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Publication DateCardiovascular research, ISSN 0008-6363, 10/2017, Volume 113, Issue 12, pp. 1433 - 1440
Noncoding RNAs | MicroRNAs | Pulmonary hypertension | Right ventricular remodelling | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Ventricular Dysfunction, Right - genetics | Humans | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | MicroRNAs - metabolism | Hypertrophy, Right Ventricular - etiology | Arrhythmogenic Right Ventricular Dysplasia - physiopathology | Arrhythmogenic Right Ventricular Dysplasia - genetics | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling - genetics | Ventricular Dysfunction, Right - physiopathology | Heart Failure - etiology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Hypertrophy, Right Ventricular - genetics | Ventricular Function, Right - genetics | Gene Expression Regulation | Heart Failure - genetics | Hypertension, Pulmonary - genetics | Heart Failure - metabolism | Genetic Markers | Disease Progression | Arrhythmogenic Right Ventricular Dysplasia - complications | Animals | Ventricular Dysfunction, Right - etiology | MicroRNAs - genetics | Hypertension, Pulmonary - complications | Index Medicus
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Loading RightsThe International Journal of Cardiovascular Imaging, ISSN 1569-5794, 4/2015, Volume 31, Issue 4, pp. 669 - 679
Heart failure | Echocardiography | Medicine & Public Health | Magnetic resonance imaging | Cardiac disease | Electrocardiography | Cardiology | Doppler | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Radiology, Nuclear Medicine & Medical Imaging | Science & Technology | Predictive Value of Tests | Hypertrophy, Left Ventricular - enzymology | Ventricular Function, Left | Ventricular Dysfunction, Right - genetics | Heart Failure - enzymology | Heart Failure - physiopathology | Hypertrophy, Left Ventricular - diagnosis | Myosin Heavy Chains - genetics | Male | Cardiomyopathy, Hypertrophic - enzymology | Ventricular Dysfunction, Right - enzymology | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Calcineurin - genetics | Hypertrophy, Left Ventricular - genetics | Ventricular Dysfunction, Left - genetics | Echocardiography, Doppler | Ventricular Dysfunction, Left - enzymology | Female | Ventricular Dysfunction, Right - physiopathology | Heart Failure - diagnosis | Hypertrophy, Right Ventricular - genetics | Disease Models, Animal | High-Throughput Screening Assays - methods | Promoter Regions, Genetic | Cardiomyopathy, Hypertrophic - genetics | Genetic Predisposition to Disease | Hypertrophy, Right Ventricular - diagnosis | Mice, Inbred C57BL | Heart Failure - genetics | Mice, Transgenic | Ventricular Dysfunction, Left - diagnosis | Hypertrophy, Right Ventricular - enzymology | Ventricular Dysfunction, Right - diagnosis | Cardiomyopathy, Hypertrophic - diagnosis | Disease Progression | Ventricular Dysfunction, Left - physiopathology | Magnetic Resonance Imaging | Phenotype | Animals | Cardiomyopathy, Hypertrophic - physiopathology | Hemodynamics | Hypertrophy, Left Ventricular - physiopathology | Calcineurin - metabolism | Ventricular Function, Right | Ventricular Myosins - genetics | Heart | Genetic engineering | Electrocardiogram | Index Medicus
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Loading RightsCardiovascular research, ISSN 0008-6363, 08/2016, Volume 111, Issue 3, pp. 184 - 193
Hypertension | Hypoxia | Pulmonary | Pulmonary heart disease | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Ventricular Dysfunction, Right - genetics | Humans | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | Male | MicroRNAs - metabolism | Hypertension, Pulmonary - prevention & control | Hypoxia - metabolism | Heart Failure - prevention & control | Hypertrophy, Right Ventricular - physiopathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Ventricular Dysfunction, Right - physiopathology | Lung - metabolism | Receptor, IGF Type 1 - deficiency | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Genetic Predisposition to Disease | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Heart Failure - genetics | Hypertension, Pulmonary - genetics | Heart Failure - metabolism | Hypertension, Pulmonary - metabolism | Hypoxia - complications | Imidazoles - pharmacology | Receptor, IGF Type 1 - genetics | Mice, Knockout | Ventricular Dysfunction, Right - prevention & control | Hypoxia - genetics | Phenotype | Animals | Heart Ventricles - physiopathology | Receptors, Somatomedin - metabolism | Heart Ventricles - metabolism | MicroRNAs - genetics | Pyridines - pharmacology | Ventricular Function, Right | Index Medicus
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Loading RightsAmerican journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 05/2013, Volume 305, Issue 4, pp. H551 - H562
Heart failure | Ubiquitin | Cardiac hypertrophy | Right ventricle | Proteasome | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Genetic Therapy | Apoptosis - drug effects | Ventricular Dysfunction, Right - genetics | Heart Failure - enzymology | Heart Failure - physiopathology | Ubiquitin - metabolism | Male | Hypertrophy, Right Ventricular - etiology | Pulmonary Artery - surgery | Ventricular Dysfunction, Right - enzymology | Heart Failure - prevention & control | Hypertrophy, Right Ventricular - physiopathology | Time Factors | Hypertrophy, Right Ventricular - pathology | Ventricular Dysfunction, Right - physiopathology | Ventricular Function, Right - drug effects | Heart Failure - etiology | Hypertrophy, Right Ventricular - genetics | Disease Models, Animal | Constriction | Proteasome Inhibitors - pharmacology | Gene Expression Regulation | Heart Failure - genetics | Mice, Transgenic | Pulmonary Artery - physiopathology | Heart Failure - pathology | Hypertrophy, Right Ventricular - enzymology | Proteasome Endopeptidase Complex - genetics | Ventricular Dysfunction, Right - prevention & control | Myocardium - enzymology | Animals | Signal Transduction - drug effects | Ventricular Dysfunction, Right - etiology | Hypertrophy, Right Ventricular - prevention & control | Hemodynamics - drug effects | Mice | Proteasome Endopeptidase Complex - metabolism | Ventricular Dysfunction, Right - pathology | Heart enlargement | Physiological aspects | Cellular signal transduction | Research | Ubiquitin-proteasome system | Health aspects | Proteins | Cardiovascular disease | Rodents | Apoptosis | Index Medicus | proteasome | heart failure | right ventricle | cardiac hypertrophy | Integrative Cardiovascular Physiology and Pathophysiology | ubiquitin
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Loading RightsCardiovascular research, ISSN 0008-6363, 03/2019, Volume 115, Issue 4, pp. 788 - 799
Remodelling | Animal models of human disease | Right ventricle | Pulmonary hypertension | Gene expression and regulation | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Adaptation, Physiological | Species Specificity | Ventricular Dysfunction, Right - genetics | Rats, Inbred F344 | Heart Failure - physiopathology | Transcriptome | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - etiology | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Ventricular Dysfunction, Right - physiopathology | Heart Failure - etiology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Hypertrophy, Right Ventricular - genetics | Disease Models, Animal | Severity of Illness Index | Signal Transduction | Gene Expression Regulation | Heart Failure - genetics | Hypertension, Pulmonary - genetics | Heart Failure - metabolism | Hypertension, Pulmonary - metabolism | Rats, Sprague-Dawley | Animals | Ventricular Dysfunction, Right - etiology | Exercise Tolerance | Myocytes, Cardiac - metabolism | Neovascularization, Physiologic | Ventricular Function, Right | Hypertension, Pulmonary - complications | Index Medicus | Original
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Loading RightsJournal of cardiovascular translational research, ISSN 1937-5387, 8/2013, Volume 6, Issue 4, pp. 623 - 639
Human Genetics | Heart failure | Functional genomics of intracardiac blood flow | Ventricular remodeling | Convective deceleration load | Medicine/Public Health, general | Biomedicine general | Ventricular chamber volumes | Biomedical Engineering | Diastolic function | Diastolic ventriculoannular disproportion | Medicine & Public Health | Cardiology | Diastolic filling vortex | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Ventricular Function, Right - genetics | Genetic Predisposition to Disease | Diastole | Models, Cardiovascular | Ventricular Dysfunction, Right - genetics | Genomics | Humans | Ventricular Function, Left - genetics | Heart Failure - physiopathology | Heart Failure - genetics | Disease Progression | Ventricular Dysfunction, Left - physiopathology | Stroke Volume | Biomechanical Phenomena | Hypertrophy, Right Ventricular - physiopathology | Phenotype | Ventricular Remodeling | Animals | Hypertrophy, Left Ventricular - genetics | Ventricular Dysfunction, Left - genetics | Ventricular Dysfunction, Right - physiopathology | Ventricular Pressure | Hypertrophy, Left Ventricular - physiopathology | Hypertrophy, Right Ventricular - genetics | Heart | Analysis | Index Medicus | heart failure | diastolic function | ventricular remodeling | convective deceleration load | diastolic filling vortex | ventricular chamber volumes | functional genomics of intracardiac blood flow | diastolic ventriculoannular disproportion
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Evidence for right ventricular lipotoxicity in heritable pulmonary arterial hypertension
American journal of respiratory and critical care medicine, ISSN 1073-449X, 02/2014, Volume 189, Issue 3, pp. 325 - 334
Heart failure | Insulin resistance | Right ventricle | Pulmonary hypertension | Respiratory System | Life Sciences & Biomedicine | Critical Care Medicine | General & Internal Medicine | Science & Technology | Cardiology. Vascular system | Heart | Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy | Biological and medical sciences | Intensive care medicine | Pulmonary hypertension. Acute cor pulmonale. Pulmonary embolism. Pulmonary vascular diseases | Medical sciences | Heart failure, cardiogenic pulmonary edema, cardiac enlargement | Pneumology | Biomarkers - metabolism | Ceramides - metabolism | Bone Morphogenetic Protein Receptors, Type II - genetics | Oligonucleotide Array Sequence Analysis | Oxidation-Reduction | Humans | Familial Primary Pulmonary Hypertension | Hypertension, Pulmonary - genetics | Mice, Transgenic | Hypertrophy, Right Ventricular - etiology | Genetic Markers | Case-Control Studies | Lipolysis | Triglycerides - metabolism | Animals | Mice | Mutation | Hypertrophy, Right Ventricular - metabolism | Hypertrophy, Right Ventricular - genetics | Hypertension, Pulmonary - complications | Index Medicus | Abridged Index Medicus | heart failure | right ventricle | Original | insulin resistance | pulmonary hypertension
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Loading RightsJournal of molecular medicine (Berlin, Germany), ISSN 0946-2716, 1/2012, Volume 90, Issue 1, pp. 31 - 43
Human Genetics | QTc interval | Myocardial fibrosis | Biomedicine | Pulmonic stenosis | Monophasic action potential duration | Right heart failure | Internal Medicine | Molecular Medicine | Genetics & Heredity | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Cardiology. Vascular system | Heart | Biological and medical sciences | General aspects | Medical sciences | Heart failure, cardiogenic pulmonary edema, cardiac enlargement | Vasodilator Agents - therapeutic use | Glucose Transporter Type 1 - metabolism | Male | Hypertrophy, Right Ventricular - drug therapy | Ranolazine | Glycogen - metabolism | Acetanilides - therapeutic use | Trimetazidine - pharmacology | Ventricular Function, Right - drug effects | Hexokinase - genetics | Fatty Acids - metabolism | Hypertrophy, Right Ventricular - metabolism | Trimetazidine - therapeutic use | Acetanilides - pharmacology | Vasodilator Agents - pharmacology | Oxidation-Reduction | Enzyme Inhibitors - pharmacology | Rats | Piperazines - therapeutic use | Enzyme Inhibitors - therapeutic use | Piperazines - pharmacology | Rats, Sprague-Dawley | Animals | Myocytes, Cardiac - drug effects | Glucose Transporter Type 1 - genetics | Glycolysis | Myocytes, Cardiac - metabolism | Hexokinase - metabolism | Physiological aspects | Biological oxidation (Metabolism) | Research | Heart enlargement | Index Medicus
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Loading RightsAmerican journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 12/2020, Volume 319, Issue 6, pp. H1459 - H1473
Mitochondria, Heart - metabolism | Mitochondria, Heart - pathology | Male | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Rats, Mutant Strains | Myocardium - metabolism | Estrogen Receptor alpha - metabolism | Female | Hypertrophy, Right Ventricular - pathology | Ventricular Dysfunction, Right - physiopathology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Disease Models, Animal | Fibrillar Collagens - metabolism | Signal Transduction | Proto-Oncogene Proteins c-jun - genetics | Kallikreins - genetics | Myocardium - pathology | Rats, Sprague-Dawley | Ventricular Dysfunction, Right - prevention & control | Animals | Estrogen Receptor alpha - genetics | Proto-Oncogene Proteins c-jun - metabolism | Fibrosis | Sex Factors | Hypertrophy, Right Ventricular - prevention & control | Mutation | Kallikreins - metabolism | Ventricular Dysfunction, Right - pathology | Ventricular Function, Right | Heart | Matrix metalloproteinases | Estrogens | Collagens | Estrogen receptors | Overloading | Matrix metalloproteinase | Tissue inhibitor of metalloproteinase 1 | Gene sequencing | Receptors | Rodents | Metalloproteinase | Adaptation | Hypertension | Phenotypes | Peptidase | Pulmonary arteries | Waveforms | Kallikrein | Ribonucleic acid--RNA | Pulmonary artery | Gelatinase B | Peptidases | Collagen | Men | Ventricle | Females | Hypertrophy | Index Medicus | adverse remodeling | right ventricle | estrogen receptor-α | pressure overload
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Loading RightsPediatric research, ISSN 0031-3998, 12/2015, Volume 78, Issue 6, pp. 634 - 640
Life Sciences & Biomedicine | Pediatrics | Science & Technology | Bronchopulmonary Dysplasia - enzymology | Phosphorylation | Superoxide Dismutase - genetics | Vascular Remodeling | Oxidative Stress | Ventricular Dysfunction, Right - genetics | Bronchopulmonary Dysplasia - pathology | Hypertension, Pulmonary - physiopathology | Bronchopulmonary Dysplasia - physiopathology | Vascular Endothelial Growth Factor A - metabolism | Ventricular Dysfunction, Right - enzymology | Hypertension, Pulmonary - chemically induced | Pulmonary Alveoli - enzymology | Bronchopulmonary Dysplasia - genetics | Hypertrophy, Right Ventricular - physiopathology | Bleomycin | Ventricular Dysfunction, Right - physiopathology | Nitric Oxide Synthase Type III - metabolism | Ventricular Dysfunction, Right - chemically induced | Hypertension, Pulmonary - enzymology | Hypertrophy, Right Ventricular - genetics | Pulmonary Alveoli - blood supply | Animals, Newborn | Genetic Predisposition to Disease | Pulmonary Alveoli - pathology | Signal Transduction | Mice, Inbred C57BL | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Hypertension, Pulmonary - genetics | Superoxide Dismutase - deficiency | Pulmonary Artery - physiopathology | Hypertrophy, Right Ventricular - enzymology | Pulmonary Artery - enzymology | Mice, Knockout | Phenotype | Animals | Bronchopulmonary Dysplasia - chemically induced | Hypertrophy, Right Ventricular - chemically induced | Ventricular Pressure | Endothelial Cells - pathology | Nitric Oxide - metabolism | Endothelial Cells - enzymology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Ventricular Function, Right | Index Medicus
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