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European heart journal, ISSN 0195-668X, 12/2011, Volume 32, Issue 24, pp. 3073 - 3080
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 04/2016, Volume 36, Issue 4, pp. 636 - 646
heart failure | cytokines | hypertrophy | inflammation | angiotensin II | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Oxidative Stress | Heart Failure - physiopathology | Male | Aortic Diseases - metabolism | Heart Failure - prevention & control | Hypertrophy, Left Ventricular - genetics | Time Factors | Ventricular Dysfunction, Left - genetics | Basigin - genetics | Myocardium - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Heart Failure - etiology | Disease Models, Animal | Fibroblasts - metabolism | Animals, Newborn | Angiotensin II - pharmacology | Hypertrophy, Left Ventricular - prevention & control | Rats | Heart Failure - genetics | Heart Failure - metabolism | Fibroblasts - pathology | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Aortic Diseases - genetics | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Fibrosis | Myocytes, Cardiac - metabolism | Hypertrophy, Left Ventricular - physiopathology | Rats, Wistar | Ventricular Function, Left | JNK Mitogen-Activated Protein Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Blood Proteins - metabolism | Hypertrophy, Left Ventricular - pathology | Inflammation Mediators - metabolism | Ventricular Dysfunction, Left - pathology | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - metabolism | Cells, Cultured | Ventricular Dysfunction, Left - etiology | Myocardium - pathology | Heart Failure - pathology | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Mechanotransduction, Cellular | Aortic Diseases - physiopathology | Animals | Aortic Diseases - complications | Basigin - metabolism | Matrix Metalloproteinases - metabolism | Index Medicus
Journal Article
Cardiovascular research, ISSN 0008-6363, 12/2012, Volume 96, Issue 3, pp. 456 - 465
Heart failure | Autophagy | Doxorubicin | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Heart | Biological and medical sciences | Medical sciences | Heart failure, cardiogenic pulmonary edema, cardiac enlargement | AMP-Activated Protein Kinases - metabolism | Microtubule-Associated Proteins - genetics | Ventricular Function, Left | Microtubule-Associated Proteins - metabolism | Heart Failure - physiopathology | Green Fluorescent Proteins - genetics | Ventricular Dysfunction, Left - chemically induced | Autophagy - drug effects | Autophagy-Related Protein-1 Homolog | Starvation - metabolism | Heart Failure - prevention & control | Hypertrophy, Left Ventricular - chemically induced | Time Factors | Hypertrophy, Left Ventricular - pathology | Adenosine Triphosphate - metabolism | Ventricular Dysfunction, Left - pathology | Protein-Serine-Threonine Kinases - metabolism | Green Fluorescent Proteins - metabolism | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Cells, Cultured | Rats | Mice, Transgenic | Heart Failure - metabolism | Antibiotics, Antineoplastic | Heart Failure - pathology | Cathepsin D - metabolism | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Stroke Volume | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Animals | Energy Metabolism | Myocytes, Cardiac - metabolism | Mice | Ventricular Pressure | Hypertrophy, Left Ventricular - physiopathology | Heart Failure - chemically induced | Starvation - complications | Index Medicus
Journal Article
BMC cardiovascular disorders, ISSN 1471-2261, 08/2017, Volume 17, Issue 1, pp. 229 - 229
Cardiac MRI | Diabetes | SGLT2 inhibitor | Left ventricular hypertrophy | Mechanistic trial | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Prospective Studies | Benzhydryl Compounds - administration & dosage | Diabetic Cardiomyopathies - etiology | Humans | Clinical Protocols | Scotland | Diabetic Cardiomyopathies - physiopathology | Benzhydryl Compounds - adverse effects | Hypoglycemic Agents - administration & dosage | Time Factors | Diabetic Cardiomyopathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Glucosides - adverse effects | Diabetic Cardiomyopathies - diagnostic imaging | Double-Blind Method | Administration, Oral | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - prevention & control | Risk Factors | Ventricular Function, Left - drug effects | Treatment Outcome | Disease Progression | Proof of Concept Study | Diabetes Mellitus, Type 2 - diagnosis | Magnetic Resonance Imaging | Glucosides - administration & dosage | Hypertrophy, Left Ventricular - physiopathology | Blood Pressure Monitoring, Ambulatory | Diabetes Mellitus, Type 2 - drug therapy | Hypoglycemic Agents - adverse effects | Research Design | Ventricular Remodeling - drug effects | Hypertrophy, Left Ventricular - diagnostic imaging | Type 2 diabetes | Complications and side effects | Heart enlargement | Dapagliflozin | Dosage and administration | Research | Drug therapy | Risk factors | Glucose transporter | Heart failure | Hypertension | Diabetes mellitus | Weight control | Magnetic resonance imaging | Sodium | Insulin resistance | Blood pressure | Ventricle | Full text | Cardiovascular diseases | Heart diseases | Hypertrophy | Index Medicus
Journal Article
Cardiovascular research, ISSN 0008-6363, 2017, Volume 113, Issue 7, pp. 760 - 769
ACE2 | ACE | APJ | Elabela | Angiotensin | Apelin | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Humans | Heart Failure - physiopathology | Apelin Receptors - deficiency | Myocardial Contraction - drug effects | Male | Heart Failure - prevention & control | Ventricular Dysfunction, Left - genetics | Myocardium - metabolism | Peptidyl-Dipeptidase A - metabolism | Hypertension - genetics | Heart Failure - etiology | Disease Models, Animal | Constriction | Hypertrophy, Left Ventricular - prevention & control | Ventricular Function, Left - drug effects | Heart Failure - genetics | Cardiotonic Agents - pharmacology | Hypertension - metabolism | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Cardiotonic Agents - administration & dosage | Signal Transduction - drug effects | Aorta - surgery | Fibrosis | Forkhead Box Protein M1 - genetics | Ligands | Hypertrophy, Left Ventricular - physiopathology | Peptide Hormones - pharmacology | Peptidyl-Dipeptidase A - genetics | Arterial Pressure | Transfection | Apelin Receptors - genetics | HEK293 Cells | Hypertension - prevention & control | Aorta - physiopathology | Angiotensin II | Peptide Hormones - administration & dosage | Hypertrophy, Left Ventricular - etiology | Hypertrophy, Left Ventricular - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Myocardium - pathology | Hypertension - physiopathology | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Infusions, Subcutaneous | Apelin Receptors - metabolism | Animals | Forkhead Box Protein M1 - metabolism | Index Medicus
Journal Article
Kidney international, ISSN 0085-2538, 02/2018, Volume 93, Issue 2, pp. 375 - 389
Journal Article
The Journal of thoracic and cardiovascular surgery, ISSN 0022-5223, 2012, Volume 143, Issue 3, pp. 656 - 664
Cardiothoracic Surgery | Cardiac & Cardiovascular Systems | Respiratory System | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Surgery | Science & Technology | Cardiology. Vascular system | Biological and medical sciences | Medical sciences | Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy | Pneumology | Ventricular Function, Left | Humans | Heart Failure - physiopathology | Heart Failure - prevention & control | Ventricular Remodeling | Hypertrophy, Left Ventricular - pathology | Myocardium - metabolism | Ventricular Dysfunction, Left - pathology | Aortic Valve Stenosis - surgery | Extracellular Matrix Proteins - metabolism | Risk Assessment | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Risk Factors | Aortic Valve Stenosis - physiopathology | Myocardium - pathology | Treatment Outcome | Heart Failure - metabolism | Heart Failure - pathology | Disease Progression | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Ventricular Dysfunction, Left - metabolism | Animals | Aortic Valve Stenosis - pathology | Heart Valve Prosthesis Implantation - adverse effects | Fibrosis | Aortic Valve Stenosis - metabolism | Ventricular Pressure | Hypertrophy, Left Ventricular - physiopathology | Heart failure | Medical colleges | RNA | Peptides | Aortic valve stenosis | Stenosis | Transforming growth factors | Heart valve diseases | Messenger RNA | Collagen | Angiotensin | Cardiology | Health aspects | Natriuretic peptides | Index Medicus | Abridged Index Medicus
Journal Article