medicus | Abridged Index Medicus
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8. Full Text Effect of Intensive Blood Pressure Lowering on Left Ventricular Hypertrophy in Patients With Diabetes Mellitus: Action to Control Cardiovascular Risk in Diabetes Blood Pressure Trial
by Soliman, Elsayed Z and Byington, Robert P and Bigger, J. Thomas and Evans, Gregory and Okin, Peter M and Goff, David C and Chen, Haiying
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 12/2015, Volume 66, Issue 6, pp. 1123 - 1129
Cornell medical index | hypertrophy, left ventricular | blood pressure | cardiovascular disease | diabetes mellitus | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiovascular Diseases - physiopathology | Follow-Up Studies | Cardiovascular Diseases - prevention & control | Humans | Middle Aged | Risk Factors | Hypertension - drug therapy | Male | Treatment Outcome | Antihypertensive Agents - therapeutic use | Hypertension - physiopathology | Cardiovascular Diseases - complications | Hypertrophy, Left Ventricular - complications | Hypertension - complications | Female | Aged | Blood Pressure - drug effects | Hypertrophy, Left Ventricular - drug therapy | Hypertrophy, Left Ventricular - physiopathology | Diabetes Mellitus, Type 2 - complications | Index Medicus
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